Lower Respiratory Disorders Flashcards

(35 cards)

1
Q

Pulmonary Embolism (PE)

A

Emboli; mobile clots that travel through circulatory system
Blockage in a pulmonary artery by an embolic thrombus (fat or air embolus, or tissue from a tumor)
Travel to ever-smaller blood vessels until it lodges & obstructs perfusion of the alveoli
More than 90% arise from deep vein thrombosis (DVT) of the legs
Usually affects lower lobes d/t high volume of blood flow
10% of patients die within the first hour
Treatment with anticoagulants reduces mortality to less than 5%

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2
Q

PE Risk Factors

A

Immobility or reduced mobility
Surgery in the last 3 months
History of DVT
Malignancy
Obesity
Oral contraceptives & hormone therapy
Heavy cigarette smoking
Prolonged air travel
Heart failure
Pregnancy
Clotting disorders

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3
Q

PE Clinical Manifestations

A

Varied & nonspecific, making it difficulty to diagnose
Dyspnea
Hypoxemia (if severe, change in mental status)
Tachypnea
Cough
Chest pain
Hemoptysis
Crackles and/or wheezing
Tachycardia
Fever
Syncope
Massive PE = hypotension & shock

Complications
Pulmonary Infarction
Death of lung tissue
May become infected & abscess may develop

Pulmonary Hypertension
from a massive or recurrent PE
Results from hypoxemia or damage to 50% of area’s normal pulmonary bed

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4
Q

PE Diagnostic Studies

A

Find under-lying cause; DVT or AFIB?)

Spiral (helical) CT scan w/ IV contrast
Most frequently used to diagnose PE
Ventilation-perfusion (V/Q) lung scan
Used for those allergic to IV contrast
Mismatch of perfusion & ventilation indicates PE

EKG monitoring
Labs:
CBC
BMP
ABG’s
PTT
PT / INR
Troponin
D-dimer
Measures fibrin fragments
Not always accurate
Chest X-Ray
Venous ultrasound
Pulmonary angiography

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5
Q

PE Collaborative Care

A

Immediate treatment as soon as PE suspected
Bedrest; Semi-Fowler’s position
02 by NC or mask
Cardiac monitoring, VS, 02 level, ABG’s, breath sounds
Fibrinolytic agent
IV Heparin drip (RN management by protocol)
Lovenox (Low-molecular-weight Heparin)
Warfarin (Coumadin)
Monitor PTT and/or PT/INR levels
Opioids for pain relief
Medications PRN anxiety
Inferior Vena Cava filter
Inserted via femoral vein, placed at level of diaphragm
Pulmonary embolectomy

Patient Teaching
Risk factors
Long-term anticoagulant therapy
Frequent blood draws for PT/INR monitoring
Coumadin adjustments
Dietary considerations

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6
Q

Pneumonia

A

Acute infection of lung parenchyma
Gram-negative bacilli
8th leading cause of death in 2018
Impacted by discovery of Sulfa & Penicillin
Despite new anti-microbial agents, morbidity & mortality still significant

Risk Factors
Immunosuppressive diseases or debilitating illness
Increased risk of aspiration
Decreased cough & epiglottal reflexes, tube feedings
Impaired muco-ciliary mechanism
Pollution
Smoking
Upper respiratory infections
Tracheal intubation
Age >65 years
Malnutrition
Inhalation of microbes (Mycoplasma/fungus)
Spread from primary infection elsewhere in body

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7
Q

Opportunistic Pneumonia

A

Altered immune system
Severe protein-calorie malnutrition
HIV / AIDS
Radiation therapy
Chemotherapy
Long-term corticosteroid therapy
Pneumocystis jiroveci (fungus)– needs a human host
Cytomegalovirus (CMV)

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8
Q

Community-Acquired Pneumonia (CAP)

A

7th leading cause of death for those over 65 years old
Pre-hospital onset or within first 2-days of admit
Highest incidence in winter
Smoking important risk factor
May present only with dyspnea & fever while lung tissue is necrotized
Viral manifestations vary:
Fever, chills
Dry, non-productive cough
Extra-pulmonary symptoms

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9
Q

Hospital-Acquired Pneumonia (HAP)

A

48-hours or more after admission
Early (5-days or less post admission)
Late (more than 5-days post admission)
Nosocomial infection with highest mortality
Pseudomonas
Enterobacter
S. Aureus
S. Pneumoniae

Risk factors:
Immunosuppression
General debility
ET Tube (VAP)
Fungal/aspiration pneumonia
Loss of consciousness
Gag/cough reflexes
Tube feedings

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10
Q

CAP

A

Gradual symptom onset
Dry cough
Headache
Malaise / fatigue
Sore throat
N/V/D (S. aureus)

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11
Q

HAP

A

Sudden onset symptoms
Fever
Chills
Productive cough
Purulent sputum
Pleuritic chest pain
Confusion or stupor in older or debilitated patient

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12
Q

Clinical Manifestations CAP/HAP

A

Cough – non-productive or productive – green, yellow, bloody
Fever/chills/shaking
Dyspnea
Tachypnea
Pleuritic chest pain
May initially appear as influenza, then respiratory sypmtoms
Confusion or stupor r/t hypoxia
Hypothermia (in elderly)
Diaphoresis, anorexia, fatigue, myalgia, headache, abd pain
Rhonchi and Crackles
Dullness to percussion over affected area

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13
Q

Diagnostic Studies CAP/HAP

A

H & P
Chest X-Ray
Vital Signs - Sp02 and ABG’s
CBC, CMP, Blood Cultures
Sputum Culture – prior to Antibiotic therapy

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14
Q

Collaborative Therapy
CAP/HAP

A

Antibiotic therapy: Multi-drug resistant pneumonia is a major problem
Increased fluid intake (3L/day)
Limit activity and rest
Antipyretics
Analgesics
02 therapy
Influenza & Pneumonia Vaccination - pneumovax

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15
Q

Complications from Pneumonia

A

Pleurisy or pleural effusion
Atelectasis
Persistent infection
Lung abscess
Empyema
Pericarditis
Endocarditis
Arthritis
Meningitis
Pneumothorax
Sepsis
Respiratory failure

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16
Q

Nursing Assessment Pneumonia

A

History
Lung cancer
COPD
Diabetes mellitus
Debilitating disease
Malnutrition
AIDS
Use of antibiotics, corticosteroids, chemotherapy, immunosuppressants
Recent abdominal or thoracic surgery
Smoking, alcoholism, respiratory infections
Prolonged bed rest

Physical Assessment
Dyspnea
Nasal congestion
Pain with breathing
Sore throat
Muscle aches
Fever
Restlessness or lethargy
Splinting affected area
Tachypnea
Tachycardia
Asymmetric chest movements
Use of accessory muscles
Crackles
Green or yellow sputum

17
Q

Nursing Considerations for Pneumonia

A

Tachycardia
Changes in mental status
Leukocytosis
Abnormal ABGs
Pleural effusion
Pneumothorax on x-ray

18
Q

Pneumonia: Patient Teaching

A

Nutrition, hygiene, rest, regular exercise to maintain natural resistance
Prompt treatment of upper respiratory infections
Offer / encourage influenza & pneumococcal vaccines – New: COVID vaccine
Reposition every 2-hours
Assist with eating, drinking & taking medications
Emphasize need to finish course of antibiotics
Teach drug-drug interactions
TCDB / “Pulmonary toilet”
Strict asepsis, hand-washing

19
Q

Lung Cancer Facts

A

Leading cause of cancer-related deaths in U.S.
Estimated 221,000 new cases diagnosed annually
High mortality, low cure rate – although treatment advancing
Early detection is important

Risk factors
Smoking – 80-90%
Pollution
Radiation
Occupational
Asbestos
Coal dust
Nickel
Uranium
Chromium
Formaldehyde
Arsenic

20
Q

Types of Lung Cancer

A

Non-small cell (NSCLC) 80%
Squamous cell
Large bronchi
Slow speed- takes 8 to 10 yrs for a tumor to reach 1 cm
Adenocarcinoma
Alveoli
Moderate speed
Most common in nonsomokers
Large cell
Rapid speed
Highly metastatic

Small cell (SCLC) 20%
Larger airways
Most malignant
Early metastasis
Associated endocrine disorders
More sensitive to chemo
Poorer prognosis
Associated with paraneoplastic Syndrome

21
Q

Diagnostic & Therapy

A

Chest X-Ray – 5% incidentally find cancer
Sputum cytology
Bronchoscopy
CT scan, MRI, PET, VATS,
Mediastinoscopy
Trans-bronchial or percutaneous fine-needle aspiration
Surgery
Radiation and/or Chemotherapy

22
Q

Clinical Manifestations

A

Early
Persistent cough
Blood tinged sputum
Dyspnea or wheezing
Chest pain
Later
Anorexia
Fatigue
Weight loss
Nausea / vomiting
Hoarseness
Palpable lymph nodes in neck or axillae

23
Q

Staging & Signs of Metastasis

A

Hoarseness
Recurrent laryngeal nerve
Dysphagia
Esophageal compression
Superior vena cava syndrome
Venous obstruction
Shortness of breath
Facial, arm, trunk swelling
Distended neck veins
Chest pain
Venous stasis

Mediastinal Lymph Node Involvement
Vocal cord paralysis
Dysphagia
Diaphragmatic paralysis
Phrenic nerve compression
Vena cava compression
Pleural effusion

24
Q

Para-neoplastic Syndrome

A

Immune response against tumor mediated by humoral factors
Hormones & cytokines excreted by tumor cells
Hypercalcemia
Syndrome of inappropriate antidiuretic hormone secretion (SIADH)
Dilutional hyponatremia
Anemia
Leukocytosis
Hypercoagulation disorders
Neurologic syndromes

25
Treatment of Lung Cancer
Radiation therapy Chemotherapy- Primary treatment for SCLC and adjuvant to surgery in NSCLC Surgery Segmental, “wedge” resection Lobectomy Pneumonectomy Immunotherapy – Can be used in people with NSCLC whose cancer starts growing again after chemo Pneumonectomy Chest tubes may, or may not be placed If placed, it is clamped & only released by surgeon to adjust volume of sero-sanguinous fluid allowed to fill space Daily CXR Position patient operative-side DOWN Pain management
26
Pneumothorax
Caused by air entering pleural cavity Positive pressure in pleural space causes lung to partially or fully collapse Increased air in pleural space equals reduced lung volume Open: opening in chest wall Penetrating trauma—sucking chest wound Closed: no external wound Suspect pneumothorax with chest wall trauma Manifestations Small pneumothorax Mild tachycardia and dyspnea Large pneumothorax Respiratory distress Absent breath sounds over affected area Diagnostic Study: Chest x-ray
27
Types of Pneumothoraces
Spontaneous—rupture of blebs Can occur in healthy or chronically ill persons (COPD, asthma, cystic fibrosis, pneumonia) Risk factors: Tall, thin, male, family history, or previous spontaneous pneumothorax Iatrogenic —medical procedures Biopsies, subclavian catheter insertion, ventilator, esophageal trauma Tension pneumothorax Accumulation of air in pleural space that cannot escape results in increased intrapleural pressure Causes mediastinal shift and hemodynamic instability: reduced venous return and reduced cardiac output Medical emergency – may be fatal if not corrected
28
Types of Pneumothoraces
Spontaneous—rupture of blebs Can occur in healthy or chronically ill persons (COPD, asthma, cystic fibrosis, pneumonia) Risk factors: Tall, thin, male, family history, or previous spontaneous pneumothorax Iatrogenic —medical procedures Biopsies, subclavian catheter insertion, ventilator, esophageal trauma Tension pneumothorax Accumulation of air in pleural space that cannot escape results in increased intrapleural pressure Causes mediastinal shift and hemodynamic instability: reduced venous return and reduced cardiac output Medical emergency – may be fatal if not corrected
29
Hemothorax and Chylothorax
Hemothorax Blood in pleural space Treat with chest tube Hemopneumothorax Blood and air in pleural space Chylothorax Lymphatic fluid in pleural space Treat conservatively or with Octreotide Refractory options: surgery or pleurodesis
30
Interprofessional Care
Dependent on severity, underlying cause and hemodynamic stability Emergency treatment—Cover wound with dressing secured on 3 sides If impaled object in place, stabilize it with a bulky dressing but do not pull it out Treatments Chest tubes with water-seal drainage Other: partial pleurectomy, stapling, or pleurodesis Tension pneumothorax Needle decompression— immediate Chest tube and water-seal drainage Inspiration: pulls dressing against wound so air cannot enter pleural space Expiration: dressing pushes out and air escapes
31
Chest Tubes and Pleural Drainage
Chest tubes Drain pleural space Reestablish negative pressure Allow lung to expand Small: air Medium: fluid Large: blood
32
Chest Tube
Collection device for fluid, air, or blood from chest cavity Three basic compartments 1st compartment or collection chamber Fluid stays in; air vents to 2nd compartment 2nd compartment or water-seal chamber Contains 2 cm of water; acts as one-way valve; air goes in, bubbles out, but can’t go back to patient 3rd compartment or suction control chamber Uses column of water to control suction from regulator Bubbling in water-seal chamber Brisk at first, eventually disappears as lung expands Intermittent with exhalation, coughing, or sneezing Tidaling in water-seal chamber Fluctuation of water with pressure changes during respiration Disappears as lung re-expands If stops suddenly, check for occlusion Suction control chamber Wet suction Amount of water in chamber (20 cm) controls suction Excess suction from source vented Usual suction order = −20 cm H2O Adjust suction until gentle bubbling in third chamber Dry suction—no water (less noise) Dial regulator to pressure; visual alert
33
Nursing Management: Chest Drainage
Drainage system Keep tubing loosely coiled Keep connections tight; taped Observe: tidaling, bubbling, air leak, fluid levels Patient’s clinical status Assess: Vital signs, lung sounds, pain Drainage amount Drainage site infection Subcutaneous emphysema Encourage: Deep breathing/Incentive spirometry Range-of-motion exercises Chest drainage Keep below chest Mark and measure drainage Report greater than 200 mL/hr in first hour and 100 mL/hr thereafter; replace unit when full Avoid overturning unit Breakage of unit Place distal end of chest tube in 2 cm water in sterile container; replace unit No milking or stripping chest tubes Dry suction chest drainage – Most common type Wet suction chest drainage Monitor: Water levels Suction at—20 cm H2O Gentle bubbling Chest tube dressings Change according to agency policy and procedure Petroleum gauze Aseptic technique Monitor for infection Document Clamping chest tubes Not advocated during transport or disconnection due to risk for tension pneumothorax May clamp briefly to change drainage unit
34
Removal of Chest Tubes
When lungs reexpanded and drainage minimal Premedicate prior to removal Valsalva maneuver during removal Apply occlusive dressing Chest x-ray Monitor for respiratory distress
35
Abnormal Lung Sounds
Crackles (rales) Snorous Wheezes (rhonchi – low pitched) Sibilant Wheezes (high pitched) Stridor