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DDS1-microbio > orofacial 2, dental caries, perio > Flashcards

orofacial 2, dental caries, perio Flashcards

(61 cards)

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1
Q

fascial space infections can come from

A
  • pericoronitis

- bacterial sialadenitis

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2
Q

two things that can result from fascial space infection

A
  • ludwig’s angina

- cavernous sinus thrombosis

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3
Q

dentoalveolar abscess can lead to

A
  • osteomyelitis
  • dry socket
  • fascial space infection
  • endocarditis
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4
Q

what can cause dentoalveolar abscess

A
  • periodontal abscess

- endodontic infection

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5
Q

periodontitis is associated with what systemic disease

A

endocarditis

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6
Q

dry socket usually occurs in which teeth

A

lower 2nd and 3rd molars

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7
Q

fascial space infections usually occur in what region

A

submandibular

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8
Q

pericoronitis

A

infected flap over tissue over 3rd molar (DO surface)

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9
Q

bacteria can spread along

A

muscle attachments

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10
Q

how do bacteria spread along muscle attachments

A

-produce hyaluronase and other matrix destructive enzymes

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11
Q

where do the bacteria target the muscles ad which teeth are effected

A

sublingual area

mandibular 2nd and 3rd molars due to

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12
Q

deep fascial space infections usually come from which teeth

A

lower 2nd and 3rd molars

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13
Q

symptoms of space infection

A

board like hardness

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14
Q

which bacteria cause space infections

A
  • normal oral flora (often the strep bacteria)

- anaerobic oral bacteria may be present

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15
Q

which Abx will you give for fascial space infection

A
  • penicillin (to kill the strep)

- clindamycin if they dont get better with pen (bc anaerobic) OR metronidazole +pen

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16
Q

dentoalveolar infections can lead to what (most commonly)

A

maxillary sinusitis

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17
Q

cavernous sinus thrombosis follows the spread of odontogenic infection along 2 pathways:

A
  • anterior facial vein

- pterygoid plexus of veins

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18
Q

symptoms of cavernous sinus thrombosis

A
  • swelling of eyelids
  • protrusion of eyeball
  • restricted movement of eyeball
  • bleeding in retina
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19
Q

ludwig’s angina description

A
  • swelling of head and neck spaces

- difficulty breathing

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20
Q

main cause of ludwig’s angina

A

cellulitis

*50-90% of cases were due to dental cource of infetion

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21
Q

teeth most commonly involved with ludwig’s angina

A

mandibular 2nd and 3rd molars

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22
Q

treatment for ludwig’s angina

A
  • parenteral abx
  • airway monitoring
  • i and d
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23
Q

non odontogenic infections of oral cavity

A
  • pericoronitis
  • bacterial sialadenitis
  • angular cheilitis
  • ulcerative mucositis
  • noma (gangrenous stomatitis)
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24
Q

pericoronitis

A
  • infection of flap of gum tissue
  • usually 3rd molars
  • pts usually older teens and early 20s
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25
bacterial sialadenitis
stone in the stensons duct (parotid duct)
26
angular cheilitis
C albicans and s aureus at edge of lips
27
ulcerative mucositis
- chemotherapy associated | - bacteria and yeasts
28
noma (gangrnous stomatitis)
- polymicrobial | - opportunistic in immunocompromised
29
bacteria that cause pericoronitis
- p intermedia - anaerobic strep - fusobacterium - A A - tannerella forsythia
30
pericoronitis can lead to
spread of infection along facial plane (if drainage is interrupted)
31
main cause of bacterial sialadenitis
s aureus (post surgery)
32
abx for bacterial sialadenitis
pen erythromycin metro
33
patients that usually get bacterial sialadenitis
- pt with xerostomia - age - medication - parkinsons
34
predisposing factors of angular cheilitis
- low B vit - dry mouth - overclosure - habits or drooling - immunosuppression - poor fitted dentures
35
ANUG
- sudden onset painful infection of gums | - common in students around march
36
cause of ANUG
- fusobacterium and spirochetes like trep denticola | - stress and immunosuppression
37
abx for ANUG
metro
38
bacteria that cause actinomycosis
- opportunistic | - cross tissue planes
39
cervicofacial actinomycosis
- Actinomyces (esp A israelii) - rock-hard induration - SPONTANOUS DRAINING OF SINUS TRACT
40
once an enamel caries reaches dentin, it
fans out and lesion gets bigger
41
what happens to biofilms when they cause disease
they become PLANKTONIC!
42
types of study designs
- cross sectional design | - longitudinal design
43
cross sectional study design
-compare microflora in health and disease at single point in time
44
longitudinal design
-compare in a single patient at different points in time
45
no single microbe has satisfied
koch's postulates for plaque-mediated disease
46
specific plaque hypothesis
only few species involed in disease (eg RED organisms)
47
non-specific plaque hypothesis
disease is outcome of overall activity of total plaque biome
48
ecological plaque hypothesis
- bacteria found in both healthy and disease | - disease is a shift in balance of resident bacteria due to change in environmental conditions
49
bacteria that cause root surface caries
- strep mutans - s sanguinis - a naeslundii
50
specific model of enamel demin
- s mutans only | - surface
51
non specific model of enamel demin
- s mutans AND Veillonella | - deep lesions
52
two things that are lost in perio
- collagen in gingiva | - bone
53
dental abscess are __ while perio is ___
acute, chronic
54
what happens to MMPs and TIMPs in perio
- activate MMPs | - degrade TIMPs
55
recognition of quorum sensing
plaque => biofilm
56
red complex organisms
-p gingivalis -t forsythia t denticola -AA
57
gingivitis
- precedes perio | - reversible
58
periodontal disease
- chronic - gingivitis but will loss of attachment between root surface, ging, and albeolar bone - bone loss - irreversible
59
perio disease is a combo of what
biofilm (direct) + host immune response (indirect)
60
advanced lesion
- comes after established lesion (red gums) - conversion of ging to perio! - "leukocte wall" is unique
61
main destruction in perio is due to
indirect mechanisms - immune response!

DDS1-microbio (10 decks)

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