Skin and coat diseases Flashcards

(41 cards)

1
Q

Pruritus

A

Intense and persistent itching

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2
Q

Dermatitis

A

Inflammation of skin

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3
Q

Erosions

A

Ulcer that goes to depth of basement membrane

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4
Q

Erythema

A

Reddening of skin caused by capillary congestion

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5
Q

Exocoriation

A

Superficial erosions or ulcers caused by scratching or abrasion

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6
Q

Furunculosis

A

Deep infection of the hair follicle leading to abscess formation with accumulation of pus and necrotic tissue

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7
Q

Hyperkeratosis

A

Thickening of the stratum corneum from an increased number of keratinised cells

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8
Q

Plaques

A

Raised flat topped lesions

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9
Q

Pustule

A

Circumscribed epidermal or dermal accumulation of purulent exudate

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10
Q

What is pyoderma?

A
  • Bacterial skin disease mainly seen in dogs

- Commonly seen as a secondary to other disorders

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11
Q

Seborrheoa

A

Increase in the scaling of the skin, with or without an increase in sebum production

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12
Q

What are the clinical signs and causes of pyoderma?

A

Clinical signs:

  • Pustules
  • Exudative lesions
  • Scabs/crusts
  • Scratching
  • Pyrexia
  • Odour
  • Loss of appetite/behavioural changes

Causes:

  • Staphylococcus intermedius
  • Proteus Pseudomonas
  • Pastuerella (cat bites)
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13
Q

What are the 3 types of pyoderma?

A
  • Surface pyoderma (skin fold dermatitis/acute moist dermatitis)
  • Superficial pyoderma (impetigo/folliculitis)
  • Deep pyoderma (furunclosis/pododermatitis)
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14
Q

What is urticaria?

A
  • Allergic dermatitis
  • Causes multiple raised reddened circumscribed areas of erythema and oedema
  • Usually pruritic
  • Oedema may extend deep into dermis and subcutaneous tissues
  • Usually seen in superficial layers
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15
Q

What are the causes and treatments for urticaria?

A

Causes:

  • Drugs sensitivity
  • Insect bites
  • Bee/wasp stings

Treatments:

  • Corticosteroids
  • Removal of reason for reaction
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16
Q

What are the causes and treatments for urticaria?

A

Causes:

  • Drugs sensitivity
  • Insect bites
  • Bee/wasp stings

Treatments:

  • Corticosteroids
  • Removal of reason for reaction
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17
Q

What is atopic dermatitis?

A

Itchy dermatitis in response to ingestion/inhalation/direct contact of antigens

18
Q

What are the diagnostics of atopic dermatitis?

A
  • Intradermal skin tests
  • Multiple allergen tests
  • Blood tests
19
Q

What are the treatments for atopic dermatitis?

A
  • Avoid contact with cause
  • Injections of allergens
  • Corticosteroids
  • Essential fatty acids
  • Antihistamines
20
Q

What clinical signs are seen with hypothyroidism?

A
  • Mainly seen in dogs and rare in cats
  • Common in middle aged animals
  • Slow regrowth of hair/seborrhoea/pyoderma/hyperpigmentation/bilateral alopecia
  • Weight gain
21
Q

What are the treatments for hypothyroidism?

A
  • Hormone drugs (eg. levothyroxine)

- Treat cause and/or secondary skin disorders (eg. antibiotic/malaseb)

22
Q

What clinical signs may be present with hyperadrenocorticism (cushings)?

A
  • Bilateral alopecia
  • Dull/dry hair
  • Skin becomes thin/inelastic (especially ventral abdominal)
  • Ascites (pot belly)
  • Veins are visible
  • Scaling of the skin
  • Comedomes (caused by follicular plugging)
  • Bruising
  • More common in dogs
23
Q

What treatment can be done for hyperadrenocorticism (cushings)?

A
  • Surgical
  • Medical
  • Radiation therapy
24
Q

What are some examples of hormonal alopecia?

A
  • Hyperthyroidism
  • Hyperadrenocorticism
  • Sertoli call tumour
  • Canine ovarian imbalance
25
What clinical signs are seen with hormonal alopecia?
- Hyperpigmented skin - Dry and brittle coat - Does not cause itching
26
What clinical signs may be seen with feline symmetric alopecia?
- Bilateral symmetrical alopecia - Alopecia moves to forelegs (below elbow above carpus) and ventrally involving two thirds of the flank - Cat can remain normal
27
What is the treatment for feline symmetric alopecia?
- Thyroid hormone replacement - Megoestral acetate therapy - Testosterone therapy (male and females)
28
What is hyperoestrogenism in male canines?
- Testicular tumour (Sertoli cell tumour) in middle/old entire dogs - Skin disorder associated is bilateral alopecia - Treatment is castration
29
What are the clinical signs of canine ovarian imbalance type I hyperoestrogenism?
- Seen in middle aged entire bitches - Bilateral symmetrical alopecia beginning in perineal and genital areas - Hyperpigmentation as disorder progresses - Enlarged teats - Seborrhoea (oily skin) and pruritus (itchy skin) may occur - Normally seen in bitches with a history of abnormal oestrus cycles - Often associated with ovarian tumours or cystic ovaries - Treatment is to spay BUT symptoms may continue for a further 6 months
30
What is type II Oestrogen-responsive dermatosis?
- Female dogs that are prematurely neutered - Alopecia in perineal/genital areas, spreading to flanks and upper legs - Teats and vulva infertile - Often incontinent - Due to lack of oestrogen production - Hormone therapy will be needed
31
What is dermatophytosis?
- A.K.A ringworm (fungal dermatitis) - Round hairless patches will crusty, scabby skin - May be sore or itchy but can also cause no discomfort
32
What is dermatophytosis?
- A.K.A ringworm (fungal dermatitis) - Round hairless patches will crusty, scabby skin - May be sore or itchy but can also cause no discomfort
33
What are the causes and treatment for dermatophytosis?
Causes: - Fungal infection (dermatophytes) - Trichophyton or microsporum families Treatment: - Isolate from other animals - Clip around lesions/remove scabs - Antifungal shampoos - Repeat treatments - Exposure to air and sun
34
What are the 4 stages of wound healing?
1. Inflammation 2. Debridement 3. Repair 4. Maturation
35
Explain how initial wound management would be carried out?
- Clean wound and remove necrotic tissue (debridement) - If suspected infection culture should be taken - Gently clean and flush wound at a pressure of 8psi (can be achieved using 18g needle) - Wounds heal better when maintained in moist environment - Ideally open wounds should be bandaged but not always possible
36
Explain how debridement can be carried out for wounds
Surgical: - Drape as would for surgery - Use scalpel to cut away necrotic tissue - Change surgical kit once all damaged tissue removed Debridement dressings: - Used in initial stages until no infection or necrotic tissue present - Do not leave on for longer than 24hrs - Egs. hydrogels, hydrocolloids and adherent dressings
37
What are the types of healing processes recognised?
1. Primary union/first intention healing 2. Secondary closure/second intention closure 3. Delayed primary union/third intention healing
38
Explain how each of the healing processes work
1. First intention healing: - Wound formed by clean incision and sutured immediately - Minimal scar formation and skin edges unite firmly 2. Second intention healing: -More significant tissue loss so not possible to close surgically -Wound is left open to heal via granulation -Use dressing and may progress to secondary closure in large wound Secondary closure: -Heavily contaminated, tissue damage -Manage as open until good granulation cover then close 3. Third intention healing: - Contaminated, manage as open until infection/FB removed - Wound left open for a few days until granulation bed forms then its sutured (secondary closure)
39
What are the use of wound drains?
- Allows removal of fluid/gas from a surgical or traumatic wound - Remove inflammatory mediators, bacteria, foreign material and necrotic tissue - Relieve pressure - Allow easy sampling of fluid during healing - Eliminate dead space
40
What are the wound drain classifications and how do they work?
Open systems: - Passive - Rely on gravity - Body movement - Pressure differentials - Overflow to remove gas/fluid Closed systems: - Active - Rely on negative pressure created by the drain
41
What are some possible complications of drains?
Higher risk of infection - Ascending infection - FB reaction to brain Vascular damage -Pressure necrosis of arterial wall Blockage - Clogged - Lose suction (active) - Failure Electrolyte imbalance -High fluid production and loss leading to metabolic derangements and hypoproteinemia