Peadiatric Hip Conditions Flashcards

(50 cards)

1
Q

when does DDH tend to present?

A

birth - 2 years

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2
Q

when does perthes disease tend to present?

A

4-8 years

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3
Q

when does SUFE tend to present?

A

10-16 years

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4
Q

how does the acetabulum develop>

A

triradiate cartilage in the middle ossifies over time

- cartilage composed of ischium, ilium and pubis

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5
Q

why is X ray not that useful in very young children? what is used instead?

A

femoral head has not yet ossified so wont be seen on X ray

US is better

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6
Q

H line on X ray?

A

horizontal line across the 2 triradiate cartilages - shows symmetry

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7
Q

what other lines can show positioning of hip on X ray?

A

2 perpendicular perkin lines - intersect the H line - upper femoral epiphysis should be mainly be in the lower left corner of the intersecting lines

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8
Q

what is DDH?

A

developmental dysplasia of the hip
developmental disorder resulting in dysplasia and possible subluxation/dislocation of the hip secondary to capsular laxity and mechanical factors

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9
Q

describe the spectrum of DDH

A

just dysplasia with shallow/underdeveloped acetabulum
subluxation
dislocation

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10
Q

where does DDH most commonly occur?

A

left hip in females (due to they way the baby lies in utero)

but can be bilateral

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11
Q

what demographics is DDH more/less common in?

A

more common in native americans and Laplanders
less common in African patients
- due to the way baby is carried after birth

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12
Q

describe the pathophysiology of DDH?

A

initial instability caused by maternal and fetal laxity, genetic laxity and intrauterine and postnatal malpositioning

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13
Q

describe the pathoanatomy of DDH

A

initial instability leads to dysplasia,dysplasia leads to gradual dislocation

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14
Q

how does DDH differ to a normally developing hip?

A

normal = correctly positioned femoral head stimulates normal head and acetabular growth
absent in DDH where hip never was or becomes subluxed/dislocated

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15
Q

what are the risk factors for DDH?

A
first borns
female
breech presentation
family history
oligohydramnios
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16
Q

how does DDH present?

A

abnormality on screening
limping - trandellenberg gait
pain later in life

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17
Q

why do DDH patients get a trandellenberg gait?

A

short lever arm means abductors need to work harder

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18
Q

how is DDH diagnosed?

A

clinical exam - leg lengths, restricted abduction, skin creases
US
radiographs

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19
Q

what 2 clinical tests can indicate DDH?

A

ortolani - can you reduce the dislocated hip back in?

Barlows - can you dislocate the hip?

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20
Q

how is early DDH treated?

A

Pavlik harness - pute femoral head into position of safety (abduction and flexion)
night time splinting for a few weeks afterwards

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21
Q

how is Pavlik harness used?

A

worn 23 hrs a day for up to 12 weeks until US is normal

22
Q

how is late DDH treated?

A

surgery
- closed reduction +/- tendonotomies + spica (cast)
- open reduction + osteotomies + spica (cast)
hip arthrogram to assess congruency during treatment

23
Q

what is reactive synovitis?

A

inflammation of the synovium, often secondary to a viral illness

24
Q

how does reactive arthritis present?

A
history of viral illness
limp with hip/groin pain
can have only referred pain to the knee
lying with hip flexed/externally rotated
pain at end range of hip movement
usually generally well
25
how is reactive synovitis diagnosed?
kochers criteria | US +/- aspiration
26
what is kochers criteria?
distinguishes between septic arthritis and reactive arthritis - higher score = more chance of it being septic arthritis
27
how is reactive synovitis treated?
self limiting | analgesia/NSAIDs
28
what is septic arthritis of the hip?
intra-articular infection of the hip joint
29
why is septic arthritis a surgical emergency?
high bacteria load causing sepsis destruction of joint due to proteolytic enzymes potential for osteonecrosis of the hip due to increased pressure
30
how does septic arthritis of the hip present?
``` sudden onset unable to weight bear due to pain hip lying flexed/externally rotated severe hip pain on passive movement usually pyrexial but can be haemodynamically stable ```
31
through what 5 mechanisms can septic arthritis occur?
direct inoculation hematogenous seeding extension from adjacent bone (osteomyelitis) continuous spread of osteomyelitis can often spread from highly vascular metaphysis
32
what are the most common causative organisms in septic arthritis?
neonates - strep infant - adults = staph aureus IV drug user = suspect pseudomonas and atypical
33
how is septic arthritis treated?
``` blood tests blood cultures kochers criteria radiographs (to rule out) US +/- aspiration ```
34
how is septic arthritis treated?
open surgical washout (with samples) | antibiotics for 6 weeks
35
what is perthes disease?
avascular necrosis of the hip
36
risk factors for perthes?
``` male 4-8 year olds lower socioeconomic class family history low birth weight second hand smoke Asian, inuit, central europe ```
37
is perthes unilateral or bilateral?
usually unilateral
38
describe the pathophysiology of perthes
osteonecrosis occurs secondary to disruption of blood supply to femoral head followed by revascularisation with subsequent resorption and later collapse creeping substitution provides pathway for remodelling after collapse
39
what are some proposed mechanisms for perthes development?
possible association with abnormal clotting factors | repeated subclinical trauma and mechanical overload lead to bone collapse and repair
40
what are the stages of perthes disease?
initial fragmentation reossification remodelling
41
what determines prognosis in perthes?
age | - younger = better
42
how does perthes present?
gradual painless limp sometimes intermittent groin/thigh/knee pain hip stiffness limp (trendelenberg gait)
43
how is perthes diagnosed?
radiograph | MRI
44
how is perthes treated?
aim to keep femoral head round whilst the process self terminates restrict weight bearing maintain ROM with physio
45
is surgery ever used in perthes?
occasionally young patients with severe disease/deformity older patients with secondary osteoarthritis
46
what is SUFE?
slipped upper femoral epiphysis | condition affecting the proximal femoral physis that leads to slippage of the metaphysis relative to the epiphysis
47
what are the risk factors for SUFE?
male obesity endocrine disorders associated with a period of rapid growth
48
how does SUFE present?
``` variable length of development groin pain (or knee/thigh) limp obligatory external rotation on hip flexion ```
49
how is SUFE diagnosed?
radiographs | MRI
50
how is SUFE treated?
surgery - pinning of the hip - +/- pinning of the other side +/- open reduction if very severe