Lp 7 Flashcards

(26 cards)

1
Q

Sensory & motor branches meet at 2 junctions?

A

-SC: reflexive movement (involuntary)
-Cerebral cortex: invitation of voluntary movement

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2
Q

-Voluntary movement?
-Helps plan movements; involved in complex motor tasks; supplies~15% of pyramidal fibres

A

1* motor cortex
&
Premotor cortex

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3
Q

-activates mm dealing w/speech?
-voluntary eye movement?

A

-Broca’s area
-frontal eye field

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4
Q

-impulses from somatosensory cortex & thalamus
-voluntary control over skeletal m Fxn

A

Stimulation

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5
Q

-located on ventral surface of cerebral cortex
-thick region ++ layers of pyramidal cells (cell bodies of UMNs)

A

1* motor cortex

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6
Q

-association neurons
-origin= frontal lobes
-long myelinated axons from pyramidal (corticospinal) tracts that project to brain stem & SC
-80% decussate at medulla
-synapse on LMNs in ventral horn

A

UMNs

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7
Q

Pyramidal cells also link to?

A

-somatosensory area
-thalamus
-premotor cortex
-basal nuclei

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8
Q

-innervation of many m fibres by same UMN (gross movement)?

-allows fro precision & fine motor coordination?

A

-polysynaptic

-monosynaptic

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9
Q

Body areas map on specific motor cortex sites
- (homunculus)

A

Somatotopy

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10
Q

-damage to areas of motor cortex
-leads to paralysis of specific mm

A

UMNL

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11
Q

R & L side of motor cortex=

A

Contralateral hemiplegia

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12
Q

?= weakness of distal flexor m groups & decrease fine motor coordination

A

Posterior aspect

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13
Q

?= weakness f larger limb & girdle mm

A

Anterior aspect

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14
Q

Only voluntary control is lost- mm still contract reflexively
-leads to spasticity

A

Lesions

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15
Q

-increase in motor tone
-leads to tetany & awkward movement

A

Spasticity

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16
Q

Temporary or permanent loss of voluntary motor Fxn

17
Q

-several large gray masses on both sides of thalamus
-contralteral control
-links circuits to premotor & primary motor cortices, & thalamus
-highly skilled behaviours
Fxn: grace movement (highly skilled motor performance) & repetitive motion patterns

18
Q

Damage to the basal nuclei leads to ABN movement patterns & lack of coordination.
Examples are?

A

-Parkinson’s disease
-Huntington’s chorea
-cerebral palsy (some forms)

19
Q

-located on posterior fossa of cranium
-unpaired central mass; 2 large cerebellum jemipheres
-composed of outer grey matter & inner white matter
-communicates w/cerebellum via thalamus
1* Fxn:
-coordinates skilled voluntary movements
-controls balance & m tone
-(with cerebrum) smoothies out movements of limbs, trunk, head & eyes
-provides constant adjustment of m contraction required of highly skilled movement

20
Q

Cerebellum receives?

A

-proprioception input
-sensory input from ears & eyes

21
Q

-coordination of specific body parts w/signals to & from cerebellum
-enables assessment of status of given body parts

22
Q

-checks what is suppose to happen w/what is actually happening & corrects via 1* motor cortex
-inhibits movement, overcoming momentum 7 stopping motion at intended point

A

Cerebellar monitoring

23
Q

Source of pyramidal cells, which are the origin of corticospinal (pyramidal) tracts; inmates contralateral voluntary movement

A

Primary motor cortex

24
Q

-helps plan movements
-selects & sequence basic motor movements into more complex task

A

Premotor cortex

25
-no direct access to motor pathways -filters out incorrect/inappropriate responses, allowing only the best responses to reach the cortex -enhance & add gracefulness to upper motor neuron-controlled movements -these background movement fxns are called associated movements
Basal nuclei
26
-provides instructions to cerebral motor cortex & subcortical motor centres, resulting in smooth, coordinated movements -responsible for proper balance & posture
Cerebellum