Midterm 2 Flashcards

(63 cards)

1
Q

Panic Disorder

A

Discrete periods of intense fear
Peaks within minutes

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2
Q

Panic disorder diagnostic criteria

A

Recurrent unexpected panic attacks - no valid reason to feel panic
Panic attacks followed by over 1 month of persistent concern or worry about additional attacks or consequences
Significant change in behaviour

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3
Q

Prevalence and course of panic disorder

A

Lifetime prevalence of 28%
Affects 1.5-3% of Canadians
Two to three time more common in women

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4
Q

Agoraphobia

A

“Fear of the marketplace”
Anxiety about being in places where escape might be difficult - particularly a fear of having panic symptoms
Often co occurs with PD
need marked fear of two or more places:
Public transit, open space, closed space, standing in line/crowds, being outside home alone
Fear of not being able to escape
Situations almost always provoke fear
Fear is out of proportion
Persistent over 6 months

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5
Q

Cognitive mod of Panic Disorder

A

People with PD pay close attention to bodily sensations
They misinterpret sensation
And engage is spiralling / catastrophic interpretations

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6
Q

GAD symptoms

A

Worry and anxiety occurring more days then not about a number of different events or activities for at least 6 months
Worry is difficult to control
Associated with 3+ of the following for 6 months:
Restlessness or feeling on edge
Easily fatigued
Difficulty concentrating or mind going blank
Irritability
Muscle tension
Sleep disturbance
Impairment or distress
Not due to substance use or other medical problems

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7
Q

Prevalence and course of GAD

A

highly comorbid with other anxiety disorder, depression, and substance use
9% of Canadian population is effected

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8
Q

Biological theories of GAD

A

GABA theory: neurotransmitter preventing neurotransmitter from firing, individuals with GAD may have deficits in GABA receptors leading to excessive firing in limbic or emotional systems
Genetic theory: GAD is inherited, general trait anxiety may increase risk

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9
Q

Cognitive theories of GAD

A

Beck: people with GAD think about threat constantly
Overpredict likelihood and cost of aversive outcomes
Under-predict their ability to cope with outcomes
Believe that worry prevents bad things from happening
Worry is negatively reinforced - allows individual to avoid negative aspect of arousal

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10
Q

Specific phobias

A

There is a phobia for everything
Criteria:
Marked fear about specific object or situation
Out of proportion to the threat
Always provokes fear
object/ situation is almost always avoided or endured with intense distress
Persistent > 6 months
impairment/ distress

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11
Q

5 main types of phobias

A

Animal type
Natural environment type - earthquakes, ocean, thunderstorms
Situational type - airplanes, cars, elevators, escalator
Blood injection-injury type - needles, seeing blood, injection
Other - literally anything else, emetophobia
Specific phobias are most prevalent psych disorder - 11%

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12
Q

Biological theories of phobias

A

Evolutionary
Biological preparedness
Disgust sensitivity -individual differences in levels of disgust to things

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13
Q

Behavioural theories of phobias

A

John B. Watson - Little Albert experiment
Avoidance – decreased anxiety – operant conditioning
Old behavioural theories thought that you had to have experience/ trauma to develop phobia

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14
Q

Social anxiety disorder

A

Specific phobia to social situations
Severely disrupts daily life
Fear of negative evaluation from others
Specify if:
Performance only: fear is related to public speaking or performing

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15
Q

Cognitive theories of Social anxiety

A

Exaggerated likelihood of negative evaluation
Exaggerated cost of negative evaluation

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16
Q

Therapies for social anxiety

A

Social skills training
CBT
Exposure
Modelling
Cognitive restructuring
CBT group therapy - can be super effective because you see modelling of behaviour, treatment is itself an exposure

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17
Q

Pharmacological treatments of anxiety

A

SSRIs - prozac, paxil, zoloft, celexa etc.
Tricyclic Antidepressants
Paxil, Tofranil
SNRIs - Effexor
Benzodiazepines - Xanax, valium
Short term anxiety relief
Physically addictive
Interfere with cognitive and motor functioning
Bupropion - NDRI norepinephrine dopamine reuptake inhibitor

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18
Q

Exposure works by

A

Stops reinforcing effects of avoidance
Allows practice of skills
Provides evidence against irrational thoughts
Habituating to a feared stimulus

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19
Q

Relaxation techniques for anxiety

A

Can’t be both relaxed and scared at the same time
PMR
Relaxation alone doesn’t work but in combination with cognitive techniques can be helpful

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20
Q

Major depressive episode

A

need at least 5 of symptoms:
1. Required: Depressed mood or anhedonia (diminished p;easure in things you used to enjoy)
2. Appetite or weight changes
3. Sleep problems
4. Psychomotor changes - agitation, fidgety, or moving/ talking slower then usual
5. Loss of energy
6. Feelings of worthlessness or inappropriate guilt
7. Concentration problems
8. Suicidality
Must cause distress and last for 2 weeks

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21
Q

Major Depression

A

Episode needs to be present
Causes distress, lasts 2 weeks
Can’t have history of mania or hypomania
Can occur in presence of loss but has to be considered carefully
Mood symptoms: Anhedonia/ sadness
Physical symptoms: weight, appetite, sleep, psychomotor
Cognitive symptoms: worthlessness, indecisiveness, suicidality

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22
Q

MDD vs Grief

A

MDD:
Persistent depressed mood
Decreased ability for pleasure
worthlessness/ self loathing
Not deserving of life, unable to cope
Grief:
Comes in waves
Moments of pleasure between waves
Thoughts are tied to loss
Self-esteem generally maintained
Thoughts of death are more joining the loss then harming oneself

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23
Q

Prevalence and course of MDD

A

1.3 million canadians in any given year will experience MDD
Lifetime prevalence is 8-12% in women, men 4-6%
One of leading causes of disability worldwide
Why 2:1 ratio for women to men:
Genetics
hormones(estrogen,progesterone)

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24
Q

PDD

A

Depressed mood lasts 2 years
At least 2 of following:
Appetite
Sleep
Low energy
Low self-esteem
Poor concentration
Feeling hopeless
Lifetime prevalence 3%
Onset typically before 21
Tends to be chronic

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25
Bipolar 1&2
Manic or hypomanic Elevated mood periods Both require three of following symptoms: Grandiosity or inflated self esteem Decreased need for sleep More talkative Flight of ideas Distractibility Increase in goal directed activities Excessive involvement in high risk activities
26
Difference between mania and hypo mania
Manic: 1 week at least OR hospitalization at any point Hypomanic: at least 4 consecutive days Hypomania does not have psychotic features, impairment in functioning or necessary hospitalization, mania could have these symptoms Duration and severity differentiate between hypomania and mania
27
Bipolar 1
1 or more lifetime manic episodes History of MDE not required but usually depression Many also have hypomanic episodes Equally common in men and women
28
Bipolar 2
One or more MDE One or more hypomanic episodes No history of manic episodes Equally common in men and women 1-year prevalence 0.3% of people
29
Cyclothymia
Numerous periods of hypomanic symptoms and subclinical depression for more then 2 years Can’t be symptom free for more than two months No MDE or Manic episodes Can develop in adolescence or early adulthood Increases risk for developing full blow MDE or manic episodes
30
Heritability estimates of mood disorders
MDD: 0.36 Bipolar: 0.75
31
Neurotransmitter theories of mood disorders
Serotonin: mood, anxiety, aggression, eating, sleeping, pain, sexual behaviour, memory Norepinephrine: regulates arousal, energy, activity, appetite - more in BP Dopamine: pleasure, reward, mood, attention, activity - low levels associated with depression
32
Bio psychosocial theories of depression
Environmental events can modify gene expression, affect brain functioning Early life stress - altered gene expression in hippocampus - greater HPA stress response in adulthood - increased vulnerability for depression
33
Cognitive theories for mood disorders
Beck’s model: depressive self schema - faulty information processing - negative thoughts about self, world , future Hopelessness model: if you attribute negative events internally, these views are stable and you think it will never improve, global thinking - implied to things beyond just negative event Interpersonal theories: How social relationships impact depression Marital dissatisfaction is strongly related to depression Lack of social and family support is related to depression Stress generation hypothesis; individuals with depression are more likely to experience negative stressful life events Excessive reassurance seeking
34
Psychosocial theories of bipolar
Stressful life events - may trigger new episodes Sensitivity to reward Changes in bodily rhythms or usual routines
35
Bio treatments for depression
SSRI’s - relief within a couple weeks, less severe side effects SSNRI’s - wellbutrin ECT - given when patient doesn't respond to treatment after 2 types of medication Relieves depression in 50-60% but 85% relapse rTMS - patient stays awake, few side effects, magnetic fields stimulating brain
36
Bio treatments for bipolar
Mood stabilizers - Lithium - ED and LD are very close so regular blood work must be done Anticonvulsants Atypical Antipsychotics Treated as chronic conditions - remain on medication through lifetime
37
Psychological treatments for bipolar
Interpersonal and social rhythms therapy - help build support and maintain routine Family-focused therapy - educating family members, learning communication CBT
38
Interpersonal theory of suicide
cognitive states that make you susceptible to suicidal ideation - thwarted belongingness and perceived burdensomeness Both cognitive states plus capability for suicide required as risk for suicide
39
Capability for suicide:
fearlessness (about death) and pain tolerance - these things are acquired over time through environment (ex. war) Plus thwarted belongingness and perceived burdensomeness
40
Treatment and prevention of suicide
Decrease burdensomeness and increase belongingness Interpersonal coping skills Challenging untrue beliefs Activities that foster connectedness Crisis intervention: Hospitalization Suicide hotlines Medication DBT:
41
Signs of suicidality
Ideation Substance use Purposelessness Anxiety Trapped Hopeless Withdrawal Anger Recklessness Mood changes
42
AN
Restriction of intake leading to low body weight Intense fear of weight gain Disturbance in body image
43
Subtypes of Anorexia
Atypical Restricting Binge eating/ Purging
44
Features of people with AN
Perfectionism High intellectual functioning fidgety/ restless Food rituals and strict rules around eating Cold High comorbidity with depression and anxiety
45
Minnesota Semi-Starvation study (1944):
Took group of men and restricted their calorie intake and required to walk a lot for several weeks and then were refed, tracking their metabolism and relating it to refeeding people after the war These men ended up exhibiting AN symptoms - effects of disorder are due to starvation and did not come before disorder
46
Medical complications of AN
constipation , cold intolerance Major organ failure cardiovascular complication Kidney and liver damage Osteoporosis Impaired immune functioning
47
Prevalence and course of AN
1–2% prevalence Least prevalent disorder 90% diagnosed are women - men often don't seek treatment Usually begins in adolescence 15-19 Often chronic course - long standing if left untreated Early intervention important Standardized mortality rate - 5.86
48
Bulimia
Recurrent episodes of binge eating Recurrent episodes of compensatory behaviour: excessive exercising, self induced vomiting, fasting, taking laxatives Must occur once per week for three month period Self evaluation heavily influenced by shape or weight These behaviors with low weight - receive diagnosis of ANBP Average or above weight - Bulimia Binge: eating an amount of food that is definitely larger than what most people would eat in a similar context - loss of control over eating during episode
49
Medical complications of BN
Not die to starvation but binging and purging Electrolyte imbalance - affects potassium and cardiac Erosion of enamel on teeth Enlarged salivary glands Ruptured esophagus Ruptured stomach
50
Binge Eating Disorder
Recurrent episodes of binge eating in the absence of compensatory behaviours Need three symptoms: Eating faster Eating until uncomfortable Eating when not hungry Eat alone due to embarrassment Feeling depressed, disgusted or guilty about overeating
51
OS feeding or eating disorders
Atypical AN: meets all criteria for AN but is not low weight Subthreshold BN Subthreshold BED Purging Disorder - no binge, just self induced vomiting to influence shape or weight Night Eating Syndrome Represents MOST cases of eating disorders
52
General prevalence of ED’s
Canadian adolescents between 11 and 20- 2.2% of boys, 4.5% of girls had current symptoms of full blown eat disorder And additional 1.1% of boys and 5.1% of girls had subthreshold eating disorder symptoms By midlife 15% of women have had an eating disorder OFSED was most common lifetime eating disorder affecting 7.6% of surveyed women Prevalence has been increasing - unsure if this is due to more cases or better detection Large proportion of individuals with ED do not seek help
53
Obsessions
recurrent and uncontrollable thoughts, impulses, or ideas that individual finds anxiety provoking r distressing
54
Compulsions
repetitive behaviours or cognitive acts that are intended to reduce anxiety
55
DSM criteria for OCD
Presence of either obsessions or compulsions Obsessions tend to be more bizarre and involve more vivid imagery then just uncontrollable worrying thoughts Thoughts that are unwanted are ignored or suppressed through efforts of neutralizing In order to be considered compulsions acts must serve purpose of alleviating anxiety Must be considered excessive or have little connection with thought or event they are trying to avoid Spending more than 1 hour a day engaged in OC acts
56
Basal ganglia and frontal cortex theory for OCD
Basal ganglia controls motor behaviour, frontal cortex responsible for higher cognitive functioning like abstract reasoning and decision making these areas are connected with loop system allowing info to travel back and forth Structural and or functional abnormalities in this brain system may be responsible for OC One study shows patients with OCD have less brain volume in frontal cortex and more brain volume in basal ganglia
57
Serotonin hypothesis of OCD
Serotonin deficiency thought to be involved cuz SSRI’s effective in treating OCD - evidence inconclusive
58
Cognitive behavioural theory of OCD
obsessions are caused by reaction to intrusive thoughts because these individuals have high personal responsibility Compulsions are reinforced when negative event doesn't occur
59
General Fear vs Anxiety Diagnosis
Distress and impairment Severe enough to lower quality of life Chronic and frequent enough to interfere with functioning
60
PTSD diagnostic criteria
Exposure to traumatic event by direct experience or learning they occurred to close person Presence of one or more of following intrusions: Recurrent memories of event Distressing dreams Disassociative reactions Physiological cues that resemble event Negative alterations in cognitions Depression symptoms
61
Pre event risk factors to PTSD
Low socioeconomic status, education, and intelligence testing Previous psychiatric history Childhood adversity
62
Dual representation theory
ways traumatic memories are stored and retrieved differently than non traumatic memories Traumatic memories may be stored and retrieved in non-verbal-sensory-based instead of verbal form like non traumatic memories These sensory memories need to be transferred into verbal memories in order to process event which can be achieved through writing and talking about event in therapy
63
Biological theories of PTSD
Dysfunction in brain areas associated with processing and responding quickly to threat Brainstem, amygdala, frontotemporal cortex HPA axis functioning Decreased cortisol and enhanced negative feedback of adrenal functioning Less volume in hippocampus - unsure if these reductions represent cause or effect