Neuropathologies In ALCD Flashcards

1
Q

Acute-Onset that Remain Stable/Improve

A

● Acute Ischemic Stroke
● Intracerebral hemorrhage
● Subarachnoid hemorrhage
● Traumatic brain injury
● Abscesses
● Encephalitis
● Acute disseminate encephalomyelitis
(ADEM)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

deals with the higher mental
processes such as thinking, decision making,
and planning.

A

Frontal Lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

deals with the processes
involved in sensory information that has to
do with taste, temperature, and touch.

A

Parietal Lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

deals with processing auditory information (e.g. mga naririnig mo,
anything regarding auditory information).

A

Temporal Lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

used for processing visual
information.

A

Occipital lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

used for the coordination of voluntary movements

A

Cerebellum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

controls the di erent cranial
nerves and motor control of the body; the other motor control,

A

Brain Stem

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

involved in production of speech sound
- Also called the Brodmann Area 44 til and 45.

A

Broca’s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

involved in Understanding of speech
- Also known as the Brodmann Area 22. It is for the comprehension of speech.

A

Wernicke’s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

controls the Movements of muscles

A

Motor Cortex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

connects Wernicke’s area to Broca’s area.

A

Arcuate Fasciculus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

There is a blockage or infarct

A

Ischemic stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

The blockage is moving

A

Embolic IS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Blood clot in specific area

A

Thrombotic Stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

happens if there’s a smaller than 1 to 1.5 cm in size;

A

Lacunar Stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

are regions of the brain that simultaneously receive blood supply from 2 types of arteries.

A

Watershed areas

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

obstruction caused by blood clot (thrombus)

A

Thrombosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

one or more heart valves does not work properly

A

Valve Disease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Contralateral, hemiparesis, sensory loss, homonymous hemianopsia, dysarthria

A

MCA stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

(R) - neglect, anosognosia, asomatognosia, aprosodia, flat effect
(L) - aphasia, alexia, agraphia, acalculia, apraxia

A

MCA stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Contralateral leg paresis, abulia, disinhibition, exec dysfunction, akinetic mutism

A

ACA stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

occipital - contralateral homonymous hemianopsia, cortical blindness (bilateral)
Medial temporal - LTM and STM deficits, behavior alteration
Thalamus - contralateral sensory loss, aphasia, exec dysfunction, decreased level of consciousness, memory impairment

A

PCA stroke

23
Q

Lesion: Sup div LMCA

A

Broca’s aphasia

24
Q

Lesion: Inferior div LMCA

A

Wernicke’s Aphasia

25
Lesion: Watershed bet LMCA and LACA
Transcortical motor aphasia
26
Lesion: Watershed bet LMCA and LPCA:
Transcortical sensory aphasia
27
RHD
Cog-comm d/o
28
Intracerebral Hemorrhage: (L) basal ganglia, internal capsule, white matter
articulatory impairment + comprehension and naming impairments ang patient
29
Intracerebral hemorrhage: L) thalamus
Naming and repetition impairments
30
Intracerebral hemorrhage: (R) thalamus
Narrative deficits
31
Amyloid angiopathy - when a amyloid proteins build up on the walls of the arteries in the brain, causing
Dementia or stroke
32
Arteriovenous malformation (AVM) - blood vessels are being tangled, nagkakaroon ng problem connecting arteries and veins causing
Abnormal connection
33
Subarachnoid Hemorrhage Possible magkaroon ng:
Aneurysmal SAH
34
CN III palsy, CN VI palsy, bilateral LE weakness, abulia, visuospatial neglect, hemiparesis, and aphasia is due to
Subarachnoid Hemorrhage
35
Can also cause aphasia or other focal neurological deficits due to blood pushing on the brain, inhibiting cortical function
Subdural Hematoma
36
Can be because of: ○ Blows to the head ○ Closed head injuries (CHI)
TBI
37
Nonspecific dull headache, mental status changes, focal neurological deficits, fever (because there is an infection)
Brain Abscesses
38
Effects: Cognitive impairments (particularly the memory), sustained attention, information processing speech, executive function
Multiple Sclerosis
39
Beta-amyloid plaques (increases) and neurofibrillary tangles (caused by the tau protein)
Alzheimer's Disease
40
Presence of slowly progressive memory impairment Also develop apraxia, agnosia, executive dysfunction, aphasia
AD
41
Dopaminergic neurons in substantia nigra Trouble processing long, complex sentences, verb generation diculties, impaired semantic priming
PD
42
3 core clinical features: resting tremor, bradykinesia, rigidity... eventually postural instability
PD
43
Gradual impairment of language production, object naming syntax, and/or word comprehension Eventually may develop cognitive deficits, parkinsonism, and behavioral diculties
PPA
44
Change in affect, lack of basic emotions, repetitive, stereotyped behaviors, changes in eating habits, executive dysfunction
Behavioral Variant Frontotemporal Dementia
45
Gait isntability, impairment of eye movements, spastic dysarthria, aphasia (nonfluent), bradykinesia, rigidity, frontal behavior changes
Progressive Supranuclear Palsy
46
Tumors in dominant hemisphere ->
Aphasia
47
Tumors is nondominant hemisphere ->
Prosody and discourse comprehension deficits
48
Exposes arteries in head and neck Opaque fluid is injected Injected sa blood to see all the arteries Ischemia-vessels seem to disappear beyond occlusion (Parang mga threads)
Angiography
49
Alterations in expected densities of brain structures
CT SCAN
50
Magnetic fields and radio frequency Picture of electromagnetic signals More sensitive than CT Non-Invasive but more time consuming
MRI
51
Electroencephalography (EEG)
Electrodes on patient’s scalp Lesion is indicated by irregular electrical activity
52
Direct measure of metabolism Radioactive tracers with oxygen and or glucose are injected into arteries
Positron Emission Tomography (PET)
53
Gold standard for identifying dysfunctional Brain tissue.
PET scan