Lecture 29 - Overview Flashcards

1
Q

What effect does exercise have on homeostasis

A

It initially disrupts it and often requires prolonged coordination of most body systems

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2
Q

During exercise what happens to the heart rate, stroke volume, cardiac output, systolic pressure and diastolic pressure

A

The heart rate, stroke volume, cardiac output and systolic pressure all increase and the diastolic pressure decreases

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3
Q

Values of heart rate, stroke volume, cardiac output, systolic pressure and diastolic pressure at rest

A
Heart rate - 70bmp
Stroke volume - ~70ml
Cardiac output -5l/min
Systolic pressure - 120mmHg
Diastolic pressure - 80mmHg
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4
Q

Values for heart rate, stroke volume, cardiac output, systolic pressure and diastolic pressure during exercise

A
Heart rate - 200bmp
Stroke volume - ~130ml
Cardiac output - 25l/min
Systolic pressure - 180mmHg
Diastolic pressure - 70mmHg
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5
Q

During exercise what does CO increase in proportion to

A

Workload

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6
Q

What is the control of the heart rate coordinated by

A

The cardiovascular control centre

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7
Q

Where is the cardiovascular control centre found

A

In the brain stem

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8
Q

What is the immediate response to exercise

A

The HR increases to deliver more oxygenated blood to the exercising muscles

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9
Q

What is the long term adaption to exercise

A

Increased strength and efficiency of the heart

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10
Q

What is SV controlled by

A

Intrinsic and extrinsic control mechanisms

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11
Q

What are the intrinsic control methods involved in SV

A

Muscular contractions compressing the veins and venoconstriction

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12
Q

What are the extrinsic controls involved in SV

A

Sympathetic stimulation and adrenaline

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13
Q

What effects do working muscles have on the local environments

A

Local Po2 falls, local Pco2 increases, local H+ concentration rises (pH falls) and muscle temperature rises

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14
Q

When local Po2 falls, local Pco2 increases, local H+ concentration rises (pH falls) and muscle temperature rises what happens

A

Metabolic hyperaemia

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15
Q

What is blood pressure proportional to

A

TPR

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16
Q

To meet the metabolic demands of skeletal muscle there is

A

A large drop in TPR brought about by vasodilation of arterioles supplying the working muscle

17
Q

What effect does decreased TPR have on diastolic pressure

A

It causes diastolic pressure to drop

18
Q

What is produced in severe exercise

A

Lactic acid

19
Q

What are the values for ventilation, VO2 and VCO2 at rest

A

Ventilation - ~6l/min
VO2 - ~0.25l/min
VCO2 - ~0.20l/min

20
Q

What are the values for ventilation, VO2 and VCO2 during exercise

A

Ventilation - ~120l/min
VO2 - ~3.5l/min (5 in trained athletes)
VCO2 - 4+

21
Q

What does H+ stimulate and what does this cause

A

H+ stimulates peripheral chemoreceptors and ventilation is excessively stimulated

22
Q

What is the O2 deficit

A

The difference between the oxygen uptake of the body during early stages of exercise and during a similar duration of steady state of exercise

23
Q

What is the O2 debt

A

The amount of extra oxygen required by muscle tissue to oxidise lactic acid and replenish depleted ATP and phosphocreatine following vigorous exercise

24
Q

What factors may increase ventilation during exercise

A

Reflexes originating from body movements, increased body temeperature, adrenaline being released and impulses from the cerebral cortex

25
What is a predictor of a persons work capacity
The determination of maximal O2 consumption
26
How can VO2 max be determined
Through exercise on a bike/treadmill where the workload is progressively increased until exhaustion and the expired air is collected during the last minutes and the %O2, %CO2 and volume of air are measured
27
What effect does regular exercise have on VO2 max
It will improve it
28
What classifications of a persons V02 max are there
Low, fair, average, good or excellent
29
During exercise what does the increase in surface area for gas exchange allow
An enhance rate of gas transfer
30
What do increased CO and an increased pulmonary BP do
They force open previously closed capillaries