OBGYN5 Flashcards

(39 cards)

1
Q

Clinical future of MGSO4 toxicity?

A

Mild/moderate/sever

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Mild?

A

hyporeflexia, headache, nausea, and flushing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Moderate

A

areflexia, hypocalcemia and somolecense

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Sever

A

respiratory depression

cardiac arrest

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Mechanism of toxicity?

A

block presynaptic ca entry–inhibit ca release

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Risk for toxicity?

A

renal failure/low U/O

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

management of toxicity

A

Stop magnesium

Give calcium gluconate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Normal MGSO4 mechanism of action?

A

block CNS ca channels–increase sizure threshold

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Therapeutic serum level?

A

4.8-8.4 meq/l–2-4x normal serum level

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Clinical future of benign ovarian mass?

A

in reproductive age

simple cystic lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Clinical future of malignant ovarian mass?

A

In postmenopausal symptom
complex cyst
chronic pain
constipation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Corpus lutetium cyst?

A

Unilateral
occur when follicle rupture
U/S –hypoechoic area with normal dopler

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Management?

A

Resolve by itself

Reevaluate after 6 week

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

pregnancy and ulcerative colitis?

A

Px exacerbates UC because of cytokine released by the placenta.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

CM?

A

hematocasia
abdominal pain
tenesmus
toxic megacolon(rarley)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Management?

A

continue tx(mesalamine, sulfasalazine, and TNF alpha inhibitor)in pregnancy but in breastfeeding continue except sulfasalazine.

17
Q

Complication?

A

IDA
IUGR
Preterm delivery

18
Q

Arteriovenous malformation?

A

GI bleeding
IDA
Ass. disease (CKD, VWD, and AS)

19
Q

Diverticulitis?

A

age >60

painless hematochezia, not an ass. with pregnancy

20
Q

Internal hemorrhoid?

A

painless hematochezia
constipation
itching

21
Q

Ovarian overstimulation syndrome?

A

Ovulation induction(clomiphene citrate)–execcve VEGF expression by ovaries–bilaterally enlarged ovaries with fluid third spacing(PE and Ascitis)

22
Q

rectovaginal fistula presentation?

A

leakage of flatus or feces(dark brown discharge) from the posterior vagina
Dark red valvey lesion(Due rectal mucosal overgrowth to form sinus)

23
Q

Risk factor?

A
Obstetric trauma
Pelvic radiation
pelvic surgery
Colonic ca
chrons disease
Diverticulitis
24
Q

Diagnosis?

A

PE
fistulography
MRI
Endosonography

25
Uterine inversion CM?
PPH Round, smooth protruded mass per vagina Lower abdominal pain Unpalpable uterus
26
Risk factors?
Macrosomia Precipitous labor Nuliparity Morbidly adherent placenta
27
mechanism?
excessive fundal pressure | excessive cord traction
28
differential?
protruding myoma
29
how to differentiate?
uterus palpable at the abdomen
30
management of UI?
Manual reversion Relaxant (nitroglycerin and terbutaline) if manual attempt faille--do then manual inversion Remove placenta and give uterotonic agent after reversion
31
Remove placenta and give uterotonic agent after reversion why?
b/c risk of further bleeding and cervical contraction which make inversion difficult consecutively.
32
Fibroid CM?
``` heavy prolonged menses pressure Sx (pelvic pain, constipation, and urinary frequency) Enlarged irregular uterus ```
33
Obstatric complication?
preterm labor abortion infertility
34
Management?
HM(preserve fertility) | hysterectomy(not want fertility)
35
spontanous pregnancy decrease with increase maternal age why?
decrease ovarian reserve
36
At which age will be a sharp decline?
age 35
37
associated hormonal finding?
increase FSH due to low estrogen prdn by the ovary
38
clinical importance related to infertility?
Infertility define > 6 month age > 35(unlike one year in age <35)
39
How to D/T with primary ovarian failure?
in POI there will be hypoestrogenic symptoms like hot flashes, vaginal dryness...