PVD & Dissection Flashcards

(38 cards)

1
Q

What effect does increase peripheral vascular resistance have on the heart

A

Increases the afterload = increase how much the heart has to work / pump against

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2
Q

What factors increase SVR

A

Atherosclerosis
HTN
Blood Viscosity
vasoconstriction / dilation

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3
Q

What is normal systolic BP

A

120mmHg

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4
Q

Where are baroreceptors located

A

carotids
aorta

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5
Q

What is elevated systolic BP

A

120-129mmHg

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6
Q

What is stage 1 HTN systolic BP

A

130-139mmHg

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7
Q

What is stage 2 HTN systolic BP

A

> 140mmHg

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8
Q

What is stage 1 HTN diastolic pressure

A

80-89mmHg

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9
Q

What is stage 2 HTN diastolic pressure

A

> 90mmHg

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10
Q

How does the body balance out blood pressure

A

baroreceptors and feedback info from those receptors

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11
Q

What do the baroreceptors stimulate with increased BP

A

Increase vagal stimulation which will cause the release of acetylcholine

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12
Q

What receptors are stimulated on the SA and AV nodes when acetylcholine is released

A

M2 = decrease HR, decrease CO and decrease BP

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13
Q

What will be inhibited during times of HTN to decrease the production of catecholamine

A

adrenal medulla

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14
Q

What happens in the kidneys during times of HTN

A

Pressure will increase within the glomerular filtration which will increase Na+/H2O secretion = decrease BP

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15
Q

What is the most common type of hypertension

A

essential

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16
Q

What factors increase HTN

A

sympathetic activation = B1 activation with increase HR = Increase CO = Increase BP

17
Q

Why does Nicotine cause HTN

A

Nicotine causes release of catecholamines

18
Q

Why does obstructive sleep apnea cause HTN

A

hypoxemia and hypercarbia cause catecholamine release

19
Q

What are hypertensive vasculopathies

A

vascular remodeling
endothelial dysfunction
inflammation

20
Q

What is malignant hypertension

21
Q

What is hypertensive emergency indicative of

A

acute end organ damage secondary to elevated BP

22
Q

What is a hypertensive urgency

A

Stable end organ disease

23
Q

What type of hypertension has no end organ damage but has highly elevated BP

A

Severe uncontrolled HTN

24
Q

What are some downstream effects of chronic HTN

A

Ischemic heart diease
AKI/CKD
Encephalopathy
Hypertensive retinopathy
atherosclerosis
SAH/Ruptured aneurysm/lacunar infart

25
Where does a dissection occur in the aorta
Separation of tunica intima and media allowing for an accumulation of blood
26
What are dissections most commonly associated with
HTN Smoking advanced age hyperlipidemia
27
What causes an aortic dissection
a supply/demand mismatch= stiffening of vascular wall = increase stress & decrease compliance, increase risk of endothelial damage = increased risk of dissection
28
What are some risks for dissection
CT disorders bicuspid aortic valve coarctation of aorta
29
What kind of trauma can cause an aortic dissection
acceleration/deceleration injuries
30
How will an aortic dissection present
abrupt onset of chest /abdominal pain described as tearing widened mediastinum pulse/ BP difference in extremities
31
What vessel layers are effected by an aortic aneurysm
all layers
32
What are the causes of aortic aneurysms
CT disorders Trauma Aortits **atherosclerosis
33
What is a ruptured AAA presentation
abdominal / back pain hypotension pulsatile abdominal mass
34
What can chronic venous disease lead to
varicose veins
35
What pressure is considered critical in compartment syndrome
10-30mmHg
36
What is ABI
ankle brachial index *take BP in both places and compare to determine the severity of PVD ankle BP/Brachial BP
37
What might a patient with critical PVD present with
persistent claudication gangrene skin breakdown
38
What is the initial treatment of claudication
walking program **allows for angiogenesis