opthalmic anatomy Flashcards

1
Q

what is pulsatile proptosis and causes

A

Pulsatile exophthalmos or pulsatile proptosis is a clinical symptom characterised by protrusion and pulsation of the eyeball that can occur due to various causes:

caroticocavernous fistulas
neurofibromatosis type 1 (with sphenoid wing dysplasia) 2
arteriovenous malformation
trauma (orbital roof fractures) 3
arachnoid cyst (rare) 4
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2
Q

how is the eye opened

A

CNIII oculomotor nerve supplies the LPS to open

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3
Q

how is the eye closed

A

CNVII facial nerve supplies the orbicularis oculi to close

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4
Q

What is retrobulbar haemorrhage

A

presence of a post septal orbital haematoma and is usually due to craniofacial trauma causing an extraconal haematoma. It may cause orbital compartment syndrome which is an ophthalmologic emergency.

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5
Q

what is orbital compartment syndrome

A

rapid rise in intraorbital volume and pressure,
reducing retinal and optic nerve blood flow. Unless urgently
decompressed, ischaemia rapidly ensues, potentially leading to
retinal infarction and permanent complete blindness

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6
Q

when to suspect orbital compartment syndrome (OCS)

examination findings

A

Any patient with an acute orbital injury is at risk of OCS.

Presenting features
rapid reduction in vision (with or without a RAPD),
tense, swollen lids, orbital congestion, and proptosis.

Other examination findings variably include periorbital haematoma,
restricted eye movements, high intraocular pressure,
(associated nausea or vomiting), chemosis and central retinal artery
occlusion.

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7
Q

what to do when you suspect OCS / treatment

A

Immediate decompression is required, with a lateral canthotomy
and cantholysis
to decompress the orbit and preserve ocular function

Immediate: lateral canthotomy (incision of the lateral canthal tendon) and lower ± upper cantholysis (disinsertion of the lateral canthal tendon) (see Box 3.1).
• If no improvement or if/worsens: IV mannitol (0.5–2g) and IV acetazolamide (500mg).

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8
Q

A common error that occurs with OCS

A

range imaging or medical treatment first,
this
delaying the only intervention which reduces the intraorbital pressure

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9
Q

when should one refer with OCS

A

Urgently refer to an orbital or maxillofacial surgeon for surgical
draining if perfusion does not return despite superior and inferior
cantholysis.

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10
Q

clinical features of retrobulbar syndrome

A
• Painful proptosis.
• reduced vision.
• resistance to retropulsion.
• elevated IOP (>35mmHg).
• relative afferent pupillary defect
• restricted extraocular movements. 
• Tight eyelids.
• retinal arterial pulsations.visual disturbance
orbital bruising and swelling
features of head and/or facial trauma
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11
Q

causes of retrobulbar syndrome

A

trauma - secondary to orbital fractures
orbital surgery
other surgery e.g. sinus or neurosurgical procedures
haemorrhage from an intraorbital vascular mass e.g. orbital vascular malformation or metastases
coagulopathies

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12
Q

what is pleomorphic adenoma of the lacrimal gland

A

(benign mixed tumor) is the most common epithelial neoplasm of the lacrimal gland.

slow growing, well-circumscribed, mass that is identical to its salivary gland counterpart

excellent prognosis for vision and long-term survival after complete surgical excision.

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13
Q

clinical features of pleomorphic adenoma of the lacrimal gland

A

slowly progressive and painless proptosis, with inferomedial displacement of the globe

visual disturbance possible with large tumours

sudden onset of pain may indicate malignant transformation

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14
Q

treatment for pleomorphic adenoma

A

Complete excision is curative, with an excellent prognosis. Incomplete excision is associated with high rates of recurrence.

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15
Q

differentials for a pleomorphic adenoma

A

other lacrimal gland masses

extralacrimal masses of the superolateral orbit such as dermoid/epidermoid cyst

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16
Q

CN III palsy presentations

A

down and out

pupils dialted

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17
Q

causes of CNIII palsy

A

increased intracranial pressure
aneurysms of PCA
vascular 2ry to diabetes, HTN - pupil sparing
cavernous sinus thrombosis

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18
Q

which cranial nerve has the longest route

A

trochlear as it emerges from the dorsal aspect of the brainstem

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19
Q

CN IV palsy

A

diplopia especially when looking down

20
Q

causes of CNIV

A

head injury or increased ICP

21
Q

what is horner syndrome

A

something impinging the sympathetic trunk

miosis
ptosis\anhidrosis

22
Q

if someone comes with painful eye, face or neck with horner’s symptoms what is the causes and treatment

A

carotid dissection also causing the visual loss

call vascular team

23
Q

weakest regions of the orbit

A

floor

medial wall

24
Q

type of proptosis when its extraconal or intraconal

A

extraconal - non axial proptosis - pushing eye sideways or vertical displacement

intraconal - axial proptosis pushes it anteriorly

25
Q

describe the surgical way of treating a squint

A

muscles can be shortened, realigned

CANNOT MOVE MORE THAN 3 MUSCLES AT A TIME

RISK ISCHAEMIA - IRIS CILIARY BODY CORNEAL LIMBUS

26
Q

whats important with 6th nerve palsy

A

false localising sign for ICP as it comes of medulla pontinary junction

27
Q

why do we have a blind spot

A

no photoreceptor cells in the optic nerve head - temporal visual field

28
Q

what is surgical emphysema

A

air/gas located in the subcutaneous tissue

29
Q

what is a chalazion

A

obstruction of the tarsal glands leading to painless swelling also called meiobian cyst

30
Q

what is a stye

A

acute infection of Moll (sweat gland) or Zeiss (sebaceous glad) or eyelash follicle

31
Q

tear path

A

lacrimal fluid entering the conjunctival sac thru duct into lacrimal lake medially into eye into lacrimal sal. nasla cavity thru nasolacrimal duct into nasopharynx

32
Q

role of tear

A

provide a smooth ocular surface for light rays to be refracted uniformly and also provide lubrication to prevent friction between ocular surfaces during lid closure.

antibacterial action

33
Q

components of a tear film

A

Surface lipid layer – secreted by the meibomian (tarsal) glands

Middle aqueous layer – secreted by the lacrimal gland and accessory lacrimal glands

Inner mucus layer – secreted by goblet cells of the conjunctiva and the epithelial cell surface

34
Q

role of the lipid layer

A

prevents drying out as it stops evaporation

35
Q

what happens if tarsal glands become obstructed

A

increase inevaporation drying th eyes out causing discomfot and grittiness
increase risk of damage and infection

36
Q

causes of dry eyes

A

obstruction in tarsal glands - increased evaporation
reduced

reduced prod of tears

37
Q

causes of red prod of tears

A

autoimmune diseases - Sjogren’s, RA

medications ie anithistamines

38
Q

what are the layers of the eyeball

A

sclera
choroid
retina

39
Q

what is the anterior chamber

A

space between the cornea and the iris

40
Q

what is the posterior chamber

A

space between the iris and the ciliary body and the lens

41
Q

what is the aqueous humour

A

ciliary body secretes this

fills the chambers of the eye

42
Q

what are the muscles of the orbit

A

levator palpebrae superioris
FOUR RECTI
two oblique

43
Q

the supplied by which CN

A

oculomotor - 3
trochlear - 4
abducen - 6

44
Q

bloody supply to orbit

A

opthalmic artery - central artery to the retina

opthalmic veins - cavernous sinus

45
Q

oculomotor palsy

A

ptosis

diplopia

46
Q

role of the cornea

A

maintaining transparency
ocular protection
refraction of incoming light