Mid-term Flashcards

(80 cards)

1
Q

F&E: what is crucial to the maintenance of homeostasis?

A

regulation of the concentration of oxygen, carbon dioxide, organic nutrients, wastes and inorganic ions

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2
Q

F&E: intracellular components

A

40% total body weight

most found in skeletal muscle cells

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3
Q

F&E: extracellular components

A

20% total body weight
compartments: interstitial [cells outside vessels], intravascular [cells w/i the blood vessel], transcellular [cells everywhere else]

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4
Q

F&E: 1 L of water= ___ lbs=___ kg

A

2.2
1
this allows for fluid loss/gain to be monitored via weight change

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5
Q

F&E: Na+

A

sodium
135-145 mEq/L
for fluid retention, neuromuscular and enzymatic functioning

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6
Q

F&E: Cl-

A

chloride
96-106 mEq/L
works w/ Na to provide hydration with a role in hydrochloric acid

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7
Q

F&E: K+

A

potassium
3.5-5.0 mEq/L
for contraction of muscles [esp/ heart muscles]

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8
Q

F&E: Ca+

A

calcium
8.6-10.2 mg/dL
most abundant mineral in the body
for muscle contraction, blood coagulation, and bone structure [as 99% of Ca+ is stored in the bones]

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9
Q

F&E: PO4-

A

phosphorous
2.4-4.4 mg/dL
combined w/ Ca+ in the crystals of bones and teeth
2nd most abundant mineral in the body
for acid-base balance as a buffer [phosphoric acid]

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10
Q

F&E: Mg+

A

magnesium
1.5-2.5 mEq/L
for neuromuscular functioning

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11
Q

F&E: HCO3

A

bicarbonate
22-26 mEq/L
for acid-base regulation

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12
Q

F&E: hydrostatic pressure

A

influenced by blood pressure and volume
arteries have a high hydrostatic pressure
it pushes fluid out of the vessel into the interstitial space
as it leaves, the H.P. decreases
as the H.P. decreases, it will fall below the colloidal osmotic pressure

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13
Q

F&E: colloidal osmotic pressure

A

exerted by plasma proteins
once the fluid is in the interstitial areas and the hydrostatic pressure is decreased, blood draws back into the vessels to be brought back into the heart
- at this point the O-carrying blood has perfused O to the tissue, so it enters the bloodstream to get oxygenated once more
in cases of malnutrition, the colloidal osmotic pressure couldn’t do its purpose in bringing blood back into the vessels b/c of the lack of essential plasma proteins

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14
Q

F&E: how much urine should be excreted?

A

1-2 mL/kg/hr

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15
Q

F&E: protein deficiency

A

causes: inadequate protein intake, protein loss, decreased protein synthesis
s/s: poor wound healing, edema [disturbance in colloidal osmotic pressure], anemia [protein carries O], fatigue, weight loss, muscle wasting [builds up muscle]
tx: diet high in amino acids, CHO [CHO used for energy leaving protein to be used for muscle gain and not energy] and protein

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16
Q

F&E: plasma-to-interstitial fluid shift

A

fluid shift from vascular compartment to interstitial compartment
causes: increased capillary hydrostatic pressure, decreased plasma protein, increased capillary permeability
s/s: increased heart rate [decrease in blood volume makes heart work harder to get blood to the major organs], decrease in B.P., decrease urine output, edema
tx: replace F and E, cautiously

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17
Q

F&E: types of edema

A
pitting
dependent
- upon gravity and position
weeping
- fluid seeps out through the skin
anascara
- edema throughout the body
other
- edema of a specific system [i.e. ascites, pleural effusion]
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18
Q

F&E: interstitial-to-plasma fluid shift

A

shift of fluid from interstitial to intravascular compartment
causes: decrease in capillary hydrostatic pressure, increases in colloidal osmotic pressure, remobilization of fluid following burns and trauma
s/s: increased B.P., large amounts of diluted urine, bounding heart beats, pleural edema, restlessness [2o to pleural edema]
tx: fluid is excreted naturally is pt. has healthy heart and kidneys, if not, use of diuretics or dialysis may be needed

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19
Q

F&E: what are primary and secondary sx of respiratory distress?

A

primary
- restlessness>increased heart rate
secondary
- crackles in the lungs, altered mental status, drop in pulse ox., use of accessory muscles

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20
Q

F&E: hyper-osmolarity

A

too many particles [of Na] or too little water which results in cell-shrinking
causes: decreased water intake, extracellular solute excess
s/s: dehydration [evidenced by increase in heart rate, thirst, poor skin turgor, dry mucous membranes/skin, decreased/concentrated urine], cell shrinkage [evidenced by altered mental status]
tx: replace water [PO or IV]

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21
Q

F&E: hypo-osmolarity

A

or water intoxication
too few particles or too much water which results in cell swelling
causes: replacing H2O and Na loss w/ only H2O, inability to excrete urine [seen in chronic renaal failure]
s/s: cerebral edema, diluted urine, increased B.P.
tx: replace loss w/ Na ad H2O [isotonic solution], utilize oral liquids w/ electrolytes

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22
Q

F&E: isotonic deficit

A

Na and H2O loss in equal proportions which do not cause size change in cells but decreases the volume of the ECF
tx: treat underlying cause, administer isotonic solution, carefully

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23
Q

F&E: what are s/s of hemorrhaging?

A

increase in heart rate, altered mental status, hypoxia, decrease in urine output

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24
Q

F&E: isotonic excess

A

Na and H2O gain in equal proportions which do not cause size change in cells but increases the volume of the ECF
s/s: pulmonary edema [causing restlessness, tachycardia, crackles in the lungs]
tx: restrict fluids, monitor fluids, diuretics/dialysis

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25
F&E: hypernatremia
an excess of Na s/s: results from cell shrinkage and fluid shifting [i.e. brain cell shrinkage causing altered mental status] tx: restrict Na intake, gradual lowering of Na to prevent cerebral edema, monitor changes in behavior
26
F&E: hyponatremia
a deficit of Na s/s: cerebral edema, diluted urine, increased B.P. tx: isotonic or hypertonic solution, restriction of water
27
F&E: hyperkalemia
an excess of K causes: pseudohyperkalemia, chronic renal failure, use of salt substitutes [major ingredient: K], K sparing diuretics, metabolic acidosis, burns [destroy K-containing blood cells] tx: K-restricted diet, calcium gluconate [does not affect K levels, used for cardiac arrhythmia's, dialysis, med.'s to reduce K [i.e. kayexalate]
28
F&E: pseudohyperkalemia
this occurs during a blood draw where the syringe hemolized [destroyed the blood cell] RBC's which caused its contents to come out the cell causing an K increase in that area the K levels in the blood would :. be high, but falsely
29
F&E: hypercalcemia
excess of Ca causes: hyperparathyroidism, immobilization, overuse of calcium products, malignancies tx: loop diuretics [promotes excretion of Ca], fluids, encourage activity, med.'s [d/o cause of condition], low Ca diet, vit. D [helps GI tract absorb Ca]
30
F&E: fluid spacing
1st- normal distribution of body water 2nd- abnormal accumulation in interstitial space [edema] 3rd- fluid is trapped and essentially unavailable and cannot go back to where it came from - it is a distributional shift in fluid in a space that does not easily exchange w. the ECF [i.e. peritonitis]
31
F&E: hypocalcemia
deficit of Ca+ causes: hypoparathyroidism, inadequate vit. D, chronic renal failure s/s: + Trousseau sign, + Chvostek sign tx: IV calcium [regularly check IV bag for calcium precipitation; flush ac and pc Ca administration], Vit. D
32
F&E: trousseaau sign
muscular contraction including flexion of the wrist and metacarpophalangeal joints, hyper-extension of the fingers, and flexion of the thumb on the palm, when a sphygmomanometer cuff is inflating to above systolic pressure for several minutes suggest neuromuscular excitability caused by hypocalcemia
33
F&E: chvostek sign
spasm of the facial muscles elicited by tapping the facial nerve in the region of the parotid gland; seen in tetany and hypocalcemia
34
F&E: tetany
characterized by spasms of the hands and feet, cramps, spasms of the larynx,, and over-active neurological reflexes indicative of hypocalcemia
35
F&E: hypermagnesemia
excess of Mg+ causes: renal failue, excessive Mg+ intake s/s: early [flushing of skin, sense of warmth, N/V] moderate [drowsiness, hypoactive reflexes, weakness] severe [neuromuscular, respiratory and cardiac depression] tx: dialysis [if caused by renal failure], remove source of Mg+, IV Ca+
36
F&E: hypomagnesemia
deficit of Mg+ causes: alcoholism, starvation, diarrhea, increased PTH s/s: hyperactive reflexes, painful muscle contraction, confusion, tetany tx: Mg+ replacement
37
F&E: hypokalemia
deficit of K+ causes: diuretics, poor intake, GI loss, metabolic alkalosis tx: K+ replacement
38
F&E: hyperphosphatemia
excess of P causes: chronic renal failure, excessive intake of phosphorous, hypoparathyroidism s/s: tingling of fingers, muscle spasms, precipitation Ca+ and P - these are symptoms that occur in hypocalcemia b/c Ca+ and P are antagonistic to one another tx: treat underlying disorder, IV Ca+ or hemodialysis [in extreme cases
39
F&E: hypophosphatemia
deficit of P causes: hyperparathyroidism, use of thiazide diuretics s/s: numbness, weakness, decreased cardiac output [decreases O2 perfusion to organs, brain, causing mental changes], mental changes [i.e. apprehension, confusion], acute respiratory failure tx: phosphorous replacement, assess Ca+ levels
40
F&E: CBC diagnostic test
complete blood count measures: WBC, RBC, platelets, Hct [#of RBC: solution], Hmg [# of O2-carrying protein on RBC] - Hct can tell us the hydration status where an elevation indicates dehydration
41
F&E: serum creatinine diagnostic test
tests kidney functioning an increase in creatinine signals liver failure this is the distinguishing factor b/w dehydration and renal failure, if there is an increase in creatinine that indicates kidney dysfunction
42
F&E: BUN diagnostic test
blood urea nitrogen 6-20 mg/dL it is a ratio tell's us the hydration status where an increase signals dehydration and vice versa urea and nitrogen are waste products :. these values tells us about the renal functioning
43
F&E: plasma protein diagnostic test
total proteins: 6.4-8.3 g/dL albumin: 3.5-5.0 g/dL indicated for pt.'s w/ edema [disturbance in colloidal osmotic pressure], in liver dysfunction [liver produces proteins->ascites]
44
F&E: routine urinalysis diagnostic test
tests urine for blood, bacteria, protein, glucose, specific gravity, pH the pH should be fluctuating constantly as the kidneys compensate for disturbances in the acid-base balance, this is a normal finding specific gravity measures urine concentration where an increase signals a concentrated urine
45
PERIOPERATIVE: reasons for surgery
diagnostic curative palliative cosmetic
46
PERIOPERATIVE: urgency of surgery
elective urgent emergent [requires life-saving measures]
47
PERIOPERATIVE: surgery impact
can cause: physical stresses - resistance to infection is lowered - organ function may be altered due to manipulation psychological stresses - fear, pain, anxiety, loss of control, body image, alterations in ADL's
48
PERIOPERATIVE: surgical risk factors
``` age nutritional status fluid and electrolyte balance general health status medications goal: to get the pt. in the most optimal position to handle the surgery to have positive outcomes ```
49
PERIOPERATIVE: legal issues
``` informed consent - regarding surgery, anesthesia, blood - pt. must be >18 y.o. to give consent -- minors may consent to their own surgery if they are emancipated witness to consent - usually the RN's role to be a witness and an advocate for the pt. emergency situations - pt. consent usually isn't obtained ```
50
PERIOPERATIVE: pre-op checklist
``` baseline vital signs provide oral hygiene, remove dentures, record loose teeth [to prevent teeth from dislodging and falling into the throat during surgery remove nail polish, cosmetics, hair pin, protheses have pt. void check name band review consent administer pre-op medications if ordered elevate side rails ```
51
PERIOPERATIVE: pre-op med.'s
anti-anxiety i.e. diazepam [valium] sedative i.e. midazolam [versed] analgesic i.e. morphine sulfate anticholinergic i.e. atropine sulfate - used to decrease oral secretion in event that intubation is needed H2-receptor antagonist i.e. zantac - used to decrease gastric acidity for pt.'s at risk for aspiration to prevent acid from damaging lungs
52
PERIOPERATIVE: members of surgical team
surgeon surgical assistant anesthesiologist or CRNA [certified registered nurse anesthetist] circulating nurse scrub nurse/surgical technologist specialty nurse, or RNFA [registered nurse first assistant]
53
PERIOPERATIVE: intra-operative care
safety positioning - once a pt. is placed under anesthesia, they will remain in the position they are placed in for the entirety of the surgery, it is imp. to prevent permanent damage to the tissues documentation surgical env't. - traffic control, infection control, sterilization of supplies inadvertent hypothermia
54
PERIOPERATIVE: universal protocol
initiated by the joint commission to eliminate surgery at the wrong site, of the wrong procedure, and on the wrong person by: - establishing a pre-op verification process - marking the operative site - performing a "time-out" immediately before starting the procedure
55
PERIOPERATIVE: ASA classifications
``` american society of anesthesiologists classifications ASA 1 healthy 2 one medical problem 3 more than one medical problem 4 severe systemic disease 5 not expected to survive w.o surgery 6 organ harvest ```
56
PERIOPERATIVE: anesthesia induction
it is the point at which anesthesia is initiated just prior to the first incisional cut
57
PERIOPERATIVE: general anesthesia
it produces a controlled loss of consciousness protective reflexes lost via total muscle relaxation can be given intravenously, orally, or through inhalation complications: CNS [emergence delirium, delayed emergence], cardiovascular [hypotension, dysrhythmias, MI], hypothermia, respiratory [laryngospasm], malignant hyperthermia
58
PERIOPERATIVE: malignant hyperthermia
it is a chain of abnormalities that occurs due to an inherited pharmacogenetic disease which degrades skeletal muscle in those w/ disorder, inhaled anesthesia is the trigger [i.e. Halothane, Enflurane, Isoflurane, Desflurane, Sevoflurane, ALL except nitrous oxide] those suspected w/ this disorder have a caffeine/halothane contracture test to confirm dx s/s: early signs [muscle rigidity, tachycardia, dysrythmias], breakdown of muscle->heat->rubor and warmth, tachypnea, pyrexia [Hallmark yet late sign] tx: discontinue anesthesia, administer 100% oxygen, administer dantrelene ASAP, cool pt.
59
PERIOPERATIVE: dermatome chart
they are used to determine the level of block [caused by regional anesthesia] for spinal and epidural access level using a sharp, dull, or cold test items
60
PERIOPERATIVE: postoperative hand-off
``` type and extent of surgical procedure type of anesthesia pt. tolerance of anesthesia and surgical procedure pt.'s allergies pathologic condition status of vital signs type and mount of IV fluids and med.'s administered incisions, dressings, tubes, drains, cathters estimated blood loss [EBL] any intra-operative complications pertinent past medical hx hand off's should be standardized ```
61
PERIOPERATIVE: post-op. respiratory assessment
``` respiratory rate breathing depth and pattern breath sounds pt.'s color use of accessory muscles O2 pulse ox. assess for atelectaasis, pneumonia, embolus - prevent via turning and encouraging to cough and deep breathe - maintain their hydration - ambulate early - encourage use of incentive spirometer ```
62
PERIOPERATIVE: post-op. circulation assessment
vital signs skin temperature peripheral vascular assessment check for bleeding
63
PERIOPERATIVE: pot-op. neurological assessment
ability to obey verbal commands level of consciousness [AAOX3] motor/sensory assessments
64
PERIOPERATIVE: post-op GI assessment
bowel sounds, flatus, B.M. - all of which would have been delayed due to the anesthesia nausea vomiting
65
PERIOPERATIVE: post-op. integumentary assessment
the surgeon ALWAYS cleans the first dressing - should the dressing become saturated in drainage, reinforce w/ additional dressing check for drainage and drains check for proper wound healing
66
PERIOPERATIVE: how to prevent thrombophlebitis
``` leg exercises while in bed early ambulation TED stocking sequential hose avoid pressure that may obstruct flow low dose heparin ```
67
IV therapy: CDC recommendation on sterile principles
IV container should be changed q24h tubing should be changed routinely q48-72h IV site dressing should be changed q48-72h check expiration of tubing, fluids, med.'s, etc. if there is a break in sterile technique, DISCARD AND START OVER
68
IV therapy: factors influencing gravity flow rate
``` height of sol'n. - the higher the sol'n. is placed above the heart, the faster the fluids will drip down patency of cannula venous spasms - due to room temp. [approx. 72o F] med.'s into 98.6o F blood size of cannula bleeding in tubing presence of local complications ```
69
IV therapy: PICC
peripherally inserted central catheters placed in the ante-cubital fossa, fed through the vein and ends at the superior vena cava there are low complication rate and it is less expensive no B.P. or blood draws allowed on the extremity w/ a PICC line
70
IV therapy: CVAD
central venous access device types: tunneled, non-tunneled, port indications: med. administration, nutrition, blood samples/transfusions, conditions [renal failure, burns, chemotherapy] care: x-ray ac starting fluids, sterile dressing change, observe sit for s/s infection or systemic complications complications: catheter occlusion, embolism, infection, pneumothorax, catheter migration
71
IV therapy: infiltration
this occurs when fluid from the IV leaves the bloodstream and invades the deep tissue space this occurrence may not be preventable the after-effects of infiltration can be s/s: pain, swelling, COLD - cold distinguishes infiltration from any other rx that have cause local edema tx: stop IV, pull catheter out, determine whether another IV needs to be put in [based on what med.'s the pt. was receiving
72
IV therapy: phlebitis
it is inflammation of the vessel the catheter is still w/i the vessel and administering sol'n. which is irritating the vessel s/s: warmth, red streak outlining the vessel that is inflamed tx: take catheter out
73
IV therapy: pyrogenic rx
contaminated set-up of IV caused sepsis s/s: abrupt rise in temp., severe chills, shaking, increase in HR and RR, headache tx: stop the IV, KVO [keep vein open] - KVO b.c of vessel constriction and for need to administer emergency med.'s
74
IV therapy: air embolism
air in the line s/s: chest pain, SOB, decrease B.P., increase H.R., cyanosis, anxiety, confusion tx: administer O, place pt. in left-lateral modified tradelenberg [air moves up :. air would move towards feet and away from heart and lungs]
75
IV therapy: circulatory overload
s/s: increase B.P., distended neck veins, S.O.B. | tx: administer diuretic, slow down rate of infusion, increase H.O.B., KVO
76
IV therapy: speed shock
caused by administration of IV push med.'s s/s: dizziness, chest tightness, flushed face, irregular pulse - these can occur in less than a minute
77
IV therapy: hyperal
hyperalimentation is a type of TPN indicated for pt.'s w/ GI disturbances, burns, cancer, malnourishment composed of calories [in dextrose form, electrolytes, protein, vitamins, minerals - individualized for a specific pt.'s needs each day
78
IV therapy: intralipids
it is an isotonic, fat emulsion substance that comes in concentrations of 10-20-30% provides calories in small volumes contains soybean and egg phospholipids the only sol'n. that can be given w/ hyperal s/s pf hypersensitivity rx: tachycardia, N/V, fever, itching, chills contraindicated in pt.'s w/ egg allergies or risk of fat embolism
79
IV therapy: complications of TPN infusions
hyperglycemia - due to the body's inability to get used to the high concentration of dextrose hypoglycemia - due to sudden seizure of TPN infection - due to high concentration of dextrose altered electrolyte, mineral, vitamin balances
80
IV therapy: PCA pumps
enables the pt. to self-administer med. on PRN basis programmed according to medical orders - dosage, time intervals b/w doses, and lock-out intervals] records the amount of med.'s received and the number of requests by the pt. - this assessment, done by the nurse, can be given to the physician w/ intention to alter the order