Lecture 5 : Why We Stretch Flashcards
(62 cards)
5 factors that limit our range of motion
- active structures
- passive structures
- viscosity
- central nervous system
- discomfort
what are active structures that limit our ROM and they are the only part of the body that can actively ….
muscle fascicles, active tissues in muscle are the only parts of the body that can actively change their tension and length
what are passive structures that limit our ROM and how can their length change
tendons, aponeuroses, joint capsules and ligaments contribute to ROM restrictions with passive muscle elongation
can only change length or tension when we passively put load on them
how does viscosity effect flexibility (ground substance : what is it and what does it do)
ground substance in connective tissues is a gel like substance which mechanical properties change based on temperature and other things
this can increase or decrease resistance to movement
how does the central nervous system affect ROM
a highly activated central nervous system could increase muscle tonus and with a less relaxed muscle inhibit flexibility
how does the discomfort affect ROM
pushing a joint to its maximum ROM can be uncomfortable, the ability to tolerate pain may allow some individuals to stretch further than others
what are easily stretched muscles called and what are examples
biarticular muscles e.g hamstrings, gastroc, rectus femoris etc
what are anatomically impossible muscles to stretch called and what are examples
monoartciular muscles e.g tibialis anterior, masseter, intercostals etc
myofibrils can be stretched to what length without damage
twice their resting length without damage
what is titin and what is it stretching ability
curly protein bands that sit at the end of your muscle fibrils
massive stretching ability
what is fibronectin and what does it do
protein that links to the surface fibre of the muscle, tends to stimulate cells on the surface of the fibre
what length of the muscle is the point from which we can usually develop the most active force
at resting length is where the most binding of actin and myosin occurs
what occurs beyond resting length of the muscle in terms of actin and myosin and force generated
beyond this length we start elongating, fewer actin and myosin are connected, we are no longer able to produce as much force actively
passive tension increases as active force is reduced
what allows us to maintain a certain amount of force production as the muscle elongates and we aren’t able to produce as much force actively
passive tension increases
how to train the active component of muscle
resistance training
how to train the passive and active component of muscle
eccentric movements
what does the evidence say about stretchiing increasing the range of motion
yes acute and chronic stretching has been shown to increase the range of motion in hamstring and gastroc
but there are trivial results regarding impact to muscle stiffness
what are the two acute effects of stretching
- decrease viscosity
- alter muscle tone
what are the thixotropic effects of acute stretching and how this decreases viscosity
thixotrophy is a time dependent property of materials that become less viscous (more fluid like) when moved or stressed but return to more gel like state when left undisturbed
in the context of muscles, tendons and connective tissues thixotrophic effects influence …
stiffness, flexibility and resistance to movement
without movement, tissues become ….. due to ….
become stiffer, due to the gel like behaviour of intracellular and extracellular components, including actin myosin cross bridges and connective tissue ground substance
how does the term move it or lose it link to ground substance
ground substance in tissues becomes stiff with periods of inactivity
as viscosity decreases what happens to hysteresis
hysteresis also decreases (tissues become more efficient)
in the loading and unloading curve (stress and strain) what does the area under the curve indicate
amount of energy being returned to the body