Stroke symposium Flashcards

(53 cards)

1
Q

How many people have a stroke each year?

A

Over 100000 people in the UK

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2
Q

What is the lifetime risk for males of a stroke?

A

1 in 6

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3
Q

What is the lifetime risk for females of a stroke?

A

1 in 5

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4
Q

What is the cost of stroke to society?

A

£26billion

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5
Q

List some impacts of having a stoke

A
Lack in confidence
Fear of another stroke 
Difficult to talk about the stroke 
Feel friends and family treat them differently
Unable to care for family
Considered breaking up with partner
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6
Q

What are the symptoms of stroke?

A

Face
Arms
Speech

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7
Q

How many neurons die a minute in stroke?

A

1.9 million neurons

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8
Q

What is found in the clinical assessment of stroke?

A

Sudden onset of focal neurological or monocular symptoms
Symptoms and signs should fit within a vascular territory
Negative symptoms rather than positive symptoms

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9
Q

What examination is used in stroke?

A

Quick assessment of systems

Standardised score

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10
Q

Describe the classic presentation of an ACA stroke

A

Colateral lower limb

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11
Q

Describe the classic presentation of a left MCA infarct

A

Dysphasia, right sided weakness/numbness

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12
Q

Describe the classic presentation of a right MCA infarct

A

Neglect, left sided weakness/numbness

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13
Q

Describe the classic presentation of a brainstem infarct

A

May involve diplopia, visual field defect, facial weakness, colateral limb weakness/numbness, incoordination

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14
Q

What are the causes of a hemorrhagic stroke?

A

Anomalies in vessel arrangement - aneurysm

Arteriovenous anomaly

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15
Q

What are the causes of an ischaemic stroke?

A

Thrombosis

Emboli

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16
Q

How is an ischaemic stroke diagnosed?

A

CT scan

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17
Q

If there is an infarct what is shown on CT?

A

Attenuation

Different colours of the brain matter

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18
Q

Where is the characteristic hypertensive haemorrhage found?

A

In the centre of the brain

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19
Q

Describe critical ischaemia

A

High metabolic demand of the brain - no glucose stores
Physiological blood flow 50ml/100g/min
<20ml/100g/min - electrical function stops - neurons still alive potentially salvageable - reversible ischemia - limited time
<10ml/100g/min - neuronal death within minutes, irreversible ischaemia - cerebral infarction

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20
Q

What is CT perfusion imaging used for?

A

Tells which areas are salvageable and determine the timing of the stroke

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21
Q

What does the length of the clot tell you?

A

Efficacy of thrombolysis

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22
Q

What is stent retrieval?

A

Way of regaining blood supply

Mechanical thrombectomy

23
Q

Why is an MRI useful?

A

Determines cerebral oedma and sites of infarction quicker

24
Q

What drug is used?

25
What are the risk factors of stroke?
``` Smoking Diabetes High blood pressure Obesity Stress Hyperlipidemia LDL ```
26
What tests are done to determine what caused the clot?
Heart - echo (PFO), ECG (AF), ultrasound bifurcation (carotid stenosis)
27
What is used to prevent the carotid stenosis causing more problems?
Stent and remove the atherosclerotic plaque
28
Describe stroke pathophysiology
Endothelial injury - Increased vascular permeability, leukocyte adhesion Accumulation of lipoproteins - Including LDL and its oxidised forms Monocyte adhesion to the endothelium - Followed by migration into the intima and transformation into macrophages and foam cells. Platelet adhesion Factor release - From activated platelets, macrophages inducing smooth muscle cell recruitment. Smooth muscle cell proliferation Lipid accumulation
29
List the 5 types of ischaemic stroke
``` ●Large artery atherosclerosis ●Cardioembolism ●Small vessel occlusion ●Stroke of other determined etiology ●Stroke of undetermined etiology ```
30
What is vital in care after a stroke?
```  Nursing and medical staff  Functional and movement disability – OT/physiotherapy  Communication and swallowing function – S&LT  Nutritional support – Dieticians  Social service ```
31
What is the role of the speech therapist in acute stroke management
Initial screen of swallow on admission - NG/NBM/PEG Swallow therapy Initial language/speech screen Follow up language therapy MDT working and support Discharge planning and hand over to rehab or community teams
32
What happens in the community by the speech therapist?
``` Follow up on communication and swallow Usually at patients home Therapy Link to community support 6 month review Getting them back to hobbies ```
33
What is dysphagia?
Difficulty swallowing An impairment at any of the 4 stages of swallowing: (pre-oral, oral, pharyngeal ,Oesophageal) Can lead to aspiration pneumonia, malnutrition, dehydration, choking and death. Assessed at bedside via observation and palpation and/or through instrumental techniques such as Videofluroscopy or Fiberoptic Endoscopic Evaluation of Swallow (FEES). Can have a negative impact on mood and social participation
34
What is aphasia?
Difficulty talking
35
List some motor speech disorders
Dysarthria | Apraxia of speech
36
How can we help with complications of post stroke symptoms?
- Adaptation of posture - Environmental changes e.g. reducing distractions - Modifying diet and fluids e.g. use of thickener in drinks - Educating family/carers e.g. feeding techniques - Compensatory strategies e.g. head turn, chin tuck- Swallow rehab exercises such as Massako manoeuvre for weak soft palate. - Adaptive equipment e.g. cutlery with large handles, plate guards, bolus limiting cups- Feeding at risk decisions
37
What do you look out for in dysphagia?
Coughing whilst eating or drinking Eyes watering, shortness of breath, choking episodes, reduced sats just after swallowing New and/or recurrent chest infections (predominantly right sided consolidation, may indicate aspiration pneumonia)
38
What are the risk factors for dysphagia?
dependent on feeding and oral care, poor positioning, reduced dentition, comorbidities e.g. COPD, frailty, alertness, cognition
39
What is aphasia?
An acquired language impairment Can affect any of the 4 modalities of language: - speech/ auditory comprehension - reading/ writing Can have a devastating impact on the individual’s Quality of Life (QoL) and wellbeing
40
Which part of the brain is involved in aphasia?
Usually associated with damage to the left cerebral hemisphere
41
People with communication impairments may find it difficult to ...
``` take part in a conversation  talk in a group or noisy environment  read a book or magazine or road sign  understand or tell jokes  follow the television or radio  write a letter or fill in a form  use the telephone use the internet  use numbers and money  say their own name or the names of their family  unable to express their immediate needs or ideas or words  go out. ```
42
Describe aphasia expressive difficulties
``` word finding difficulties non-fluent output short, staggered sentences difficulty with past/present, he/she may miss words out ```
43
Describe aphasia receptive difficulties
Long muddled sentences Use of non-words e.g. ‘ploof’ for ‘dog’ Unable to understand what others are saying May not be aware of impairment
44
What does aphasia therapy focus on?
Specific impairments (usually 1:1) e.g. use of past tense - Functional communication e.g. practising how to order a coffee and then completing the task or learning compensatory strategies e.g. gesture and writing - Social participation e.g. developing communication techniques in group settings with family and friends or training carers to support communication.
45
What is dysarthria?
Difficulty in speaking resulting from a weakness or loss of control of the muscles used to make the sounds of speech, e.g. lips, tongue, soft palate, larynx Speech can sound slurred or unintelligible Varies from individual to individual – some people may not be able to form any words at all and other people may have only slightly imprecise articulation
46
What is apraxia of speech?
An inability to control the muscles used to form words. When the messages from the brain to the mouth are disrupted, the person cannot move his or her lips or tongue in the correct manner to make letter sounds. Speech is uncoordinated and effortful. Many sound and word errors e.g. ‘kitchen’ for ‘chicken’ Often very frustrating for the person
47
What is cognitive communication disorder?
Result from damage to frontal regions of the brain (predominantly right-sided) The frontal lobes are particularly important for cognitive communication skills because of their role in the brain's 'executive functions', including planning, organisation, flexible thinking and social behaviour. People with CCD may: - talk too much or not enough - interrupt conversation - jump from topic to topic - give too much or not enough eye contact - Over or under-share - Show impulsive behaviours such as spending lots of money online in one go - Have reduced facial expression
48
List some tips for communicating with people with communication difficulties
``` Don’t pretend you understand Write down key words Say one thing at a time Relax – be natural Recap – check you both understand Don’t rush – slow down, be patient Draw diagrams or pictures Reduce background noise Ask what helps, e.g. some people would prefer that others don’t finish their sentences for them Use pen and paper ```
49
What is post stroke rehabilitation
``` EARLY INDIVIDUALISED FOCUSED and SPECIFIC COMPLEX ONGOING ```
50
List some post stroke problems that impact of therapy
``` Joint/muscle stiffness Loss of muscle length Contractures Tissue Breakdown/Pressure Sores Unstable Shoulder Respiratory Complications Urinary Problems Pain Circulatory Problems Depression and Anxiety Osteoporosis CV Deconditioning Hygiene difficulties Oedema Constipation ```
51
What is hypertonia?
Made up of 1. Neural component 2. Biomechanical component CNS Damage - Direct result of blood alteration, ischaemia/haemorrhage, causing neuronal change and disordered information to be sent via corticospinal pathways. 2. Biomechanical - Muscle shortening and lengthening for cross bridges in muscle fibres depending on direction of pull
52
What is spasticity?
Resistance in 1 direction Characteristic posture changes Sensitive to sensory input
53
What is rigidity?
Resistance in all directions No static postural changes Not Sensitive to sensory input Cogwheel = rigidity plus tremor