Quiz #2 Flashcards

(92 cards)

1
Q

Stressor

A

-Event that creates demands
-Causes fear when viewed as threatening

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2
Q

Stress response

A

Person’s reactions to demands

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3
Q

Extraordinary stress and trauma

A

Can play a central role in certain psychological disorders

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4
Q

Where are features of arousal and fear set?

A

The hypothalamus

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5
Q

What 2 important systems are activated when arousal and/or fear set in?

A
  1. Autonomic Nervous System (ANS)
    An extensive network of nerve fibers that connect to the central nervous system (brain and spinal cord) to all other organs of the body
  2. Endocrine system
    A network of glands throughout the body that release hormones
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6
Q

Sympathetic nervous system

A

The nerve fibers of the ANS that quicken the heartbeat and produce other changes experienced as arousal

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7
Q

Parasympathetic nervous system

A

The nerve fibers of the ANS that help return bodily processes to normal

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8
Q

Two pathways by which ANS and endocrine system produce arousal/fear reactions

A
  1. Sympathetic nervous system pathway
  2. Hypothalamic-pituitary-adrenal pathway (hypo. signals the pituitary gland, which stimulates the adrenal cortex to release corticosteroids (stress hormones) into the bloodstream
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9
Q

DSM-5 lists trauma and stressor-related disorders as:

A
  1. Acute stress disorder
  2. PTSD
  3. Dissociate disorders
    -Dissociative amnesia
    -Dissociative fugue
    -DID
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10
Q

Acute stress disorder

A

Symptoms begin within 4 weeks of event, last for less than one month

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11
Q

PTSD

A

Symptoms may begin either shortly after event, or months or years afterward (80% of all cases for acute stress disorder develop into PTSD)

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12
Q

Both acute and PTSD checklist

A

-Person is exposed to a traumatic event—death or threatened death, severe injury, or sexual violation
A person experiences at least one of the following intrusive symptoms:
Repeated, uncontrolled, and distressing memories
Repeated and upsetting trauma-linked dreams
Dissociative experiences such as flashbacks
Significant upset when exposed to trauma-linked cues
Pronounced physical reactions when reminded of the event

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13
Q

What triggers acute disorder and PTSD?

A

Combat
Shell shock; combat fatigue
Psychological distress after combat
Disasters, accidents, and illnesses
Natural and accidental disasters; medical illnesses; epidemics
Earthquakes, floods, tornadoes, fires, airplane crashes, and serious car accidents
Victimization
Sexual assault and rape

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14
Q

Treatment for acute and PTSD

A

General goals
End lingering stress reactions
Gain perspective on painful experiences
Return to constructive living
Treatments
Antidepressant drugs
Cognitive-behavioral therapy
Mindfulness-based techniques
Exposure techniques; prolonged exposure: VIRTUAL REALITY
Eye movement desensitization and reprocessing (EMDR)
Couple or family therapy
Group therapy

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15
Q

Dissociative amnesia

A

Inability to recall important information, usually of an upsetting nature, about one’s life
Memory loss much more extensive than normal forgetting and is not caused by physical factors
Often the amnesia episode is directly triggered by a specific upsetting event
Checklist
Person cannot recall important life-related information, typically traumatic or stressful information. Memory problem is more than simple forgetting.
Leads to significant distress or impairment
Symptoms are not caused by a substance or medical condition

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16
Q

Types of dissociative amnesia

A

Localized: Most common type; loss of all memory of events occurring within a limited period
Selective: Loss of memory for some, but not all, events occurring within a period
Generalized: Loss of memory beginning with an event, but extending back in time; may lose sense of identity; may fail to recognize family and friends
Continuous: Forgetting continues into the future; quite rare in cases of dissociative amnesia

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17
Q

Dissociative Fugue

A

Extreme version of dissociative amnesia
People not only forget their personal identities and details of their past, but also flee to an entirely different location
May be brief or more severe
For some, fugue is brief—a matter of hours or days—and ends suddenly.
For others, the fugue is more severe: People may travel far from home, take a new name and establish new relationships, and even enter a new line of work; some display new personality characteristics.
The majority of people regain most or all of their memories and never have a recurrence.

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18
Q

DID

A

Two or more distinct personalities (subpersonalities) develop
Each has unique set of memories, behaviors, thoughts, and emotions
Sudden movement from one subpersonality to another (switching) : triggered by stress
Women diagnosed three times more often than men
At any given time, one of the subpersonalities dominates the person’s functioning.
Usually one of these subpersonalities—called the primary, or host, personality—appears more often than the others.
The transition from one subpersonality to the next (“switching”) is usually sudden and may be dramatic.
Most cases are first diagnosed in late adolescence or early adulthood.
Symptoms generally begin in childhood after episodes of abuse.
Typical onset is before age 5.

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19
Q

Checklist for DID

A

Person experiences a disruption to his or her identity, as reflected by at least two separate personality states or experiences of possession
Person repeatedly experiences memory gaps regarding daily events, key personal information, or traumatic events, beyond ordinary forgetting
Leads to significant distress or impairment
Symptoms are not caused by a substance or medical condition

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20
Q

DID: Mutually amnesic relationships

A

Subpersonalities have no awareness of one another.

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21
Q

DID: Mutually cognizant patterns

A

Each subpersonality is well aware of the rest.

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22
Q

DID: One-way amnesic relationships

A

Most common pattern; some personalities are aware of others, but the awareness is not mutual. Those who are aware (“co-conscious subpersonalities”) are “quiet observers.”

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23
Q

DID: How do subpersonalities differ?

A

Subpersonalities may differ in features as basic as age, sex, race, and family history.
It is not uncommon for different subpersonalities to have different abilities, including being able to drive, speak a foreign language, or play an instrument.
Subpersonalities may have physiological differences, such as differences in autonomic nervous system activity, blood pressure levels, and allergies.

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24
Q

Depersonalization-derealization disorder

A

DSM-5 categorizes this as a dissociative disorder, but not as one characterized by the memory difficulties found in the other dissociative disorders
Central symptom is persistent and recurrent episodes of depersonalization and/or derealization

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25
Depersonalization
Feeling separation from own body Seeing self from inside out; doubling Having mechanical, dreamlike, dizzy feelings Awareness that perceptions are distorted
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Derealization
Feeling external world is unreal and strange Changing object shape or size May see other people as robots
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Depression
Low, sad state marked by significant levels of sadness, lack of energy, low self-worth, guilt, or related symptoms
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Mania
State or episode of euphoria or frenzied activity in which people may have an exaggerated belief that the world is theirs for the taking
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Depressive disorders
Group of disorders marked by unipolar depression
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Unipolar depression
Depression without a history of mania
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Bipolar disorder
Disorder marked by alternating or intermixed periods of mania and depression
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Learned helplessness
Depression occurs when people believe they have no control over life’s reinforcements and assume responsibility for this helpless state
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Attribution-helplessness theory
Modified learned helplessness theory Internal (global and stable) attribution of present lack of control --> feel helpless to prevent future negative outcomes --> depression
34
Behavioral activation
therapists work systematically to increase number of constructive and rewarding activities and events in a client's life
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Symptoms of mania
typically experience dramatic and inappropriate rises in mood. Emotional symptoms Active, powerful emotions in search of an outlet Motivational symptoms Need for constant excitement, involvement, companionship Behavioral symptoms Very active—move quickly; talk loudly or rapidly; flamboyance is not uncommon Cognitive symptoms Show poor judgment or planning; may have trouble remaining coherent or in touch with reality Physical symptoms High energy level—often in the presence of little or no rest
36
Manic episode
For 1 week or more, person displays a continually abnormal, inflated, unrestrained, or irritable mood as well as continually heightened energy or activity, for most of every day **Flamboyance: flashy clothes, giving out large sums of money, involved in dangerous activities
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Bipolar I disorder
Occurrence of a manic episode Hypomanic or major depressive episodes may precede or follow the manic episode
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Bipolar II disorder
Presence or history of major depressive episode(s) Presence or history of hypomanic episode(s) No history of a manic episode
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Hypomania
Milder form of mania : Found in BiPolar II Diagnosis Found to also make the week before a menstrual cycle more difficult for woman
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Hypomania symptoms
higher, happier mood than usual irritability or rude behavior -- agitation overconfident higher activity or energy levels than usual without a clear cause powerful feeling of physical and mental wellbeing more social and talkative than usual stronger desire for sex than usual need to sleep less than usual
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Cyclothymic Disorder
Numerous periods of hypomanic symptoms and mild depression symptoms Symptoms continue for two or more years, with normal moods for days or weeks in between May evolve into bipolar I or bipolar II
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Bipolar disorder treatment: Lithium
Very effective in treating bipolar disorders and mania Determining the correct dosage for a given patient is a delicate process Too low = no effect Too high = lithium intoxication (poisoning)
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Bipolar disorder treatment: Other mood stabilizers
Some patients respond better to other drugs or to combinations of drugs
44
Suicide
Self-inflicted death in which one makes intentional, direct, and conscious effort to end one's life. One of the leading causes of death in the world; not officially classified as a mental disorder in DSM-5
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Parasuicide
A suicide attempt that does not result in death
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Death seekers
Clearly intend to end their lives
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Death initiators
Intend to end their lives b/c they believe that the process of death is already under way
48
Death ignorers
Do not believe that their self-inflicted death will mean the end of their existence - believe trading present life for a better or happier existence - cult, children, delusional
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Death darers
Have ambivalent feelings about death and show this in the act itself
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Sub-intentional death
Indirect, covert, partial, or unconscious roles in their own death Drug, alcohol, tobacco, physical fighting, medication mismanagement, self injury behavior
51
Self-injury or mutilation (self-harm)
Purposely injuring self by skin cutting, burning, etc.
52
Stressful events/situations: Immediate stressors
Loss of loved one through death, divorce, or rejection Loss of job or significant financial loss Natural disasters
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Stressful events/situations: Long-term stressors
Social isolation Serious illness Abusive or repressive environment Occupational stress
54
Factitious Disorder (munchausen syndrome)
Self-imposed: false creation of physical or psychological symptoms, or deceptive production of injury or disease, even without external rewards Presentation of oneself as ill, damaged, or hurt
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Munchausen syndrome by proxy
Imposed on another False creation of physical or psychological symptoms, or deceptive production of injury or disease, in another person, even without external rewards Presentation of another person (victim) as ill, damaged, or hurt
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Malingering
intentionally feigning illness to achieve some external gain, such as financial compensation or time off from work
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Factitious disorder characteristics
Poor social support or relationships and little family life Extensive medical treatment in childhood Grudge against medical profession Employment as nurse, lab technician, or aide
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Somatic symptom disorder
At least one upsetting or repeatedly disruptive physical (somatic) symptom An unreasonable number of thoughts, feelings, and behaviors regarding this symptom Physical symptoms usually continue to some degree for more than 6 months
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Somatic symptom disorder characteristics
Repeatedly check body for abnormalities, frequent health care visits that don't relieve your concerns or that make them worse, fearing symptoms are serious when there is no evidence
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Psychophysiological disorders
Affected persons have a medical condition Psychological factors negatively affect the medical condition Affect the course of the medical condition Provide obstacles to the treatment of medical condition Pose new health risks Trigger or worsen the medical condition
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Typical psychophysiological disorders:
Ulcers Asthma Insomnia Chronic headaches Migraine headaches Hypertension
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What factors contribute to psychophysiological disorders?
Biological: defects in ANS, other weak body systems Psychological: certain needs, attitudes, emotions, or coping styles cause people to overreact repeatedly to stressors
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Anorexia Nervosa
Individual purposely takes in too little nourishment, resulting in body weight that is very low and below that of other people of similar age and gender Individual is very fearful of gaining weight, or repeatedly seeks to prevent weight gain despite low body weight Individual has a distorted body perception, places inappropriate emphasis on weight or shape in judgments of herself or himself fails to appreciate the serious implications of her or his low weight
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Restricting type of anorexia
Lose weight by cutting out sweets and fattening snacks, eventually eliminating nearly all food Show almost no variability in diet
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Binge-eating/purging type
Lose weight by forcing themselves to vomit after meals or by abusing laxatives or diuretics May engage in eating binges
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Anorexia cont.
Key goal is becoming thin --Thinking is distorted Driving motivation is fear Preoccupation with food occurs Usually have a low opinion of their body shape Potential psychological problems Medical problems
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Bullimia nervosa
First repeated binge eating episodes Then repeated performance of ill-advised compensatory behaviors (e.g., forced vomiting) to prevent weight gain Symptoms take place at least weekly for a period of 3 months Inappropriate influence of weight and shape on appraisal of oneself
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Binge
an episode of uncontrollable eating during which a person ingests a very large quantity of food 1–30 episodes per week and 2,000–3,400 calories per episode Followed by extreme self-blame, shame, guilt, depression, and weight gain fear Often carried out in secret
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Compensatory behaviors
Disorder is also characterized by inappropriate compensatory behaviors Vomiting; purging Laxative or diuretics use Compensatory behaviors effectiveness Some temporary positive effects Caloric bingeing effects not undone
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Binge eating disorder
Recurrent binge eating episodes that include at least three of these features: Unusually fast eating Absence of hunger Uncomfortable fullness Secret eating due to sense of shame Subsequent feelings of self-disgust, depression, or severe guilt Binge-eating episodes take place at least weekly over the course of 3 months Absence of excessive compensatory behaviors
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Biological factors for eating disorders
-Suspected that certain genes may leave some people particularly susceptible to eating disorders -Relatives of people w/ eating disorders are up to 6 times more likely than other people to develop the disorders themselves -If one twin has a disorder, the other twin often develops it too -Hypothalamus: brain structure that helps regulate bodily functions; including eating/hunger Lateral hypothalamus (LH): brain region produces hunger when activated Ventromedial hypothalamus (VMH): depresses hunger when activated
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Drug
Any substance other than food affecting our bodies or minds, including alcohol, tobacco, and caffeine
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Substance intoxication
Cluster of changes in behavior, emotion, or thought caused by substances (DSM-5)
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Substance use disorders
Maladaptive behavior patterns and reactions caused by repeated substance use Presence of at least 2 of the following symptoms within a 1-year period Substance is often taken in larger amounts or over a longer period than intended Unsuccessful efforts or persistent desire to reduce or control substance use Much time spent trying to obtain, use, or recover from the effects of substance use Failure to fulfill major role oblig. at work, school, or home as a result of repeated substance use
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Tolerance
Need for increasing doses of substances to produce desired effect
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Withdrawal
Unpleasant and sometimes dangerous symptoms occurring with drug stopping or cutting back
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Group most likely to have substance use disorders?
American Indians
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Opioids
Include natural (opium, heroin, morphine, codeine) and synthetic (methadone) compounds Opioids create these effects by depressing the CNS producing pleasurable and calming feelings Injection seems to be the most common method of use, although other techniques have been increasing in recent years. An injection quickly brings on a “rush”—a spasm of warmth and ecstasy that is sometimes compared with orgasm. This spasm is followed by hours of pleasurable feelings (called a “high” or “nod”).
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Dangers of opioid use
Most immediate danger is overdose Opioid overdose closes down the respiratory center in the brain, paralyzing breathing and causing death. Ignorance of tolerance Getting impure drugs Infection from dirty needles and other equipment
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Stimulants
increase the activity of the central nervous system (CNS) Most common stimulants Cocaine Amphetamines Caffeine
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Cocaine
Most powerful known natural stimulant Produces a euphoric rush of well-being Increases the supply of dopamine at key neurons throughout the brain as well as norepinephrine and serotonin levels Can be snorted, injected, or smoked High doses of cocaine can produce, cocaine intoxication symptoms include mania, paranoia, and impaired judgment. Cocaine-induced psychotic disorder: hallucinations and/or delusions As the stimulant effects of the drug subside, the user experiences a depression-like letdown, popularly called “crashing.”
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Freebasing Cocaine
almost 100% pure cocaine in a rock form.
82
Crack cocaine
the only difference between cocaine and crack is crack has its hydrochloride removed, increasing the melting point and making the drug smokable
83
Hallucinogens (psychedelic drugs)
Produce powerful changes primarily in sensory perception (trips) Natural hallucinogens Lysergic acid diethylamide (LSD) Mescaline Psilocybin MDMA (Ecstasy)
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LSD (lysergic acid diethylamide)
One of most powerful hallucinogens derived from ergot alkaloids (fungus) Brings on a state of hallucinogen intoxication (hallucinosis) Increased / altered sensory perception, psychological changes, and physical symptoms Hallucinations and/or synesthesia Effects wear off in about 6 hours Self-injury, bad trips, flashbacks Users may experience a “bad trip”—the experience of enormous unpleasant perceptual, emotional, and behavioral reactions. Another danger is the risk of “flashbacks,” which can occur days or months after last drug use
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MDMA
Ecstasy, Molly Stimulant produces hallucinogenic effects; provides an energy boost and strong feelings of connectedness. Dangers Immediate psychological problems, cognitive impairment, unpleasant and potential dangerous physical symptoms
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Cannabis
Produced from varieties of hemp plants Hashish: Solidified resin of the cannabis plant Marijuana: Mixture of buds, crushed leaves, and flowering tops Major active ingredient: tetrahydrocannabinol (THC) Potency influenced by environmental conditions When smoked, produces a mixture of hallucinogenic, depressant, and stimulant effects, known as cannabis intoxication Most of the effects last 2 to 6 hours At low doses, the cannabis user feels joy and relaxation. May become anxious, suspicious, or irritated This overall “high” is technically called cannabis intoxication. At high doses, cannabis produces odd visual experiences, changes in body image, and hallucinations.
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Polysubstance use (combinations of substances)
Taking more than one drug at a time
88
Cross-tolerance
Can reduce the symptoms of withdrawal from one drug by taking the other
89
Synergistic effects:
create greater effect when same type of drugs are combined
90
BAC (Blood alcohol concentration)
Extent of the effect of ethyl alcohol is determined by its concentration in the blood
91
Levels of BAC impairment
BAC = 0.06: Relaxation and comfort BAC = 0.09: Intoxication BAC > 0.55: Death Most people lose consciousness before they can drink this much.