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RISK AND RESIL- Week 7-11 test Flashcards

(109 cards)

1
Q

Neurodevelopmental disorders

A

A group of disorders that arise during the developmental period, before adulthood.

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2
Q

Why do neurodevelopmental disorders occur?

A

Improper developmental of the central nervous system, as a result of interplay between enviromental and genetic factors.

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3
Q

Autism Spectrum Disorder

A

A neurodevelopmental condition. Effects cognitive, sensory and social processing. Present from early childhood.

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4
Q

Three functional levels of autism

A

Level 1: requiring support (difficulty initiating social interactions, organsiation and planning issues)

Level 2: requiring substantial support (social interactions limited to narrow special interests, repetitive behaviours)

Level 3: Requiring very substantial support (severe verbal deficits in verbal and non verbal communication, distress/difficulty changing actions)

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5
Q

Autism Epidemiology

A

Us prevalence: 1/54
NZ identification: 1/102
Gender: 3:1 (m:f)

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6
Q

Why is ASD presented more in males than females?

A

Girls more socially aware, masking and camouflaging and diagnostic bias

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7
Q

Neuro correlations with ASD

A

Regions including cerebral cortex, limbic system, and cerebellum are all affected.

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8
Q

DSM-5 Diagnostic criteria for ASD

A

A: Persistent deficits in social communication and social interaction across many contexts.

A1: Deficits in social emotional reciprocity (monotone voice, initiating conversations, back and fourth convo)

A2: Deficits in nonverbal communicative behaviours used for social interactions (difficulty in non verbal communication)

A3: Deficits in developing, maintaining and understanding relationships

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9
Q

Theory of Mind (ToM)

A

The cognitive ability to understand that others have thoughts, beliefs and, desired and intentions that differ from there own.

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10
Q

At what age does theory of mind typically develop?

A

Around age 3 to 4

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11
Q

What are examples of theory of mind challenges in ASD?

A
  • Difficulty understanding sarcasm or irony.

-Trouble with social cues like body language and tone

-Challenges with perspective taking

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12
Q

How is ToM assessed?

A
  • False belief tasks
    -Sally and Anne scenario
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13
Q

What are the DSM-5 core diagnostic criteria for ASD?

A

-Persistent deficits in social communication and interaction
-Restricted, repetitve patterns of behaviours, interests, or actitivities
-Symptoms present in early developmental period
-Symptoms cause significant impairment
-Not better explained by intellectual disability alone

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14
Q

What are the example of restricted/repetitive behaviours in ASD?

A

-Repetitive movements
-Inflexible routines
-Intense interests
-Unusual sensory responses

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15
Q

What is sensory processing disorder in ASD?

A

Atypical responses to sensory input (e.g., hyper- or hypo- sensitivity to sounds, lights, textures)

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16
Q

Common comorbidities with ASD?

A

Intellectual disability

Language disorders

ADHD

Anxiety and depression

Specific learning disorders

Suicide risk

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17
Q

When can ASD be diagnosed?

A

As early as 1 year old, based on developmental milestones such as smiling, babbling, and gesture use.

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18
Q

What are some early signs of ASD?

A

No joyful expressions by 6 months

No babbling by 12 months

No pointing or showing by 12 months

No words by 16 months

Loss of skills at any age

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19
Q

What does the “development and course” of ASD look like?

A

Lifelong difficulties

Individuals often develop coping strategies over time

May mask or camouflage symptoms

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20
Q

What is the impact of ASD diagnosis?

A

Diagnosis helps identify impairments

Important to recognize strengths and adaptive traits

Can help tailor support and interventions

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21
Q

What is the best practice for ASD assessment?

A

Early assessment and intervention

Conducted by trained professionals (e.g., psychologists, pediatricians)

Interdisciplinary approach

Across multiple settings

Uses interviews, observations, developmental history

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22
Q

What is trauma in children?

A

Trauma is the emotional, psychological, and physiological residue from stressful or threatening experiences. It can be from single or repeated events.

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23
Q

What are the three E’s of trauma?

A

Event(s), Experience of event(s), and Effect. Trauma is defined by what happened, how it was experienced, and its lasting effects.

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24
Q

What is the DSM-5 criteria A for PTSD?

A

Exposure to death, serious injury, or sexual violence via direct experience, witnessing, learning of a close person’s trauma, or repeated exposure to details.

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25
What is complex PTSD?
It includes PTSD symptoms plus difficulties in emotional regulation, negative self-beliefs, and trouble with relationships. Not a separate DSM diagnosis.
26
What is reactive attachment Disorder (RAD)?
A condition where children fail to form healthy attachments due to early neglect or mistreatment. Diagnosed before age 5, after age 9 months.
27
What is the ACE study?
A landmark study linking childhood adversity (abuse, neglect, household dysfunction) to long-term health outcomes like heart disease and depression.
28
What is polyvictimization?
Experiencing multiple different types of trauma, which has greater impact than repeated instances of one type.
29
What are common types of child maltreatment?
Physical abuse, sexual abuse, emotional abuse, neglect, exposure to domestic violence, and bullying.
30
What are signs of PTSD in children?
Nightmares, physical symptoms (headaches, stomach aches), traumatic play, reenactments, and emotional withdrawal.
31
What are protective factors against trauma?
Secure attachment, intelligence, stable home, prosocial relationships, and safe neighborhood during childhood.
32
What is TF-CBT?
Trauma-Focused Cognitive Behavioral Therapy. It combines CBT with family support to address trauma through coping skills, education, and exposure.
33
What is ADHD?
A neurodevelopmental disorder characterized by inattention, hyperactivity, and impulsivity that impacts daily functioning and well-being.
34
What are the subtypes of ADHD?
ADHD-I (Inattentive), ADHD-H (Hyperactive/Impulsive), ADHD-C (Combined type).
35
What are the key characteristics of ADHD-I?
Often seen as spacey or apathetic, commonly mistaken for anxiety or mood disorder, more prevalent in girls and individuals with ASD.
36
What are key characteristics of ADHD-H?
Includes obvious hyperactivity and impulsiveness, more likely to be diagnosed, involves impaired response inhibition.
37
How common is ADHD?
Globally ~7.6% in children and 5.6% in teens. More diagnosed in boys during childhood, but levels out in adulthood.
38
Why is ADHD underdiagnosed in girls?
Girls may present less obvious symptoms and are often diagnosed only after secondary issues appear.
39
Can ADHD persist into adulthood?
Yes. Symptoms may change form but persist. Adult diagnosis often follows missed childhood signs due to masking or misunderstanding.
40
What causes ADHD?
It is highly heritable (60–70%). Involves brain structure and function differences, and environmental risk factors like prenatal exposure or trauma.
41
What are common comorbidities with ADHD?
Anxiety, mood disorders, conduct disorder, ASD, learning disorders, substance use disorders.
42
What are the treatment options for ADHD?
Stimulants (e.g., Ritalin, Vyvanse), non-stimulants (e.g., Atomoxetine), and non-pharmacological interventions (e.g., behavioral therapy).
43
What is Barkley's theory of ADHD?
ADHD is a delay in self-regulation and executive functioning by about 30%, not just a difference in attention span.
44
What executive function challenges are linked with ADHD?
Issues with planning, organizing, time management, remembering instructions, and shifting attention.
45
What is emotional dysregulation in ADHD?
Quick emotional reactions, difficulty calming down, often misdiagnosed as mood disorder; not currently in the DSM.
46
What are the sleep challenges in ADHD?
Up to 80% of adults with ADHD have sleep issues, which exacerbate ADHD symptoms.
47
How is ADHD portrayed in media?
Social media, especially TikTok, increases awareness but also risks overgeneralization and self-diagnosis. Accurate representation is vital.
48
What is oppositional disorder (ODD)?
ODD involves persistent defiant, disobedient, and hostile behavior toward authority
49
What is Conduct disorder (CD)?
involves repetitive severe antisocial and aggressive behaviors.
50
What are common symptoms of ODD?
Loses temper, easily annoyed, argumentative, defies rules, deliberately annoys others, blames others, spiteful/vindictive (DSM-5 criteria: 4+ for 6 months).
51
What is the prevalence of ODD and CD?
Globally, ODD is ~3.6% and CD is ~2.1%. In NZ, rates among Māori 16–18 years old are 11.3% vs 4% for non-Māori.
52
How do aggression types differ?
Physical (hitting), verbal (yelling), and relational (exclusion); Proactive (planned) vs Reactive (impulsive) aggression.
53
What is the difference between childhood-onset and adolescent-onset CD?
Childhood-onset (before 10, more boys, severe, persistent); Adolescent-onset (more balanced gender, less severe, better prognosis).
54
What are callous-unemotional (CU) traits?
Low empathy, restricted affect, interpersonal callousness; associated with severe, stable antisocial behavior.
55
What is Patterson’s Coercion Theory?
Antisocial behavior develops through coercive parent-child interactions reinforced by negative patterns.
56
What are Rothbart's temperament dimensions?
Reactivity (arousal, intensity) and Self-Regulation (emotional control, adaptability, persistence).
57
How does social information processing relate to aggression?
Aggressive children misinterpret social cues, assume hostile intent, generate fewer solutions, and favor aggressive responses (Crick & Dodge model).
58
What are common comorbidities with ODD/CD?
ADHD, anxiety disorders, OCD, depression, autism.
59
Are ODD and CD seperate conditions?
Yes. While CD can follow ODD, many with CD did not have prior ODD, and most with ODD do not develop CD.
60
What are effective treatments for CD and ODD?
Early, intensive, and tailored interventions; focus on parenting, social skills training, and emotion regulation; particularly important for CU traits.
61
What are the core symptoms of depression in children?
Persistent sadness, loss of enjoyment, and irritability.
62
What is the average episode length and recurrence rate of depression?
Average episode is 7-9 months; 40% recur within 2 years, 60% by adulthood.
63
What are common symptoms of depression in teens?
Anhedonia, hopelessness, sleep/appetite changes, suicidal ideation, drug use.
64
What are the key risk factors for childhood depression?
Parental depression, family conflict, low SES, abuse, chronic stress.
65
How does suicide relate to depression in adolescents?
60% of depressed youth have suicidal thoughts; 30% attempt; girls more likely than boys.
66
What distinguishes fear from anxiety?
Fear is a response to immediate threat; anxiety is anticipation of future threat.
67
What are the three response systems in anxiety?
Physical (e.g. increased heart rate), Cognitive (e.g. worry), and Behavioural (e.g. avoidance).
68
What is seperation anxiety disorder (SAD)?
Excessive anxiety about separation from caregivers, common in young children.
69
What is specific phobia?
Irrational fear of a specific object or situation lasting 6+ months, causing impairment.
70
What is Social Anxiety Disorder?
Fear of social situations and performance, with onset in adolescence; common in girls.
71
How do genetics influence anxiety?
About 1/3 of childhood anxiety symptoms are genetic; linked to serotonin/dopamine systems.
72
What is resilience in human development?
Resilience refers to factors that protect individuals from developing psychopathology despite the presence of risk factors.
73
What is a risk factor in psychology?
Any condition or circumstance that increases the likelihood of developing psychopathology.
74
What is a vulnerability factor?
A factor that intensifies an indivdual's response to risk.
75
What is a protective factor?
A characteristic or condition that reduces the likelihood of negative outcomes despite risk.
76
What does positive psychology focus on?
Optimal human functioning, aiming to promote factors that help individuals and communities thrive.
77
Who is a key founder of positive psychology and what did he develop?
Abraham maslow; devloped the theory of human motivation (Maslow’s Hierarchy of Needs).
78
How does positive psychology differ from traditional psychology?
It focuses on strengths and flourishing, while traditional psychology focuses on deficits and dysfunction.
79
What is misconception #1 about deficits?
Fixing what's wrong does not automatically lead to well-being; absence of mental illness ≠ presence of mental health.
80
What is misconception #2?
Effective coping isn’t just reducing negative states—it’s about managing stress effectively.
81
What is misconception #3?
A deficit focus alone does not prevent problems. Prevention requires understanding why some people thrive.
82
Who is Michael Ungar?
A researcher on resilience and Principal Investigator at the Resilience Research Centre (Dalhousie University).
83
What does Ungar argue is essential for resilience?
Supportive environments that foster change, survival, and flourishing—not just individual traits like grit.
84
What are the 9 things all children need for resilience (Ungar)?
Structure, Consequences, Parent-child connection, Strong relationships, Powerful identity, Sense of control, Sense of belonging, Fair treatment, and Physical/psychological safety.
85
What are types of neurodevelopmental disorders
Intellectual disabilities, communication disorder, autism spectrum disorders (ASD), Attention deficit hyperactivity disorder (ADHD), specific learning disorders (SLDs), motor disorders.
86
What are the two main domains of ASD?
Social communication impairment and restricted/repetitive behaviours.
87
What does altered brain connectivity mean in the context of Autism Spectrum Disorder (ASD)?
It refers to both reduced (hypo-) and increased (hyper-) communication between brain regions, especially in networks involved in social functioning like the temporo- and frontolimbic areas.
88
Which brain regions are commonly affected in individuals with ASD?
The cerebral cortex, limbic system, and cerebellum often show structural and functional differences in people with ASD.
89
What is the “social brain network” and how is it relevant to ASD?
It includes regions like the temporal and frontal lobes and the limbic system, which are involved in social understanding and are often functionally altered in ASD.
90
Stressor
any event—either witnessed firsthand, experienced personally, or experienced that increases physical or psychological demands on an individual
91
Childhood trauma
may be related to a specific event * death of a loved one, a natural disaster, and other adverse childhood events
92
maltreatment
neglect or abuse of a child
93
What is trauma?
Trauma is the emotional, psychological and physiological residue left over from heightened stress that accompanies experience of threat, violence and life changing events
94
Types of traumatic events
Complex trauma * Family violence * Medical trauma * Natural disasters * Community and school violence * Neglect * Physical abuse * Sexual abuse * Traumatic grief * Refugee and war zone trauma
95
What system is activated immediately during stress?
The catecholamine system/sympathetic nervous system, triggering the fight-or-flight response.
96
What does the HPA axis do in the stress response?
It regulates longer-term stress by releasing cortisol through a chain reaction from the hypothalamus → pituitary → adrenal glands.
97
What hormone is released by the HPA axis during stress?
Cortisol
98
How can trauma affect the brain’s stress system?
Trauma can alter HPA axis activity, disrupt emotional brain circuits, and increase risk for mental health disorders like PTSD and depression.
99
What early experiences are known to disrupt the HPA axis?
Abuse, neglect, or deprivation during childhood.
100
What system shows more consistent changes after trauma—neuroendocrine or neuroanatomy?
Neuroendocrine (e.g., HPA axis changes) is more consistently affected.
101
How does trauma affect the HPA axis?
It can lead to a blunted cortisol response and disrupted cortisol patterns throughout the day.
102
What brain changes are associated with childhood trauma?
Smaller prefrontal cortex, increased amygdala reactivity, and disrupted brain connectivity.
103
What cognitive functions may be affected by maltreatment?
Working memory, attention, processing speed, language, visual-spatial and motor skills.
104
Q: What kind of trauma most often leads to CPTSD?
Prolonged or repeated trauma from which escape is difficult or impossible, such as childhood abuse or captivity.
105
What must be present for someone to be diagnosed with CPTSD?
What must be present for someone to be diagnosed with CPTSD?
106
What is the core feature of Reactive Attachment Disorder (RAD)?
Emotionally withdrawn behavior toward adult caregivers, including failure to seek or respond to comfort.
107
List two domains of functioning affected by ASD according to DSM-5-TR.
Social communication & interaction; Repetitive behaviors/restricted interests
108
What are the three symptom domains of ODD in DSM-5-TR?
Irritable/angry mood, argumentative/defiant behavior, vindictiveness
109
The Yerkes-Dodson Law
suggests performance increases with arousal to a point, after which too much anxiety decreases performance.