MPR Study Session Flashcards
Where is CHE synthesized?
liver
An increase in CHE would cause
nephrotic syndrome and heptaocyte recovery
A decrease in CHE would cause
liver disease
Organophosphates are what type of inhibiter?
suicide inhibiter, irreversibly binds to CHE
How would you treat an exposure to organophosphates?
Atropine Sulfate (prevents Ach from binding to AchR)
Where our Aspartate Transaminases produces
Cardiac cells, liver, and blood cells
In infants, would you be alarmed with high levels of AST?
no, physiological levels increase in infants and hemolysis
Increase ASTs would stem from
MI, acute hepatitis,
modern increase in liver cirrhosis
Alanine Transaminases produced
skeletal muscle, liver
Increase in ALT would cause
Acute hepatitis,
modern levels chl. jaundice
Where are Alkaline Phosphates produced
bile duct and bone
Increases in ALP would suspect
bone disease and bile duct disease
After a high fat meal, what would increase?
ALP levels
If a woman is pregnant, what levels would increase AST, ALT, or ALP?
ALP levels would increase
Acid phosphate is produced where
majority in the prostate
What is the tumor maker for prostate cancer?
PSA
In the heart, what are the types of LDH?
1 and 2
In the liver, what are the types of LDH?
4 and 5
CK-BB produced where?
brain
CK-MB produces in the ?
heart
GGT is produced where ?
bile duct
Amylase and lipase is produced
pancreas
First enzyme to elevate in acute MI
troponin
second enzyme to raise in acute MI
CK-MB
