Neuro Monitoring Flashcards

1
Q

detects decreases in perfusion and ultimately ischemia based on reductions in electrical brain activity

A

EEG

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2
Q

CBF average

A

50 mL/100 gm/min but can vary regionally from 30-300 mL/100 gm/min

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3
Q

CBF < 25 mL/100 gm/min =

A

slowing of EEG

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4
Q

CBF ≈ 15-20 mL/100 gm/min =

A

isoelectric EEG (internal cell)

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5
Q

CBF < 10 mL/100 gm/min =

A

↓ cell integrity, irreversible injury

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6
Q

CPP of this will show EEG changes

A

CPP < 50 torr

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7
Q

CPP of this shows ↓ cell integrity, irreversible damage

A

CPP < 25 tor

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8
Q

what does an EEG monitor?

A

electrical signals of the cerebral cortex

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9
Q

Readings of EEG can be misinterpreted with what 4 pathologies?

A

hypothermia,
hypercapnia,
electrolyte imbalance,
volatile anesthetic agents resemble ischemic ∆s

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10
Q

affects brain focally and asymmetrically

A

ischemia

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11
Q

affects brain symmetrically

A

anesthesia

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12
Q

Major rhythm seen in normal
resting relaxed awake adults. stops in eye
opening and mental exertion.

A

alpha 8-12 htz

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13
Q

Fast activity, alert, eyes open,

concentrating, anxious or busy thinking.

A

Beta - 13-40 Hz

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14
Q

Slow activity, considered abnormal
in awake adults. Can indicate encephalopathy,
subcortical lesions, Normal in young children.

A

Theta - 4-7 Hz

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15
Q

slowest frequency and highest
amplitude. Subcortical lesions and
encephalopathy, hydrocephalus. Normal in babies.

A

Delta - 1-3 Hz

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16
Q

Anesthetic effects on EEG Barbiturates and Benzodiazepines

A

accentuate frequency then

decrease it.

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17
Q

Opioids Anesthetic effects on EEG

A

slow frequency, increase amplitude

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18
Q

Inhalation anesthetics effects on EEG

A

both frequency and amplitude are reduced

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19
Q

flat line EEG associated with anesthesia

indicative of decreased metabolic oxygen demands and neuroprotective qualities.

A

Isoelectric state:

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20
Q

conscious recall or

remembering exact events of previous experiences

A

Explicit (conscious) memory

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21
Q

movement and ability
to respond to commands without specific conscious
recall of events (some call awareness w/o recall)

A

Implicit (unconscious) memory

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22
Q

also known as “recall” is defined as a
consciousness(explicit memory) under general
anesthesia with subsequent recall of the experienced
events

A

awareness

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23
Q

paralysis of un-anesthetized patients
occurring when patients are given NMB prior to
anesthesia (out of sequence, mislabeling)

A

Awake paralysis

24
Q

risk factors for awareness

A

< 60 y.o., ASA 1-2, female, elective surgery

25
3 primary reasons for awareness?
1. light anesthesia 2. pt requires an increased level of anesthetic 3. anesthetic delivery problems
26
Signs of “Light” Anesthesia and possible | awareness/recall [none reliable]
``` • Minor patient movement • Tearing and sweating • Autonomic nervous system changes –Tachycardia –Hypertension –Increased respiratory rate and depth if spontaneous respiration ```
27
drugs that Mask physiological responses to light anesthesia
–Amphetamines –Beta blockers –Calcium channel blockers -NMB
28
what vitamin interferes with anesthesia levels?
vit C
29
Reducing the Risks of Awareness Claims
- premedicate with amnestic drugs - benzo, scopalamine | - MAC >.7 monitoring expired
30
Quantitative EEG index assessing level of | consciousness during sedation or anesthesia
Patient State Analyzer Array PSArray
31
what regions of brain does PSArray monitor?
Analyzes anterior and posterior and bilateral | regions of the brain
32
Analysis of raw EEG data to derive a formula-driven | numerical value indicative of level of consciousness
BIS
33
3 things BIS takes into account?
–Amount of slow wave content (beta ratio) –Amount of bicoherence of all frequency pairs –Amount of burst suppression present (ratio)
34
General anesthesia is a BIS reading of
40-60
35
BIS value of 0 represents
flat line EEG
36
BIS value > | 70 is associated with
increased probability of | recall
37
Arguments against relying on BIS
``` hypothermia shivering unipolar cautery ketamine and N2O head trauma patient position ```
38
Electrical signals produced in response to | various stimuli by the nervous system to monitor neuronal pathway
Evoked Potentials
39
4 types of evoked potentials
somatosensory (SSEP), motor | MEP), auditory (BAEP), and visual (VEP
40
how t general anesthetics affect Evoked Potential Monitoring
inhibit neurotransmission of impulses
41
produce a dose-dependent increase | in SSEP latency and decrease in amplitude
volatiles
42
IV anesthetics affect SSEP ___ inhaled anesthetics
less than
43
meds that increase SSEP amplitude
Etomidate and Ketamine
44
have little to no affect on SSEP amplitude
opioids
45
nitrous affect on SSEP amplitude?
60% decreased SSEP, but had no effect on latency
46
decrease SSEP
reduction in blood flow
47
Assesses corticospinal tracts that are not monitored by SSEP
Motor Evoked Potentials (MEP)
48
Monitors motor response to nerve stimulation.
EMG
49
Damage to the sensory pathway will display a
decreased | signal amplitude and an increased latency
50
optimal MAC?
0.5-0.7 MAC
51
valuable indicators of depth of anesthesia.
The rate, rhythm, and muscles used for respiration
52
explain ether eye signs
The gaze becomes disconjugate during stage II and becomes congruent again when stage III is entered.
53
No substantive changes in respiration, heart rate, pupillary activity, reflexes or muscle tone.
Stage I: Analgesia and Disorientation
54
Respiration: irregular, breath holding/apnea, gasping. Reflexes: hyperactive. Muscle Tone: hyperactive, often thrashing extremities.
Stage II: Delirium, Agitation, and Excitement
55
``` Medullary Paralysis (Moribund) Progressive cardiovascular collapse ```
stage IV