Neuromus Disorders 3 Flashcards Preview

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Flashcards in Neuromus Disorders 3 Deck (45):

Peripheral Neuropathies Causes

Single: trauma, pressure paralysis, forcible overexten of joint, hemorrhage into nerve, exposure to cold/radiation or ischemic (restriction of blood flow) paralysis. Multiple: collagen vascular disease, metabolic diseases (DM) or infectious agents (Lyme). Other: alcohol, nutritional deficiencies, malignancy, toxic agents/microorganism


Peripheral Neuropathies Symptoms

A syndrome of sensory/motor/reflex/vasomotor symptoms. Pain, weakness, parasthesis (pins/needles) in distribution of affected nerve


Guillan-Barre Syndrome

Unknown etiology but may occur after infectious d/o, surgery or immunization. Affects both sexes/any age. Onset of recovery is 2-5wks after inital symptoms


Long term prognosis of Guillan-Barre Syndrome

50% exhibit mild neurolog deficits, 15% exhibit residual fx'al deficits, 80% are ambulatory after 6mo and only 5% die of complications


Guillan-Barre Syndrome Symptoms

Acute, rapidly progressive form of polyneuropathy characterized by symmeteric muscular weakness and mild distal sensory loss/parasthesias. Weakness always most predominant. Sensory symptoms are relatively mild. Deep tendon reflexes are lost/sphincter control spared.


Myasthenia Gravis

Diseased caused by autoimmune attack on ACT receptor of postsynaptic neuromus junction; considered disorder of neuromus transmission. Onset at any age: most often older men/younger women. Usually progressive & death may occur from respiratory comps.


Myasthenia Gravis Symptoms

Characterized by episodic mus weakness - chiefly mus innervated by CNs. Ptosis, diplopia, muscle fatigue after exercise, dysarthria, dysphagia, prox limb weakness. Symptoms fluctuate over the day. Deep tendon reflexes/sensation in tact. Ability to relapse w quadriparesis


Post-Polio Syndrome

Some MN infected w Polio virus die, others survive. Recovered develop new terminal axon sprouts that reinnervate muscle cells. After years of stability, motor units breakdown cause muscle weakness. Typ occurs 15yrs after Polio recovery. Slow to progress and good prognosis.


Post-Polio Syndrome Symptoms & Tx

New onset of weakness, easily fatigued, muscle pain, joint pain, cold intol, atrophy, loss of fx'al skills > Bracing, stretching/exercise programs


Multiple Sclerosis (MS)

Slowly progressive CNS disease characterized by patches of demyelination of brain and SC. Occurs most often between 20-50yrs. Overall prognosis is variable w unpredictable disease course > multiple CNS lesions & at least 2 episodes of neurolog disturbance. remissions and exacerbation


MS Symptoms

Onset usually insidious (gradual but harmful). Parasthesias in one+ extremities/trunk/face. Weakness of hands/legs. Visual/emotional/cog disturbances. Balance loss/vertigo. Bladder dysfunction. Sensorimotor findings.


MS Patterns

Relapsing remitting, secondary progressive, primary progressive, progressive relapsing


Eval for Neurolog D/o

Determine sensory/motor dysfunction strengths: paralysis/weakness, GM/FM, spasticity, sensory, postural control, ROM, MMT, skin integrity, foundation visual skills, pervasive impairments, psychosoc. Impact of deficits on ADLs/occs


General Intervention/Tx for Neurolog D/o

Positioning, pressure reduction, postural control, motor learning approaches, motor control retraining/relearning, specific ADL training/retraining/adaptations, AT, splinting for contractures, fam/caregiver edu, cog retraining, visual skills retraining, sexual dysfunction help, B&B training, skin care edu, DME, sensory re-edu, community re-integration, return to work/work hardening


Work Hardening Program

Highly structured, goal oriented, individualized treatment program designed to maximize the indiviual's ability to return-to-work.


Fast Pain

Transmitted over A Delta fibers. Processed in SC dorsal horn lamina. Crosses to excite lat spinothalamic tract & terminates in BS reticular formation. Functions for discrimination of pain/localization


Slow Pain

Transmitted over C fibers. Processed in SC lamina. Crossed to excite ant spinothalamic tract. Terms in BS reticular formation > excites Reticular Activating System (RAS)/ Functions for diffuse arousal, affective & motivational aspects of pain


Acute Pain

Sharp pain, sympathetic changes (increased HR, BP, pup dilation, sweating, hyperventilation, anx, escape/protective behaviors


Chronic Pain

Pain that persists beyond usual healing course. Symptoms present for longer than 6mo for which underlying pathology is no longer identifiable or may never have been present


Neuropathic Pain

Pain as result of lesions in some part of nervous system; usually accompanied by some degree of sensory deficit.


Thalamic Pain

Continuous, intense, occurring on contralat hemiplegic side > result of stroke involving vent postlat thalamus; poor rehab potential


Complex Regional Pain Syndrome Type I

Formerly known as Reflex Sympathetic Dystrophy (RSD).; pain maintained by efferent activity of sympathetic nervous system. Characterized by abnorm burning pain, hypersensitivity to light touch & sympathetic hyperfunction (sweating/coldness) > usually associated w traumatic injury


Complex Regional Pain Syndrome Type II

Formerly known as Neuralgia. Pain occurring along branches of a nerve.


Herpes Zoster (Shingles)

Acute, painful mononeuropathy caused by varicella virus. Vesicular eruption and marked inflam of post root gang if affected SN or sensory gang of CN - vent root involvement (motor weakness). Can last 10days- 5wk > pain may persist for months


Psychosomatic Pain

origin of pain experience is d/t mental/emo disorder


Referred Pain

Pain arising from deep visceral tissues that is felt in body region remote from the site of pathology resulting in tenderness & cutaneous hyperalgesia (abnorm heightened pain sensitivity)


Postural Stress Syndrome (PSS)

Chronic mus lengthening/shortening that causes postural malalignment and stress to soft tissue > check for during pain eval


Eval for Chronic Pain

Hx, localization, nature of pain (constant/intermittent), subject pain intensity scale, physical assessment, PSS, autonomic changes (sympathetic activity), abnorm mvmts, degree of suffering, fx'al changes, emo changes, precrip drug misuse, dependence on health care system, responsiveness to pain, motivation/affective components


Chronic Pain Tx

Edu on contributing factors, edu on responding adaptively to pain behaviors, develop strategies/techniques to manage pain (relaxation training), refer to other professionals, estab realistic daily activity program, improve overall fx'al capacity, fam edu


Sensory Processing D/o

Subtle, prim subcorticala, neural dysfun w impaired processing of sensory info and modulation of multisensory systems


Dunn's Model of Sensory Processing Symptom Classification

classified according to interaction of sensory stim that are needed to stim behavioral response. High/low threshold & passive/active response


Sensory Modulation D/o

Sensory Overresponsitivity, Sensory Underresponsitivity or Sensory Seeking/Craving


Sensory Based Motor D/o

Includes underlying sensory discrim d/o as well as possible sensory mod d/o: dyspraxia (developmental coordination disorder variation) or sensory-based postural disorders


Eval for Sensory Processing D/o

Hx, fam/teacher interview, informal observation followed by formal assessment of clinical observations, standardized testing


Seizure D/o vs Epilespy

Epi= chronic state of recurrent seizures. Seizure D/o= temp disturbance in brain activity causing a group of nerve cells to fire excessively, interfering with norm brain fx


Seizures are often associated w these other conditions

O deprivation, severe head injury or brain hemorrhage, CP, stroke, brain tumor, other neurolog d/o, hydrocephalus, metabolic d/o, infections, meningitis, encephalitis, rubella


Prim Gen Seizures]

Begin w widespread involv of both sides of brain


Partial Seizures

Involv of smaller/localized area > can still spread within seconds/mins to gen (secondary gen seizure)


Tonic-Clonic Seizures (grand-mal)

Most common type of seizure d/o in children. Brief warning/aura (numbness, taste, smell). Tonic phase includes LOC, stiff of bod, heavy/irreg breathing, drooling, skin pallor, occasional incontinence. Clonic phase includes alternating rigidity/relax of muscles. Following= Postictal State which includes drowsiness, disorientation or fatigue


Myoclonic-Akinetic Seizures

*Not the same as infantile. Brief, invol jerking of extem w or w/o LOC, include loss of tone - difficult to control.


Petit-Mal Seizures

Absent seizures. Typically between 4-12y/o, LOC w/o loss of mus tone. Rapid blinking or staring into space - does not fall but does not recall episode


First Aid Seizure Procedures

Remain calm and if status elepticus call for immediate med attn, remove all dangerous objects from area, do not interfere w mvmts, raise bed rails, make sure nothing is in the mouth, turn to side if aspiration risk (recovery position), allow seizure to happen while protecting head, monitor for improved mental state


Call for med attn during seizure if

Individual's first, in H2O, if has second seizure, if consciousness is not regained after 5-10min, if seizure last 5min or more, if individual is diabetic or pregs


Post Seizure Care

Allow individual to rest/sleep, inform parents/caregiver, observe safety precautions


Seizure Eval and Intervention

Assess/intervene for DD as necessary. Observe all med/safety precautions. Doc/report any seizure activity, med side effects or behavioral changes.