lecture 5/6 Flashcards

1
Q

most anterior part of cerebellum vermis

A

nodule

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2
Q

function cerebellum

A

receives proprioception
coordinates balance
eye hand coordination
predicts consequences of movement

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3
Q

T/F cerebellum is coordinator and predictor of corticol output

A

true

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4
Q

T/F cerebellum not needed for skilled manipulation of mm

A

false, its critical

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5
Q

T/F cerebellum used for skilled manipulation of mental concepts

A

t

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6
Q

cerebellum mutism

A

not able to speak, can’t predict

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7
Q

information the cerebellum gets

A

cortical
proprioception
vestibular

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8
Q

vestibular information

A

project to paravermis / flocculonodular lobe
position head/ body in space
orient eye movements during locomotion

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9
Q

proprioceptive information

A

ia, II fibres from mm to
Ib from golgi tendon organs

from clarkes column

terminate in anterior lobe /vermis

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10
Q

anterior lobe associated w

A

limbs

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11
Q

vermis associated w

A

limb

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12
Q

cortical information

A

cortex projects to continue nuclei

fine motor UE, hand dexterity / eye coordination

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13
Q

olivocerebellar fibers

A

afferents from olive project to entire cerebellum cortex

climbing fibres

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14
Q

outflow from cerebellum

A

via deep cerebellar nuclei
dentatorubrothalmic tract
to vestibular nuclei / olives

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15
Q

dentatorubrothalmic tract

A

from dentate to red to thalamus to cortex

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16
Q

archicerebellum =

A

vestibulocerebellum

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17
Q

paleocerebellum =

A

spinocerebellum

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18
Q

neocerebellum =

A

cerebrocerebllum

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19
Q

vestibulocerebellum

A

flocculonodular lobe and paravermis

feedback to vestibular nuclei / SC

trunk control

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20
Q

spinocerebellum

A

vermis and anterior lobe

feedback to cortex/ SC

extremities

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21
Q

cerebrocerebllum

A

posterior lobe

topographical of extremity
eye movement
speech
coordination of movement

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22
Q

cerebellar loops

A

vestibulocerebllear
spinocerebellar
cerebrocerebllar

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23
Q

what do cerebellar loops allow

A

coordinated balance smooth movement

anticipation of movement

predictions that feedback to the loop

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24
Q

what fine tunes ongoing movements

A

spinocerebllar loop

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25
information from __ to pontine uncle cross over to
cortex | contralateral cerebellum
26
red nucleus
relay nucleus
27
T/F olives project to all of the cerebellum
true
28
what calculates feed forward loops
olives
29
what does automation of cortical output
spinocerebellar loop
30
midline cerebellar disease
gait difficulty imbalance abnormal head posture oculomotor dysfunction
31
lesion to flocculonodular node
truncal ataxia | nystagmus
32
lateral cerebellar disease
posterior lobe function ``` cerebellum mutism eye / speech movement coordinate movements dysmetria dysdiokinesiea ```
33
dysmetria
over n under shoot
34
dysdiokinesiea
rapid agonist antagonist movements
35
lesions to anterior lobe
affects spinocerebllear input | gait ataxia
36
ethanol is toxic to what
purkinje cells
37
ethanol effects which lobe
anterior
38
is ethanol and gait ataxia reversible
usually
39
when is ethanol gait ataxia not reversible
chronic alcoholic
40
T/F cerebellum related to emotion / cognition
yes
41
cerebellum emotion / intellectual processing
judge facial expression language (grammar adjustments) neuropsychiatric manifestions
42
neuropsychiatric manifestions
exaggerated or diminution of repose to environment in automatic processing of emotions
43
T/F cerebellum effects communciton with non motor cognitive association areas
yes
44
interaction between cerebellum / cognitive cortical networks
interaction with cerebral executive control | interaction with default mode network
45
function basal ganglia
modulate voluntary motor activity
46
three circuits in basal ganglia
motor associative limbic
47
motor circuit
controls body and eye movements direct/indirect pathway
48
associative circuit
higher level cognitive function (planning)
49
limbic circuit
emotional / motivational processing
50
activity in basal ganglia encodes for
decision to move direction/ amplitude of movement motor expression of emotion efficiency of movement (procedure)
51
T/F thamalus under acute inhibition when we are not moving
false chronic
52
release inhibtion model components
direct and indirect pathway
53
direct pathway
release tonic inhibition of thalamus | leads to more execution of motor cortex
54
indirect pathway
inhibit output from thalamus | less execution of motor cortex
55
input to
caudate and putamens
56
output from
globus pallidus
57
what pathway facilities target orientated movements
direct
58
which pathway impacts potentially competing movements
indirect pathways
59
T/f direct and indirect pathways are simultaneous
true
60
T/F direct and indirect pathways do not provide fine balance for cortical output
false, they do
61
diseases related to lesions in basal ganglia
parkinsons ballism huntingtons tourettes, OCD
62
parskinsons
inhibition of motor output weaker direct pathway decrease in movement and facial expression
63
ballism / huntingtons disease
excessive motor output
64
T/f basal ganglia related to nearo disorders
yea Tourettes and OCD
65
ballism
sudden uncontrolled flinging of movements of extremities
66
underlying cause of ballism
stroke of contralateral subthalmic nucleus
67
hemi ballism
loss indirect pathway more involuntary motor output can't suppress the extra movements
68
huntingtons
degeneration of striatum hyperkinetic movement disorder direct n indirect pathway effected
69
parts of striatum
caudate and putamen
70
damage to striatum effects
direct n indirect pathway
71
hypokinesia akinesia and hypminmia loss or decrease associated with what disease
parkinson
72
parsons does what to inhibition
inhibits it less (so more left over) | excited the inhibition (adding even more)
73
t/f damage to substance nigra related to Parkinson's
yes
74
how do basal ganglia interact w rest of brain
the three circuits
75
mask face or rich in dopaminergic neutrons what circuit
limbic
76
are all basal ganglia circuits working seperate or at once
at once