Sprains Strains Flashcards

(36 cards)

1
Q

Neuro soft tissue

A

nerve roots, trunks, peripheral nerves

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2
Q

contractile soft tissue

A

muscles, tendons

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3
Q

Inert soft tissue

A

(ligaments, capsules, fascia)

Bone
Bursa
Joint capsule
Synovium
Meniscus

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4
Q

Nerve Injury Can Be Caused By

A
  • Compression
  • Stretch or traction
  • Friction
  • Cooling or freezing
  • Thermal/electrical
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5
Q

Lower Motor Neuron Lesions Presentation

A
  • Flaccidity
  • Hyporeflexia or areflexia
  • Hypotonicity
  • Fasciculations
  • Muscle weakness/atrophy (specific to nerve)
  • Positive pathological reflex (1 side)
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6
Q

UMN Lesion Presentation

A
  • Spasticity
  • Hyperreflexia
  • Hypertonicity
  • Muscle weakness
  • Extensor plantar response (bilateral)
  • Absent or reduced superficial reflex
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7
Q
  • West Point Ankle Sprain Grading System
    • Grade I
A
  • Location of Tenderness
    ATF Ligament
  • Edema and Ecchymosis
    Slight and Local
  • Weight-Bearing Ability
    Full or partial
  • Ligament Damage
    Stretched
  • Instability
    None
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8
Q
  • West Point Ankle Sprain Grading System
    • Grade II
A
  • Location of Tenderness
    ATF and CF Ligament
  • Edema and Ecchymosis
    Moderate and Local
  • Weight-Bearing Ability
    Difficulty without crutches
  • Ligament Damage
    Partial tear
  • Instability
    None or slight
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9
Q
  • West Point Ankle Sprain Grading System
    • Grade III
A
  • Location of Tenderness
    ATF, CF, and PTF Ligaments
  • Edema and Ecchymosis
    Significant and diffuse
  • Weight-Bearing Ability
    impossible (too painful)
  • Ligament Damage
    Complete tear
  • Instability
    definite
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10
Q
  • Soft Tissue Lesions: Stages of Inflammation and Repair
    • Acute Stage - Reaction and Inflammation
A

4-6 days

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11
Q

Soft Tissue Lesions: Stages of Inflammation and Repair
Subacute Stage - Proliferation, Repair, and Healing

A
  • Usually lasts 10 to 17 days (14 to 21 days after onset of injury)
  • May last up to 6 weeks or longer in tissues with limited circulation, such as tendons
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12
Q

Soft Tissue Lesions: Stages of Inflammation and Repair
Chronic Stage - Maturation and Remodeling

A

May last 6 weeks to one year depending on the tissue involved and amount of tissue damage

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13
Q
  • Soft Tissue Lesions: Response to Therapist Exam
    • “Irritability” of tissue:
A

reflects stages of inflammation and repair

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14
Q
  • Acute Stage - Phase I (Maximum Protection)
    • Tissue Response: Inflammation
A
  • Cellular
  • Vascular
  • Chemical
  • During the second to fourth days after tissue injury, the inflammation begins to decrease, the clot starts resolving, and repair of the injured site begins.
  • Myofibroblastic activity begins about day 5, causing scar shrinkage (contraction).
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15
Q

Subacute Stage— Phase II (Moderate Protection/Controlled Motion)
- Tissue response: Proliferation, repair, and healing

A
  • 10 to 17 days (14–21 days after onset of injury), but may last up to 6 weeks
    • Noxious stimuli are removed, and capillary beds begin to grow into the area
    • Synthesis and deposition of collagen
      • Fibroblastic activity, collagen formation, and granulation tissue development increase
      • The fibroblasts produce new collagen, and this immature collagen replaces the exudate that originally formed the clot
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16
Q

Depending on the size of injury, wound closure usually takes

A

5 to 8 days in muscle and skin and 3 to 6 weeks in tendons and ligaments.

17
Q

Subacute Stage— Phase II (Moderate Protection/Controlled Motion)
During this stage of healing, the immature connective tissue that is produced is

A

thin and unorganized. It is extremely fragile and easily injured if overstressed.

18
Q
  • Chronic Stage— Phase III- Minimum to no protection/return to function
    • Tissue response: Maturation and remodeling
A

Scar retraction from activity of the myofibroblasts is usually complete by day 21 and the scar stops increasing in size

19
Q

From day 21 to day 60, there is a predominance of

A

fibroblasts that are easily remodeled. Want to avoid adhesions/contractures!

20
Q

The maturation and remodeling of the scar tissue occur as collagen fibers become

A

thicker and reorient in response to stresses placed on the connective tissue.

21
Q

Remodeling time is influenced by factors that affect the density and activity level of the

A

fibroblasts, including the amount of time immobilized, stress placed on the tissue, location of the lesion, and vascular supply.

22
Q
  • Cumulative Trauma: Chronic Recurring Pain
    • Tissue response from repetitive stress: chronic inflammation
      Causes of chronic inflammation:
A
  • Overuse, cumulative trauma, repetitive strain
  • Reinjury of an “old scar”
  • Contractures or poor mobility
23
Q

MSK Pain

A

Sharp, superficial

■Associated with movement

■Aggravated by mechanical stress

■Associated with trauma or overuse

■Generally lessens at night

24
Q

Systemic Pain

A

■Deep, aching throb

■Reduced by pressure

■Constant waves of pain & spasm

Not affected by movement

■Associated with jaundice, migratory arthralgias, skin rash, fatigue, weight loss, low- grade fever, weakness, signs of infection

■Progressive symptoms\

■Disturbs sleep

25
Pain with activity that decreases with rest usually indicates
mechanical pain from something being pinched, stretched, or contracted (tightened).
26
Pain and stiffness in the morning, which improves with activity, usually indicates
chronic inflammation & edema that decrease with activity.
27
Pain that is not affected by rest or activity usually indicates
bone pain, organic/systemic disorders, or other severe pathology.
28
Pain and aching that increase as the day progresses usually indicate
congestion (swelling) in the joint from the joint being overstressed.
29
Patterns of Inert Tissue Lesion
- Look for capsular patterns - Pain and limited ROM in all directions - Pain and excessive or limited ROM
30
Causes of Active Movement Abnormalities
Pain Muscle weakness Muscle paralysis Muscle spasm Tight or shortened structures Altered length-tension relationships Neuromuscular factors Joint-muscle interaction
31
Abnormal End Feels
- Muscle spasm - Spasticity - Capsular - Bone to bone - Empty - Springy block
32
Non Capsular Patterns
- Movement in one direction limited (e.g., impingement) - Internal derangement (e.g., meniscus) - Extra-articular lesion (e.g., adhesions)
33
A noncapsularpattern of restricted hip motion may be a sign of
pathology other than a joint problem associated with osteoarthritis
34
Empty end feel can be an indicator of
potentially serious disease such as infection or neoplasm. Empty end feel is described as limiting pain before the end range of motion is reached but with no resistance perceived by the examiner.
35
Vascular disorders such as Arterial Insufficiency-
pain is often throbbing in nature and exacerbated by activity. Look for Risk factors!
36
Cancer can cause
METS to the Hip- such as Prostate, Testicular, Breast and Ovarian cancer