Lecture 3: CV & R Anatomy & Physiology Flashcards

1
Q

arteries

A

carry blood away from the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

veins

A

carry blood to the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

capillaries

A

site of exchange of substances

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

blood

A

contains hemoglobin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Pathway

A

arteries to arterioles to capillaries to venules to medium veins to large veins

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Electrical Conduction System of the Heart

A

-SA node- heart’s pacemaker, works on auto-rhythmicity, initiates impulses spread
Inernodal pathways
AV Node- continues electrical impulses at the AV bundle to the bundle branches
AV bundle-
Purkinje fibres
Right bundle branch
Left bundle branch

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Slight delay between atrial and ventricle contraction comes from

A

AV node

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

SA node to

A

atrial contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

AV node to

A

ventricular contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

ECG

A

Electrocardiogram

-graphic representation of the electrical activity in the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Normal Heartbeat

A

range=60 to 100bpm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Fast Heartbeat

A

tachycardia

-over 100bpm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Slow Heartbeat

A

bradycardia

-less than 60bpm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Irregular heartbeat

A

Arrhythmia

-number of different causes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

P-wave

A

activation/depolarization of the atria= contraction o the atria that pushes blood into the ventricles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

QRS comples

A

ventricular contraction/depolarization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

T-wave

A

Re-polarization of the ventricles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Difference between P-wave and QRS complex

A
  • ventricular contraction is much more forceful a contraction
  • QRS complex hides the re-polarization of the atria
  • re-polarize atria at the same time
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

BP is higher..

A

in systemic circulation

-aorta, large arteries, small arteries arterioles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

BP is lower

A

in systemic circulation
-capillaries, venules, small veins, large veins, venae cavae
in pulmonary circulation
-pulmonary arteries, arterioles, capillaries, venules, pulmonary veins

21
Q

Respiratory System

A

responsible for exchange of O2 and CO2 between air and blood

22
Q

COnducting portion

A

nose to bronchioles

23
Q

Respiratory Portion

A

actual gas exchange between bronchioles and alveoli

24
Q

Contraction and Expansion of Thoracic Cage

A
  • high pressure to low pressure gradient
  • must elevate rib cage
  • contraction of intercostals and diaphragm
25
Accessory Muscles
Inspiration- sternocleido mastoid, serratus anterior, pec minor, scalenes (increase chest cavity size) Expiration- transverse thoracis, obliques, rectus abdominus (compress abdominal cavity)
26
Acute CV response to Resistance Training
^ CO, SV, HR, O2 uptake, SBP, BF to active muscles | -largely dependent on intensity and volume of exercise, muscle involvement, rest period length
27
When is SBP highest?
Sticking point accluding blood flow -valsalva manoeuvre
28
When are CO, SV, & HR highest?
at the end of exercise
29
Reactive hyperemia
^ BF after exercise is finished
30
Chronic CV responses to RT
-Resting HR, no change or decrease (sedentary more change than fit person) -Resting BP no change or decrease slightly (sedentary more change than fit person) -SV increases in absolute magnitude -Cholesterol & LDLs may not change or slightly decrease while HDL's may increase
31
Chronic CV Responses to RT
not effective in improving VO2 max - no change in capillary density - no O2 extraction improvement - very high blood lactate concentrations - low intensity high volume training may increase capillarization and improve O2 extraction
32
What type of RT may improve O2 extraction
``` cross fit plyos circuit training aerobic endurance training ```
33
Chronic Ventilatory Response to RT
- unaffected or only moderately improved by anaerobic training - tidal volume increases - breathing frequency increase - improved ventilation efficiency (how much O2 we get out of each breath)
34
Acute CV response to Aerobic Training
^CO, SV, HR, SBP, BF to active muscles, decrease DBP
35
Stroke Volume
regulated by teh end diastolic volume and action of catechoamines (epi, norepi, dopamine, ^BP&HR, fight or flight response) -amount of blood available to pump @end of diastole
36
Frank Starling Mechanism
force of contraction is a function of the length of the fibres of the muscle wall -more blood returning to heart=more stretching of wall=^contraction force
37
What muscles are suppressed during exercise?
- digestive & immune system - blood flow to active muscles is increased by dilation of local arterioles, but decreased to other organ system via constriction of arterioles
38
Acute Respiratory Response to Aerobic Training
^O2 delivery to tissues, CO2 return to lungs, minute ventilation (frequency and tidal volume) and diffusion capacities
39
Breathing rate at rest
12-15 breaths per minute
40
Breathing rate during exercise
35-45 breaths/min
41
Chronic Responses to Aerobic Training
- ^max CO & VO2max - slower resting and submax HR - increased capillarization - improved ventilation efficiency - increased O2 extraction - OBLA occurring at higher percentage of aerobic capacity
42
OBLA
Onset of blood lactate accumulation - ^ during ^duration and intensity of aerobic metabolism - more lactate being produced than what is being removed
43
Altitude Acute Physiological Changes
-hyperventilation -increased HR -increased CO @ >1200m
44
Acclimatization effects
- rates return to normal - ^RBC production, ^ Hb - ^ O2 diffusing capacity - ^capillarization - pH balance * acclimatization is VERY important 10-14 days
45
Hyperoxic Breathing
breathing O2 rich gas mixtures during rest or following exercise - ^amount of O2 carried by blood - likely more helpful for individuals at higher altitudes or those with respiratory illnesses/disorders - help ^SpO2% @ sea level SpO2 is already high
46
Blood Doping- how to
artificially increases RBC mass - infusion of own RBC - infusion of someone else's RBC - EO erythropoetin (stimulates RBC production)
47
Effects of Blood doping
- Max O2 uptake increases | - improved altitude performance, thermoregulation (heat and cold stress) due to more O2 carrying capacity
48
Risks of Blood Doping
``` flu like symptoms ^BP Stroke heart attack thrombosis pulmonary embolism it is also ILLEGAL ```