MSK Injuries in Children and Adolescents Flashcards

(91 cards)

1
Q

Age 12-15 boys

A

Second growth spurt

Excessive stress on musculoskeletal system that isn’t present in mature skeleton

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2
Q

Dominant biological process first 20 years of life

A

Growth

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3
Q

Difference between child and mature adult

A
Epiphyseal plates
Growth spurts
Bone malleability
Apophysites
Articular cartilage
Muscle development
Frequency and variety fo sport
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4
Q

Epiphysis

A

End of bone

Covered in articular cartilage

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5
Q

Metaphysis

A

Adjacent to epiphyseal plate
Undergoes growth in adolescents
Softer

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6
Q

Diaphysis

A

Shaft of long bone

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7
Q

Epiphyseal plates

A

Developing skeleton
Site of weakness
Susceptible to sheer forces- are of bone weaker

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8
Q

Epiphyseal plates + growth

A

Growth occurs and forms cell matrix

Then calcifies to become skeletal bone

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9
Q

Growth spurts

A

Changes in balance between bone and muscle length
Change in coordination + biomechanics
Effect on energy levels

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10
Q

How do kids grow

A

Bone length changes

Soft tissue adapts to that

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11
Q

Bone malleability

A

Metaphysis of bone is softer
Absorbs greater energy
Bones less brittle- less likely to shatter
Less dense and more porous bone

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12
Q

Bone in adolescents properties

A

Less brittle
Less dense
More porous
Susceptible to diff. form of fractures e.g. greenstick

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13
Q

Apophysite

A

Bony attachment site of a tendon

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14
Q

Pelvic apophysites

A
Iliac crest
ASIS
AIIS
G. Troc
L. Troc
Ischial Tub
Pubic symph
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15
Q

Iliac crest is attachment site of

A

Gluteals

TFL

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16
Q

ASIS is attachment site of

A

Sartorius

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17
Q

AIIS is attachment site of

A

Rec Fem

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18
Q

G. Troc is attachment site of

A

Glute Med/Min

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19
Q

L. Troc is attachment site of

A

Psoas/Iliacus

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20
Q

Ischial Tub is attachment site of

A

Add magnus
Biceps femoris
Semi tend
Semi memb

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21
Q

Pubic Symph is attachment site of

A

Rectus abdominus via inguinal ligament

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22
Q

Apophysitis

A

Inflammation of tendon attachment onto bone

Mature skeleton- area of weakness in bone itself not the MTU

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23
Q

Articular cartilage is

A

Site of development and remodelling of adolescent bone
Thicker than in adults
Greater ability to remodel

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24
Q

Articular cartilage remodelling

A

Can be damaged more easily
Thicker and less mature
But has a chance to mature

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25
Articular Cartilage layers (top to bottom)
Articular surface Superficial Tangential Zone (10-20%) Middle Zone (40-60%) Deep Zone (30%)
26
Osteochondritis Dessicans (OCD)
When blood supply is cut off Cartilage starts to degenerate More common in children Damage can be caused by trauma or overuse
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Joint stability
Lower in developing skeleton Less muscle development More ligament laxity- incomplete cross bridge formation Less core stability
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Inflammatory Conditions
``` Juvenile RA/SLE Reactive arthritis hx Ex Bloods Urine Joint aspiration Management ```
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Cardiovascular changes- compared to fully mature athlete
``` Lower systolic BP Lower SV Increased maximal HR Lower cardiac output Increased RR Less anaerobic power Screening ```
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Environmental changes
``` greater body SA to mass Lower sweating rate More skin SA to gain/lose heat Rate of heat acclimatisation lower Problems in hot and cold environments More regular drinks breaks, subs waiting inside ```
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Ethics
``` Autonomy Beneficence Confidentiality Do no harm Equity ```
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Important conditions
``` Fractures Hip + groin complaints Back pathology Traction apophysitis Joint instability ```
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Growth plate fracture
Salter-Harris classification Treatment depends on type Can be complicated
34
Growth plate fracture healing
``` Depends on: severity age which growth plate type ```
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SALTER stands for
``` S- straight across A- above L- lower TE- through everything R- cRush ```
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Salter Harris classification
``` 1-5 All are complicated Type 5 worst GP effect--> affects growth e.g. leg length discrepancy GP can become inactive ```
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Type 1
A complete physeal fracture with or without displacement
38
Type 1 treatment
Rarely will have to be put back in place | Normally just need a cast- all things still intact- unless damage to blood supply, should grow normally once healed
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Type 2
A physeal fracture that extends through the metaphysis, producing a chip fracture of the metaphysis, which may be very small
40
Type 2 treatment
Most common | Typically have to be put back in place surgically and immobilised to allow growth to continue
41
Type 3
A physeal fracture that extends through the epiphysis
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Type 3 treatment
Occurs rarely Usually lower end of tibia Surgery sometimes necessary Outlook/prognosis for growth is good if blood supply to separated bit still intact
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Type 4
A physeal fracture plus epiphyseal and metaphyseal fractures
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Type 4 treatment
Surgery needed to restore bone back and perfectly align growth plate If not perfect alignment achieved when placed back or during growth, prognosis for growth is poor
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Type 5
A compression fracture of the growth plate
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Type 5 treatment
Uncommon Occurs when bone is crushed Prognosis poor Growth likely to be stunted in that portion only --> biomechanical imbalances
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Greenstick Fracture
One side broken, one side bent- like breaking branch Reduced Casted 6 weeks Usually occur in metaphysis
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Buckle fracture
``` 'Torus' fracture Incomplete fracture as one side of bone Buckles without disrupting other side --> outside intact, inside fractured 5-11 years FOOSH Pain that lasts couple of hours, child doesn't want to use arm Quicker healing time 3 week cast ```
49
Hip/groin pain
``` Traumatic Apophyseal injuries Avascular necrosis of hip Perthe's diseases Slipped upper femoral epiphysis Most common boys 5-12 4th most common injury affecting footballers 3rd longest absence from sport (after fracture + ACL injury) ```
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Hip/groin pain mechanism of injury
Torque/twisting with opposing forces Conjoint tendon pulls up and rotates trunk Adductor pulls down and rotates upper leg Opposing forces disruption of muscles at their insertion Imbalance between weak abdominal muscles in relation to strong leg muscles
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Perthe's disease
Reduced circulation to femoral head and vitamin deficiencies affecting bone formation and development Avascular necrosis During revascularisation bone is soft
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Perthe's disease epidemiology
``` Boys 5-12 Painful hip Limp X ray/bone scan/MRI ```
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Perthe's disease x ray reasoning
Hip pain + limp + under 18
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Slipped Upper Femoral Epiphysis (SUFE)
Femoral head slips posteriorly | Due to weakness of growth plate- sheer force across GP
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SUFE Epidemiology
During periods of accelerated growth Antalgic gait/limb Leg short and externally rotated Surgery
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Avascular necrosis of hip
Ball/socket joint Articular cartilage Damage --> collapse/flattening of femoral head blood supply is through neck of femur, so is shut off
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Avascular necrosis of hip causes
NOF Dislocation ETOH systemic cortisone
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Avascular necrosis of hip symptoms
``` Pain on weightbearing Pain- lower abdo, groin, testicle Weakness running/cutting/side-steps Sit ups Coughing/sneezing ```
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Avascular necrosis treatment
Conservative | Surgical-decompressing femoral head
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Blood supply age 8-10 head of femur
The artery to head of femur (goes through ligament to head of femur/teres) is lost and the medial femoral circumflex takes over
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Back pain in young athletes
``` Spondylolysis Spondylolisthesis Lumbosacral sprain Scoliosis Scheuermann's disease Osteomyelitis Congenital abnormalities Ankylosing spondylitis Juvenile RA Malignancy ```
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Spondylolysis
Defect on pars interarticularis 6% general pop 50% sporting back pain Repetitive hyperextension
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Spondylolysis occurs commonly in
L4/5 or L5/S1
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Spondylolysis history
Sport related pain Can also be asymptomatic, and found incidentally on imaging Football, cricket bowlers, gymnastics, weightlifting Daily activity related pain Rest pain
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Spondylolysis history also ask
``` Morning stiffness Multiple joint pain/swelling Night pain Neuro symptoms Systemic symptoms ```
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Spondylolysis examination
``` Observation SIF ROM Pain on lumbar extension, single leg extension or extension combined with rotation Slump test ```
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Spondylolysis investigations
X ray- AP, lat, oblique SPECT CT MRI
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Spondylolysis treaatment
Relative rest from extension or aggravating activities Analgesia Rehab- core, flexion activities, hamstring stretches, aerobic fitness, sports specific Bracing
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Traction Apophysitis'
Inflammation of site of tendinous attachment
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Traction Apophysitis' examples (overuse injuries)
``` Osgood Schlatters Severs Sinding Larsen Johansson Little league elbow Iselins ```
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Severs
Calcaneum | Achilles tendon
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Sinding Larsen Johansson
Inf. pole patella | Patella tendon
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Little league elbow
Med. epicondyle | Wrist flexors
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Iselins
5th metatarsal | Peroneus brevis
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Avulsion fractures
Bone v soft | Avulsion fracture
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Osgood Schlatters
Inflammation at site of patella tendon attachment at tibial tubercle In adults --> superior and inferior patella tendinopathy
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Osgood Schlatters clinical findings
``` TOP tibial tubercle Protruded tibial tubercle Pain on resisted knee extension/squatting Pain on passive knee flexion Restricted hams ability ```
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Osgood schlatters RFs
``` Biomechanical issues: poor quads flexibility poor hams flexibility growth spurt increased Q angle patella alta overpronated feet knee valgum Relationship between growth and load ```
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Severs
Inflammation at site of calcaneal growth plate
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Severs biomechanical factors
Overpronation/valgus at ankle | Stiff forefoot
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Severs clinical finding
TOP calcaneal growth plate Pain and restriction on DF stretch Pain on resisted PF/calf raise
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Treatment and rehab of traction apophysitis
Rest Treatment of inflammation address biomechanical factors improve movement factors
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Patellofemoral instability
Patella will always dislocate laterally | Joint laxity
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Patella instability mechanism
Patellar alignment maintained by fibrous structures Lateral pull vs medial pull Subluxation if partial loss of patella femoral joint congruity Dislocation is complete loss of joint congruity
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Patella instability biomechanical RFs
``` Shallow femoral trochlea Hypoplastic lateral femoral condyle Patella shape Patella alta Poor VMo strength ```
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Patella dislocation
One of medial structures fail Medial patella femoral ligament detached at femoral attachment, then pulled out by ITB Avulsion fracture at chondro-osseus junction
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Patella dislocation treatment
X ray and orthopaedic review Brace vs no brace progressive knee flexion Main aims of rehab are to strengthen VMO whilst limiting tension of lateral structures
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Osteochondritis Dissecans S and S
``` Vague history joint pain in children normally full ROM Effusion Palpation of joint line will be tender Locking/giving away may be reported Wilson's sign ```
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Osteochondritis dessicans
Separation of articular cartilage from subchondral bone | Avascular fragment can result in loose body
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RFs for OD in knee
``` Trauma (50%) Male sex overuse due to sport Familial 10% ligamentous weakness genu valgum/varum meniscal lesions in knee ```
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Anthropometric measurements
Height and weight measurements- 1 per month Screen those with significant growth Assess for growth related pathologies- moree than 1cm/month Use to manage load