PSYCHOPATHOLOGY Flashcards

(10 cards)

1
Q

Definitions of abnormality

Deviation from social norms

A

When someone violates rules or expected behaviour of a group or society
evaluation:
-norms are culturally relative.We label others according to our standards.E.G hearing voices is normal in africa
+useful in different contexts
-definition could lead to human rights abuse.Used to control minority groups

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2
Q

Definitions of abnormality

statistical infrequency

A

Numerically uncommon,defining something as ‘normal’ or ‘abnormal’ depending on how far away it is from the mean, median or average
Evaluation:
+ real life application.Can be used to diagnosed disorders
- unusual characteristics can be positive

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3
Q

deviation from abnormality

failure to function adequately

A

-inability to cope with the demands of everyday life
Rosenhan criteria:
personal distress:person is upset/depressed
violation of moral standards:breaking laws/unwritten social rules
observer discomfort:behaviour which makes other people feel uncomfortable
evaluation:
+considers patients perspective
-involves subjective judgment

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4
Q

definitions of abnormality

deviation from ideal mental health

A

a criteria needed to be reached for a psychological well-being.The less people meet this, the more abnormal
Jahodas criteria:
personal autonomy:independent of others
positive attitude towards the self:having high self esteem
self actualisation:growth and realisation of one’s potential.Best version of yourself
evaluation
-sets up unrealistically high expectations for mental health
-bias towards individualistic cultures
+covers broad range of criteria

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5
Q

behavioural explanation for phobias

A

Two process model (Mowrer)
-phobias are learnt through classical conditioning
NS is paired with UCS which causes fear UCR.After a while becomes CS leading to fear CR.Generalisation of fear to other stimuli
Phobias are maintained through operant conditioning
-negative reinforcement.Avoiding phobia causes relief of anxiety
LITTLE ALBERT STUDY
procedure:association of white rats (NS) with loud noise(UCS) lead to unpleasant response (UCR)
findings:white rats became CS leading to fear (CR)
generalised to similar stimuli (white furry objects)
evaluation:
+application to therapy
-doesn’t explain development of all phobias
-phobias don’t always follow trauma.Dinardo et al found that not everyone bitten by dogs develops a phobia of dogs.
-ignores cognitive aspects

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6
Q

behavioural approach to treating phobias

Systematic desensitisation

A

-based on classical conditioning
-gradual exposure to stimuli
counter conditioning:learning new association
Reciprocal inhibition:two conflicting emtims csnt co-exist.Relaxed and anxious
1.relaxation techniques
2.anxiety hierarchy
3.desensitisation
EVALUATION:
+Evidence:Gilroy et al followed patients who had SD for spider phobia with 3, 45 min sessions.At 3 and 33 months SD group were less fearful
+can work do a diverse range of patients.EG-people with learning difficulties will find this easier than flood etc.Not as traumatic
-time and cost.Takes a long time and people might not have the time/money to do this

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7
Q

behavioural treatments for phobias

Flooding

A

-immediate exposure to stimulus
-occurs over 1/2 sessions long hours
extinction:phobia goes away as they realise that the phobic stimuli is harmless
EVALUATION:
+cost effective:cheap and quick
-less effective for some types of phobias.social phobias involve cognitive aspects so CBT may be better suited
-treatment isn’t that ethical.Can be really traumatic for some patients

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8
Q

Biological explanation for OCD

Genetic

A

GENETIC
-candidate genes:specific genes are linked to OCD.EG-Gene 9
-polygenic:development of OCD is not 1 but a few
-Aetiologically heterogeneous:one group of genes may cause it one person but another group of genes cause it in another
Evaluation:
+supporting evidence:Nestadt et al:reviewed twin studies+found 68% of MZ twins shared OCD compared to 31% of DZ twins
-too many candidate genes been identified:Taylor identified 230 candidate genes.Less predictive value:likelihood of getting OCD,even if they have a few might not get OCD
-doesn’t account for environmental factors:Cromer et al found that over half of patients with OCD had experienced trauma+OCD was worse in those with multiple traumas.Also supported diathesis stress model (vulnerability)

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9
Q

Biological explanation for OCD

NEURAL

A

-neurotransmitters are responsible for relaying info from one neuron to another
-low levels of serotonin, mood relevant info doesn’t take place.results in mood being effected
-high levels of dopamine
-brain scans have enabled people to compare normal brain patterns with abnormal brain patterns.EG-malfunctioning of temporal lobes:involved with decision making
EVALUATION:
+evidence:Soomro et al found that SSRIs work in reducing symptoms of OCD
-correlation not causation.Just because we issues with certain areas of the brain doesn’t mean it causes OCD
- Co-morbidity:two conditions together.people with OCD usually have depression.Which involves disruption of serotonin levels-may be caused by depression not OCD

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10
Q

Biological approach to treating OCD

drug therapy

A

Drug therapies:SSRIS
aim to increase the levels of serotonin in the synapse
-antidepressant drug,20-60mg 3-4 weeks
-combining SSRIs with CBT is most effective treatment
Evaluation:
+drug therapies are effective:Soomro et al:reviewed 17 studies of comparing SSRIs to Placebos and found that patients had improved when using SSRIs
+drugs are cheap and not disruptive.Compared to other treatments this is cheaper and not time consuming->benefits some patients
-Drugs have side effects->side effects may be worse than actual disorder
-evidence may be bias:Goldacre believes evidence found for drug therapies is bias as it’s funded by the drug companies

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