Lecture 29 Flashcards

1
Q

what is responsible for strength in the first weeks

A

neural adaptation

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2
Q

is neural adaptation specfic to training?

A

highly - train one muscle and get very little transfer to another

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3
Q

increased agonist activation via:

A

decreased neural inhibitory reflexes
increased motor unit recruitment, and/or
increased motor unit firing frequency

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4
Q

which things modify agonist-antagonist reflex control?

A

stretch reflex and Golgi tendon organ.

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5
Q

what does strech reflex do

A

primes muscles to increase force output. inherently increase tension in muscle and connective tissues.

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6
Q

the run down on stretch reflex.

A

muscle is stretched - muscle spindles activated - muscle contracted to prevent over stretching - increase muscle activation and RFD>

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7
Q

what happens if muslce used wile “primed”

A

stretch shorten cycle

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8
Q

what is SSC

A
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9
Q
A
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10
Q

what does the Golgi tendon do?

A

restrict high laods of tension

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11
Q

step by step of the golgi tendon

A

muscle contraction above threshold - Golgi tendon organs activated - muscle contraction inhibited to prevent excessive force.

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12
Q

how might golgi tendon be down-regulated

A

high impact training.

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13
Q

what is the monosynaptic stretch reflex

A

around 50 ms. only happens in stretch reflex. between the muscle spindle activation anf muscle contracted to prevent over-stretching

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14
Q

what is plyometrics

A

jump training. high explosive exercise that uses the short shortening cycle.

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15
Q

what are you training with plyos

A

high velocity and RFD

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16
Q

how does plyo increase speed.

A

increase ability tolerate high-stretch loads, increase muscle power outlet through stored elastic energy and stretch reflex

17
Q

neuromuscular adaptation to high-intensity training.

A

Increase Max power
increase Muscle strength
increase Size (Hypertrophy)
increase Selective hypertrophy Type II
(triangle) Type of myosin
increase Neural activation (RFD)
increase Na+ K+ ATPase activity

18
Q

Metabolic Adaptation

A

increase in all:
ATP & PC stores
  Creatine kinase activity
  ATPase activity
  PFK activity
  Buffering capacity

19
Q

how much do metabolic adaptations increase

A

20-40% in high intensity training.

20
Q

other adaptations to short duration, high intensity trainingn

A

increase efficieny of movement
- neural adaptatipn
increase pain tolerance.

21
Q

what energy systems does HIIT stimulate.

A

ALL.

22
Q

intense cellular metabolic and mechanical stress in HIIT.

A

can stimulate fat oxidative capability and aerobic power as effectively as much larger volume of regular aerobic training.

23
Q

HIIT local and systemic adaptation

A

high vascular shear stress and cardiac load. increase CVS, but more care with progression and use more “clinical model’
time efficient

24
Q

HIIt for endurance athletes

A

useful in minor (<20%) training time, espically late-phase training.

25
Q

HIIT for RHIE athletes

A

essential but also normal part of training.

26
Q

benefits of aerobic adaptations from interval training

A

simultaneously maximal contribution of all energy systems.
increase reliance on ATP supply aerobically (so less anaerobic demands)

27
Q

positive effect of resistance training.

A

difficult to identify how decline with aging. large individual differences in strength and training.

28
Q

strength training in older adults.

A

reducing falls (RFD important): daily function, heat, glucose control blood pressure and bones.
less absolute strength gains.
but same relative (%) gains.
also help retain precision of force control.

29
Q

how is there less absolute strength gain in strength training in older people.

A

less cell signalling
local acute inflammatory

30
Q

what does interval training help with

A

all types of fitness. tick every box