Lecture 15 - Case Studies Flashcards

1
Q

What can you assume if damage causes sensory or motor changes on both sides of body?

A

Damage has to be to CNS

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2
Q

Dermatome of umbilicus?

A

T10

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3
Q

If we know the level of damage of a spinal cord nerve, how do we know in between which vertebrae the damage occurred? Why?

A

2 to 3 less than the nerve number because the spinal cord stops elongating during development before the vertebral column does AND because of curvature

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4
Q

Can position sense be tested for on the trunk?

A

NOPE

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5
Q

Why is hyperexflexia seen with UMN damage?

A

Because afferent nerve fibers will sprout to fill synapses vacated by degenerating UMN fibers

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6
Q

Why is the Babinski sign lost during infancy?

A

Myelination of the lateral corticospinal tract

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7
Q

How could a Babinski sign re-appear in adulthood?

A

Loss of lateral corticospinal tract

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8
Q

Why is recovery due to NS damage so poor in adults?

A

Because adults’ NS has very limited capacity for regrowth of damaged pathways due to inhibitory molecules such as Nogo-A

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9
Q

What can a large lesion of the left internal capsule of the brain result in?

A

Paralysis of the right lower face, arm, and leg

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10
Q

What is the internal capsule in the brain mad of?

A
  1. Geniculate fibers

2. Corticobulbar tract

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11
Q

What input do muscles of the upper face receive?

A

Bilateral corticobulbar innervation

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12
Q

What input do muscles of the lower face receive?

A

Contralateral corticobulbar innervation

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13
Q

What is dysarthria? Eg?

A

Faulty speech because of difficulty performing movements needed for speech

Eg: damage to corticobulbar tract

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14
Q

What is aphasia?

A

Faulty speech

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15
Q

What can disconnect the somatosensory thalamus from the sensory cortex?

A

Interruption of the internal capsule

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16
Q

What is hemianopsia?

A

Visual field loss on the left or right side of the midline

17
Q

What is homonymous hemianopsia? What can this be due to?

A

Visual field loss on the same side of both eyes

Can be due to large capsular lesion that extends far enough posteriorly to interrupt the optic radiation