Red and Blue Lesions Flashcards Preview

Oral Pathology > Red and Blue Lesions > Flashcards

Flashcards in Red and Blue Lesions Deck (40):
1

Reasons that lesions appear red

Dilation of blood vessels
Increase blood supply
Hemorrhage soft tissues
Thin epithelium
Epithelial erosion

2

Diascopy positive lesions

Hemangioma
Sturge-Weber syndrome
Hereditary hemorrhagic telamgiectasia
CREST syndrome

3

Diascopy negative lesions

Sub mucosal hemorrhage
Thrombocytopenia
Infectious mononucleosis

4

Another name for congenital hemangioma

Strawberry nevus

5

Causes hemangioma versus vascular malformation

He

6

What is the appearance of hemangioma of bone

Multi

7

Pathogenesis of hemangioma

abnormal endothelial cells

8

Pathogenesis of vascular formations

abnormal blood vessel development

9

T/F hemangioma has a bruit present with it

FALSE

10

T/F hemangiomas do not involute spontaneously

FALSE

11

Describe how hemangioma of bone would look

Multilocular, sunburst pattern
Delicate trabeculae
Root resorption
Cortical expansion

12

What areas of the body are affected by Sturge-Weber?

The brain, face, and intraorally

13

What nerve us affected by Sturge-Weber

Trigeminal nerve with facial lesions along the nerve
Usually UNILATERAL

14

Neurological effects of S-W Syndrome

Mental retardation, hemiparesis, and seizures
Intracranial calcification of leptomeningies

15

Intral oral lesions of S-W Syndrome

Very red
Ipsilateral oral mucosa involvement

16

Cause of Hereditary Hemorrhagic Telangiectasia (HHT)

Abnormal dilation of terminal vessels

17

Presentation of HHT

Intranasal EPITAXIS
Telangiectasa of vermillion, tongue, and buccal mucosa

18

What are varix?

Abnormally dilated veins

19

Where are varicosities multiple and solitary?

Multiple: ventral and lateral tongue
Solitary: vermilion and bucal mucosa

20

Etiology of angina bullosa hemorrhagica

Trauma
Not immunologically related

21

Pathogenesis of pyogenic granuloma

Reactive hyperplasia of vascularized granulation tissue

22

Clinical presentation of pyogenic granuloma

Usually solitary, circumscribed red nodule

23

Usual populations with Pyo. G

Women
During pregnancy
During hormonal changes (puburty)

24

Locations of pyogenic granuloma

Gingiva
Tongue
Labial mucosa

25

DD of PG

The "P's"
Peripheral giant cell granuloma
Peripheral ossifying fibroma
Metestatic tumors

26

Tx of PG

Remove the cause (local plaque or calculus)
Surgical excision
Occasionally lesions recur

27

Cause of Periph. GCG

Trauma or irritation

28

Location of Periph. GCG

Gingiva anterior to the first molar. Exclusively on the gingiva

29

How are PyoG and Periph GCG differentiated

The presence of giant cells on PGCG

30

How would an erythroplakia look intraorally and who would it be on?

Well-defined macule or plaque on the floor of mouth, tongue or soft palate
Older men (50-70) usually have it

31

DD of erythroplakia

Nonspecific mucositis
Candidiasis
Vascular lesion

32

Histopathology of erythroplakia

40% will show sever dysplasia
50% will be SCC

33

Tx of erythroplakia

Biopsy to confirm it.
Excise it.
Long term follow up

34

Name one type of intravascular neoplasm

Kaposi's sarcoma

35

Tx of KS

Surgical excision, low-dose radiation

36

DD of KS

Erythroplakia
Hemangioma
Melanoma
Pyogenic granuloma

37

Type of Anemias

Plummer-Vinson syndrome
Pernicious anemia

38

Plummer-Vinson syndrome cause

Iron deficiency

39

Plummer-Vinson syndrome effects

Dysphagia
Erythema and papillary atrophy
Angular cheilitis
BALD TONGUE

40

Perniciious anema cause

Intrinsic factor and Vitamin B12 deficiency