NEW AFTER 1 WEEK-14 Flashcards

(62 cards)

1
Q

DUAL RULE RRNA

A

CATALYTIC AND STRUCTURAL

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2
Q

STRCTURES OF TRNA

A

ACCEPTOR ARM,D ARM, TPSI ARM,V LOOP AND ANTICODON

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3
Q

TPSI ARM CONTAIN ?

A

RICH IN THYMINE AND PSEUDOURACIL

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4
Q

ARM D CONTAIN ?

A

DIHYDROOURADINE

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5
Q

rRNA PROCESSING PRO

A

1- RNASE 3 CLEAVE COLMPLEMENTERY AROUND 1 AND 23
2- RNASE E : 5S REMOVE

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6
Q

rRNA PROCESSING EUK

A

1- 45S TO 41 S REMOVE 5 PRIME
2- 41S SPLIT TO 20S AND 32 S
3-3 PRIME OF THE 20 S REMOVE FROM 18S
SEPERATE THE 28 ADN 5.8

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7
Q

AMINOACYL TRNA SYNHTHESASE

A

TRANSFER AA TO TRNA

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8
Q

ISOACCEPTOR

A

DIFFERNT ANTOCODON SAME AA
SAMEE maniacyl trns synthesase

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9
Q

nucleotidyl traSNFERASE wnyzme vs aminoacyl trna synthesase

A

1- transfer cca to the 3 prime of the trna in eyk
2- transfer aa to trna

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10
Q

ALPHA SUBUNIT OF THE RNA POL

A

CTD BIND TO DISTAL ELEMENTS
NTD PUT THE COMPONENTS OF THE RNA POL THOGHTER

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11
Q

RNASE

A

REMOVE 5S FROM PRECURSORE rRNA

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12
Q

RNASEP

A

REMOVE THE 5 PRIME IN THE TRNA IN BOTH EUKARYOTES AND PROKARYOTES

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13
Q

….S IS PRE-rRNA IN EUK

A

45

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14
Q

C/D BOX SNORNA

A

SMALL NUCLEOLAR
METHYLATION

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15
Q

H/ACA BOX SNORNA

A

SMALL NUCLEO
PSEUDOURIDILATION

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16
Q

WHAT WE ADD IN THE CAPPING PROCESS

A

WE ADD M7G FORMING THE 5 PRIME 5 PRIME TRIPHOSPHATE BOND

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17
Q

ADIIDTION OF THE CAP ENZYMES

A

1- RNASE TRIPHOASPHATASE : GMAMMPHOPHATE THE 5 PRIME

2- GUANYLYL TRANSEFERASE : ADD GMP

3- METHYLTRANSFERASE : ADD METHYL FROMT THE S ADENOSYL METHONINE TO 7 OF TH N IN G

4- ANOTHER : ADD METHL TO 2 PRIME OH OF THE NUCLEOTIDE OF THE TRANSCRIPT

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18
Q

WHEN OCCUR THE CAPPING

A

ERALT TRANSCRIPTION
20-40 NT

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19
Q

WHEN POLY A OCCUR

A

POLY A RICH SIGNAL SEQUENCE POLY ADNYLATIONSIGNAL
CPSF + CSTF + CF1 +CF2+ POLY A POLYMERASE

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20
Q

2 COMPLEX IN THE POLY A

A

CPSF AND CSTF

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21
Q

CPSF

A

CLEAVAGE AND POLY A SPECIFICITY FACTOR
BIND TO AAUAAA POL A SIGNAL

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22
Q

CSTF

A

CLEAVAGE STIMIULATION FACTOR
BIND TO GU OR U RICH SITE INCREASE EFFCIENCY OF THE POLY A

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23
Q

SHORT / LONGTHENEING THE POLY A

A

RNASE , CYTOSOLIC POLY A

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24
Q

Bloom syndrome

A

Helicase BLM in a mutated form is the cause of the
Bloom syndrome (recessive inheritance)
Tipically, in patient’s cells there is an excess of
homologous recombination between sister chromatids

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25
CONJUGATION
SEX BACTERIA CROSS OVER YES RECOMBINATION YES
26
TRASNFORMATION
FROM THE MEDIUM RECOMBINATION YES
27
TRASNDUCTION
VIRUS RECOMBIANTION YES
28
intergenic suppression IN EU
second mutation is in a different geneoften due to gene interactions)
29
ntergenic suppression IN PRO
change results in a tRNA anticodon capable of pairing with the stop codon
30
Mutation frequency
proportion of individuals in a population t
31
Mutation rate
probability that a particular mutation will o
32
(STRs
short tandem repeats,
33
RFLP
restriction fragment length polymorphism
34
mutation models
inifinte allel= each mutation 1 allel infinite site allel= each mutation just 1 site no chance for back mutation or recurrecnt mutation stepwise mutation = add / delete the same rate by 1 unit indel=insretion./deeletion finit sit mutation = mtation same site diffent times cpg
35
controle theinteraction and associaton of the ribosome
nitiiaion factors
36
formyl trna block... formation
peptie bond
37
MOST COMPACT DISTRIBUTION OF THE EXON
VERTEBRATESS
38
REPLICATOR
ORIC/ORGIN OF THE EPLICATION
39
COTROL ELEMNETS IN THE REPLICATION
REPLICATOR/ORIGIN \ND TERMINATION SITE
40
EXTRA CHROMOSOMAL REPLICON
ORGANELLAR PLASMID PHAE/VIRAL DNA
41
REPRESSOR OF THE LAC OPERON
TETRAMER BIND TO 2 SIDE FORM THE LOOP BIND TO THE OPERON AS DIMER N TERMINAL BIND TO DNS AND THE ALLOLACTOSE C TERMINAL BIND TO ALPHA HELIX WHEN THE INDUCER BIND WE CAN SEE ALLOSTERIC TRANSITION
42
INDUCER /COREPRESSOR IN LAC OPERON
ALLOLACTOSE - INDUCER
43
LAC A CODE FOR ?
TRANSACETYLASE (ACETYL TO BETA GALACTOSIDASE 0
44
beggining and donwstream of introns
UPSTREAM/ 5 PRIME /BEGINNING: GU DOSNTREM/3 PRIME/THE END : AG
45
RLFP
RESTRICTION FRAGMENT LENGTH POLYMORPHISM
46
LONGEVITY
e BALANCE between the advantage of having an appropriate immune response (↑ inflammation) and the disadvantage of chronic inflammation during ageing.
47
Resilience
s at the core of life and is interpreted as the harmonic assemblage of the biochemical processes that are aimed at maintaining the identity, integrity, and autonomy of individual organisms against the perturbations induced by both internal and external environments.
48
apoe
related to anatgonstic pleitropy fertility high but the alzihmer high as well
49
brca
feterility high but risk of breast cancer
50
AOUNT OF ENERGY BY ATP HYDROLYSATION
8 KCAL/MOL
51
PUROMYCIN
mimic aminoacyl trna
52
mimic aminoacyl trna
PUROMYCIN
53
each release factor in euk corrospond to what release factor in bacteria
erf1 = rf1 and rf2 erf3=rf3
54
kirromycin
not release of the EF-TU
55
CHLORAMPHENICOL
INHIBITOR OF PEPTIDE BOND FORMATION - PROK
56
CYCLOEXIMIDE
INHIBITOR OF PEPTIDE FORMATION EUK
57
FUSIDIC ACID
RELEASE OF EF-G AND GDP
58
RIBOSWITCH TPP
THUAMINE MONOPHOSPHATE VIT B1 SYNTHESIS
59
RELA PR
STRINGENCY FACTOR
60
TERMINATION IN THE RNA POL 1
TTF1 BIND AND BLOCK TRNASCIPTION
61
STRINGENT FACTOR
REL A
62
ppGpp PPPGPP
GUANOSINE TETRAPHOPHATE GUONOSINE PENTAPHOPSHPHATE