RUMEN Digestion Flashcards

(44 cards)

1
Q

Which side do you assess the rumen?

A

Left side

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2
Q

What is the main function of the rumen?

A

Fermentation of foodstuffs to produce…:
- Volatile fatty acids (VFA’s)
- Microbial proteins (synthesize by the bacteria)
- B Vitamins
(FERMENTATION: CORE FUNCTION OF THE RUMEN)

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3
Q

How do we mesure the feeding of ruminants?

A

With the dry matter (you DO NOT include the water component):
E.g.: 15 Kg DM –> Easier to give recommandation as a vet!

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4
Q

What does cellulose and hemicellulose turn into?

A

CARBOHYDRATES
Cellulose: starch + pectin
Hemicellulose: sugars

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5
Q

How is the carbohydrates metabolized?

A

BY THE BACTERIA IN THE RUMEN!

- Breaks them down into hexose –> 2 pyruvate –> Acetate, Butyrate, proprionate

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6
Q

What are the 2 big categories of carbohydrates?

A

Cell contents = non-fiber carbs or non-structural carbs (NFC/NSC) –> easily digested!

Cell wall = structural carbs = neutral detergent fiber (NDF)

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7
Q

What are the 2 components of NDF? Are they easily digestible like the NFC/NSC? Both digestible as easily?

A
  • Hemicellulose
  • Acid detergent Fiber: cellulose and lignin –> not digestible, need to limit those!

–> They are not as easily digested as NFC/NSC, they require specialize mechanisms to digest them!

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8
Q

Crude proteins is the name given to the protein being digested. What are the 2 main categories and subcategories of each? Are they all really able to be digested…?

So what are the metabolizable protein…?

A

RUMEN DEGRADABLE: (microbial protein synthesis)

  • Non-proteins N fraction A
  • AA & proteins Fraction B

RUMEN UNDEGRADABLE: (usually supplemented by the owner)

  • AA & Proteins = absorption in the SI
  • -> Part indigestible: lost in heat damages, complexed

It is the proteins that the ruminants can actually utilize.
- Rumen digestable and some rumen undigestable, but of those are lost!

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9
Q

What does NFC/NSC stimulate?
What does NDF stimulate?

For a balance rumen fermentation, what is the % of NDF, NFC and crude protein (CD) do you want to target? And why?

A

NFC/NSC: stimulate microbial digestion, therefore VFA production –> 40%

NDF: stimulates chewing, therefore saliva production (buffer) –> 30%

CP –> 17%

This formula will….

  • increase VFA production = increase production (via NFC)
  • Increase rate VFA production = decrease pH (via NFC again and lower % of NDF but not too much to keep buffer)
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10
Q

Is there a portion of the protein in the fecal material?

A

Yes! But need to minize it!

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11
Q

What is the main source of protein? Rumen digestable or undigestible?

A

Rumen degradable!

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12
Q

When bovins are eating they are ingesting big particles and later on (when not eating), what are they doing to make them smaller?

A

They are ruminating and fermenting the food. Bringing back food in the mouth to grinding them even more, and fermentation in the rumen.

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13
Q

What is a major source of lubrication? What is the other function of that thing?

A

Saliva! IMPORTANT Buffer substance! = key element for rumen function!!!!

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14
Q

If you change the diet of the ruminants, you have to do it….

A

gradually!!! The rumen is always adapting to the diet!

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15
Q

What does decreasing the size of the food does?

A

It increase the digestibility! Smaller piece = more efficient digestion!

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16
Q

How is the homoestasis of rumen maintained?

A
  • Sources of buffer (epithelium, saliva)
  • Effect of diet: VFA absorption
  • Adaptation of the rumen all the time!
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17
Q

Is the epithelium of the rumen important?

A

Yes! The rumen epithelium acts as a buffer!

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18
Q

Why is a decrease of water supply will disturb the rumen function? How much acid does saliva absorbs (neutralize)?

A

Because it will decrease the saliva = decrease buffer HCO3- in the saliva.
Saliva neutralize 50% of acids produced!

19
Q

So, now, what are the 3 element of constitutive control of Rumen pH?

And, what are the 3 element of inducible control of Rumen pH? (what you have control on)

A

Constitutive:

  • VFA production
  • Buffer from rumen epithelium
  • Organic acid outflow

Inducible:

  • VFA absorption
  • Buffer from saliva
  • Rumen microbiome
20
Q

How does the rumen adapt? To what is it adapting?

A

Increase rumen mass and papillae size!!! It adapt to a change in diet, for example, in this case, it to an increase in dry matter intake.

21
Q

Important to keep a precise pH range, how do ruminants do it?

A

Buffer inside the saliva!!
Major source of bicarbonate! + lubricate the food!
120L per day!
Neutralize 40-50% of acid produced
Need more water when producing milk = more water!

22
Q

How do you ensure a good amount of saliva production?

A

Provide clean and ad lib water!

23
Q

What can rumination time influence?
What can influence rumination time?

What affect the dry matter intake (DMI)?

A
  • -> Increase ruminating = increase rumen pH (protect from acidosis)
  • -> Increase NDF/peNDF = increase ruminating = increase saliva production = more buffer (also protect from acidosis)

NDF and peNDF affect DMI!

24
Q

What is the main source of carbs use?
What does an increase of NFC/starch does?
Why is the rate of degradation of those NFC important? How do you control it?

A

CORN!
- Increase NFC/startch = decrease rumen pH

The rate of degradation is important for a good absorption and you can control it by fine chopping Alf silage and corn silage.

25
What is the difference between dry corn and high moisture corn (HMC)?
Dry corn (finely chopped): decrease rumination time, decrease the pH, increase NDF, decrease starch HMC: increase rumination time, increase the pH, decrease NDF, increase starch
26
What happens in acute acidosis? | In what situation does it usually happen?
MAIN PROBLEM WHEN NOT FED GOOD DIET! Metabolic status defines by decreased blood pH and bicarbonate, caused by overproduction of ruminal lactate acids! Usually: Over eat grains = overload rumen with grains = lactic acidosis
27
Why do we call a dead rumen, a rumen that is in acute acidosis?
Because bacteria of the rumen have been killed = there's no longer fermenting, no longer moving, no sounds!
28
What accumulate in the rumen and blood? Can this thing acute outside the rumen?
LACTATE! Yes, it can be inside the rumen and systemic! | - Rumen and blood pH low!!
29
In general, how will the pH will change?
It will change depending on the time of the day, feeding time, type of diet. < 5.5 for > 3 hours/day < 5.8 for > 5 hours/day
30
What is the difference between the acute rumen acidosis and SARA (sub-acute rumen acidosis)?
SARA: is most common in dairy cow and it is not associated with an accumulation of lactate acids inside the rumen.
31
What is the sub-acute rumen acidosis (SARA)? What cause it?
Characterized by extended periods of depressed ruminal pH below 5.5-5.6. SARA results from excessive VFA production that exceeds the ability of the ruminal papillae to absorb them. Usually from... - Increase starch digestion (Less dry matter intake) = easily fermentable = increase in VFA = decrease of pH + decrease of ruminal contraction + mucosal damage of rumen
32
True or false. The rumen epithelium can change in dairy cattle with SARA.
TRUE!
33
In diagnosing SARA, should you take in consideration the sex and the calving/transition period?
YES!
34
What are the inflammatory consequences in SARA?
- Increase LPS in the rumen and portal vein - Increase LPS binding protein in blood - Increase cytokines in portal vein and liver - Increase acute phase proteins!
35
True or false. The rumen condition cannot affect the cecum lower down stream.
FALSE! Yes it can affect the epithelium of the cecum just like the one in the rumen!
36
What are other systemic effects of SARA?
- Laminitis: walk with a curved back because of pain hook (laminar corium inflammed) - Then become acute --> Liver abscesses = if one of the abscess burst = Caudal vena caval syndrome = sudden death syndrome!
37
How do you know the rumen is "acid", low pH?
- Rumenocentesis --> Sampling: percutaneous aspiration or pH meter. Test 8 hours post feeding in TMR herd or 3 hours post-concentrate feeding in component fed herd.
38
You can have an acute, chronic or subacute, and long term acidosis. What is the CS of each?
ACUTE: laminitis, founder, hopelessly off-feed, sell immediately CHRONIC or SUBACUTE: a little off feed, poor performers LONG TERM EFFECT: rumenitis, liver abscesses
39
How do you know if you think a herd is affected by SARA? What tells you that you need to take corrective measures?
Take 15 cows that are fed the same diet, housed similarly and distinguish 1st lactation vs Older. - -> If 4/15 rumen samples pH 5.5 = take corrective action - -> It is not an estimate of prevalence * * Reasonable accuracy!
40
What is the devise that can help with monitoring rumen pH by farmers?
pH meter!
41
What is the ideal rumen pH?
IDEAL: pH 6.0-6.4
42
What high tech devise can help to know if your ruminant are sick?
Accelerometers: mesure position and number of steps (if decrease = decrease activity, lameness. Rumination monitors
43
What high tech devise can help to know if your ruminant are sick?
Accelerometers: mesure position and number of steps (if decrease = decrease activity, lameness. Rumination monitors
44
How do you prevent acidosis? 3 technics, what are they? Explain each one of them.
- Diet formulation: assessment of grains (rate starch digestion), grass quality (forage quality and availability) - Supplement: - -> maybe supplement hay for high risk period - -> Ionophores: control gram+ bacteria, increase feed efficiency, no effect on rumen pH - Temperature management: thermal neutral zone 40-68 degrés F (hot weather decrease rumination, altered feeding patterns)