Overview Of Acquired Aphasia And Cog D/O Flashcards

(53 cards)

1
Q

Lesion in Broca’s area(BA 44, 45, inferoposterior portion of frontal lobe)

A

Broca’s Aphasia

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2
Q

deficit in formulating and processing syntax

A

Agrammatism

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3
Q

Broca’s Aphasia speech

A

Telegraphic speech: missing function words; mostly content words

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4
Q

problems with word finding

A

Anomia

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5
Q

Lesions in multiple areas: frontal, parietal, temporal areas receiving MCA blood supply

A

Global Aphasia

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6
Q

Communication is carried out largely through gesture, tone of voice, and facial expression

A

Global Aphasia

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7
Q

Transcortical Motor Lesion

A

Lesion in the anterior watershed area of left frontal lobe, extending to prefrontal areas (BA 6, 8, 9, 10 ,46)

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8
Q

Intact repetition compared with broca’s aphasia

A

Transcortical Motor

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9
Q

lesion of mixed transcortical

A

likely multifocal lesions in frontal and temporal watershed regions

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10
Q

Intact repetition compared with global aphasia

A

Mixed transcortical

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11
Q

WA lesion

A

Lesions in Wernicke’s area (BA 22, superior temporal lobe)

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12
Q

Signs of WA

A

● Fluent
● Neologisms: new words
● Paraphasias
● Relatively intact syntax than nonfluent aphasias
● Logorrhea

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13
Q

Transcortical sensory lesions

A

Lesion in angular gyrus/area surrounding the Wernicke’s area, excluding the Wernicke’s area itself (BA 39); and posterior portion of middle temporal gyrus (BA 37) [watershed regions of temporal lobe]

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14
Q

Intact repetition compared with Wernicke’s aphasia

A

Transcortical sensory

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15
Q

Conduction Aphasia lesion

A

Lesions in arcuate fasciculus (BA 40, within supramarginal gyrus)

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16
Q

Conduction Aphasia symptoms

A

● Impaired repetition (more di culty with longer and moe complex stimuli)
● Good comprehension and spontaneously production of spoken and written language
● Awareness of errors
● Common phonemic paraphasias

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17
Q

Lesion in angular gyrus

A

Anomic aphasia

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18
Q

Signs of anomic aphasia

A

● Comprehension and syntactic production
are relatively spread
● Circumlocutions, use of generic terms

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19
Q

Any form of aphasia due to damage to RH instead of LF in a person who is right-handed

A

Crossed Aphasia

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20
Q

Lesion below the cortex (thalamus, basal ganglia, cerebellum)

A

Subcortical Aphasia

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21
Q

● Frontal lobe is likely a ected during TBI
● Left and right orbital frontal lobe injury →
frontal lobe symptoms

A

FL syndrome

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22
Q

● Executive function deficits: challenges with
self-regulation, reasoning, making judgements and decisions, goal setting, planning, strategizing, being aware of strengths and weakness, organizing, sequencing, allocating attention, inhibiting inappropriate behaviors
● Pragmatic deficits: problem with social use of language

23
Q

Perceptual and Attentional Deficits
● Left hemispatial deficits
● Prosopagnosia

24
Q

A ffective Deficits
● Di culty expressing emotions
● Di culty recognizing emotions of others
● Depression
● Apparent lack of motivation

25
Communicative Deficits ● Difficulty with word retrieval ● Impaired auditory-verbal comprehension ● Reading and writing deficits ● Impaired prosodic features of speech ● Difficulty with pragmatics ● dysarthria
RHD
26
Cognitive deficits ● Disorientation ● Impaired attention ● Difficulty with memory ● Poor integration of info ● Difficulty with logic, reasoning, planning, and problem solving ● Impaired comprehension of inferred meanings ● Difficulty understanding humor
RHD
27
● Prosopagnosia ● Anosognosia ● Anosodiaphoria ● Constructional impairments ● Memory deficits ● Topographical and disorientation geographical ● Planning, problem solving, and organizing deficits
RHD
28
● Progressive diuse brain atrophy and accumulation of beta-amyloid plaques and neurofibrillary tangles ● Gradual onset ● Memory, attention, Efs
AD
29
● AKA Ischemic dementia ● 2nd most common ● Caused by blood supply problems to brain ● Stepwise progression (due to series of strokes or TIAs) ● There is evidence of multi-infarct dementia (multiple focal lesions)
VASCULAR DEM.
30
3rd most common, M>F Abnormal protein (alpha-synuclein) deposits Confusion, variable states of awareness and alertness, memory loss, ANS problems, visual hallucinations Many have neuromuscular problems common to PD (muscle rigidity, tremors, balance problems)
DLB
31
Atrophy of anterior frontal and temporal lobes Most likely to occur in ages <65 (Onset typically in 40-60s) Symptoms determined by associated functions of aected specific areas of brain PPA and certain type of movement disorders are also associated with some forms of FTD EL and RL deficits Behavioral variant FTD
FTD
32
Hereditary condition Chorea, psychiatric and cognitive-linguistic problems Poor language organization, anomia, emotional lability
Huntington’s Disease
33
Cortical atrophy caused by thiamine (vitamin B1) deficiency, most commonly due to chronic alcohol abuse STM and LTM deficits and confabulation
Korsako ’s Syndrome
34
Rare, rapidly progressive, degenerative viral disease Common bodily protein, prion, forms into misshapen configuration that destroy brain cells Rapid loss of cog and linguistic abilities and cortical and cerebellar muscular coordination, mood changes
CreutzfeltJacob Disease
35
Challenges in EFs, pragmatic abilities, attention, memory HIV-associated MCI is mild version of this condition
AIDS Dementia Complex
36
Ongoing loss of language abilities in the face of relatively preserved cognitive abilities Caused by neurodegenerative disease
PPA
37
has insidious onset, rather than sudden onset
PPA
38
symptoms get progressively words, rather than improve or stabilize
PPA
39
tend to be linguistic (e.g. word-finding) in the face of relatively intact cognitive abilities
PPA
40
● Challenges in wordfinding (esp. confrontational naming) and comprehension (even at single-word level) ● Verbal output, syntax relatively good ● Tends not to have concomitant motor speech impairments
SEMANTIC PPA
41
Problems with word finding, especially in spontaneously conversation
Logopenic PPA
42
● Diculty with syntax (esp. in expression relative to comprehension) ● Often have concomitant AOS ● AKA Nonfluent PPA
AGRAMMATIC PPA
43
what is neuroplasticity?
Capacity of the nervous system for change
44
What is intrinsic learning?
Internally, there will be change in their practice patterns because of their feedback mechanisms
45
What is extrinsic learning?
provide them with strategies and activities that will stimulate them in the perspective of motor speech. We’re going to provide them with motor experience to learn speech motor movements and to develop their PRAXIS and execution.
46
6 guiding frameworks of Mayo Clinic
1. ICF 2. Motor Speech Treatment Hierarchy 3. Neuromuscular Treatment Principles 4. Motor Learning Principles 5. Treatment Process 6. EBP
47
International Classification of Function (ICF) general groupings are:
1. Health Condition 2. Body Functions and Structures 3. Activity and Participation 4. Contextual Factors
48
Domain: Oral and Auditory Mechanisms, Task:
Task: 1. Oral structure exam. 2. Cranial nerve exam 3. Assessment of speech processes 4. Hearing test 5. Case history
49
Domain: Speech like oro-motor function, Task:
1. DDK 2. Single word sample 3. Polysyllabic words 4. Non-word repetition test
50
Domain: speech accuracy, task:
1. CS sampling 2. Consistency 3. Stimulability
51
Domain: prosody, task:
Ax of prosody
52
Domain: RL/EL, phonological awareness, task:
Task: Language/Cognitive Skills
53
Domain: related cognitive skills, task:
Task: speech perception testing, verbal working memory