Skin and Sensory (Final) Flashcards

(92 cards)

1
Q

Contact Dermatitis

A

• acute inflammatory reaction triggered by direct exposure to an irritant or allergen-producing substance; not contagious or life threatening
• varies in severity depending on substance, are affected, exposure extent, and individual sensitivity
• usually resolves in 2-4 weeks

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2
Q

Irritant Contact Dermatitis

A

doesn’t involve the immune system but triggers the inflammatory response

produces a similar reaction to a burn

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3
Q

Irritant Contact Dermatitis Causes

A

chemicals
plants
body fluids
rubber gloves
soaps

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4
Q

Irritant Contact Dermatitis Manifestations

A

erythema
edema
pain
pruritus
vesicles

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5
Q

Allergic Contact Dermatitis

A

sensitization occurs on first exposure and subsequent exposures produce type IV cell-mediated hypersensitivity

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6
Q

Allergic Contact Dermatitis Causes

A

metals
chemicals
adhesives
cosmetics
plants

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7
Q

Allergic Contact Dermatitis Manifestations

A

appear 24-48 hrs after exposure

pruritus
erythema
edema
small vesicles

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8
Q

Atopic Dermatitis (Eczema)

A

chronic inflammatory condition with an inherited tendency; may be accompanied by asthma and allergic rhinitis

most common in infants and usually resolves by early adulthood; characterized by remissions and exacerbations

exact cause unknown but may result from immune system malfunction (similar to hypersensitivity reactions ie IgE elevation)

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9
Q

Atopic Dermatitis Complications

A

secondary bacterial skin infections
neurodermatitis (permanent scarring and discoloration from chronic scratching)
eye problems (conjunctivitis)

may affect any area but pattern exhibited tends to be age specific

YOUNG CHILDREN: FACE, SCALP, HANDS, FEET
OLDER CHILDREN/ADULTS: KNEES AND ELBOWS

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10
Q

Atopic Dermatitis Manifestations

A

may be worsened by environmental factors like food allergens, airborne allergens, Staphylococcus aureus colonizations on skin, topical products, sweating, and rough fabrics

red to brownish-gray skin patches
pruritus, which may be severe especially at night
vesicles
thickened (lichenified), cracked or scaly skin
irritated, sensitive skin from scratching

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11
Q

Urticaria (Hives)

A

raised erythematous skin lesions (welts)

occurs when histamine release is initiated by these substances or conditions

usually short lived and harmless

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12
Q

Urticaria Causes

A

Type I hypersensitivity reaction often triggered by food (shellfish, nuts) and medicine (antibiotics)

emotional stress
excessive perspiration
diseases (autoimmune, leukemia)
infections (mono)

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13
Q

Urticaria Manifestations

A

welts that blanch and pruritus
diffuse welts may grow large, spread and fuse together
can impair breathing if around face and progress to anaphylaxis and shock

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14
Q

Psoriasis

A

common chronic inflammatory condition that affects skin cell life cycle, specifically keratinocytes

cellular proliferation is significantly increased, causing cells to build up too rapidly on skins surface; buildup leads to thickening of dermis and epidermis because dead skin cells cannot shed fast enough

may take days-weeks for symptom to emerge during flare ups

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15
Q

Psoriasis Causes

A

exact cause unknown, but thought to be multifactorial

environmental factors
trauma
infections
obesity
excessive alcohol
certain meds
genetic
immunologic factors
autoimmune process in which T lymphocytes mistake normal skin cells as foreign

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16
Q

Psoriasis Onset

A

family tendency observed; severity varies (weakened immune system)

most frequently between 15-35 years and may be sudden or gradual

REMISSIONS AND EXACERBATIONS; MAY ALSO HAVE ARTHRITIS

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17
Q

Psoriasis Exacerbation Causes

A

bacteria or viral infections in any location
dry air/dry skin
skin injuries
certain meds (antimalaria agents, beta blockers, lithium)
stress
too little/too much sunlight
excessive alcohol

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18
Q

Psoriasis Papules

A

begins as small red papule, often on elbows, knees and trunk but can appear anywhere

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19
Q

Erythrodermic

A

intense erythema that covers a large area

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20
Q

Guttate

A

small pink-red spots

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21
Q

Inverse

A

erythema and irritation that occurs in armpits, groin, and skin folds

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22
Q

Plaque

A

thick, red patches covered by flaky, silver white scales (MOST COMMON)

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23
Q

Pustular

A

white blisters surrounded by red, irritated skin

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24
Q

Psoriasis Manifestations

A

pruritus
genital lesions
joint pain or aching (if arthritis present)
nail changes (thickening, yellow brown spots, pits on nail surface, separation of nail from base)
dandruff

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25
Infectious Integumentary Disorders
skin infections are common organisms gain access through breach in skin or mucous membranes which triggers inflammation **can occur in any skin layer or structure, may be acute or chronic, severity varies, resolves with treatment**
26
Bacterial Infections
can be caused by any of the normal flora, mild to life threatening staphylococcus and streptococcus genera are common culprits
27
Folliculitis
bacterial infection involving hair folllicles **tender, swollen areas that form around hair follicles often on neck, boobs, butt and face**
28
Furuncles
bacterial infection beginning in hair follicles and then spread into surrounding dermis; most common on face, neck, axilla, groin, butt, back **starts as a firm, red painful nodule that develops into a large painful mass which frequently drains large amounts of purulent exudate**
29
Carbuncles
a cluster of furuncles
30
Impetigo
common and highly contagious bacterial infection; can spread throughout body through self transfer of exudate can occur without apparent skin breach but typically arises from break in skin **typically caused by staphylococci, which produce a toxin that attacks collagen and promotes spread**
31
Impetigo Manifestations
lesions usually begin as small vesicles that enlarge and rupture, forming characteristic honey colored crust pruritus lymphadenopathy
32
Cellulitis
bacterial infection that occurs deep in the dermis and subQ tissue; **appears as a swollen, warm and tender area of erythema** usually results from direct invasion of pathogens through break in skin especially where contamination is likely or spreads from existing infection
33
Cellulitis Manifestations and Complications
**M:** indicators of infections (*fever, leukocytosis, malaise, arthralgia** **C:** necrotizing fasciitis, septicemia, septic shock
34
Necrotizing Fasciitis
a rare but serious bacterial infection that can aggressively destroy skin, fat, muscle and other tissue **typically results from highly virulent strain of gram-positive, group A beta-hemolytic streptococcus that invades through a minor cut or scrape; *bacteria release toxins that directly destroy tissue, disrupt blood flow and break down tissue***
35
Necrotizing Fasciitis Wound
first sign may be a small, reddish painful area that quickly evolves into a painful bronze or purple colored patch center of lesion may become black and necrotic with exudate wound may grow in less than an hour
36
Necrotizing Fasciitis Manifestations and Complications
**M:** fever, tachycardia, hypotension, confusion **C:** gangrene, multi system organ failure, shock
37
Herpes Simplex Type 1
typically affects lips, mouth and face beginning in childhood; can involve the eyes leading to conjunctivitis can result in meningoencephalitis transmitted by contact with infected saliva, primary infection may be asymptomatic
38
HSV 1 Pathogenesis
1. after primary infection, virus remains dormant in sensory nerve ganglion to the trigeminal nerve until is is reactivated 2. reactivation may result from infection, stress, immunosuppression, or sun exposure (*painful blisters or ulcerations that are preceded by burning or tingling sensation*) 3. lesions may resolves spontaneously within 3 weeks, but healing can be accelerated with the administration of oral or topical antiviral agents
39
Herpes Zoster (Shingles)
varicella-zoster virus, appears in adulthood years after a primary infection of varicella in childhood **virus lies dormant on a cranial/spinal nerve dermatome until activated years later; virus affects this nerve only giving the condition its typical unilateral manifestations**
40
Shingles Manifestations
pain paresthesia red/silvery vesicular rash that develops in along over the area innervated by affected nerve (*nose sit of the head or torso*) extremely sensitive skin pruritus **rash may persist for weeks to months**
41
Shingles Complications
neuralgia blindness **vaccines are available to prevent both varicella and herpes zoster**
42
Rubella (German/3-Day Measles)
RNA virus enters bloodstream through respiratory route; mild in most children
43
Rubella Manifestations
enlarged cervical and postauricular lymph nodes low grade fever headache sore throat runny nose cough faint pink to red maculopapular rash caused by virus dissemination to the skin
44
Rubeola (Red Measles)
highly contagious acute viral disease of childhood (*droplet transmission*) no symptoms in incubation period (7-12 days)
45
Rubeola Manifestations
high fever malaise enlarged lymph nodes runny nose conjunctivitis barking cough rash that develops over head and spreads distally over the trunk, extremities, hands and feet characteristic pinpoint white spots (**Koplik**) found over buccal mucosa
46
Roseola
herpesvirus 6 or 7 infection; 6 months to 2 yrs of age *intubation of 5-15 days followed by sudden onset of fever that lasts 3-5 days* **after fever, erythematous macular rash that lasts about 24 hrs (usually doesn’t require treatment)**
47
Tinea (Ringworm/Athletes Foot)
parasitic infection that causes several types of superficial fungal infections typically manifests as a **circular, erythematous rash accompanied by pruritus and burning**
48
Tinea Capitis
the scalp; common in school aged children hair loss at site is common
49
Tinea Corporis
involving the body
50
Tinea Pedis
involving the feet especially the toes
51
Tinea Unguium
involving the nails, typically the toenails *begins at the tip of one or more nails and then usually spread to other nails* **turns nails white and then brown causing them to thicken and crack**
52
Scabies
result of mite infestation male mites fertilize the females then die, female mites burrow into epidermis laying eggs over a period of several weeks through series of tracts and die after that
53
Scabies Process
1. larvae hatch from eggs and migrate to skin surface 2. larvae burrow in search of nutrients and mature to repeat cycle 3. burrowing appears as small, light brown streaks on skin 4. burrowing and fecal matter left by the mites triggers the inflammatory process, leading to erythema and pruritus **transmission of close contact**
54
Pediculosis (Lice Infestation)
small brown insects that feed off human blood females lay nits on hair shaft close to scalp (*appear as white, small, iridescent shells on hair*) after hatching, the life bite and suck on blood bite size develops as highly pruritic macule or papule CLOSE CONTACT!!
55
Skin Cancer
most frequent cancer in the US; more prevalent in *males, whites, fair complexion, family history* early detection is crucial
56
Basal Cell Carcinoma
most common; develops from abnormal growth of cells in lowest layer of epidermis rarely metastasizes
57
Squamous Cell Carcinoma
involves changes in the squamous cells found in the middle layer of epidermis
58
Melanoma
melanocytes; least common type but most serious often metastasizes
59
Skin Cancer Suspicious Features
ABCD (larger than 6 mm) any skin growth that bleeds or will not heal any skin growth that changes in appearance over time
60
Conjunctivitis
caused by viruses (most common), bacteria (staphylococcus, gonorrhea, chlamydia), allergens (pollen, dust), chemical irritants, trauma **can generate edema, pain, blurry vision, photophobia**
61
Viral vs Bacterial Conjunctivitis
viral: **watery, mucus like exudate** bacterial: **yellow-green exudate** HIGHLY CONTAGIOUS THROUGH DIRECT CONTACT
62
Conjunctivitis Risk Factors
wearing contact lenses contaminated makeup ophthalmic meds **allergens and irritants typically produce redness, itching and excessive tearing**
63
Acute Otitis Media
infection of middle ear; common in young children due to eustachian tubes being narrower, straighter and shorter and an immature immune system begins as viral upper respiratory infection; more common in winter **fluid collection from viral infection provides prime medium for secondary bacterial growth, usually streptococcus pneumoniae and haemophilus influenza**
64
Acute Otitis Media Risk Factors
childcare in group settings feeding infants in supine position environmental smoke exposure pacifier use orofacial deformities history of allergic rhinitis **fluid accumulation in middle ear due to adenoid enlargement, usually due to inflammation**
65
Acute Otitis Media Complications
effusions rupture of tympanic membrane scar tissue formation conductive hearing loss mastoiditis cholesteatoma meningitis osteomyelitis
66
Acute Otitis Media Manifestations
ear pain crying or irritability rubbing or pulling at ear mild hearing deficits sleep disturbances red and bulging tympanic membrane indications of infection purulent or clear exudate from external ear canal (if tympanic membrane ruptures) n/v diarrhea headache
67
Glaucoma
group of eye conditions that lead to damage to the optic nerve; *caused by increased intraocular pressure and decreased blood flow to the optic nerve* **pressures inside the eye can climb when the outflow of aqueous humor becomes blocked or production of aqueous humor increased to an abnormal level (*increased pressures cause ischemia and degeneration of the optic nerve*)** SECOND LEADING CAUSE OF BLINDNESS (diabetic retinopathy is #1)
68
Open-Angle Glaucoma
most common type; intraocular pressure may increase gradually over an extended period of *RISK FACTORS: FAMILY HISTORY AND AA** **because vision changes are gradual, can be overlooked or misdiagnosed as presbyopia**
69
Open-Angle Glaucoma Manifestations
painless, insidious, bilateral changes in vision (*tunnel vision, blurred vision, halos around lights, decreased color discrimination*) loss of peripheral vision (tunnel vision) patients describe vision as looking through a straw or curtains are closing
70
Closed-Angle Glaucoma
result of a sudden blockage of aqueous humor outflow; can be acute, subacute or chronic and typically unilateral **without treatment, acute and narrow angle glaucoma can cause blindness**
71
Closed-Angle Glaucoma Causes
traum, sudden pupil dilation (*exposure to bright light after prolonged darkness*) prolonged pupil dilation (*meds for eye exams*) emotional stress
72
Closed-Angle Glaucoma Manifestations
**usually sudden and worsening quickly** sudden and severe eye pain headache n/v nonreactive pupil redness haziness of cornea vision changes (*halos around lights*)
73
Cataracts
opacity or clouding of the lens; can occur as a congenital condition or develop later on may affects one or both eyes and doesn’t always affect eyes symmetrically
74
Cataracts Risk Factors
family history advancing age smoking UV light exposure (*natural or artificial*) metabolic conditions (*diabetes mellitus*) certain meds (*corticosteroids*) eye injury
75
Cataracts Manifestations
cloudy, fuzzy, foggy or filmy vision color intensity loss diplopia impaired night vision gradually progressing to impaired day vision halos around lights photosensitivity frequent changes in eyeglass or contact lens prescriptions
76
Macular Degeneration
deterioration of the macular area of the retina caused by impaired blood supply to the macula that results in cellular waste accumulation and ischemia
77
Macular Degeneration Risk Factors
**ADVANCING AGE** family history being female and white smoking increased UV light exposure decreased carotenoid intake high fat diet cardiovascular disease hypertension obesity
78
Macular Degeneration Manifestations
DRY FORM: **blurry vision with loss of central vision** WET FORM: **distortion of straight lines, dark spots in central vision, sudden loss of central vision**
79
Ménière’s disease
disorder of the inner ear that results from endolymph swelling which stretches the membranes and interferes with the hair receptors in the cochlea and vestibule peak incidence is 20-50 years
80
Ménière’s Disease Risk Factors
**cause is unknown but mag be metabolic disturbances, hormonal imbalances, autoimmune, head injuries, otitis media, syphilis** allergic rhinitis alcohol abuse stress fatigue certain meds (*aspirin*) respiratory infections
81
Ménière’s Disease Manifestations
attacks typically occur in waves of acute episodes that last several months followed by brief periods of relief (**triggered by changes in barometric pressure; includes intermittent episodes of vertigo, tinnitus, unilateral hearing loss, and a sensation of fullness**) n/v diarrhea headache uncontrollable eye movement **C: PERMANENT HEARING LOSS**
82
Retinal Detachment
acute condition that occurs when retina separates from supporting structures **as vitreous humor collects underneath, the retina peels away from the underlying choroid; these detached areas may expand over time and the retina becomes ischemic and stops functioning causing vision loss**
83
Retinal Detachment Pathogenesis
1. vitreous humor leaks through retinal tear and accumulates underneath retina 2. leakage can also occur through tiny holes where the retina has thinned due to aging or other retinal disorders 3. less commonly, fluid can leak directly underneath retina without a tear or break
84
Retinal Detachment Causes
spontaneous severe nearsightedness trauma diabetes mellitus inflammation degenerative aging changes scar tissue
85
Retinal Detachment Manifestations
typically painless flashes of light in peripheral visual field blurred vision floaters darkening vision (*like curtain drawing across a visual field*)
86
Tinnitus
hearing abnormal noises in the ear may be described as a ringing, buzzing, humming, whistling, roaring or blowing
87
Tinnitus Associations
presbycusis exposure to excessive noise cerumen impaction otosclerosis ménière’s disease stress head injury acoustic neuroma atherosclerosis hypertension carotid stenosis arteriovenous malformation caffeine ototoxic meds (*many antibiotics, aspirin, chemo, diuretics*)
88
Vertigo
illusion of motion; NOT THE SAME AS DIZZINESS n/v
89
Peripheral Vertigo
there is a problem with vestibular labyrinth, semicircular canals or vestibular nerve
90
Peripheral Vertigo Causes
certain meds (*amino-glycoside antibiotics*) head injuries ménière’s disease nerve compression infections inflammation
91
Central Vertigo
there is a problem in the brain (brain stem or cerebellum)
92
Central Vertigo Causes
arteriosclerosis certain meds (*anti seizure agents and aspirin*) alcohol migraines MS seizures