Lecture 12: Skin and Soft Tissue Infections Flashcards

1
Q

What do skin and soft tissue infections involve?

A

Invastion of the skin and soft tissue

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2
Q

Cardinal sign of a SSTI?

A

Host inflammatory response followed by the manifestations including fever and rapid progression of lesions

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3
Q

How do we classify SSTI?

A

Depth of infection

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4
Q

List some risk factors for SSTI’s

A

DM, cirrhosis, bite wounds, neutropenia, reptiles, hot tubs, fish tank, water, drug abuse

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5
Q

A patient presents with a eruption of flaccid pustules which form a honey-coloured crust. These lesions are highly contagious and it is seen in children. What is this?

A

Epidemic impetigo

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6
Q

What organisms cause epidemic impetigo?

A

Group A strep, staph aureus

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7
Q

How is epidemic impetigo treated?

A

Antibiotic therapy

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8
Q

What is a rapid spreading infection of deep skin layers that involves superficial dermal lymphatics that lead to swelling?

A

Erysipelas

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9
Q

What can erysipelas be a a source of?

A

Bacteremia and has the potential for a systemic infection

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10
Q

What causes erysipelas?

A

Streptococcus progenies (group A strep)

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11
Q

What does cellulitis mean?

A

Refers to a deeper infection with acute spreading in the subcutaneous layer

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12
Q

What does cellulitis tend to follow?

A

Local infection or trauma

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13
Q

List the organisms which may cause cellulitis?

A

Staph aureus, streptococcus progenies, pseudomonas aeruginosa, gram negative bacilli- organisms depend on the sire of the wound

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14
Q

Furuncles

A

Boil

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15
Q

What causes boils?

A

ALWAYS staphylococcus aureus

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16
Q

Carbuncle

A

Multiple furuncles

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17
Q

How is a furuncle treated?

A

Cloxacillin

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18
Q

Decubitus ulcers

A

Bed wound

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19
Q

What type of organisms lead to bed wounds?

A

COlonozied with mixed potential pathogens or non-pathogens

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20
Q

How are bed wounds treated?

A

Nursing care

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21
Q

Clean

A

No significant contamination (i.e. heart surgery)

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22
Q

Clean contaminated

A

Through mucous membrane, GU, Resp tract

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23
Q

Contaminated

A

Colon, accident

24
Q

Dirty/infected

A

Greatest risk of infection

25
What should you tell the lab if a client suffers from a bite wound?
The details of the situation
26
What can bites lead to?
Cellulitis, abscess, deep tissue infections
27
If a client has a clenched fist and is bitten here....
Do not ignore this as there is a high risk of deep infection as the skin over the knuckles is tight and there is a TON of bacteria in a human's mouth
28
What is fasciitis?
A rapidly progressing cellulitis with extensive necrosis of the subcutaneous tissue as a result of toxin producing agents
29
What is the most common agent of flesh eating disease?
Streptococcus progenies (group A strep)
30
How do you diagnose flesh eating disease?
Rapid clinical followed by gram stain and culture confirmation
31
Give an example of a dermatophyte
Tinea or ringworm, infections in the hair nails, and skin
32
Where can infections pop up in the body in dermatophytes?
Anywhere in the body
33
What causes tinea/rignworm?
Yeast infection of dermatophytic fungi
34
How is this diagnosed in the lab?
Skin/nail scrapping sent to the lab, calcoflour stain and a +/- fungal culture is performed
35
What do dermatophytes produce?
Keratinase which breaks down keratin
36
Which sex of scabies burrow into the skin and lay eggs?
Females
37
What are we able to see on the skin's surface with a scabies infestation?
The tracks from this microscopic mite
38
What are the three "easy's" of lice?
Spreads, diagnosed, controlled
39
How many types of herpes simplex virus are there?
Two
40
Facial (cold sores) herpes
Type 1
41
Genital herpes
Type 1 or 2
42
How does transmission happen in herpes?
Direct contact
43
How is herpes diagnosed
Clinical but with genital it may require the virus culture or molecular detection
44
How is herpes treated?
With antiviral drugs which can shorten healing time if taken early
45
Talk about recurrence and herpes viruses
Common, with genital this will usually diminish in a year and some people never have recurrences
46
Varicella Zoster Virus
Chicken pox and shingles
47
Vaccine for VSZ
Vaccine is safe and effective, live vaccine which has led to decreased hospitalizations and death by 95%
48
What is shingles?
Varicella zoster reactivated which is diagnosed clinically
49
How does the VSZ virus persist?
In a latent state in the nerve ganglia in the face and trunk
50
List the microbes found: in mild infections (above the waist)
Staph Aureus, strep pyogenes
51
List the microbes found: Infections of hand and head
S. Aureus S. Pyogenes Haemophilus influenzae
52
List the microbes found: severe infections (above the waist) without special considerations
S aureus, S pyogenes
53
List the microbes found: Mild infections below the waist
S aureus, S pyogenes, coliform species possible
54
List the microbes found: Severe infections below the waist
Escherichia coli, Enterococcus species, other coliform species, S aureus, S pyogenes
55