Wound assessment and healing Flashcards

(38 cards)

1
Q

Acute inflammatory phase

A

Healing occurs in 2 to 3 weeks, usually leaving no residual damage
Neutrophils are predominant cell type at site of inflammation

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2
Q

Sub-acute inflammatory phase

A

Has same features as acute inflammation but persists longer

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3
Q

Chronic inflammatory phase

A

May last for years
Injurious agent persists or repeats injury to site
Predominant cell types involved are lymphocytes and macrophages
May result from changes in immune system (e.g., autoimmune disease)

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4
Q

Inflammatory Response

A

Final phase of inflammatory response is HEALING

Healing has 2 major components
Regeneration
Repair

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5
Q

Wound Healing: Regeneration

A

Replacement of lost cells and tissues with cells of same type
Stable cells regenerate in response to injury
Liver, bone, kidney & pancreas

Constantly dividing cells that rapidly regenerate
Skin, bone marrow, lymphoid organs, mucous membrane cells of urinary, reproductive, and GI tracts

Permanent cells do not divide
Neurons replaced by glial cells or stem cells
Skeletal & cardiac muscle are repaired with scar tissue

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6
Q

Wound Healing: Regeneration

A

Replacement of lost cells and tissues with cells of same type
Stable cells regenerate in response to injury
Liver, bone, kidney & pancreas

Constantly dividing cells that rapidly regenerate
Skin, bone marrow, lymphoid organs, mucous membrane cells of urinary, reproductive, and GI tracts

Permanent cells do not divide
Neurons replaced by glial cells or stem cells
Skeletal & cardiac muscle are repaired with scar tissue

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7
Q

Wound Healing: Repair

A

Repair
Healing as a result of lost cells being replaced with connective tissue
More common than regeneration
More complex than regeneration
Occurs by primary, secondary, or tertiary intention

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8
Q

Primary Intention

A

Initial phase
Lasts 3 to 5 days
Edges of incision are aligned
Blood fills the incision area, which forms matrix for WBC formation
Acute inflammatory reaction occurs
May initially be covered with dry dressing

Granulation phase
Lasts 5 days to 3 weeks
Fibroblasts migrate to site
Wound pink and vascular
Surface epithelium begins to regenerate

Maturation phase & scar contraction
Begins 7-days after injury & continues for several months/years
Fibroblasts disappear as wound becomes stronger
Mature scar forms

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9
Q

Secondary Intention

A

Wounds occurring from trauma, ulceration & infection
Large amounts of exudate
Wide, irregular margins
Extensive tissue loss
Edges cannot be approximated
Results in more debris, cells, exudate
Wound care depends on etiology & type of tissue in wound

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10
Q

Tertiary Intention

A

Delayed primary intention due to delayed suturing of the wound
Example: A contaminated wound left open & sutured closed after infection is controlled

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10
Q

Tertiary Intention

A

Delayed primary intention due to delayed suturing of the wound
Example: A contaminated wound left open & sutured closed after infection is controlled

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11
Q

Risks Factors for Delayed Healing

A

Nutritional deficiencies
Inadequate blood supply
Corticosteroid drugs
Infection
Smoking
Mechanical friction on wound
Advanced age
Obesity
Diabetes mellitus
Poor general health
Anemia

Complications:
Adhesions
Contractures
Dehiscence
Evisceration
Excess granulation tissue
Fistula formation
Infection
Hemorrhage
Hypertrophic scars
Keloid formation

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12
Q

Pressure Ulcers

A

Graded or “Staged” according to deepest level of tissue damage:
Stage I (minor) to Stage IV (severe)
Un-stageable

Slough and/or eschar must be removed for accurate staging
EXCEPTION:
Stable (dry, adherent, intact) eschar on the heels serves as “the body’s natural (biologic) cover” & should not be removed

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13
Q

Stage I pressure ulcers

A

Intact skin
Areas of non-blanchable redness (in lighter skin individuals)
In dark-skinned patients after applying light pressure, look for an area that’s darker than the surrounding skin or that’s taut, shiny, or indurated (hardened)

If you suspect a skin area is becoming damaged, use the light from a camera flash system to enhance your visualization of dark skin; with the patient’s permission, take a series of digital images each day to document changes in wound color, size, and depth. Check for localized changes in skin texture and temperature.

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14
Q

Stage I – Dark Skin

A

Early signs of skin damage include induration, bogginess (less-than-normal stiffness), and increased warmth at the injury site compared to nearby areas. Over time, as tissues become more damaged, the area becomes cooler to the touch.
Possible indicators
Skin temperature
Tissue consistency
Pain
May appear red, blue, or purple in patients with darker skin tones

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15
Q

Stage II

A

Partial-thickness loss of dermis
Shallow open ulcer with red pink wound bed
Presents as an intact or ruptured serum-filled blister
May also present as a blood-filled blister

16
Q

Stage III

A

Full-thickness skin loss
Damage or necrosis of subcutaneous tissue
May extend to, but not through underlying fascia
Presents as a deep crater with possible undermining
Depth varies by anatomic location

17
Q

Stage IV

A

Full-thickness loss extending to muscle, bone, or supporting structures
Bone, tendon, or muscle may be visible or palpable
Undermining & tunneling may occur

Slough or eschar may be present on some parts of the wound bed.

18
Q

Stage IV

A

Full-thickness loss extending to muscle, bone, or supporting structures
Bone, tendon, or muscle may be visible or palpable
Undermining & tunneling may occur

Slough or eschar may be present on some parts of the wound bed.

19
Q

Wound Assessment

A

Admission skin assessment
On admission from PACU
At regular intervals
Identify factors that may delay healing

Nursing Diagnosis
Impaired skin integrity
Impaired tissue integrity
Risk for infection

20
Q

Wound Assessment

A

Admission skin assessment
On admission from PACU
At regular intervals
Identify factors that may delay healing

Nursing Diagnosis
Impaired skin integrity
Impaired tissue integrity
Risk for infection

21
Q

Wound Classification

A

Cause
Surgical or nonsurgical
Acute or chronic
Depth of tissue affected
Superficial, partial thickness, full thickness
Color
Red
Yellow
Black
May have two or more colors

22
Q

Wound Measurement

A

Made in centimeters
Head to toe
Side to side
Depth (if any)
Tunneling?
Undermining?

23
Q

Tunneling

A

Movement when cotton-tipped applicator is placed in wound

24
Undermining
Presence of “lip” when cotton-tipped applicator is placed in wound Charted in respect to a clock with 12 o'clock being toward the patient's head
25
Wound Documentation
Location Type Stage if pressure ulcer Measurements (Height, width, depth) Undermining? Tunneling? % granulation tissue, slough, eschar? Drainage (amount, color, quality, odor?) Dressing / treatment Patient tolerance, pain?
26
Method of Wound Care Depends On:
Causative agent Degree of injury Patient’s condition Purposes of wound management Cleansing Treating infection Protecting clean wound from trauma Sutures / fibrin sealant help closure Drains may be inserted Topical antimicrobials / antibacterials should be used with caution
27
Nursing Implementation
Red Wounds Protection Gentle cleaning PRN Yellow wounds Absorb exudate Cleanse wound surface Hydrocolloid dressings Black Wounds Debridement of nonviable tissue (“Eschar”)
28
Types of Debridement
Surgical Mechanical Wet-to-dry (moist) dressings Irrigation Whirlpool Autolytic Semi-occlusive Occlusive Transparent films Hydrocolloids (Duoderm) Enzymatic Topical application of enzyme ointments / creams Covered with moist dressing Collagenase (Santyl) Papain / Urea (Panafil)
29
Types of Debridement
Surgical Mechanical Wet-to-dry (moist) dressings Irrigation Whirlpool Autolytic Semi-occlusive Occlusive Transparent films Hydrocolloids (Duoderm) Enzymatic Topical application of enzyme ointments / creams Covered with moist dressing Collagenase (Santyl) Papain / Urea (Panafil)
30
Negative Pressure Wound Therapy
Vacuum-assisted closure “Wound VAC” (K.C.I.) Suction removes drainage and speeds healing Monitor: Serum protein Fluid & electrolytes Coagulation studies
31
Negative Pressure Wound Therapy
Vacuum-assisted closure “Wound VAC” (K.C.I.) Suction removes drainage and speeds healing Monitor: Serum protein Fluid & electrolytes Coagulation studies
32
Hyperbaric Oxygen Therapy
Delivery of 100% oxygen 1.5 to 3 times normal atmospheric pressure 10 to 60 treatments (90 to 120 minutes) Allows O2 to diffuse into serum Stimulates angiogenesis Kills anaerobic bacteria Increases killing power of WBCs & certain antibiotics Accelerates formation of granulation tissue
33
Nutritional Therapy
High protein High carb High vitamin Moderate fat
34
Psychological Implications
Fear of scar / disfigurement Concern about drainage or odor
35
Infection Prevention/Control
Don’t touch recently injured areas Keep environment free of possibly contaminated items Prophylactic antibiotics? Wound cultures - Concurrent swab specimens obtained from wound exudate
36
Z-technique
Rotate swab as wound is swabbed margin to margin in a 10-point zigzag
37
Levine’s technique
Rotate swab over a 1cm² area with sufficient pressure to express fluid