Ophthalmic Disorders Flashcards

(166 cards)

1
Q

COMMON EYE COMPLAINTS

A

Redness
Pain
Foreign body sensation
Photophobia
Dryness
Watery eyes
Decreased visual acuity
Loss of vision
Double vision
Conjunctival discharge
“Spots,” “floaters,” “flashing lights”

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2
Q

Eye redness diseases

A

Acute conjunctivitis
Acute Anterior Uveitis (iritis)
Acute Angle-Closure Glaucoma
Corneal Trauma or Infection

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3
Q

what can cause ocular discomfort?

A

Pain (trauma, infxn, rapid increase in IOP)

Dry eye (lacrimal gland hypofunc. related to systemic disorders or drugs)

Watery eyes (inadequate tear drainage related to obstruction of lacrimal drainage sys or malposition of lower lid, reflex tearing due to disturbance of corneal epithelium)

Photophobia (keratitis, iritis, albinism, aniridia, cone dystrophy, fever due to systemic infection)

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4
Q

Strabismus

A

Ocular misalignment

Only one eye fixates on an object

Present in ~4% of children
“Ocular instability of infancy” - unsteady ocular alignment often present in normal newborns during first few months of life, typically goes away after 4-6 months

Risk factors = family history, preterm/low birth weight, other ocular conditions, certain neuromuscular conditions

Can also be acquired due to cranial nerve palsies or orbital mass, fracture, or thyroid eye disease

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5
Q

Prefix describes

A

direction of eye deviation

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6
Q

Eso

A

inward

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7
Q

Exo

A

outward

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8
Q

Hyper

A

upward

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9
Q

Hypo

A

downward

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10
Q

Suffix describes

A

conditions under which it is present

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11
Q

Phoria (latent strabismus)

A

strabismus that is present only when binocular fusion is disrupted (i.e. when one is covered)

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12
Q

Tropia (manifest strabismus)

A

strabismus that is present when there is no disruption of binocular fusion

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13
Q

Most common causes - Strabismus
Esodeviations =

A

DASIA

accommodative esotropia, idiopathic infantile esotropia, Duane syndrome, abducens palsy, sensory esotropia

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14
Q

Most common causes - Strabismus
Exodeviations =

A

intermittent exotropia

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15
Q

Most common causes - Strabismus
Hyperdeviations =

A

trochlear nerve palsy

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16
Q

Most common causes - Strabismus
Hypodeviations =

A

fracture of orbital floor or wall, thyroid-related ophthalmopathy, Brown syndrome, oculomotor palsy, trochlear nerve palsy, congenital fibrosis of extraocular muscles

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17
Q

Strabismus test

A

Corneal light reflex (Hirschberg test)

Cover test (detects tropia) - looking at uncovered eye

cover/uncover test (detects phoria)

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18
Q

cover test

A

Detects tropia (manifest strabismus)

In this test, the child is asked to visually fixate on a target at distance or near. The examiner briefly covers one eye while observing the opposite eye for movement

No movement is detected when covering either eye if the child has normal ocular alignment (orthotropia).

Manifest strabismus (tropia) is present if the eye that is not occluded with the cover test shifts to refixate on the target when the fellow previously fixating eye is covered

repeat on each eye

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19
Q

Cover/uncover test

A

Detects phoria (latent strabismus)

Do not have to do if cover test reveals tropia

the child is asked to visually fixate on a target at distance or near. A cover is placed over one eye for a few seconds, and then it is rapidly removed. The eye that was under the cover is observed for refixation movement

If a phoria is present, this previously covered eye will shift back into the orthotropic (straight-ahead) position to reestablish sensory fusion with the other eye

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20
Q

Strabismus complications

A

amblyopia (in up to half of younger children with strabismus)

Diplopia (in acquired strabismus in patients > 8 years old

Secondary contracture of extraocular muscles

Adverse psychosocial and vocational consequences

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21
Q

when to refer (strabismus)

A

Constant strabismus at any age

Intermittent manifest strabismus after 4-6 months of age

Persistent esodeviations after 4 months of age

Abnormal corneal light reflex test or cover test

Deviation that changes depending on position of gaze

Diplopia or asthenopia

Parental concern about ocular alignment

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22
Q

Corneal light reflex (Hirschberg test)

A

shining a light onto the child’s eyes from a distance and observing the reflection of the light on the cornea with respect to the pupil. The location of the reflection from both eyes should appear symmetric and generally slightly nasal to the center of the pupil

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23
Q

Treatment (strabismus)

A

Non-surgical = glasses, contacts, occlusion therapy, visual training exercises

Surgical = recession, resection and transposition of extraocular muscles

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24
Q

Amblyopia - what it is

A

Functional reduction in visual acuity caused by abnormal visual development early in life

50% of cases related to strabismus

predominately unilateral (defined as as ≥ 2 line difference in visual acuity between eyes)

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25
Amblyopia risk factors
prematurity, small for gestational age, first-degree relative with amblyopia, neurodevelopmental delay
26
Amblyopia MCC of
ped visual impairment (1-4% children) One of the reasons we assess vision in preschool-age children! Vision screening at age 3, 4, and 5 Refer to ophthalmology/optometry if suspected
27
Leukocoria
White pupillary reflex
28
Leukocoria common causes
Retinoblastoma! Cataract, coats dz, persistent fetal vasculature, vitreous hemorrhage, ocular toxocariasis, Hereditary retinal dysplasia Retinal detachment, Coloboma, Astrocytic hamartoma
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Retinoblastoma
Most common primary intraocular malignancy of childhood accounts for 10-15% cancers that occur w/i first year of life median age at dx = 18-20 months typically presents at leukocoria in a child <3 years
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Retinoblastoma other S/S
Strabismus! Nystagmus; red, inflamed eye; decreased vision; FH
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Retinoblastoma Treatment
deadly if untreated, tx is very successful (>95% 5-year survival rate) multidisciplinary approach - chemo - surgery - cryotherapy - laser photoablation
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Retinitis Pigmentosa
Causes progressive loss of night and peripheral vision
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Retinitis Pigmentosa
group of inherited dystrophies = progressive degeneration and dysfunction of retina Primarily affects photoreceptor and retinal pigment epithelial function May occur alone or as part of a syndrome May be inherited or occur sporadically
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Retinitis Pigmentosa - Ophtho findings
Attenuation of retinal blood vessels Waxy pallor of optic disc Intraretinal pigmentation in a bone-spicule pattern
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Retinitis Pigmentosa - Treatment
no great tx currently
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Conjunctivitis
Inflammation of the mucous membrane that lines the surface of the eyeball and inner eyelids
37
Conjunctivitis - 3 types
Viral Bacterial Allergic
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Conjunctivitis - Mode of transmission:
direct contact of contaminated fingers or objects to other eye or other persons
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Conjunctivitis - symptoms
foreign body sensation scratching/burning itching photophobia sensation of fullness around eyes
40
Viral conjunctivitis
MCC = adenovrius May or may not be part of viral prodrome followed by adenopathy, fever, pharyngitis, URI
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Viral conjunctivitis usually sequential
bilateral dz spreads easily
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Viral conjunctivitis presentation
Conjunctival injection with WATERY or mucoserous discharge Burning, sandy, gritty feeing in one eye May have crusting in the morning and scant mucus throughout the day May see small amount of mucoid discharge when pulling down lower lid on exam, but usually profuse tearing +/- follicular appearance of tarsal conjunctiva
43
Viral conjunctivitis management
no specific tx lasts up to 2 weeks; may get worse before better (consult pts on this) Artificial tears and cold compresses may help discomfort
44
Viral conjunctivitis made up story
Adens kid FOLLed 2 weeks ago at the Sandy shore. His kids EYES (both eyes watery) filled with water because it he just had a viral infection. I told that kid it may get worse before it gets better. It was COLD that day anyway.
45
Bacterial Conjunctivitis MC organisms
Staph aureus, Strep pneumo, Haemphilus species, Pseudomonas (contact lens wearers), Moraxella "Some Say Hairy People that wear contacts Make bacterial conjunctivitis)" other causes gonococcal conjunc. and chlamydial conjun. "they have gonorrhea and chlaymdia and are very contagious"
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Bacterial Conjunctivitis other factors
usually unilateral highly contagious
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Bacterial Conjunctivitis clinical presentation
Purulent eye discharge Eyelid matting Mild discomfort Mild blurring of vision
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Bacterial Conjunctivitis - management
Usually self-limited Lasts 10-14 days if untreated Topical antibiotics may hasten remission May consider depending on back-to-school considerations Erthryo ointment or Bactrim (TMX-SMP) drops Contact lens wearers must always be treated! (fluoroquinolones drop such as ofloxacin or ciprofloxacin)
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Made up story Bact. Conjunc.
Some Say Hairy People that wear contact lens Make bacterial conjunctivitis in one eye. They have Gonorrhea and Chlamydia so they are very Contagious. They discharge constantly and pray on a Mat that is goes away. Luckily is doesn’t hurt too bad and they just have mild blurry vision, this should be a Self lesson. Maybe a 2 week lesson. I they are in school they can have antibiotic topicals because we don’t want them to come BACk with red eyes (erythromycin) and if they do wear contact lens they should be treated with eye drops Of Course (ofloxacin and ciprofloxacin). She needs new makeup anyway and forget about them contacts until the discharge is gone for another 24.
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Contact users for Bact. Conjunc.
ciprofloxacin vs ofloxacin Don't wear contacts until no longer red and no discharge 24 hours after taking meds; change makeup esp. things like eyeliner
51
Gonococcal Conjunctivitis
usually acquires thru genital secretions ophthalmic emergency!
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Gonococcal Conjunctivitis - clinical presentation
Copious purulent discharge
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Gonococcal Conjunctivitis - Diagnosis
confirm with stained smear and culture of discharge
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Gonococcal Conjunctivitis - Management
Single dose ceftriaxone IM May add topical erythro/bacitracin Also treat for chlamydia (doxy) Consider other STIs
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Chlamydial Conjunctivitis
Trachoma = chronic keratoconjunctivitis caused by recurrent infection with Chlamydia trachomatis
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Chlamydial Conjunctivitis MC in
children Typically asymptomatic May have eye redness, discomfort, light sensitivity, and mucopurulent discharge Recurrent episodes cause bilateral follicular conjunctivitis, epithelial keratitis, corneal vascularization, scarring of tarsal conjunctiva Scarring of tarsal conjunctiva leads to entropion and trichiasis
57
Chlamydial Conjunctivitis - Adult inclusion conjunctivitis
Chronic, indolent conjunctivitis caused by certain serotypes of C. trachomatis Concurrent asymptomatic urogenital infection typically present Usually unilateral follicular conjunctivitis lasting weeks-months that has not responded to topical antibiotics
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Chlamydial Conjunctivitis - Diagnosis
Giemsa or DFA stain of conjunctival smears or culture of swabbed specimen
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Chlamydial Conjunctivitis - Treatment
Single dose of azithromycin PO surgery to correct lid deformities
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Allergic Conjunctivitis
Usually associated with atopy: Eczema, asthma, allergic rhinitis May be seasonal (hay fever); usually in spring/summer Bilateral
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Allergic Conjunctivitis - Clinical Presentation
Itching Conjunctival hyperemia and edema (chemosis) Watery discharge Cobblestoning of upper tarsal conjunctiva Itching
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Allergic Conjunctivitis - Management
Avoid known allergens Avoid rubbing Cool compresses Artificial tears Avoid contact lenses Topical therapy (see next slide) Oral H1 antihistamines
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Examples of antihistamines for Allergic Conjunc.
Olopatadine (OTC) - usually 1st line + oral antihistamine
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Vasoconstrictor/antihistamine combo (allergic conjunc)
Usually not recommended b/c after using can make eyes red Naphazoline and pheniramine (OTC)
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Mast cell stabilizers (allergic conjunc)
can be used if initial antihistamine not effective enough Rx only Cromolyn sodium Nedocromil
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Big Picture - Bacterial Conjunc.
unilateral or bilateral Discharge most prominent symptom Eye often "stuck shut" in morning No other symptoms "red eye"
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Big Picture - Viral Conjunc.
Unilateral or bilateral Sensation of grittiness, burning, or irritation Crusting on lid margin in morning May be part of viral URI w/ associated nasal congestion or pharyngitis "red eye"
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Big Picture - Allergic Conjunc.
Typically bilateral Itching Sensation of grittiness, burning, or irritation Crusting on lid margin in morning May have other allergic symptoms, such as nasal congestion, sneezing, cough, wheezing "red eye"
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Bacterial Conjuctivits - Tx
self limited topical abx may shorten symptom duration and required for contact lens wearers
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Viral Conjunctivitis - Tx
Self limited topical antihistamines and/or topical decongestants warm or cool compresses
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Allergic Conjunctivits - Tx
minimizing exposure to allergen topical lubricants cool compresses topical or systemic antihistamines
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Dacryocystitis
Infection of lacrimal sac secondary to obstruction of nasolacrimal duct Thought to be related to chronic inflammation resulting in fibrosis within the duct uncommon
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Dacryocystitis Pathogens
S aureus, S epidermis, Pseudomonas aeruginosa or anaerobic organisms
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Dacryocystitis - Clinical Presentation
Tearing/discharge Inflammation, pain, swelling, tenderness beneath medial canthal tendon in area of lacrimal sac May be able to express purulent drainage through lacrimal puncta by applying pressure to sac
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Dacryocystitis - Diagnosis
Gram stain/culture of drainage
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Dacryocystitis - Treatment
Antibiotics based on gram stain/culture results Surgical correction
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Keratoconjunctivitis Sicca
AKA Dry eye disease Multifactorial loss of hemostasis of tear film Can have significant impact on visual acuity, social/physical functioning, and workplace productivity
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Keratoconjunctivitis Sicca- Risk factors
Advanced age Female sex Hormonal changes Systemic diseases (DM, Parkinson’s, Sjogren’s) Contact lens wear Systemic/ocular medications Nutritional deficiencies (vitamin A) Decreased corneal sensation Ophthalmic surgery Low-humidity environments
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Keratoconjunctivitis Sicca - Management
Artificial tears Discontinue offending medications if possible Warm compresses
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Made up story - KCS
Keep Congress Stupid - they cant see what's going on , no social life, and especially no workplace productivity. They are just so DRY. Most are old, some women with hormone changes (yikes), some had eye surgery and have to wear contacts and take medication for. They just need to have WARMer hearts or DISCONTINUE working. Cry me a river (of artificial tears)
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HSV Keratitis
Ability of virus to colonize trigeminal ganglion leads to recurrences that may be precipitated by fever, excessive exposure to sunlight or immunodeficiency Typically unilateral
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HSV Keratitis - Clinical Presentation
Dendritic (branching) corneal ulcer!!! Lid, conjunctival or corneal ulceration
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HSV Keratitis - Diagnosis
Fluorescein stain and examination with cobalt blue light
84
HSV Keratitis - Treatment
Topical or oral antivirals - Trifluridine drops, ganciclovir gel, acyclovir ointment (10-14 days) - Acyclovir, valacyclovir PO Avoid topical corticosteriods (leave to ophtho) - CS can promote viral replication
85
Bacterial Keratitis MC pathogens
staph, strep, Pseudomonas aeruginosa, Moraxella species, other gram neg bacilli
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Bacterial Keratitis - risk factors
Contact lens wear (especially overnight) Corneal trauma
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Bacterial Keratitis - Clinical presentation
Foreign body sensation Difficulty keeping eye open!! Central corneal ulcer +/- hypopyon (WBC in anterior chamber and fall to bottom = emergency!) Mucopurulent discharge
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Bacterial Keratitis - Treatment
Urgent ophtho referral Topical fluoroquinolones applied hourly for first 48 hours
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Herpes Zoster Ophthalmicus
freq involves ophthalmic division of trigeminal nerve Involvement of tip of nose or lid margin predicts involvement of the eye Potentially sight-threatening
90
Herpes Zoster Ophthalmicus - Clinical Presentation
Periorbital burning/itching Vesicular rash that becomes pustular, then crusts Conjunctivitis, keratitis, episcleritis, anterior uveitis +/- elevated IOP
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Herpes Zoster Ophthalmicus - Tx
Urgent ophtho referral High dose oral antivirals (Acyclovir, valacyclovir, famciclovir x 7-10 days)
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Periorbital Cellulitis
AKA preseptal cellulitis Infection of soft tissues anterior to orbital septum Does not involve orbit or other ocular structures May be confused with orbital cellulitis Generally mild, rarely leads to serious complications May arise from sinusitis or local trauma
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Periorbital Cellulitis - Pathogens
If related to sinuses or nasopharynx = S pneumo, Moraxella, H influenzae If arising from trauma or other skin infection = Staph aureus, Strep pyogenes
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Periorbital Cellulitis - Clinical Presentation
Unilateral ocular pain Eyelid swelling and erythema Does NOT affect visual acuity or extraocular movements
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Periorbital Cellulitis - Treatment
Empiric abx Amoxicillin/clavulanate Consider admission in children < 1 year old, severely ill, or unsure if orbit is involved
96
Orbital Cellulitis
Infection involving contents of orbit (fat and extraocular muscles) Can be vision/life-threatening MC in children Typically caused by infection of paranasal sinuses
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Orbital Cellulitis - Clinical presentation
Fever Proptosis Restriction of extraocular movements Pain/swelling with redness of lids Decreased visual acuity
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Orbital Cellulitis - Diagnosis
CT/MRI Culture if purulent drainage on exam
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Orbital Cellulitis - Treatment
Urgent IV abx to prevent optic nerve damage and spread of infection Antibiotic choice depends upon pathogen Empiric = vancomycin + ceftriaxone +/- surgical drainage of paranasal sinuses/orbital abscess
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Orbital Cellulitis - Complications
Cavernous sinus thrombosis Intracranial extension Vision loss Death
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Big picture - Preseptal Cellulitis
Eyelid swelling w/ or w/o erythema Eye pain/tenderness may be present chemosis rarely present Fever and leukocytosis may be present
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Big Picture - Orbital Cellulitis
Eyelid swelling w/ or w/o erythema Eye pain/tenderness Pain w/ eye movements Proptosis usually but may be subtle Ophthalmoplegia +/- diplopia may be present vision impairment may be present Chemosis and leukocytosis may be present Fever usually present
103
Blepharitis
Chronic bilateral inflammatory condition of lid margins - Anterior = involves lid skin, eyelashes and associated glands - Posterior = results from inflammation of meibomian glands Common cause of recurrent conjunctivitis
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Blepharitis - Clin Pres
Irritation, burning, itching Anterior = “red-rimmed” eyes with scales clinging to lashes Posterior = hyperemic lid margins with telangiectasias, inflamed meibomian glands
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Blepharitis - Tx
Anterior = warm compresses, eyelid cleansing with baby shampoo, antibiotic ointment for acute exacerbations Posterior = Regular meibomian gland expression, warm compresses
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Chalazion
Common granulomatous inflammation of a meibomian gland that may follow an internal hordeolum
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Chalazion - Clin Pres
Hard, NONTENDER swelling on upper or lower lid Redness and swelling of adjacent conjunctiva
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Chalazion - TX
Warm compresses Incision and curettage if not resolved in 2-3 weeks Corticosteroid injection may be effective Typically improve over several months- refer to ophtho for IL corticosteriod injection or surgical removal
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Hordeolum (stye)
Acute infection/abscess of either meibomian gland (internal) or gland of Zeis or Moll (external) MC due to staph aureus
110
Hordeolum - Clin Presentation
Localized red, swollen, TENDER area of upper or lower lid
111
Hordeolum - Tx
Warm compressions Incision if not resolved in 48 hours Topical antibiotic ointment applied every 3 hours during acute stage Generally improves w/i 1-2 weeks
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Hordeolum - complications
Internal hordeolum may lead to generalized cellulitis of lid
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Chalazion - risk factors
Rosacea, posterior blepharitis
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Hordeolum - risk factors
Rosacea, seborrhiec dermatitis, use of eye make-up
115
Cataracts
Opacities of crystalline lens Usually bilateral Leading cause of blindness worldwide MCC = age
116
Cataracts - other causes
Congenital Traumatic Systemic disease (DM) Topical, systemic, or inhaled corticosteroid treatment Uveitis Radiation exposure
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Cataracts - risk factor
smoking cigarettes
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Cataracts - clinical presentation
Progressive blurring of vision Flare in bright lights or night driving
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Cataracts - Tx
Surgery (improves visual acuity in 95% of cases) Topical eye drops to dissolve or prevent cataracts are being experimented
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Cataracts - Prevention
Multivitamin/mineral supplement High dietary antioxidants
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Chronic Glaucoma
Gradually progressive excavation (“cupping”) of optic disk with progressive loss of vision (slight visual field loss to complete blindness)
122
3 types of Chronic glaucoma
Open- angle (Elevated IOP due to reduced drainage of aqueous fluid through trabecular meshwork) Angle-closure (Obstruction of flow of aqueous fluid into anterior chamber) Normal-tension (Normal IOP but same pattern of optic nerve damage)
123
Chronic Glaucoma Dx requires consistent and reproducible abnormalities in at least 2/3 parameters:
Often first suspected at routine eye test - Optic disk cupping = an increase or asymmetry between the two eyes of the ratio of diameter of optic cup to diameter of whole optic disk (ratio > 0.5 or asymmetry > 0.2) - Visual field abnormalities (central vision remains good until late in disease) - Intraocular pressure = Normal range 10-21 mmHg
124
Chronic Glaucoma - Screening targeted
Affected first-degree relative Diabetes mellitus Older individuals with African or Hispanic ancestry Long-term use of corticosteroids
125
Chronic Glaucoma - Tx
Prostaglandin analog eye drops - Latanoprost, bimatoprost Alpha-2-agonist, topical carbonic anhydrase inhibitors can be used in addition - Brimonidine, brinzolamide Laser therapy/surgery Open-angle glaucoma = trabeculectomy Angle-closure glaucoma = iridotomy/iridectomy
126
Acute Angle-closure Glaucoma (Primary)
Results from a closure of preexisting narrow anterior chamber angle Closure of angle precipitated by pupillary dilation
127
Acute Angle-closure Glaucoma (Secondary)
Does not require preexisting narrow angle May occur with anterior uveitis, dislocation of lens, hemodialysis, or various drugs
128
Acute Angle-closure Glaucoma - Clin Pres
Same symptoms but diff management Extreme pain Blurred vision Halos around lights +/- nausea, abdominal pain Red eye, cloudy cornea, moderate dilated pupil that is nonreactive to light IOP > 50 mm Hg Hard eye on palpation
129
Acute Angle-closure Glaucoma - Tx
Initial treatment is reduction of IOP with IV acetazolamide + topical medications
130
Acute Angle-closure Glaucoma - Tx Primary
Topical 4% pilocarpine q 15 mins x 1 hour, then four times a day Cataract extraction (definitive; sometimes done first-line) Laser peripheral iridotomy Consider prophylactic laser peripheral iridotomy to unaffected eye
131
Acute Angle-closure Glaucoma - Tx Secondary
Treat underlying cause
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Acute Angle-closure Glaucoma - Prognosis
Results in permanent visual loss within 2-5 days if not treated
133
Macular Degeneration
Age-related is leading permanent visual loss in older population Prevalence progressively increases over age 50 Slight female predominance
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Macular Degeneration - Risk factors
Family history HTN Hyperlipidemia CVD Farsightedness Light iris color Cigarette smoking
135
Macular Degeneration - 2 types
Wet Dry
136
Macular Degeneration - Clin Pres
Drusen Hard drusen = discrete yellow subretinal deposits Soft drusen = paler and less distinct Central vision loss “Dry” = gradually progressive bilateral visual loss “Wet” = more rapid and severe onset of visual loss
137
Macular Degeneration - Tx
Dry = no specific treatment Wet = rehabilitation including low-vision aids; VEGF inhibitors (Ranibizumab, bevacizumab, aflibercept, brolucizumab) Stop smoking Vitamin supplements can reduce progression = vitamins C and E, zinc, copper, carotenoids
138
Retinal Detachment 3 types
Rhegmatogenous (most common) Tractional Exudative
139
Retinal detachment - Rhegmatogenous
One or more peripheral retinal tears or holes Usually results from posterior vitreous detachment related to degenerative changes in vitreous Can also be caused by penetrating or blunt trauma Often occurs in people > 50 years of age
140
Retinal detachment - Tractional
preretinal fibrosis (as in proliferative retinopathy due to diabetic retinopathy or retinal vein occlusion) OR complication of rhegmatogenous retinal detachment
141
Retinal detachment - Exudative
accumulation of subretinal fluid , (neovascular age-related macular degeneration or secondary to choroidal tumor
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Retinal Detachment - Clin Pres
Rapidly progressive visual field loss Floaters Photopsias Retina may be seen elevated in vitreous cavity with an irregular surface on ophthalmoscopic exam
143
Retinal Detachment - Tx
Laser photocoagulation Pneumatic retinopexy = (expansile gas injected into vitreous cavity and patient’s head is positioned to facilitate apposition between gas and the hole, which permits reattachment of retina Vitrectomy, direct manipulation of retina, internal tamponade of retina with air, expansile gas, or silicone oil
144
Corneal Abrasion
Defect in epithelial surface of the cornea - mechanical trauma Can be traumatic (related to foreign body or contact lens) or spontaneous
145
Corneal Abrasion - Clin Pres
Severe eye pain Photophobia Foreign body sensation preventing opening of eye
146
Corneal Abrasion - Dx
Clinical Can be confirmed on fluorescein stain Evert eyelid to assess for presence of retained foreign body
147
Corneal Abrasion - Management
Removal of foreign body Topical antibiotics Cycloplegics for large abrasions (inhibit miotic response to light) Oral/topical NSAIDs
148
Globe Rupture
Occurs following blunt eye injury
149
Globe Rupture - Clin Pres
Decreased visual acuity Relative afferent pupillary defect Eccentric or teardrop pupil Increased or decreased anterior chamber depth Extrusion of vitreous External prolapse of uvea Tenting of cornea or sclera at site of injury Low IOP
150
Globe Rupture - Management
Emergent ophtho consult! Pain control, IV abx Surgical repair
151
Subconjunctival Hemorrhage
May occur spontaneously or with Valsalva associated with coughing, sneezing, straining, or vomiting Generally asymptomatic
152
Subconjunctival Hemorrhage - Dx
confirmed by normal visual acuity and absence of S/S
153
Subconjunctival Hemorrhage - Prognosis
Blood generally resorbs over 1-2 weeks (may seem to increase on second day) No specific treatment
154
Hyphema
Blood in the anterior chamber Common complication of blunt or penetrating eye injury Can result in permanent vision loss
155
Hyphema- Clin Pres
Vision loss Eye pain Photophobia Anisocoria N/V if elevated IOP Often accompanied by corneal abrasion
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Hyphema _ Dx
Clinical Must exclude open globe
157
Hyphema - Management
Eye shield Bed rest/dim lighting Elevate HOB to 30 degrees Topical pain control Close monitoring by ophtho
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Pterygium
Fleshy, triangular encroachment of conjunctiva onto the cornea Usually associated with prolonged exposure to wind, sun, sand, and dust Often bilateral Occurs more frequently on nasal side of the conjunctiva May become inflamed and may grow
159
Pterygium - Tx
rarely required indicated when growth threatens vision
160
Pinguecula
Yellowish, elevated conjunctival nodule in area of palpebral fissure Common in people > age 35 years Often bilateral Occurs more frequently on nasal side of the conjunctiva Rarely grows but may become inflamed
161
Pinguecula - Tx
rarely required artificial tears beneficial
162
Entropion
inward turning of eyelid
163
Entropion Occurs occasionally in
older people as result of degeneration of lid fascia May follow extensive scarring of conjunctiva and tarsus
164
Entropion - Tx
surgery if lashes rub cornea; botulinum toxin injections may help
165
Ectropion
outward turning of eyelid Common with advanced age
166
Ectropion - tx
surgery if excessive tearing, exposure keratitis, or cosmetic problem