MSK Flashcards by Claudia Zizzo

WHAT IS THE DEFINITION OF JIA

A CHRONIC INFLAMMATORY JOINT DISEASE

PERSISTENT JOINT SWELLING

IN ABSCENSE OF INFECTION OR OTHER DEFINED CAUSES

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WHAT ARE THE SUBSTYPES OF JIA

OLIGOARTHRITIS

EXTENDED OLIGOATHRITIS

POLYARTHRITIS

SYSTEMIC

PSORIATIC

ENTHESITIS RELATED

UNDIFFERENTATED

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A CHILD COMES IN WITH

MORNING STIFFNESS AND PAIN
STIFFNESS AFTER REST
INTERMITTENT LIMP
LOW MOOD
POOR BEHAVIOUS
JOINT SWELLING
+/- SALMON PINK RASH

JIA

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WHAT ARE COMPLICATIONS OF JIA

CHRONIC ANTERIOR UVEITIS

FLEXION CONTRACTURES

GROWTH FAILURE

OSTEOPOROSIS

AMYLOIDOSIS

VALGUS DEFORMITY

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HOW DO YOU MANAGE JIA

EDUCATION AND SUPPORT

PHYSIO

NSAID

JOINT INJECTIONS (IF OLIGOARTHRITIS)

METHOTREXATE (POLYARTHRITIS)

PULSED IV CORTICOSTERIOIDS

METHYLPREDNISOLONE FOR SYSTEMIC ARTHRITIS

BIOLOGICS

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DESCRIBE THE ARTICULAR PATTERN OF EXTENDED OLIGOARTHRITIS

4< JOINT

AFTER 6M

ASSYMETRICAL JOINT DISTRIBUTION

ANA +VE

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DESCRIBE THE ARTICULAR PATTERN + LAB RESULTS OF OLIGO ARTHRITIS

1-4 JOINTS

MOST COMMONLY KNEE, ANKLE, WRIST

+/-ANA +VE

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DESCRIBE THE ARTICULAR PATTERN + LAB RESULTS POLYARTHRITS

SYMMETRICAL JOINTS DISTRIBUTION WITH HIGH FINGER INVOLVEMENT

1-6Y: RF -VE

10-16Y: RF +VE (POOR PROGNOSIS)

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WHAT DOES AMYLIODOSIS CAUSE

PROTEINUREA

RENAL FAILURE

HIGH MORTALITY

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WHY DOES OSTEOPOROSIS OCCUR IN JIA

REDUCED WEIGHT BARING

STEROIDS

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WHAT IS THE TREATEMENT OF JIA OSTEOPOROSIS

CALCIUM

VIT D

WEIGHT BARING EXCERSISES

DECREASE STERIODS

BISPHOSPHONATES

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WHY DO FLEXION CONTRACTURES OCCUR

FROM HOLDING THE JOINTS IN THE MOST COMFORTABLE POSITION

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WHAT ARE THE COMMON COMPLICATONS IN JIA FLEXION CONTRACTURES

JOINT DESTRUCTION REQUIRING REPLACEMENT

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HOW DO YOU TREAT CHRONIC ANTERIOR UVEITIS

CORTICOSTEROID EYEDROPS

MYCROLITIC EYE DROPS

CORTICOSTEROID INJECTION

SYSTEMIC CORTICOSTEROIDS

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WHAT IS CHRONIC ANTERIOR UVEITIS

INFLAMMATION OF THE ANTERIOR PART OF THE EE

VERY COMMON COMPLICATION OF IBD AND ARTHRITIS THAT CAN LEAD TO VISUAL IMPAIREMENT

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DESCRIBE THE ARTICUALR FINDINGS OF PSORIATIC ARTHRITIS

ASYMMETRICAL DISTRIBUTION

DACTYLISIS

PSORIASIS

NAIL PITTING

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WHAT IS THE PRESENTATION OF SYSTEMIC ARTHRITS

AND LAB FINDINGS

OLIGOARTHRITIS/POLYARTHRITIS

PAIN IN MUSCLES

ACUTE ILLNESS

SALMON PINK RASH

RAISED WCC

RAISED PLATELETS

RAISED INFLAM MARKERS

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WHAT ARE FEATURES ONLY FOUND IN POLYARTHRITIS FROM 10-16Y

RHEUMATOID NODULES

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HOW DO YOU DIAGNOSE SEPTIC ARTHRITIS

BLOOD CULTURES

BLOODS

JOINT ASPIRATE

XRAY

BRI

BONE SCAN

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HOW DOES THE PRESENTATION OF SEPTIC ARTHRITIS DIFFER IN INFANTS

PEUDOPARALYSIS

CRY IF JOINT IS MOVED

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WHAT IS THE PRESENTATION OF SEPTIC ARTHRITISIN CHILDREN

ERYTHETEMATOUS

ACUTELY TENDER

REDUCED ROM

ACUTELY UNWELL AND FEBRILE CHILD

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WHAT PATHOGENS COMMONLY CAUSE SEPTIC ARTHRITIS

STAPH. AUREUS

HiB

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WHAT IS TRANSIENT SYNOVITIS

MOST COMMON CAUSE OF HIP PAIN IN CHILDREN OFTEN ACCOMPNIED BY A VIRAL INFECTION

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WHAT IS THE PRESENTATION OF TRANSENT SYNOVITIS

SUDDEN ONSET OF PAIN

LIMP

DECREASED ROM

REFFERRED KNEE PAIN

AFEBRILE/LOW GRADE FEVER

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WHAT INVESTIGATIONS WOULD YOU USE TO DIFFERENTIATE TRANSIENT SYNOVITIS FROM SEPTIC ARTHRITIS

BLOOD CULTURES JOINT ASPIRATION

OUTLINE THE DIFFERENCES BETWEEN SEPTIC ARTHRITIS AND TRANSIENT SYNOVITIS

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HOW DO YOU MANAGE TRANSIENT SYNOVITIS

BED REST +/- SKIN TRACTION

WHAT DSEASE CAN HAVE TRANSIENT SYNOVITIS AS A PRECURSER

PERTHES

WHAT IS SEPTIC ARTHRITIS

A SERIOUS INFECTION OF THE JOINT SPACE WHICH CAN LEAD TO BONE DESTRUCTION

HOW DO YOU TREAT SEPTIC ARTHRITIS

IV ABX \> PO ABX SURGIAL LAVAGE IMMOBALIZATION OF JOINT MOBALISATION

WHAT ABX WOULD YOU USE IN SEPTIC ARTHRITIS FOR S. AUREUS

FLUCLOXACILLIN OR CEPHALEXIN

WHAT ABX WOULD YOU USE IN SEPTIC ARTHRITIS FOR HiB

CEFTRIAXONE

WHAT IS HIP DISPLASIA

AN ABNORMALITY OF THE HIP JOINT WHERE THE SOCKET DOESNT FULLY COVER THE BALL

WHAT ARE THE RISK FACTORS WHICH PREDISPOSE YOU TO HIP DYSPLASIA

FAMILY HISTORY SERTAIN SWADDLING TECHNIQUES BREECH BIRTH

WHAT IS THE PRESENTATION OF HIP DYSPLASIA

ASSYMPETRICAL LEG LENGTH AND SKIN FOLDS LIMP SENSATION OF INSTABILITY GROIN/HIP PAIN

HOW DO YOU DIAGNOSE HIP DYSPLASIA

NIPE EXAM US XR

WHAT IS THE TREATEMENT OF HIP DISPLASIA

CAST +/- HI TRACTION SURGERY PHYSIO MAY RESOLVE SPONTANEOUSLY

WHAT IS THE DEFINITION OF OSTEOMYELITIS

AN INFECTION OF THE METAPHYSIS OF LONG BONES

WHERE DOES OSTEOMYELITIS NORMALLY OCCUR

DISTAL FEMOUR PROXIMAL TIBIA

WHAT ARE COMMON PATHOGENS OF OSTEOMYELITIS

STAPH. AUREUS STREP HiB SALMONELLA -SICKLE CELL TB - IMMUNOCOMPRIMISED

WHAT IS THE PRESENTATION OF OSTEOMYELITIS

PAIN PSEUDOPARALYSIS SWELLING AND TENDERNESS ERYTHETEMATOUS WARM

HOW WOULD YOU INVESTIGATE OSTEOMYELITIS

BLOOD CULTURES BLOODS: RAISED WCC, ESR, CRP X RAYS

WHAT RADIOLOGICAL FINDINGS WOULD YOU HAVE IN OSTEOMYELITIS

SOFT TISSUE SWELLING SUBPERIOSTEAL NEW BONE FORMATION

HOW WPULD YOU TREAT OSTEOMYELITIS

PARENTERAL ABX\>IV\>PO SURGICAL DRAINIGE LIMB IN SPLINT EARLY MOBILISATION

WHAT IS KÖHLER DISEASE

A RARE BONE DISEASE AFFECTING CHILDREN AGED 6-9Y THERES NECROSIS OF THE NAVICULAR BONE DUE TO DECREASED BLOOD SUPPLY

WHAT IS THE PRESENTATION OF KOHLER DISEASE

PAIN AND SWELLING AT MIDDLE PART OF FOOT LIMP TENDERNESS AT MIDDLE FOOT

HOW WOULD OU INVESTIGATE KOHLER DISEASE

X RAY

HOW WOULD YOU TREAT KOHLER DISEASE

REST +/- CAST ANALGESIA

WHAT ARE DISCOID MENISCI

A RARE HUMAN ANATOMIC VARIENT AFFECTS LATERAL MENISCI OF THE BONE

DESCIBE THE WATANABE CLASSIFICTAIONS

INCOMPLETE - SLIGHTLY DEFORMED COMPLETE - FULLY ROUNDED WRISTBERG - LOCK OF POSERIOR MENISCI

WHAT IS THE PRESENTATION OF DISCOID MENISCI

PAIN CLICKING MECHANICAL LOCKING

WHEN DO DISCOID MENISCI PRESENT

ADOLESCENCE

HOW DO YOU DIAGNOSE DISCOID MENISCI

XRAY MRI

HOW WOULD YOU TREAT DISCOID MENISCI

SURGERY PHYSIO

WHAT IS OSGOOD SCHLATTERS

INFLAMMATION OF POSTERIAL LIGAMENT AT TIBIAL TUBEROSITY

WHAT RF PREDISPOSE YOU TO OSGOOD

IMPACT SPORTS OVERUSE

WHAT IS THE PRESENTATION OF OSGOOD

PAIN IN FRONT LOWER PART OF KNEE WORSE ON USE

HOW WOULD YOU DIAGNOSE OSGOOD

SYMPTOMATICALLY

HOW DO YOU TREAT OSGOOD

PHYSIO BRACE SURGERY

WHAT IS SLIPPED CAPITAL FEMORAL EPIPHESIS

DISPLACEMENT OF EPIPHESIS OF FEMORAL HEAD POSTERIOINERIORLY

WHAT CONDITIONS ARE ASSOCIATED WITH OSGOOD

HYPOTHYROIDISM HYPOGONADISM

WHAT IS THE PRESENTATION OF SLIPPED CAPITAL FEMORAL EPIPHESIS

LIMP HIP PIAN REFERRED KNEE PAIN DECREASED ROM

WHAT INVESTIGATIONS ARE REQUIRE FOR SLIPPED CAPITAL FEMORAL EPIPHESIS

X RAY

HOW WOULD YOU MANAGE SLIPPED CAPITAL FEMORAL EPIPHESIS

SURGICAL PIN FIXED IN SITU

WHAT IS PETHES DISEASE

AVASULAR NECROSIS OF THE CAPITAL FEMORAL EPPHESIS DUE TO AN INTERUPTED BLOOD SUPPLY

DESCRIBE THE PHYSIOLOGICAL PATHWAY OF REVOVERY FROM PETHES DISEASE

NECROSIS REVASULARISATION REOSSIFICATION

WHAT IS THE PRESENTATION OF PETHES DISEASE

HIP/KNEE PAIN LIMP SIMILAR TO TRANSIENT SYNOVITIS

WHAT IS THE MANAGEMENT OF PERTHES

IF IDENTIFIED EARLY BED REST + TRACTION OTHERWISE SURGERY

WHAT IS A POSSIBLE CONSEQUENCE OF PETHES DISEASE

INCREASED RISKOF DEGENERATIVE ARTHRITIS

WHAT IS OSTEOGENESIS IMPERFECTA

A GROUP OF DISORDERS OF COLLAGEN METABOLISM CAUSING BONE FRAGILITY CAUSING BOWING AND FREQUENT FRACTIONS

WHAT ARE THE TWO TYPES OF OSTEOGENESIS IMPERFECTA

TYPE 1 - AUTOSOMAL DOMINANT TYPE 2 - SEVERE AND LIFE THREATNING

WHAT IS THE PRESENTATION OF T1 OSTEOGENESIS IMPERFECTA

CHILDHOOD FRACTURES BLUE SCLERA IN 50% HEARING LOSS

WHAT IS THE PRESENTATION OF T2 OSTEOGENESIS IMPERFECTA

LETHAL IN PERINATAL PERIOD FRACTURES PRESENT BEFORE DEATH

HOW DO OU DIAGNOSE OSTEOGENESIS IMPERFECTA

X RAYS DNA + COLLAGEN TESTING US - FETAL PRESENTATION OFTEN IN T2

WHAT IS THE THREATEMENT OF OSTEOGENESIS IMPERFECTA

PHYSIO REHAB BISPHOSPHONATES (PALANDOMIC ACID IV) SURGERY

WHAT IS RICKETS

A FAILURE IN MINERALISATION OF THE GROWING BONE (OSTEOID)

WHAT IS OPTEOMALACIA

FAILURE TO MINERALISE MATURE BONE

WHAT ARE THE TYPES OF RICKETS

NUTRITIONAL VIT D DEPENDANT VIT D RESISTANT

WHAT NUTRITIONAL DEFICIENCIES CAN CAUSE RICKETS

VIT D CALCIUM PHOSPHURUS

WHAT ARE THE TWO TYPES OF VITAMIN D RICKETS

TYPE 1: DECREASED VIT D HYDROXYLASE TYPE 2: DEFECTIVE VIT D RECEPTOR AND PRODUCTION OF 1,25 DIHYDROXY VIT D

WHAT ARE THE TWO TYPES OF VIT D RESISTENT RICKETS

X LINKED : DECREASED RENALPHOSPHUSRUS TRANSPORT /ABSORBTION = HYPOPHOPHOATREMIC HEREDIATRY : DECREASED PHOSPHATE REABSORBTION WITH RAISED LEVELS OF CALCITOL = HYPOPHOPHOATREMIC + HYPERCALCEAMIC

WHAT ARE CAUSES OF RICKETS

NORTHERN LATITUDES DIETARY MATERNAL VIT D DEFICIENCY COELIAC PANCREATIC INSIFFICIENCY CHOLESTATIC LIVER DISEASE CHRONIC LIVER DISEASE HEREDITARY X LINKED FANCONI SYNDROME

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