MKSAP Cardiovascular Flashcards

(38 cards)

1
Q

Diagnosis of non-ST-elevation MI

A

Characteristics: chest pain at rest, absence of ST elevation on ECG, and elevated MI biomarkers

  • ST depression is often seen.
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2
Q

Diagnosis of acute pericarditis

A

Three classic features: 1. pleuritic chest pain, 2. friction rub, 3. diffuse concordant ST-segment elevation on ECG

  • Chest pain that’s worse when supine.
  • Often can see PR segment depression.
  • Fever is often present.

Friction rub: 3 components - atrial systole, ventricular contraction, and rapid ventricular filling; squeaky, scratchy and high-pitched

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3
Q

ST-elevation MI treatment

A
  • PCI is the preferred treatment above thrombolytics.

- Most effective if completed within 12 hours of the onset of chest pain.

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4
Q

Contraindications to thrombolytic therapy

A
  1. prior intracerebral hemorrhage
  2. ichemic stroke within 3 months
  3. suspected aortic dissection
  4. active bleeding
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5
Q

Aortic dissection

A
  1. severe-onset chest pain radiating to the back
  2. BP differential between arms
  3. Murmur of aortic regurgitation
  4. Widened mediastinum of CXR
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6
Q

Treating RVMI

A
  • Volume expansion with normal saline

Physical exam findings: Classic triad - 1. hypotension, 2. clear lung fields, 3. elevated estimated central venous pressure

ECG: ST-segment elevation of right-sided leads

Treatment: Reperfusion therapy, IV fluids, possibly inotropic support with dobutamine if IV fluids are not sufficient (second-line due to risk of worsening infarction due to increased O2 demand)

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7
Q

Noncardiac, GERD chest pain

A
  • Symptoms can present with radiation and can last minutes to hours (even 18 hrs)
  • Stress test pretty much r/o cardiac ischemia making an empiric trial of PPIs reasonable.
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8
Q

Diagnosis of Third degree AV block

A
  • Complete absence of atrial impulses to the ventricle
  • Most common caused of marked bradycardia w/ ventricular rates usually 30-50.

Causes: Lyme carditis (acute-onset, high-grade AV conduction defects occasionally associated with myocarditis)

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9
Q

First-degree AV block

A

PR interval greater than 0.2 sec

- Often associated with a soft S1

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10
Q

Diagnosis of panic disorder

A
  • Symptoms peak within 10 minute of onset and usually last from 15-60 minutes

Treatment: CBT and SSRI

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11
Q

Pheochromocytoma

A

Classic triad: 1. sudden severe headaches, 2. diaphoresis, and 3. palpitations

Other symptoms: pallor, hyperglycemia, weight loss, arrhythmias, catecholamine-induced cardiomyopathy

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12
Q

Mobitz type II second-degree heart block

A
  • Associated with disease of the conduction system (bundle-branch block, etc.)

Treatment: pacemaker

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13
Q

Treatment of worsening symptoms of chronic stable angina

A
  • Increase beta blocker dosage
  • Beta blockers should be titrated to achieve a resting HR of approximately 55 to 60 bpm and approximately 75% of the HR that produces angina w/ exertion.
  • consider coronary angiography only after angina persists despite maximal medical therapy.
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14
Q

Ranolazine

A
  • Used to treat chronic stable angina

- Only added to baseline therpy that includes a beta-blocker, calcium channel blocker, and long-acting nitrate.

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15
Q

Diagnosis of PE

A

Symptoms: chest pain, dyspnea, asymmetric leg edema, elevated CVP, tachypnea, and tachycardia

Diagnosis: CT pulmonary angiography

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16
Q

Echocardiogram during chest pain

A
  • Normal wall motion excludes coronary ischemia or infarction.
17
Q

Ascending aortic dissection

A
  1. acute aortic regurgitation
  2. MI
  3. cardiac tamponade
  4. hemopericardium
  5. hemothorax or exsanguination
18
Q

Descending aortic dissection

A
  • Splanchnic ischemia, renal insufficiency, lower extremity ischemia, or focal neurologic deficit due to spinal cord ischemia
19
Q

Evaluation os suspected CAD

A
  • Exercise stress test

- Especially useful in pts w/ intermediate probability of CAD w/ a normal baseline ECG, who are able to exercise

20
Q

Adenosine nuclear perfusion stress test contraindication

A
  • Significant bronchospastic disease.
21
Q

Atrial flutter

A
  • multiple P waves in a sawtooth pattern w/ 2:1 ventricular conduction.
  • Most noticeable in inferior leads.
  • Sawtooth patternshows negative deflections in inferior leads, but positive deflection in V1.
22
Q

SA node dysfunction

A
  • AKA sick sinus syndrome
  • Symptomatic sinus bradycardia (in between episodes of tachycardia due to a fib) and tachycardia-bradycardia syndrome
  • A. fib is the most common tachyarrhythmia observed

Can be due to sinus arrest, sinus exit block, and sinus bradycardia

Treatment: pacemaker implantation

23
Q

Ventricular tachycardia

A
  • Wide-complex QRS tachycardia

- Rate 140-250

24
Q

LDL cholesterol in CAD

25
Atrial fibrillation
Rate between 350 and 600
26
Multifocal atrial tachycardia
- Associated with chronic lung disease w/ three of more P-wave configurations on ECG.
27
Atrioventricular reentrant tachycardia
AKA Wolff-Parkinson White syndrome | - Short PR interval (
28
Indications for coronary revascularization
Pt has chronic stable angina + one or more of these: 1. Angina pectoris refractory to medical therapy 2. a large area of ischemic myocardium 3. high-risk coronary anatomy (left main stenosis or 3 vessel disease) 4. Significant stenosis w/ reduced LV systolic function
29
Unstable angina treamtment
Admission to the CCU and IV heparin and nitroglycerin
30
Torsades de pointes
polymorphic ventricular tachycardia | - Rate of 200-300
31
Ventricular tachycardia
- Any wide complex QRS should be considered to be this unless proven otherwise
32
Cannon waves
- large a waves in jugular pulsations | - Signifies AV dissociation
33
Indications for inta-aortic balloon pump
1. ACS w/ cardiogenic shock unresponsive to medical therapy 2. acute mitral regurgitation secondary to papillary muscle dysfunction 3. ventricular septal rupture 4. refractory angina
34
Pathologic Q waves
-Width greater than 1mm and depth >25% the heighth of the QRS.
35
Always comes before CABG
Coronary angiography
36
Increased vagal tone
- Associated with inferior wall MI
37
Ventricular septal defect
- New systolic murmur, hypotension, and respiratory distress 1-3 days following an MI. - Palpable thrill.
38
New onset murmur following MI
mitral regurgitation or ventricular septal defect