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Flashcards in Neuromus Disorders Deck (38):

Central Cord Syndrome

Resulting from hyperexten injuries and presenting as more UE deficits vs LE


Brown Sequard Syndrome (hemi-section)

Causes disruption of descending lat cotricospinal tracts, ascending dorsal column and ascending lat spinothalamic tracts; result is ipsilat paralysis, position sense, discrim touch & contral lat loss of pain/temp


Ant Cord Syndrome

Caused from flex injuries; motor fx, pain, pinprick & temp sensation are lost bilat below lesion; proprioception & light touch are preserved


Post Cord Syndrome

Least common; injury to post columns results in proprioceptive loss; pain/temp/touch are preserved; Motor fx preserved to various degrees


Conus Medullaris Syndrome

Injury of SC & lumbar nerve roots resulting in LE motor & sensory loss & an areflexic (flaccid, storing lots) B&B > if lesion is in sacral segs reflexes may be preserved


Cauda Equina Syndrome

Injury at L1 level & below resulting in LMN lesion; flaccid paralysis w no spinal reflex activity; areflexic B&B


Autonomic Dysreflexia

Abnorm response to noxious stim that results in extreme rise in BP, pounding headache & profuse sweating > deemed Med EMERGENCY if not reversed by quickly moving the stim


Prevention for Autonomic Dysreflexia

Edu pt/cg frequent pressure relief principles; ensure compliance w intermittent catheterization; practice well balanced diet habits; ensure med compliance; edu on signs/causes and initiate first aid procedures


Medical Mngt For SCI

Prevention for further damage via stabilization; traction/rest for unstable injuries; diuretic prescrip to decrease inflam; bladder care; ulcer prevention; control of autonomic dysreflexia/orthostatic HTN, prevention of thrombus formation & tx for heterotopic ossification


CP cause

Injury and/or disease prior to, during or shortly after birth resulting in brain damage and secondary neurolog & muscular deficits - lack of O, intracranial hemorrhage, meningitis, chronic alcohol abuse, toxicosis, infections, genetics, endocrine or metabolic disorders


CP is noprogressive however

Deformities & contractures could develop; may be accompanied w seizure, ID/DD or behavioral disorders


Spastic CP

Lesion of motor cortex will result in spasticity w flexor/extensor imbalance; Hypertonia: increased muscle tone or Hyperreflexia: increased intensity of reflex responses


Dyskinetic CP

Lesion in BG results in fluctuations in muscle tone: Dystonia (excess or inadequate muscle tone), Athetosis (writhing invol mvmts more distal), Chorea (spasmodic invol mvmts more prox w lack of co-contractions)


Ataxic CP

Lesion in cerebellum results in hypotonia & ataxic mvmts characterized by lack of stability so coactivation is difficult > results in more prim mvmt patterns; Gross Motor Fx Classification & Manual Ability Classification used



Mono: 1 ex; Hemi: UE/LE on same side; Para: LEs; Quad: all ex; Di: less UE and greater LE fx'al impairment


CP Complications

50% seizures, lang/cog deficits in 50-75%, visual impairments in 40-50%, feeding disturbances & diminished sensation



Deviation of how one eye aligns with other



Reflexive response of eyes triggered by head mvmts


Myopia, Hyperopia & Presbyopia

M= nearsightedness; H= farsightedness & P= decreased elasticity of lens resulting in diff w accomodation when focusing on objects nearby and when shifting focus from near to far



Invol, nonrepetitive but occasionally stereotyped mvmts affecting distal, prox and axial musculature in varying combo - most rep of BG disorders



Brief & rapid contraction of muscle or muscle group



Brief rapid, invol mvmts often resembling fragments of normal motor behavior. Tend to be stereotyped and repetitive but no rhythmic



Brief, purposeless, invol mvmts of distal ex and face; usually considered to be manifestation of dopaminergic overactivity in BG



Results in sustained abnorm postures and disruptions of ongoing mvmt resulting from alterations of muscle tone - maybe be generalized or focal



Lack of coord while performing vol mvmts - may appear as clumsiness/instability. Mvmts are jerky/disjointed



Characterized by invol flinging motions of ex; Mvmts are often violent and have wide amplitudes of motion - continuous & random & can involve prox/distal muscles on one side of the body


Parkinson's Disease (PD)

Hypokinetic CNS mvmt disorder this is idiopathic (arises spontaneously), slowly progressive and degenerative; onset usually after 40; rate of deterioration ranges from 2-20yrs


PD Symptoms

Begins insidiously w resting pill-rolling tremor of 1 hand; cardinal signs include tremor, rigidity, resistance to passive motion that is not velocity dependent; akinesia, postural instability, festinating gait, retropulsion (falling backwards) or propulsion (falling forward), mask face & micrographia (tiny handwriting)


Hoehn and Yahr's 5-stage scale

Used for PD. Stage I: unilat tremor, rigidity, akinesia (loss of vol mvmt) w min or no fx impair. Stage II: bilat tremor, rigidity, akinesia w or w/wo axial signs, I with ADL & no balance impair. Stage III: worsening sypmts, 1st sign of impaired righting reflex, onset of disability with ADL - can lead I lift. Stage IV: req help w some/all ADL, unable to live alone w/o some A, able to walk/stand unaided. Stage V: confined to wc/bed; max A


Spina Bifida

Etiology is unknown; it is the failure of the spinal columns vert arches to fully form to enclose/protect neural tube > may result in protrusion of neural tube (could be d/t lack of folic acid); Detected prenatally


Spinda Bifida Occulta

Body malformation w separation of vert arches of one or more vert w no external manifestations - may not be discovered until late childhood. * usually doesnt result in any symptoms


Occult Spinal dysraphism (OSD)

When ex manifestations such as red birthmark, patch of hair, dermal sinus , fatty benign tumor or dimple covering site are present


Spina Bifida Cystica

Exposed pouch comprised of SC and meninges


Spina Bifida Cystica w meningocele

Protrusion of a sac thru the spine containing CSF and meninges but does NOT include SC * usually doesnt present symptoms


Spina Bifida Cystica w myelomeningocele

Protrusion of a sac thru the spine containing CSF and meninges as well as SC or nerve roots. Results in sensory & motor deficit occurring below level of lesion & may result in LE paralysis or deformities, incontinence (lesions of S2-4), decubitius ulcer or DVT


Occult Spinal dysraphism (OSD) Symptoms

May result in SC being split (dyplomyelia) or tied down & tethered (disatematomyelia) which may lead to neurolog damage and developmental abnorm as child grows


Tethered Cord Syndrome

Occurs in tail end of SC when cord is stretched as a result of compression, being trapped w fatty mass/scar tissue, developmental abnorm or injury


Tethered Cord Syndrome Signs

Hairy skin patch, hemangioma (blood vessels that form a birthmark) and/or dimple of lower spine. Diff w B&B, gait, deform of feet, LBP, sclerosis may result and can go undiagnosed until one of these symptoms are found