Lecture8 Flashcards

1
Q

List the 3 components of emotion

A

Physiological changes (not always conscious), subjective feeling & associated behaviour (e.g. facial expression)

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2
Q

Define emotion

A

A feeling state characterised by physiological arousal, expressive behaviours, & a cognitive interpretation

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3
Q

What are some physiological components of emotion?; Expressive components?;
Cognitive components?

A

Heart rate, breathing & sweating;
Facial expressions, body movements & voice;
Beliefs & appraisals

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4
Q

What learnt factors depend on how we control our emotions?

A

Personality, past history/culture, situational factors & mood

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5
Q

How does the autonomic system deal with states of arousal?

A

Sympathetic - fight or flight; parasympathetic - calming down; enteric - visceral (butterflies/sick to stomach)

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6
Q
The sympathetic nervous system dilates...; 
Inhibits...; 
Increases...; 
Stimulates...; 
Secretes...; 
Relaxes...
A
Pupils; 
Salivation, digestion & genitals; 
Respiration & heartbeat; 
Glucose release; 
Adrenaline & noradrenaline; 
Bladder
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7
Q

The parasympathetic system constricts…;
Stimulates…;
Slows…;
Contracts…

A

Pupils;
Salivation, gall bladder, digestion & genitals;
Respiration & heartbeat;
Bladder

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8
Q

What physiological responses are typically measured on a polygraph?

A

Galvanic skin response; pulse; blood pressure; breathing & fidgeting

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9
Q

What is the theory about the order of emotions & responses according to folk psychology?

A

Perceived event (stimulus) leads to emotional experience (e.g. fear response) which leads to physiological & behavioural changes (autonomic arousal)

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10
Q

What is the James-Lange theory about the order of emotions & responses?

A

Perceived event (stimulus) leads to physiological behaviour (arousal) which leads to emotional experience (e.g. conscious feeling of fear);

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11
Q

In regards to the James-Lange theory, William James says…;
Carl Lange says…

A

Physiological arousal causes emotion;

Physiological arousal IS the emotion

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12
Q

What are some problems with the James-Lange view?

A

Visceral responses are not specific enough to determine particular emotions, they would take too long to cause emotion, they can occur without emotions & emotions can occur without visceral responses

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13
Q

How did Maranon find that visceral responses can occur without emotions?;
How did Cannon find the same thing?

A

Injecting subjects with adrenaline produced physiological responses but no clear emotional states;
Disconnecting viscera from CNS by removing SNS in cats has no effect on emotional expressions

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14
Q

What is the Facial Feedback-hypothesis about emotions?; What’s an example of the strong version of this?;
What’s the weak version?

A

Expressing a particular emotion puts us into the corresponding emotional state (similar to James-Lange); First you laugh, then you infer “that’s funny” or “I’m happy”;
Facial expression modulates emotion (i.e. fake it till you make it)

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15
Q

How was the Facial Feedback-hypothesis tested?

A

By asking participants to contract/relax facial muscles important for expressing emotion, then testing intensity of emotion when corresponding muscle groups are active

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16
Q

Why did Duchenne stimulate facial muscles to create grotesque facial expressions?;
What marks a Duchenne smile?

A

To find out which muscle groups are involved in genuine expressions;
Lines around & under the eyes, raised cheeks, naso-labial folds & corners of mouth pulled back & up

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17
Q

What is the Corrugator muscle involved in?
What does the Orbicularis Oculi muscle control?
The Zygomaticus muscle?

A

Frowning;
The eye when smiling;
The mouth when smiling

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18
Q

How does a facial EMG measure expression?;
Where do positive emotions increase activity?;
Where do negative emotions increase activity?

A

Electrodes placed on the face record activity in various muscles;
In the cheeks;
In forehead & brow areas

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19
Q

What evidence did Hennenlotter et al. find about botox injections?

A

Injecting botox into frown muscles decreases activity in brain regions that process emotions (amygdala & brain stem)

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20
Q

What did Havas et al. find about botox injections on subjects’ reading?

A

Slow reading of angry & sad sentences but not happy sentences

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21
Q

Physiological states & expressions / behaviour can modulate emotions, so it’s worth trying to…

A

Fake it until you make it

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22
Q

What is the Cannon-Bard theory about the order of emotions & responses?;
Therefore, emotions are…

A
Perceived event (stimulus) processed in subcortical brain activity leading to both physiological & behavioural responses (autonomic arousal) & emotional experience (conscious feeling); 
Separate from ANS response & behaviour
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23
Q

Give an example of step 1 of Cannon-Bard’s Thalamic / Hypothalamic theory of emotion

A

Dog growls (stimulus); thalamus is activated & sends simultaneous messages to cerebral cortex (I am afraid - emotion) & SNS (My heart is beating fast, I’m breathing hard - bodily changes)

24
Q

What’s step 2 of the Cannon-Bard theory?;

A

Hypothalamus evaluates the emotional relevance of environmental events

25
What's step 3A of the Cannon-Bard theory? | Step 3B?
Cortex conscious experience - projections from hypothalamus to cortex mediate conscious experience; Brain stem emotional response
26
Beginning at the olfactory bulb, name the main areas of the Limbic System in a clockwise direction
Cingulate gyrus, thalamus, fornix, hypothalamus, mamillary body, hippocampus, amygdala
27
What progress was made with rhesus monkeys when assessing emotional behaviours?
Fear conditioning
28
In classical conditioning, US is...; UR is...; CS is...; CR is...
Unconditioned Stimulus (hardwired); Unconditioned Response; Conditioned Stimulus (learnt); Conditioned Response
29
What evidence reveals that the amygdala is important for fear learning?; What do fMRI scans show?
Damage to the lateral nucleus of the amygdala interferes with fear conditioning; Amygdala activity changes during conditioning, & these correlate with thalamus activity but not cortical activity
30
What brain region is important for differential conditioning?; What about contextual conditioning?
Cortex; | Hippocampus
31
How is context conditioning applied?
No distinct CS, environment serves as CS; context A: CS-US, context B: CS (2 different environments)
32
What's an example of differential conditioning?
Animal receives electro shock upon tone in box A but not in box B
33
Explain LeDoux's Low Road to emotional processing
Sensory input to the thalamus straight to amygdala - activation of emotions before cognitive processing (unconscious); amygdala controls physiological & behavioural emotional response - no cortex involved
34
Explain LeDoux's High Road to emotional processing
Sensory impulses sent from the thalamus to the neocortex for cognitive processing (perceptions & interpretations), then activation of emotions in amygdala (modulated response)
35
From the thalamus, the amygdala signals a threat which triggers the hypothalamus. What is the process of neurotransmitter activity from this point?; What occurs simultaneously?
Hypothalamus releases corticotropin-releasing hormone (CRH) which activates the pituitary gland & secretes adrenocorticotropic hormone (ACTH) & cortisol is released (stress hormone); Activation of adrenal gland, which releases adrenaline / noradrenaline (increasing sympathetic response)
36
What did Feinstein et al. find when testing patients with focal bilateral amygdala lesions for fear?;
No conditioning to aversive stimuli, failure to recognise fearful faces & absence of fear when exposed to fear-provoking stimuli
37
How did Feinstein et al. test for fear responses with 3 amygdala lesioned patients?; What were the results?; What did the controls show differently?
The 3 patients & 12 controls inhaled CO2; Fear or panic was triggered in all 3 patients & physiological response was the same or heightened; Anticipatory fear response (they could predict)
38
As well as for conditioning to fear, the amygdala is important for...; What isn't it necessary for?; Who's theory can't explain fear response?; Brain structures that by-pass the amygdala can also...
Anticipation or detection of threat; Experiencing emotions (fear/panic); LeDoux's; Evoke fear
39
Provide 4 explanations as to why extinction is not "passive forgetting"
Spontaneous recovery (emergence of conditioned fear some time after extinction); renewal (reappears in a different context); reinstatement (emergence after encounter with CS alone after successful extinction); rapid reacquisition (faster than learning)
40
What happens with active suppression of fear responses during extinction (learning)?
CS becomes ambiguous
41
What happens at a neural level with associative learning?
Changing connection strengths between neurons (synaptic weights); changing wiring between neurons (plasticity)
42
According to Hebbian's learning rule, what is Synaptic strength?; What is plasticity?
A synapse between 2 neurons is strengthened when the neurons on either side of the synapse (input & output) have highly correlated outputs (both activated or both inhibited); What fires together wires together - neurogenesis & changes in wiring of synaptic connections
43
Name 2 ways of changing synaptic weights
Coincident activity in pre- & post-synaptic neuron strengthens connection; modulatory interneuron can strengthen connections when activity coincides with pre-synaptic neuron
44
What do neuromodulators do?; What are 2 important neuromodulators?; They don’t necessarily depolarize a cell directly but they...
Modulate firing rates (sensitivity) of enervated neurons; Dopamine & Serotonin; Increase their sensitivity & have longer-lasting effects than neurotransmitters
45
Extinction is an active process involving what?; | What doesn’t happen during extinction?
Changes in synaptic weights (similar to learning); | True unlearning of fear response (it’s latently there)
46
Give 2 examples of research approaches to assess detection of threat vs. threat learning
Detection: visual search task; learning: resistance to extinction (conditioning to threatening vs. non-threatening CS+, then compare extinction for threat CS+ & non-threat CS+)
47
What did Seligman & Maier find in their Learned Helplessness study with dogs?; What did Hiroto find in the equivalent human experiment with loud noise?
Even though the dogs could escape electric shocks by jumping over a partition, 80% of the group 2 dogs never jumped; The same results - those in uncontrollable noise condition didn’t learn to move the lever & stop the noise
48
Learned helplessness is also known as...; How is this linked to depression?; What is learned helplessness an example of?
Retardation of learning, learning transfer or consequences of learning; From learning that outcomes are uncontrollable; How past learning / experience directly influences how a situation is perceived & how this perception leads to certain emotions / mood states
49
Give examples of how attributions determine the development of helplessness
Stable v.s unstable attributions > chronic vs. acute helplessness; global vs. specific attributions > broad vs. narrow helplessness; internal vs. external attributions > lower vs. higher self-esteem
50
Explain the Schachter & Singer 2-factor theory of emotion
Event (stimulus) > physiological changes (autonomic arousal) > cognitive labels (appraisal) > emotional experience & behaviour (conscious feeling)
51
In the 2-factor theory, what equals emotion?; Emotion only occurs if...; How can this be tested?
Arousal + cognition; Body is aroused; a reason for arousal is located; the labeling of arousal determines emotion; arousal without cognition leads to no emotion; By making people attach a wrong cognitive label (misattribution) to arousal state
52
In the experiment where participants were injected with Epinephrine (adrenaline), & either informed or misinformed about arousal effects followed by friendly or angry confederate, what were the results?
In euphoric condition, misinformed were happiest & informed the least happiest or angriest; informed group attributed arousal to the drug & were least effected by the context
53
In Dutton & Aron's misattribution experiment, where participants crossed either an unsafe or safe bridge, followed by a survey with attractive experimenter, who were more likely to call her & ask for a date?; How could this be interpreted?
Those who crossed the unsafe bridge (50%) with only 12.5% of those who crossed the safe bridge; People crossing the scary bridge misattributed arousal (fear) to the experimenter (attraction)
54
Attributions can determine...; What can arise from misattribution?; What's a similar theory to this?
The emotions we form or experience; Misguided emotions; Cognitive appraisal theory (cognition causes arousal)
55
Explain the order of different theories of emotion over history
Focus on ANS, visceral responses & behaviour; brain structure & localisation (thalamus, hypothalamus & amygdala); conditioning to alter emotional responses to particular stimuli (& extinction); cognitive appraisal in defining emotions (or arousal)