Nervous system Flashcards

(186 cards)

1
Q

Brain

controls
generates

CNS

A

controls center of nervous system-

generates thoughts, emotions and speech

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2
Q

Hemispheres-

what kinds/lobes
each hemisphere
ie

CNS

A

Left & Right & 4 lobes-

each hemisphere receives sensory and motor impulse from opposite side of body

(Ie right hemisphere receivers impulse from left side of body

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3
Q

Left hemisphere controls what

CNS

A

Left side controls language

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4
Q

Right hemisphere controls what

CNS

A

non verbal perceptual function

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5
Q

Brainstem consists of:

CNS

A

midbrain

pons

medulla oblongata

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6
Q

Midbrain function

CNS

A

center for auditory and visual reflexes

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7
Q

Pons function

CNS

A

controls respirations

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8
Q

Medulla oblongata-

important role in

CNS

A

important role in cardiac rate, blood pressure, respirations and swallowing

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9
Q

CSF

liquid/derived from where
forms
helps

CNS

A
  • CSF is a clear colorless liquid derived from blood plasma

, forms a cushion for brain tissue and protects the brain and spinal cord from trauma.

Helps nourish brain and remove waste products

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10
Q

Spinal Cord-

protected by
serves as

CNS

A

protected by vertebrae,

serves as center for conducting messages to and from brain

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11
Q

Meninges-

protective
covers

CNS

A

protective tissue membrane

covers the brain and spinal cord

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12
Q

Peripheral nervous system

links
consists. of

A

Links the central nervous system with rest of body

Consists of Nerves, sensory receptors, motor receptors

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13
Q

Spinal nerves

contain
31 pairs-
C
T
L
S
C

A

contain both sensory and motor fibers

31 pairs- named by locations-
cervical (8)
, thoracic (12)
lumbar (5),
sacral (5)
, coccygeal 1

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14
Q

Reflexes-what are they

A

rapid, involuntary motor responses to a stimulus

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15
Q

Olfactory nerve #

function

A

1

sense of smell

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16
Q

Optic nerve #

function

A

2

vision

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17
Q

Oculomotor nerve #

function

A

3

eyeball movement
raining of upper eyelid
contraction of pupil
proprioception

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18
Q

trochlear nerve #

function

A

4

eyeball movement

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19
Q

Trigeminal nerve #

function

A

5

sensation of upper scalp, upper eyelid, nose, nasal cavity, cornea and lacrimal galena

sensation of palate, upper teeth, check, top lip, lower eyelid

chewing

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20
Q

Abducens nerve #

function

A

6

lateral movement of eyeball

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21
Q

Facial nerve #

function

A

7

movement of facial muscles

secretions of lacrimal,, nasal, submandibual and sublingual glands

sensation of taste

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22
Q

acoustic nerve #

function

A

8

sensation of equilibrium

sensation of taste

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23
Q

glossopharngyleal nerve #

function

A

9

swallowing

gagreflex

sensation of taste

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24
Q

vagus nerve #

function

A

10

swallowing

regulation of cardiac rate

regulation of respirations

digestion

sense of taste

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25
accessory nerve # function
11 movement of head and neck
26
hypoglossal nerve # function
12 movement of tongue for speech and swallowing
27
autonomic nervous system regulates what
internal environment of the body
28
sympathetic division what is primary trasnmitter prepares for what autonomic nervous system
norepinephrine is primary neurotransmitter of sympathetic division- prepares the body to handle harmful or stressful situations
29
sympathetic division how will body present autonomic nervous system
dilated pupils, increased heart rate/force of heart contraction, increased glucose release diaphoresis
30
Parasympathetic Division- what is primary trasmitter operates when autonomic nervous system
acetylcholine is primary transmitter, operates during non stressful situations,
31
Parasympathetic Division- what happens when stimulated autonomic nervous system
constriction of pupils, decreased heart rate vasoconstriction of coronary arteries
32
What do diagnostic tests of nervous system used for nurse is responsible for-(explain/ assess/ signed/ sup)
Help to support diagnosis of specific disease or injury Nurse is responsible for : Explaining procedure & special considerations Assessing medications Signed consent Supporting patient
33
Diagnostic Tests of the Neurologic System
Carotid Duplex CT Scan –used for suspected stroke or looking for blockage/ hemorrhage EEG Lumbar Puncture –regularly address dressing site, encourage inc of oral fluids, encourage acetaminophen for back/headaches MRI Myelogram PET scan
34
Health Assessment Interview consider what- interview-questions? what testing
Consider genetic influences-many neurologic diseases have a genetic component due to inheritance of a gene or gene mutation, Interview- questions to ask?--- ask about family history, health problems related to neurological disorders, history of disorders like- seizures, ALS,MS, Parkinson, tremors, past history of dizziness, headaches, trauma, or injury Genetic testing, counseling if data collected indicates risk factors or alterations
35
Neurological Physical Assessment
Mental Status, cranial nerve assessment, sensory assessment, motor function, reflexes Vitals, assesses level of consciousness, pupillary response, strength, sense pain/touch
36
Glasgow Coma Scale what is it assessment of what score of 15 is what
–objective tool used to describe extend of impaired consciousness, Assessment of eyes open, best motor response, best verbal response Score of 15 indicates patient is alert and oriented; highest level of functioning
37
Decorticate manifestations arms wrists legs feet
you will see upper arms are close to sides, wrists and fingers are flexed, legs internally rotated feet plantar flexed
38
Decorticate cause
posture that occurs with lesions of cortical spinal tract
39
Decerebrate manifestaitons arms wrists feet
arms pronated and extended, wrists that are flexed feet plantar flexed,
40
Decerebrate cause
posture that occurs with lesions/injures to brain stem-midbrain or pons
41
Aphasia-
defective/ absent language function
42
Dysphonia –
change in tone of voice
43
Dysarthria
–difficulty speaking
44
Dysphagia
–difficulty swallowing
45
Anosmia
– inability to smell
46
Nystagmus
involuntary eye movement
47
Ataxia
lack of coordination/ clumsiness
48
Flaccidity
muscle tone is decreased or absent causing extremities to drift in strokes or loss of tone d/t consequences of stroke
49
Spasticity
–muscle tone increased- tightening of muscles-seen in MS and cerebral palsy
50
Consciousness
individual is aware of self/environment and they respond appropriately
51
altered level of consciousness looking at
Level of consciousness Pupillary response Oculomotor response Motor response Breathing
52
AEIOU altered loc
alcohol, epilepsy, insulin, opium, uremia(high levels of urate in blood)
53
T-I-P-S-S what causes altered loc
tumor, injury, psych, stroke, sepsis
54
Full consciousness- Decreased LOC
alert and orientated, can comprehend
55
Confusion- Decreased LOC
patients are unable to think rapidly or quickly
56
Disorientation- Decreased LOC
not aware, nor orientated to time place or person,
57
Obtunded Decreased LOC
- lethargic but react to stimuli and drift back to sleep
58
Stupor- Decreased LOC
briefly affected by painful stimuli
59
Semicomtatose- Decreased LOC
do not move spontaneously, stimuli may cause moaning, but withdrawal right away
60
Coma Decreased LOC
- unarousable, make no attempt to withdrawal
61
Deep coma Decreased LOC
- absence of reflexes
62
what is a seizure abnormal may result
Its an Abnormal electrical activity may result in abrupt altered stated of function
63
epilepsy partial seizure generalized seizure
Epilepsy- seizure disorder, chronic Partial Seizure  may affect only part of brain Generalized Seizure  affects all of brain
64
MRI & CT- Seizures- Diagnosis
Abnormalities in brain
65
EEG- Seizures- Diagnosis
helps to localize any brain lesions and confirm diagnosis
66
Lumbar Puncture- nursing considerations assess encourage backpain/headahce Seizures- Diagnosis
assess spinal fluid for CNS infections Nursing Considerations??  assess dressing site encourage increase of oral fluids acetaminophen for back pain/ headache
67
labs Seizures- Diagnosis
Look at Blood count, electrolytes, blood urea, glucose
68
Antiepileptic drugs dont cure-rather they limits Seizures- Medications
- don’t cure; rather they: reduce/ control activity- it will raise the seizure threshold, which limits the spread of abnormal activity in the brain
69
Goal->seizure meds protect reduce allow focus need
protect from harm, reduce/prevent activity-will be lethargic , allow pt to rest, focus on safety and positioning if another seizure, need to lay on side
70
Seizures- Medications
Phenytoin phenobarbital, valproic acid lamotrigine gabapentin
71
Seizures- Medications considerations: minimum dosing not always frequent
Pt will be on for minimum of three years – if Seizure free, withdrawal may be considered by reducing one drug at a time over several weeks/months Dosing-need to take exact dose Not always well tolerated-ca cause decreased alertness, headache, dizziness, Frequent blood monitoring-make sure they have decreased blood levels, need to do liver function test
72
health promotion seizures what care stress importance of vehicles family teaching
Follow up care, Stress importance of taking, scheduling, considering and continuing medications even if no seizure activity Vehicles –may be prohibited and reinstated after prover has cleared them Family teaching-first aid
73
positioning during episode positioning after episode
Positioning during episode cushion- head if seizure, loosen anything tight around neck, turn head and body to side and call for assistance Positioning after episode –let them rest, but lay on side in case of another seizure
74
ineffective airway clearence-seizures what happens what pools pt airway interventions-clothing/ turn/ dont put/ may need
Tongue falls back, obstruct airway –Gag reflex depressed Secretions pool Patent airway interventions: risk for aspiration- loosen clothing, turn to side, do not put anything in mouth and may need oxygen
75
anxiety/knowledge-seizures provide support identify medical identification watching avoiding
Provide support –emotional, mental, physical Identify safe activities –identify safe activities when shower or bathing Medical identification-bracelet or card watching Triggers/aura Avoiding alcohol/coffee
76
heachace pain where results from
Pain within the cranial area Result from ->benign or pathologic conditions such as stress, tension or disease process
77
Migraine what is it ignited by accompanied by
- recurring primary headache, initiated by trigger, accompanied by neurologic dysfunction
78
Cluster Headache s/s
severe or burning, poking sensation in or directly around the eye causing the eye to water
79
headache based on-> diagnostic tests to rule out structural disease processes->
Based of history & physical, symptoms, precipitating events Diagnostic tests to r/o structural disease processes mri, xray, eeg, lumbar puncture
80
Sumatriptan binds rapidly effective medication headache
binds with serotonin receptors rapidly effective when given in onset of migraine
81
Once migraines are in progress meds
narcotics/antiemetics may be prescribed
82
what helps to decrease incidence of headaches when wake up no avoid
when wake up, eat meals, get regular exercise,, no artificial sweeteners or msg Avoid stimulates & food high in tyramine  avoid smoked meats, beers, alcohol, chocolate or caffeine
83
Alzheimer's- Overview
Form of dementia- progressive, irreversible deterioration of intellectual functioning
84
stages 1-7 alzheimers
Stage 1- no cognitive impairment Stage 2- Very mild decline Stage 3-Mild cognitive decline Stage 4- Moderate cognitive decline Stage 5- Moderately severe cognitive decline Stage 6- Severe cognitive decline Stage 7- Very severe cognitive decline
85
diagnosis of Alzheimers
No specific diagnostic test- can have postmortem examination of brain tissue History and physical, identifying if reversible or treatable
86
Alzheimer medications-> use what meds
Medications slow the progression of cognitive decline Donepezil (), memantine ()
87
Priorities in care alzheimers top priority risk for injury con daily caregiver
Top Priority: optimize environment to patients functional level Risk for injury- SAFETY!!- need protective measure to keep them safe as they decline Continuity of care Daily routines Caregiver, role strain
88
Multiple Sclerosis- what is it
What is it? A chronic disease of the CNS (Brain, nerves, spinal cord)
89
Multiple Sclerosis- patho response destroyed slows/distorts
autoimmune response, the myeline sheath that protects nerve fibers are destroyed, demyelination slows and distorts impulse transmission
90
manifestations for MS vary-> manifestations
Manifestations vary- depending on area of nervous system involved Sensory loss, visual deficits, weakness, paresthesia, ataxia, vertigo
91
Multiple sclerosis exacerbations remission end result
Exacerbations- manifestations present, remission- manifestations not obvious end result- total loss of function
92
Diagnostic Test MS
MRI –may show lesions Cerebral spinal fluid analysis –lymphocytes indicating an immune response or increased igg CT Scan-may reveal atrophy in white matter lesion
93
One of the following criteria has to be met MS two history slowly
Two or more exacerbations separated by 1 month or more and lasting more than 24 hours, followed by recovery A history of repeated exacerbations and remissions with out without complete recovery; progression of symptom severity Slowly increasing manifestations for at least 6 months
94
multiple sclerosis medications are used for treat modify interrupy surgery ->
1. treat manifestations 2._modify course of disease 3-interrupt progression of disease surgery ->indicated for severe spasticity and deformity
95
Immunomodulators- meds given for monitor assess/ rotate report Multiple sclerosis
interferon, given to prolong onset of disability, monitor liver function tests, cbc assess / rotate injection sites report any feelings of suicide/self harm
96
Adrenocorticosteroid- type of meds given to treat avoid doses labs take when Multiple sclerosis
"prednisone”- given to treat exacerbations of ms, suppress immune system, avoid crowds taper doses labs- inc glucose take in morning
97
Muscle Relaxants- meds receives/supress can have change maintain Multiple sclerosis
“baclofen, diazepam, -relieves muscle spasms, suppress cans reflexes can have sedative effects-monitor fall precautions, change positions slowly maintain safety-fall precautions
98
Immunosuppressant- meds what does assess watch for protect against increase labs Multiple sclerosis
imuran, methotrexate, suppress immune system assess anemia/fatigue watch for bleeding, protect against infection increase fluids labs- CBC and urine
99
Multiple sclerosis watch what fatigue-> components Nursing Care: Health Promotion
watch Nutrition, Dietary needs, Weight Fatigue ->low blood counts, medications that cause sedative effect, poor nutrition, overall muscle weakness Psychosocial components
100
Multiple sclerosis fatigue rehab assess arrange ask perform avoid releive refer
assess fatigue arrange daily activites to promote rest ask patents for priotites perform taks in morning avoid extreme temps receive pain refer to apporpatite professionals
101
Parkinson Disease- degenerative characterized by
Degenerative neurologic disorder- progressive Characterized by tremors, muscle rigidity, bradykinesia (slow movement), postural instability
102
Tremor manifestations parkinsons
at rest, starts on one side and progresses to both, affects dexterity and fine muscle control
103
Rigidity & Bradykinesia manifestations parkinsons
slow movements, making passive and active movements difficulty, difficulty starting, continuing and coordinating movements –shuffling gait
104
Abnormal posture manifestations parkinsons
flexion of head makes It difficulty to maintain upright position
105
Autonomic & Neuroendocrine manifestations parkinsons
elimination problems, blood pressure issues and skin changes
106
Mood & Cognition manifestations parkinsons
high incidence of depression rt dimension
107
Sleep disturbances-> manifestations parkinsons
difficulties falling asleep and staying asleep
108
diagnosis of Parkinson's cardinal signs
H&P, having 2/3 cardinal signs- tremor at rest, bradykinesia, rigidity
109
is there cure meds do what Pt/ot/st Parkinson's
No cure; medications improve debilitating manifestations PT/OT/ST-focusing on strength, tremor control and activities that strengthen and control muscles on affected side
110
Dopamine Precursors- Carbidopa- Levodopa- how does it work makes what improves /decreases Parkinson's meds
 carbidopa makes more levodopa for the brain –which improves mobility and decreased muscle rigidity and tremors
111
Dopamine Precursors-Carbidopa-Levodopa nursing repsosiblites -s/e Psychological reactions Parkinson's meds
s/e- nausea vomiting, orthostatic hypotension Psychological reactions- hallucinations and vivid dreams
112
Dopamine Agonists-Ropinirole//Pramipexole mimics/increases equals How does it work?? Parkinson's meds
Mimics role of dopamine in brain and increases effects of levodopa, which equals a reduction of symptom fluctuation
113
Dopamine Agonists-Ropinirole//Pramipexole nursing repsobilites Parkinson's meds
Can cause compulsive behaviors
114
Anticholinergics-Benztropine blocks used to ease how does it work Parkinson's meds
Blocks excitatory action of acetylcholine , used to ease side effects like drooling, tremors and rigiity
115
Anticholinergics-Benztropine nursing responsibilities mouth can have changes assess for increase check Parkinson's meds
Dry mouth, can have. Urinary retention mentation changes assess for other meds that have anticholinergic effect inc fluids check I and o
116
Parkinson's preventative measure of complications
Malnutrition, falls, skin breakdown, constipation, safety
117
parkinsons education how to take incrase slow limit increase
how to improve sleep take meds increase protein slow positional changes limit alcohohol increase fluids
118
Amyotrophic lateral sclerosis (ALS) what is->rapid muscle aka incidence
Rapid progressive and fatal degenerative neurologic disease Muscle weakness and wasting, fatigue AKA lou Gehrigs disease Incidence high in men vs women, middle to late age
119
survival after onset ALS
2-5 years dt repository failure and decreased repository function
120
complications of ALS
communication dysfunction , muscle dysfunction needed for respiratory support swallowing issues
121
ALS- Musculoskeletal Manifestations
fatigue, progressive muscle weakness, twitching of muscles
122
ALS- Respiratory Manifestations
difficultly clearing airway, complications such as pneumonia, respiratory failure and often death is due to decreased function
123
ALS- Nutritional Manifestations
difficulty chewing, aphasia ultimately malnutrition
124
ALS- emotional Manifestations
 loss of control can lead to depression
125
goals for als
r/t decreasing complications r/t respiratory status & mobility
126
Risk for disuse syndrome ALS at risk for altered cannot move
At risk for developing problems associated with bedrest Altered nutrition and hydration status Cannot move and reposition self, at risk for  risk for breakdown, reposition every 2 hours
127
Stroke/CVA Emergency condition->results from
neurologic deficits result from sudden decrease in blood flow to area of brain
128
risk factors for stroke/cva
Hypertension, a fib, hyperlipidemia, sleep apnea, smoking, family history, obesity
129
Ischemic stroke thrombotic stenosis embolic
Thrombotic-blockage dt blood clot fat, stenosis dt plaque embolic stroke dt a fib
130
Hemorrhagic stroke
blood vessel ruptures, spilling blood into surrounding area
131
what is used for stroke when looking for blocking/ hemorrhage
ct scan
132
Manifestations stroke onset left hemisphere right hemisphere FAST glucose->
Sudden in onset, focal and usually one-sided Left hemisphere– right sided deficits, drifts Right hemisphere- left sided deficits , drifts FAST- Face, Arm, Speech (aphasia), Time-quick assessment, monitor repository status, pulse ox and treat as needed Low glucose can mimic sign of stroke
133
Visual Complications: Hemianopia Homonymous Hemianopia Complications Related to Stroke
Hemianopia- Loss of half of visual field of one or both eyes Homonymous Hemianopia- visual loss in the same half of the visual field of each eye
134
Cognitive & behavioral Complications: agnosia-> Consciousness: loss decreased poor Complications Related to Stroke
Agnosia-> inability to identify objects or people Memory loss, decreased attention, poor judgemen
135
behavior changes-> Consciousness-> Complications Related to Stroke
Inability to control emotions Consciousness-> ranging from mild – coma
136
Aphasia- inability common Communication Disorders Complications Related to Stroke
inability to use or understand language; common with CVA
137
Expressive can can only Communication Disorders Complications Related to Stroke
- can understand what is being said, can only respond verbally in short phrases and can be garbled
138
Receptive cant speech Communication Disorders Complications Related to Stroke
- can not understand spoken world or written word, speech is fluent but inappropriate in content
139
Mixed/global- dysfunction Communication Disorders Complications Related to Stroke
dysfunction in both understanding and expression
140
stroke may cause change in Elimination Disorders Complications Related to Stroke
Stroke may cause loss of sensation that trigger elimination Change in bowel elimination –watch bowel regimen and if they need anything
141
Urinary frequency, urgency or incontinence – offer how often/why
offer means of urinary elimination via urinal, commode – do this every 2 hrs to make urinary schedule and prevent skin breakdown
142
Stroke interrupts produces Motor Deficits Complications related to Stoke
Stroke interrupts component of relay system produce effects to opposite side in which structure/condition occurs
143
Hemiplegia Hemiparesis Complications related to Stoke
Hemiplegia- paralysis of left or right side of body Hemiparesis- weakness of left or right side of body
144
Assistive device safety Complications related to Stoke
how to move walker, how to move weak/affected side first followed by strong side
145
Neglect syndrome- cannot interventions-utilize/ promote/ dressing Complications related to Stoke
cannot integrate and use perceptions from affected side of body Interventions: utilize and encourage unaffected side use Promote use of assistive devices Dressing-dress affected side first
146
Diagnosis stroke
Complete history and physical- neuro exam ct scan
147
Medications stroke used to treat gabapentin
used to treat during acute phase, to prevent further thrombosis, increase cerebral blood flow gabapentin can be given to prevent seuizures
148
Fibrinolytic therapy- TPA (tissue plasminogen activator) converts given need contraindicated
Converts plasmogin to plasmin resulting in breaking of the clot Given within.3 hrs of onset Need a ct scan to confirm its not hemorrhagic Contraindicated in recent head injury
149
surgery for stroke helps repair removal
help restore blood flow, repair vascular damage, removal of clot
150
Medication management & Prevention what drugs drugs do what Nursing considerations???? daily prevent seizures w/ stroke
Clopidogrel //ticlopidine Drugs to prevent clot formation/vessel occlusion Nursing considerations???? Look for bleeding, epistaxis, Daily low dose aspirin Prevent Seizures- Gabapentin
151
Stroke nursing education exercises use what adl check bladder sit eating keep
ROM exercises use unaffected arms for aDL check for pocketing of food bladder training with kegels sit upright when eaating keep objects on unaffected side
152
Conjunctivitis- what is it Eye Conditions/Disorders
inflammation of conjunctiva
153
Conjunctivitis- diagnosis Eye Conditions/Disorders
culture and sensitivity Fluorescein stain Conjunctival scraping
154
Conjunctivitis-why is accurate diagnosis important Eye Conditions/Disorders
important be use other potentially vision threatening conditions can cause red eyes
155
Conjunctivitis-health promotion wash do nont avoid contact when inflamed Eye Conditions/Disorders
wash hands before administering meds do not share towels or makeup avoid rubbing/scrathing contact lens care reduce lighting and wear sunglasses and dont use contact when onflammed
156
Cataracts-what is it Eye Conditions/Disorders
clouding of lens of eye
157
Cataracts manifestations color visual difficulty Eye Conditions/Disorders
color discrimination is impaired visual acuity down, affecting close and distance difficulty adjusting to dark/light
158
Cataracts c a t a r a c Eye Conditions/Disorders
Congenital Aging Toxicity Accidents Radiation Altered metabolism Cigarette smoking
159
cataracts post surgery eye do not place in avoid
eye patches/sheilds do not touch/ scratch/ squeeze place in semi/ fowlers avoid coughing, sneezing, straining
160
Glaucoma-what is it Eye Conditions/Disorders
optic neuropathy increase intraocular pressure
161
Glaucoma-open angle manifestaitons Eye Conditions/Disorders
Open-angle glaucoma is painless, with gradual loss of visual fields
162
Glaucoma-closed angle manifestaitons pain / colored abrupt Eye Conditions/Disorders
eye pain nausea vomiting colored halos abrupt decrease in visual acuity
163
Glaucoma-diangoses Eye Conditions/Disorders
tonometry Fundoscopy Gonioscopy Visual field testing
164
Macular Degeneration-what is it Eye Conditions/Disorders
degeneration of retina
165
Macular Degeneration-manfiestations central straight initially Eye Conditions/Disorders
central vision is blurry straight lines appear wavy/ distorted initially is one eye
166
Macular Degeneration-nursing care large increase magnify visual Eye Conditions/Disorders
large print increase lighting magnifying glass visual aids
167
Macular Degeneration-health promotion reduce risk w quit vitamins Eye Conditions/Disorders
reduce risk w/ omega 3 quit smoking vitamins c and e slow progression
168
Retinal Detachment- seperation _or_
Separation of the retina of the eye from the pigmented vascular layer Trauma or spontaneously
169
Retinal Detachment Manifestations
floaters, spots, curtain is drawn across visual fields
170
Early intervention vital will need Retinal Detachment- overview
vital to preserve sight, will need surgery
171
Positioning the patient-eyedissorder important lateral position so
important until intervention can take place! Lateral positioning on affected side Position so affected eye is inferior- this allows posterior portion of eye to place pressure on detached area; bringing retina closer in
172
Otitis externa inflammation most prevelant
inflammation of the ear canal, “swimmer’s ear” _most prevalent in people who spent a lot of time in water
173
Otitis externa etiology
Bacterial infection , fungal infection, local hypersensitivity reaction, trauma
174
Otitis externa Manifestations:
Feeling of fullness in ear, pain, drainage, inflamed and edematous ear canal
175
Otitis externa Treatment: cleansing anti meds
Cleansing ear canal, local or systemic antibiotics, meds for pain/itching may have topical steriod__
176
Otitis media: middle ear commonly infection dysfunction
inflammation or infection of middle ear, most commonly in infants and children Upper respiratory infection eustachian tube dysfunction
177
Serous otitis media auditory tube impairing
auditory tube is obstructed for prolonged time, impairing pressure equalization(popping in ear)
178
Acute otitis media- edema prevents causing
edema of auditory tube prevents drainage of middle ear causing mucus and fluids to accumulate
179
Otitis media- manifestations
Mild to severe pain Elevated temperature Diminished hearing, dizziness, vertigo, tinnitus Tympanic membrane red and inflamed or dull and bulging
180
Otitis media- treatment
Short course of steroids Antibiotics- finish course Symptomatic treatment: analgesics, antipyretics Surgery
181
Mastoiditis- Mastoid process
portion of the temporal bone that lies adjacent to the middle ear. Contains the mastoid sinuses.
182
Mastoiditis- infection effective if treatment is ineffective
Infection of acute otitis media generally extends into the mastoid sinuses. Effective treatment of otitis media usually eliminates infection of the mastoid sinuses. If treatment is ineffective = acute mastoiditis
183
Mastoiditis- Manifestations develop when recurrent tenderness possible others
usually develop 2-3 weeks after an episode of acute otitis media Recurrent earache and loss of hearing Tenderness over mastoid process, possible redness and inflammation Fever, tinnitus, headache, profuse ear drainage
184
mastoiditis treatment
Aggressive antibiotic therapy Mastoidectomy if at high risk for spread of infection to the brain
185
Meniere’s Disease- manifestations
; vertigo, unilateral hearing loss, tinnitus
186
Meniere’s Disease- Treatment/Medications- rest diet meds surgery
Bedrest during vertigo attacks, safety/help Low sodium diet to reduce pressure Medications- diuretics, depressant, antivertigo/antiemetic Surgery- shunt to eliminate pressure