Managing cardiac arrest Flashcards

(51 cards)

1
Q

what is a cardiac arrest?

A

effective cessation of the heart

- no circulation & no oxygen delivered

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2
Q

how do you recognise somebody is having a cardiac arrest?

A

1) unresponsive
2) not breathing normally
3) no pulse

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3
Q

why is the majority of cardiac rhythm shockable (i.e. able to be defibrillated?

A
  • as presenting rhythm in most cases is VF/VT

= majority is potentially reversible

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4
Q

what 2 things commonly precede cardiac arrest?

A
  • hypotension

- hypoxia

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5
Q

when should defibrillation be given, if appropriate?

A

= within 3 minutes

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6
Q

what are most problems of cardiac arrest caused by?

A

1) airway
2) breathing
3) circulation
= oxygen delivery problems

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7
Q

how can you improve SaO2 oxygen delivery factors?

A

1) SaO2

= clear airway

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8
Q

how can you improve [Hb= for oxygen deliver?

A

= transfusion trigger, treat anaemia

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9
Q

how can you alter heart rate for oxygen delivery?

A

= atropine or B-stimulant (e.g. ephedrine) for bradycardia, pace

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10
Q

how can you alter stroke volume to improve oxygen delivery?

A

1) preload = IV fluids, raise legs

2) afterload
= excess afterload (HBP) use vasodilators
= reduce after load (e.g. septic shock) use vasoconstrictors

3) contractility = treat cause

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11
Q

what other factors affect oxygen delivery?

A

= BP

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12
Q

how would you manage a cardiac arrest patient?

A

ABCDE

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13
Q

how can you check for SaO2 and [Hb] levels?

A

SaO2
= pulse oximetry on finger
= arterial blood fas (GOLD STANDARD)

[Hb]
= part of full blood count
- bedside (e.g. hemocue)

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14
Q

how would you check heart rate?

A
  • pulse
  • pulse oximetry
  • ECG monitor with sound
  • arterial BP monitor
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15
Q

how would you calculate B{?

A

BP = CO x TPR

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16
Q

what are 4 causes of obstruction which causes AIRWAY problems?

A

1) CNS depression = tongue
2) lumen blocker = blood, vomit, foreign body
3) swelling = trauma, infection, inflammation
4) muscle = laryngospasm., bronchospasm

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17
Q

how would you recognise airway obstruction (SpO2)?

A
  • talking
  • difficult breathing, distressed, choking
  • shortness of breath
  • noisy breathing = stridor, wheeze, gurgling
  • see saw respiratory pattern using accessory muscles
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18
Q

how would you treat airway obstruction (SpO2)?

A
  • airway opening
    e. g. head tilt, chin lift, jaw thrust, suction
  • simple adjuncts
  • advanced techniques
    e. g. LMA, tracheal tube
  • oxygen (increase FiO2)
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19
Q

what are causes of BREATHING problems (SpO2)?

A

1) airway problems

2) decreased respirator drive
- CNS depression

3) decreased respiratory effort
- muscle weakness, nerve damage, restrictive chest defect, pain from fractured ribs.

4) lung disorders
- pneumothorax, haemothorax, infection, acute exacerbation COPD, asthma, PE, ARDS

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20
Q

how would you recognise breathing problems (SpO2)?

A

1) look - respiratory distress, accessory muscles, cyanosis, respiratory rate, chest deformity, consciousness level
2) listen - noisy breathing, breath sounds
3) feel - expansion, percussion, tracheal position

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21
Q

how would you treat breathing problems (SpO2)?

A
  • oxygen
  • airway
  • treat underlying cause to improve breathing
    e. g. drain pneumothorax
  • support breathing
    e. g. ventilate with bag mask
22
Q

what are primary and secondary causes of circulatory problems ([Hb]/CO)?

A

Primary

  • Acute coronary syndromes
  • Dysrhythmias
  • Hypertensive heart disease
  • Valve disease
  • Drugs
  • Hereditary cardiac diseases
  • Electrolyte / acid base
  • abnormalities
  • Electrocution

Secondary Asphyxia

  • Hypoxaemia
  • Blood loss
  • Hypothermia
  • Septic shock
23
Q

how would you recognise circulatory problems?

A

[Hb], CO = HR x SV

  • general exam = distres, palor
  • indications of organ perfusion
    = chest pain, mental state, urine output
  • blood pressure
  • pulse, tachycardia, bradycardia
  • peripheral perfusion = capillary refit time
  • bleeding, fluid losses, JVP, CVP
24
Q

how do you treat circulatory problems?

A

1) ensure airway, breathing O2
2) IV/IO access, take bloods
3) treat cause
- fluid challenge
- inotropes/vasopressors
- oxygen, aspiring, nitrates, morphine for ACS
4) haemodynamic monitoring

25
how do you recognise disability?
``` Treatment - ABC = treat underlying cause = blood glucose - if < 3mmol-1 give glucose = consider lateral (recovery) position = check drug chart ```
26
what heart rhythm is shockable and non-shockable?
SHOCKABLE = VF = pulseless VT NON-SHOCKABLE = asystole = pulseless electrical activity (PEA)
27
why is ventricular fibrillation shockable, whats its ECG appearance?
- bizarre irregular waveform - no recognisable QRS complex - random frequency and ampltitude - uncoordinated electrical activity - coarse/fine
28
describe ventricular tachycardia, whats its ECG appearance?
Monomorphic VT - broad complex rhythm - rapid rate - constant QRS morphology Polymorphic VT - torsade de pointes
29
what is pre-cordial thump?
= only use if defibrillator is not immediately available AND monitored VF/VT cardiac arrest
30
what is a defibrillator?
use of electrical current to "reset" heart electrical rhythm with hope that regular rhythm will recur.
31
why are pads used in defibrillation?
to minimise interruptions
32
After delivery of shock, then immediate CPR, check for 2 minutes. If VF/VT persists what would you do?
1) deliver 2nd shock 2) CPR for 2min 3) deliver 3rd shock 4) CPR if VF/VT persists Adrenaline 1mg IV
33
when should adrenaline and amiodarone be given?
ADRENALINE - after 3rd shock then after alternate shocks (every 3-5mins) AMIODARONE - after 3rd shock
34
why is asystole non shockable, describe its ECG appearance?
= effectively flat line - absent ventricular QRS activity - atrial activity (P wave) may persist - rarely a straight line trace, its a wave line
35
how should you treat asystole?
- adrenaline 1mg IV soon as | - every 3-5mins thereafter (every 2 CPR cycles)
36
why are pulseless electrical activity non-shockable? describe its ECG appearance?
- ECG normally associated with an output
37
how would you treat pulseless electrical activity?
- exclude/treat reversible causes - adrenaline 1mg IV as soon as possible - every 3-5mins thereafter, every 2 cycles
38
what dose of adrenaline and amiodarone should be given? and what does adrenaline act as?
Adrenaline 1mg Amiodarone 300mg - alpha vasoconstrictors - B inotropic
39
what are the 4Hs and 4Ts?
= potential reversible causes H's - hypoxia - hypovolaemia - hypo/hyperkalaemia/metabolic - hypothermia ``` T's = thrombosis - coronary/pulmonary = tension pneumothroax = tamponade - cardiac = toxins ```
40
how would you treat hypoxia?
= airway and ventilation 1) secure airway - tracheal tube - suprglottic airway device, e.g. LMA/iGel 2) once airway is secured, don't interrupt chest compressions for ventilation 3) avoid hyperventilation
41
what are advantages & disadvantages of mouth to mask ventilation?
ADVANTAGES; - avoids direct person to person contact - decreases potential for cross infection - allows oxygen enrichment LIMITATIONS: - maintenance of airtight seal - gastric inflation
42
advantages and disadvantages of self-inflating bag?
ADVANTGES; - avoids direct person to person contact - all O2 supplementation = up to 85% - can be used with facemark, LMA, combitube, tracheal tube ``` DISADVANTAGES - when used with a facemark; = risk of inadequate ventilation = risk of gastric inflation = need 2 persons for optimal use ```
43
advantages of disadvantages of laryngeal mask airway (LMA)?
ADVANTAGES ; - Rapidly and easily inserted - Variety of sizes - More efficient ventilation than facemask - Avoids the need for laryngoscopy DISADVANTAGES - No absolute guarantee against aspiration - Not suitable if very high inflation pressures needed - Unable to aspirate airway
44
what are problems of insertion of tracheal tube?
- incorrect placement | - delays CPR or defibrillator
45
how would you treat hypovolaemia?
IV fluids
46
how would you treat hypo/hyperkalaemia/metabolic?
- correct accordingto U&Es/blood gases
47
how would you treat thrombosis, tension pneumothorax, cardiac tamponade and toxins?
thrombosis = thrombolysis tension pneumothorax = needle thoracentesis cardiac tamponade = needle cardiocentesis toxins; = specific treatment/antidote
48
when should you stop CPR?
= when return of spontaneous circulation (ROSC) | - when seems useless, time, diagnosis, pre-arrest conditions or DNR/DNAR
49
what are 4 factors of post-cardiac arrest syndrome?
1) post cardiac arrest brain injury 2) post cardiac arrest Myocardial dysfunction 3) systemic ischaemia / re-perfussion response 4) persistant precipitating pathology
50
what should be done immediately after post resuscitation?
1) 12 lead ECG 2) reliable IV access 3) intra-arterial BP monitor 4) target SBP > 100mmHg 5) fluid (crystalloid) normovolaemia 6) consider inotrope/vasopressor
51
in unconscious adults with risk of spontaneous circulation after arrest be temperature managed to?
32-36degrees.