Spinal Cord Compression Flashcards

1
Q

describe the corticospinal tract

A

2 neurone tract
UMN = from motor cortex > anterior grey horn
decussates at medulla
ipsilateral tract
LMN = anterior horn cell > site of action

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2
Q

features of UMN lesion?

A

increased tone
no real muscle wasting
hyperreflexia
no fasciculation

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3
Q

features of LMN lesion?

A

decreased tone
muscle wasting
fasciculation
diminished reflexes

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4
Q

describe spinothalamic tract?

A

carried info on pain, temp and crude touch
contralateral tract
decussates at spinal level

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5
Q

describe dorsal columns

A

carried info on fine touch, proprioception, vibration
ipsilateral tract
decussates at medulla

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6
Q

which spinal joint is most commonly affected in rheumatoid arthritis?

A

C1/C2

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7
Q

what is cord transection?

A

conceptual term meaning spinal cord is injured
can be complete (all motor and sensory modalities below level of injury affected)
can be incomplete (some power/sensation preserved)

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8
Q

how does cord transection present?

A

hypotension (due to loss of sympathetic outflow below lesion)
initially a flaccid areflexic paralysis/”spinal shock” (no reflexes, floppy paralysis)
classic UMN signs develop later (spastic paralysis, hypereflexes etc)

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9
Q

how does borwn sequard syndrome present?

A

ipsilateral motor level affected
ipsilateral dorsal column sensor level affected
contralateral spinothalamic sensory level affected

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10
Q

how does central cord syndrome generally present?

A
usually an elderly person after a fall
cant move hands
band of numbness/pins and needles over tops of hands
distal upper limb weakness
cape-like spinothalamic sensory loss
normal lower limbs and dorsal column
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11
Q

what usually causes a central cord syndrome?

A

hyperflexion or extension injury to already stenotic neck

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12
Q

why does ischaemia often occur in central cord syndrome?

A

pinching of medial spinal cord = most vulnerable part to ischaemia due to blood supply

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13
Q

describe sensory loss in central cord syndrome?

A

not true sensory loss as there isn’t complete loss of sensation everywhere below injury, only at that level

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14
Q

how does chronic spinal cord compression present?

A

same as acute but UMN signs predominate

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15
Q

most common areas of spine injured in trauma?

A

cervical

- most mobile

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16
Q

commonest cause of spinal tumours?

A

metastases

17
Q

what causes extradural tumours?

A

metastases (Lung, breast, kidney, prostate)

18
Q

what causes intradural tumour?

A

extramedullary (meningioma, schwannoma)

intramedullary (astrocytoma, ependymoma)

19
Q

how can tumours damage spinal cord?

A

can slowly compress

can cause acute compression by collapse or haemorrhage

20
Q

features of spinal stenosis?

A

osteophyte formation
bulging of IV discs
facet joint hypertrophy
subluxation

21
Q

causes of spinal cord infection?

A
epidural abscess (bloodborne staph, TB)
surgery or trauma
22
Q

what types of bleeding can cause spinal cord compression?

A

epidural
subdural
intramedullary

23
Q

causes of bleeding leading to spinal cord compression?

A

trauma
bleeding diatheses
anticoagulants
AVM

24
Q

how is spinal cord trauma managed?

A

immobilise (collar, board, blocks etc)
investigate (CT/X-ray, MRI)
decompress and stabilise (surgery, traction, external fixation)
methylprednisolone (acute inflammation decompression but generally not used due to side effects)

25
how are metastatic tumours causing spinal cord compression managed?
IV dexamethasone in acute presentation radiotherapy usually chemotherapy if chemosensitive (multiple myeloma etc) surgical decompression and stabilisation if needed but try to avoid
26
how are primary spinal tumours managed?
mainly excised
27
how is infection in the spine managed?
antimicrobial therapy surgical drainage (always drain pus) stabilisation where required
28
how is haemorrhage in spinal canal managed?
reverse anticoagulation | surgical decompression
29
how is degenerative disease managed?
surgical decompression +/- stabilisation