Random Clinical Flashcards

(34 cards)

1
Q

Causes of raised JVP (>4cm)

A
Heart Failure
Cardiac Temponade
Restrictive pericarditis 
Fluid overload e.g. renal disease
Superior vena cava obstruction
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2
Q

Interpret the following CSF results. Given are the tests and normal ranges.

Appearance (clear), White cells (<5), predominant white cell (all mononuclear), protein (0.2-0.4), glucose (>60%)

a) 
cloudy &amp; viscous
900
mononuclear 
5
 <30%

b)

  • Clear
  • 100
  • Mononuclear
  • 0.6
  • > 60%

c)

  • Cloudy and Turbid
  • 5,000
  • Polymorphs
  • 8
  • 35%
d) 
Fibrin web 
400
Mononuclear 
0.4
30%
A

a) Is TB meningitis
- WCC Less high than normal bacterial men but raised 50-1000
- High protein
- Low glucose

b) Viral
Clear, raised white cells (10-1000),
- normal/high protein
- normal glucose

c) bacterial: cloudy & think, very high white cell count, polymorphs and very high protein

d)
Fibrin web, high WCC (not as high as bacterial)
- Low protein
- Normal/low protein

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3
Q

Bacteria in Newborns

A

group B strep
E. coli
listeria monocytogenes

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4
Q

Bacteria in young children

A

N. meningitidis
Strep pneumonia
Haemophilus influenza

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5
Q

Bacteria in teens/adults

A

N. meningitides

Strep pneumonia

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6
Q

Virus

A

VZV, enterovirus, HSV, HIV, mumps

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7
Q

Fungal

A

Cryptococcus neoformans

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8
Q

Additional Tests after LP

A
  • culture: grow bacteria
  • PCR for virus
  • Electrophoresis: oligoclonal bands (MS)
  • Acid-fast stain TB
  • Xanthochromia/bilirubin: subarachnoid
  • Cytology: malignant cells
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9
Q

Subarachnoid Haemorrhage in LP

A

Usually blood stained, normal white blood cells,

Red cells high, normal or high protein, glucose normal or high

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10
Q

Treatment for TB

A

RIPE

Rifampicin: 600-900mg
Isoniazid: 15mg/kg
Pyrazinamide: 2.5g
Ethambutol 30mg/kg

All PO 3 times a week

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11
Q

What cause of large bowel obstruction appears as a large coffee bean on AXR?

A

Sigmoid Volvulus

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12
Q

What width should the large and small bowel be on AXR

A

Large= 5cm (except the caecum which is 8cm)

Small= 3cm

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13
Q

Name 3 ways of identifying pneumoperitoneum on XR

A

CXR: air under diaphragm (gastric air bubble under left is normal)
Rigler’s sign: see both sides of bowel wall (normally only inner wall is viable) contrast of air inside bowel
Football sign: round area of air, mostly found in neonates.

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14
Q

Causes of perforation

A
obstruction
gallstone disease
inflammatory conditions (Crohn's)
Appendicitis 
Trauma
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15
Q

What does thumb printing of the bowel on AXR suggest?

A

Oedema of the bowel wall, occurs in inflammatory bowel disease and ischaemic colitis

If you see an enlarged colon, question if it inflammatory bowel disease-related toxic megacolon (particularly UC)

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16
Q

Talk through how to interperate an abdo x ray

A
  1. Projection
  2. Patient details
  3. Technical Adequacy (entire abdo)
  4. Obvious abnormalities
  5. Systematic review
    - Foreign bodies
    - Assess bowel (large then small, size, abnormalities, extra or intraluminal content (air is black, faeces are mottled grey
    - Liver, spleen, gallbladder (size, gallstones (most radiolucent))
    - Abdominal aorta (calcifications, aneurysm, if suspecting dissection look for psoas muscle shadow (normally present)
    - Kidney stones
    - Bones (pelvic & hip& spine)
  6. Summary: key findings, diagnosis, management plan * differentials
17
Q

Example of describing AXR for small bowel obstruction in OSCE.

A

This is an AP supine abdominal radiograph. From the identifying markers, I would like ensure it is the correct patient and check the date. A view of the entire abdomen are included in this film.

There are multiple loops of small bowel obstruction. It is the small bowel due to the central distribution & valvulae commiventes.

There is no evidence of hernia or previous surgery. There is no evidence of extraluminal air. The abdominal aorta is not visible and the bladder seems a normal size. There are no apparent bony abnormalities.

In summary, this is an abdominal radiograph showing small bowel obstruction with no evidence of perforation. I would like to arrange an erect chest X-ray to look for free air under the diaphragm.

Differentials for the cause of small bowel obstruction would be adhesions, neoplasia, incarcerated hernias and strictures.

18
Q

Management of small bowel obstruction

A
  • NBM
  • Drip & Suck IV access for IV fluids & NG tube
  • Bloods: FBC, U&E, LFT, CRP, clotting, group & save (prepare for theatre)
  • Erect CXR
  • Urgent surgical review for further imaging to surgical intervention
19
Q

Causes of blood on urinalysis

A

Haematuria or in women form menstrual peroid

20
Q

Ketones

A

↑DKA & starvation

NB:↑Glucose, metabolic acidosis & ↑ketones for DKA
If no ketones with high glucose = HONK hyperosmolar non ketotic state

  • ask patient if they are deliberately trying to lose weight & when they last ate
21
Q

Nitrites & leukocytes

A

↑ suggest bacterial infection

leukocytes are non-specific
Nitrites only produced by gram negative bacteria. Some infections will be negative.

22
Q

protein

A

↑renovascular, glomerular, tubulointersitial disease, pre-eclampsia & hypertension

Can be benign (exercise, postal)

In nephrotic syndrome (low albumin, oedema, raised cholesterol & proteinuria) must measure protein loss in 24 hours

23
Q

Glucose

A

↑diabetes: especially HONK or DKA

can be raised in pregnancy

24
Q

Specific gravity

A

Concentrating and diluting status of the kidney
↑dehydration, HF, liver failure, syndrome of inappropriate ADH (SIADH)
↓diabetes insipidus & ↑fluid intake

25
Urine pH
useful for UTI & calculi (stones) - UTI symtoms + alkaline = infection with urea splitting organism - Acidic calculi made of uric acid & cystine. Alkalinisation of the urine is an important part of the management of these patients (potassium citrate)
26
Management of renal stones
- Diclofenac (NSAID) - IV fluids - Antibotics (cefuroxime) Refer to urology
27
What other test can you do on urine
pregnancy test microscopy, culture & sensitivity (UTI) Protein electrophoresis test for microalbuminuria (DM)
28
ABG interpretation
1. Ask about patients current clinical status. On oxygen, two 2. Is the patient hypoxia - >10kpa on air - 10kpa< that FiO2 - <10kpa= hypoxic <8kpa =severely hypoxic- - Type 1 or 2 RF: look at pCo2. If >6kpa= Type 2 3. pH. Metabolic (abnormal HCO3-)or Resp (abnormal CO2)?? 4. PaCO2: does it fit with pH (high=lowpH) If doesn't make sense (↓CO ↓PH) then think metabolic - compensation 5. HCO3- (mops H+): low pH= low (acidosis) 6. Base Excess: - high= ↑level of HCO3, metabolic alkalosis or compensated respiratory acidosis -Low= metabolic acidosis or compensated respiratory alkalosis
29
Respiratory acidosis
inadequate ventilation = CO2 retention ↓pH ↑ CO2 Respiratory depression (e.g. opiates) Guillain-Barre – paralysis leads to an inability to adequately ventilate Asthma Chronic obstructive pulmonary disease (COPD) Iatrogenic (incorrect mechanical ventilation settings)
30
Respiratory Alkalosis
hyperventilation ↑pH ↓CO2 Anxiety – often referred to as a panic attack Pain – causing increased respiratory rate Hypoxia – resulting in increased alveolar ventilation in an attempt to compensate Pulmonary embolism Pneumothorax Iatrogenic (excessive mechanical ventilation)
31
Metabolic Acidosis
1. Increased acid production or ingestion 2. Decreased acid excretion/GI or renal HCO3- loss ↓pH ↓HCO3- ↓BE
32
Anion Gap
= Na+ - (Cl-+HCO3-) ↑ anion gap = ↑acid production or ingestion ``` Diabetic ketoacidosis (↑ production) Lactic acidosis (↑ production) Aspirin overdose (ingestion of acid) ``` ↓anion gap= ↓acid excretion or loss of HCO3- GI loss of HCO3— diarrhoea, ileostomy, proximal colostomy Renal tubular acidosis (retaining H+) Addison’s disease (retaining H+)
33
Metabolic alkalosis
↓hydrogen conc.
34
Complications of chronic alcoholism
``` Hypertension Wernickes encephalopathy Alcoholic Dementia Liver cirrhosis pernicious anaemia malnutrition vit b deficiency hepatic carcinoma ```