Midterm Flashcards

(37 cards)

1
Q

What is VINDICATE?

A

A DDx list for why someone is having neurological Sx

Vascular

Infection/ inflammatory

Neoplasm

Degenerative / dysfunction

Intoxication

Congenital

AI / allergy

Trauma

Endocrine/ metabolic

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2
Q

What are the parts of a neurological exam? (8)

A

Hx

Mental status exam

Posture, gait, station

Coordination

Cranial nerve exam

SMR exam

Provocative tests

Ancillary tests

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3
Q

What are the parts of the mental status exam? (3)

A

Eval level of consciousness (attn & alertness)

Eval signs of dementia (cognitive)

Eval language and speech function

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4
Q

How do you eval level of consciousness?

A

Glasgow Coma Scale (GCS)

  1. Eye opening
  2. Best verbal response
  3. Best motor response
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5
Q

What are the 5 parts of the signs of dementia?

A

Loss of memory

Disorientation

Loss of intellectual fxn

Loss of abstract thinking

Loss of judgement

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6
Q

How do you eval for signs of dementia?

*hint: mnemonic

A

FOGS

Family story

Orientation (person, place, time)

General information (memory)

Spelling/ calculations

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7
Q

What are the 6 pieces of evaluating for language and speech function?

A

Spontaneous speech Comprehension Naming (word finding) Repetition Reading Writing

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8
Q

How do you test abstract though and judgement?

A

Ask patient to list difference between orange and apple

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9
Q

What is the language center in the frontal lobe of the dominant hemisphere and what does it do?

A

Broca’s center

Coordinates motor aspect of language

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10
Q

What is the language center in the temporal lobe of the dominant hemisphere and what does it do?

A

Wernicke’s center

Interprets the understanding/receptive aspect of language

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11
Q

What occurs when the language centers or their connections in the brain are damaged?

A

Aphasia

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13
Q

What is the connection between Broca’s and Wernicke’s called?

A

Accurate fasciculus

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14
Q

What are the 4 types of aphasia?

A

Broca’s aphasia Wernicke’s aphasia Conductive aphasia Global aphasia

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15
Q

Describe Broca’s aphasia

A

Expressive/motor aphasia Patient has difficulty speaking and is usually aware of deficit. Patient can comprehend language, and so is able to do tasks when asked. Therefore they are aware of deficit and frustrated.

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16
Q

Describe Wernicke’s aphasia

A

AKA Receptive/sensory aphasia Patient cannot comprehend or repeat language. Cannot follow oral or written commands, but can imitate examiner’s actions when prompted by a gesture. Language expression is preserved, so patient can speak though it is nonsensical words (neologisms). Patient may not be aware of deficits because their comprehension is disrupted.

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17
Q

What is global aphasia?

A

All aspects of brain’s language centers are damaged. As seen in large strokes of the internal carotid artery. No comprehension No expression No repetition

18
Q

What CN nuclei are in the pons?

A

5,6,7,8 in the pons (8 also spans into the medulla)

19
Q

How do you test visual acuity?

A

S Ellen chart

20
Q

How do you test visual fiends?

A

Confrontation: cover 1 eye and then patient look at doc’s eye. Have doctor test the peripheral vision by starting with finger outside quadrant and then moving in until patient can detect the finger. The object is to determine whether patient’s visual field is coextensive with-more restricted than-the examiner’s.

21
Q

Where are sensory integration disorder lesions from anatomically? And what do they cause?

A

Parietal lobe Cause misperception of or inattention to sensory stimuli not he side of the body opposite the lesion, even though primary sensory modalities (e.g. touch) are intact

22
Q

What are you looking for with pupillary reaction to light?

A

PERRLA Pupils: diameter & shape Equal: ~3 mm and can differ in size from side to side ~1mm Round: should be round React to Light and : constrict briskly in response to direct light and less so when the opposite pupil is illuminated (consensual response) AccomModation: eyes converge to focus on nearer object

23
Q

Miosis (pupillary constriction) is mediated through parasympathetic fibers that originate in ___ What would cause interruption of this p/way?

A

the midbrain and travel with oculomotor nerve to the eye Interruption of this pathway (tumor) would compress oculomotor nerve as it exits brain stem and produce dilated uncreative pupil. Interruption would NOT be caused by cerebral Lesion because its a brain stem mediated response.

24
Q

Normal visual fields: __ ˚ superior __ ˚ nasally __ ˚ temporally __ ˚ inferiorly

A

Superior 50˚ Nasally 60˚ Temporally 90˚ Inferiorly 70˚

25
Q

What happens to the visual field if there is inflammation of the optic disk or optic nerve?

A

See page 13 Large central scotoma (blind spot)

26
What happens if the optic nerve is completely severed on the right side?
Total blindness of the right eye Pg 13
27
What happens if there is a perichiasmal lesion? (E.g. calcified internal carotid artery)
Right nasal hemianopia Pg 13
28
What happens if there is a lesion of the left optic tract?
Right homonymous hemianopia Pg 13
29
What is the visual field if there is partial involvement of optic radiation by a lesion in the left temporal lobe (Meyer loop). What if there is partial involvement of optic radiation by a lesion in the left parietal lobe?
A - Right homonymous superior quadrantanopia B - right homonymous inferior quadrantanopia Pg 13
30
What happens if there is a complete lesion fo the left optic radiation
Right homonymous hemianopia Pg 13
31
What if there is a posterior cerebral artery occlusion?
Right homonymous hemianopia (spares the macula) Pg 13
32
What is papilledema? What Is it caused by? What is the Hx like?
Optic disc swelling d/t increased intra-cranial pressure Usually bilateral, painless transient episodes of blurred vision Increased physiologic blind spot
33
What are signs of early papilledema
Hyperemic disc Congested veins Loss of venous pulsation Disc margins blurred Disc appears elevated Splinter hemorrhages
34
What are signs of advanced papilledema
Obliteration of cup Flame hemorrhages Soft/hard exudates Visual field loss Blindness
35
Is advanced papilledema reversible?
Not usually
36
Sx of optic nerve
Blurring, blindness Loss of peripheral vision Blind spots Pain around the eye Headaches Pupillary light reflex loss
37
What are conditions that could cause Sx of optic nerve?
Optic neuritis Retrobulbar neuritis Optic atrophy/papill. Disease of nerve, chaism, tract
38