Survival+ Response + Nervous Coordination Flashcards

1
Q

Why do organisms need to respond to changes in their environment

A

Avoid harmful environments, competition, predators
Keep in a favourable environment (correct temperature/humidity)

Grow towards/away from a stimuli
Dispersing to find new mates

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2
Q

Kinesis

A

Movement is random
Rate in change of movement

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3
Q

Taxes and the 2 types

A

Movement is directional towards or away from a stimulus

Positive taxis- towards a stimulus
Negative taxis- away from a stimulus

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4
Q

Earthworms moving away from light
What behaviour is this
List advantages of this

A

More chance of survival in soil
Les likely to dehydrate
More likely to find food and avoid predators

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5
Q

Positive tropism

A

Growth towards a stimulus

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6
Q

Negative tropism

A

Growth away from a stimulus
E.g plants grow away from light
Roots grow into soil
More likely to absorb water and mineral ions

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7
Q

What does the nervous system involve
Fast or slow?

A

Detection of stimuli by receptors
Transmission of nerve impulses/electrical by neurones
Response by effectors
Rapid response

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8
Q

What does the endocrine system involve
Fast or slow?

A

Communication by hormones
Slower and longer lasting response

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9
Q

CNS

A

Brain+ spinal chord

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10
Q

Peripheral nervous system

A

Nerves that originate from brain/spinal chord

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11
Q

Different types of neurones

A

Sensory
Relay
Motor

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12
Q

Whats the potential difference at resting potential
Whys it negative?

A

-70mV
More positive ions outside the cell than inside so inside slightly negative compared to the outside

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13
Q

How resting potential established

A

📈 conc of potassium ions inside cell
📈conc of sodium ions outside cell
Sodium potassium pump
Pumps 3 sodium ions outside the cell
Pumps 2 potassium ions inside the cell
Requiring energy released from ATP
Active transport of sodium ions outside of neurone
Different permeabillity to K+ and Na+
Membrane more permeable to K+

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14
Q

Voltage gated channel proteins in Resting neurone

A

Sodium ion voltage gates channel proteins r closed
So sodium ions cannot facilitatedly diffuse back in

Some potassium ion channel proteins are open as
Membrane more permeable to potassium ions so diffuse

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15
Q

Membrane potential in depolarisation

A

-70mV to 40mV

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16
Q

Depolarisation

A

Stimulus causes the membrane to be more permeable to Na*
Membrane reaches threshold then all Na+ voltage gated channel proteins open
Higher conc of Na+ ions outside cell than inside
So Na+ ions facilitatedly diffuse in
Higher conc of Na+ ions inside cell reverses resting potential
K+ ion voltage gates channel protein remains closed

+40mV

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17
Q

Repolarisation

A

Na+ voltage gates channel proteins close and K+ voltage gated channel proteins open
Higher conc of K+ inside cell than outside
So K+ diffuse out cell down conc gradient

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18
Q

Hyperpolarisation

A

K+ channel protein remain open for longer
Causes potential difference to go to -90mV
Sodium potassium pump restores resting potential back to -70mV

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19
Q

All or nothing response

A

Action potential only occur when membrane reaches threshold so all Na+ channel protein open
Minimum intensity of stimulus is threshold
Sub threshold= no action potential
Above threshold= action potential given

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20
Q

Refractory period

A

Limits number of impulses per second

When maximum frequency reached no further increase in info

Time taken to restore resting potential

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21
Q

Impulse transmission along the axon

A

Nerve impulse is the movement of an action potential along neurone
Action potential act as stimulus to adjacent polarised areas of the membrane and this causes the action potential to be passed along

22
Q

Synaptic transmission
Electrical impulses

A

Cannot jump the gap between neurones
Impulses transmitted between neurones across a synapse using neurotransmitters

23
Q

Synaptic transmission
Unidirectional

A

Impulses are unidirectional from
Presynaptic neurone to post synaptic neurone
Neurotransmitters are only found in the presynaptic neurone and neurotransmitter receptor proteins are only found in the post synaptic membrane

24
Q

How the synapse works

A

Action potential arrives at the synaptic knob in the presynaptic neurone and this causes Ca2+ voltage gated channel proteins to open
Ca2+ facilitatedly diffuse into synaptic knob as there is a higher conc in the synaptic cleft
Ca2+ activate enzymes which cause synaptic vesicles to move towards presynaptic membrane
Vesicle fuses with the presynaptic membrane releasing acetylcholine via exocytosis- requires ATP
Acetylcholine diffuses down a conc gradient across the cleft and binds to acetylcholine receptors in the post synaptic membrane as they are complementary
When it binds it causes Na+ channel proteins to open causing Na+ to diffuse in
If membrane reaches threshold then membrane becomes depolarised and action potential occurs (sub-threshold=no action potential)

In cholinergic synpase acetylcholinesterase hydrolyses acetylcholine into choline and ethanoic acid
Which diffuses across the synaptic cleft into preysnaptic neurone
Mitochondria releases Atp to resynthesises acetylcholine to be stored in vesicles in presynaptic neurone

25
Q

Points to remember for synpase

A

Synapse wil delay impulse slightly
(Refactory period)
Synapses prevent impulse going in wrong direction

26
Q

Neuromuscular junction

A

Synapse between a motor neurone and a muscle cell
Uses acetylcholine which binds to nicotinic cholinergic receptors

27
Q

Differences between neuromuscular junction and cholinergic synpase

A

Neuromuscular junctions have more folds so larger SA so MORE acetylcholinesterase enzymes hydrolyse acetylcholine at faster rate
More receptors on post synaptic membrane

Action potential always triggers a response in the muscle cell
( not the case for a synapse between two neurones)

28
Q

Summation
Whys it required
Two types

A

The build of neurotransmitters within the synapse
Sometimes the arrival of an impulse does not lead to sufficient release of enough neurotransmitters to depolarise the post synaptic membrane and generate action potential

Spatial
Temporal

29
Q

Fatigue

A

If the rate of transmitter release is higher than the rate at which it is being reformed- fatigue
The pre synaptic neurone cannot release enough neurotransmitters to generate an action potential in post synaptic neurone

30
Q

Excitatory neurotransmitters

A

They cause action potentials in post synaptic neurone by making the resting potential less negative so less Na+ required to reach threshold

31
Q

Inhibitory neurotransmitters

A

Cause the resting potential to be more negative hyperpolarised
Post synaptic membrane less likely to reach threshold and generate action potential

32
Q

The effects of drugs on synapses
2 ways

A

They can either
Stimulate nervous system by creating more action potentials- excitatory effect

They can create fewer action potential
Drugs may inhibit release of neurotransmitter or bind and block the receptors on post synaptic membrane

33
Q

Why are nerve impulses able to transmit an electrical impulse

A

Because there is a difference in electrical potential across the membrane

34
Q

Describe how inhibition if acetylcholineesterase affects action of synpase

A

Acetylcholine not broken down
Na+ continue to enter
Depolarisation continues. action potential

35
Q

Myelination affects rate of conduction of nerve impulse
How

A

Depolarisation only occurs at nodes of ranvier
Fewer nodes

36
Q

Binding of GABA causes Chloride ions to enter the post synaptic neurone
Explain how this inhibits transmission of nerve impulses

A

Neurone becomes more negative
More Na+ required to reach threshold
Depolarisation

37
Q

Potential across the membrane is reversed when an action potential is produced
How

A

Na+ channel proteins open
Allowing Na+ to diffuse in
Leads to depolarisation

38
Q

Other than temperature, give 2 other factors that affect speed of impusle

A

Myelination
Axon diameter

39
Q

What happens when calcium ion channel proteins close

A

Leas diffusion of calcium ions into the synaptic knob of presynaptic neurone
Synaptic vesicles dont fuse w the presynaptic neurone
So dont release neurotransmitter
So neurotransmitter doesn’t diffuse across the synapse and bind to receptor on post synaptic membrane
No action potential
No depolarisation as sodium ion channels dont open

40
Q

Damage to the myelin sheath of neurones can lead to problems controlling contracting muscles
Why

A

Action potentials travel slower
So muscles contract slower

41
Q

Na+ + K+ can only cross through the axon membrane through proteins
Why

A

Cannot pass through phospholipid bilayer
Because not lipid soluble

42
Q

After exercise some atp is used to re-establish resting potential
How

A

Sodium potassim pump
Sodium out potassium in

43
Q

Student did not carry our repeats but was able to carry our statistical test
Why

A

Collected paired data

44
Q

Resting potential -70mv to 0mV
After adding respiratory inhibitor
Why

A

Respiratory inhibitor so no ATP produced
No active transport of ions
No ner movement of sodium and potassium ions

45
Q

Explain why the speed of transmission of impulses is faster along a myelinated axon than along a non-myelinated axon

A

Myelinated axon provides electrical insulation and saltatory conduction
Depolarisation only occurs at the nodes of ranvier
However in non-myelinated axon depolarisation occurs along whole length of axon

46
Q

Features of axons that conduct impulses faster and why

A

Myelinated
Shows saltatory conduction
Large diameter so larger SA of channel proteins
Less resistance of flow of ions

47
Q

Synapses ensure that nerve impulses only travel in one direction
How

A

Neurotransmitters only stored in presynaptic neurone
Neurotransmitter receptor proteins only found in post synaptic neurone

48
Q

Simple reflex arc

A

Only involves 3 neurones

49
Q

IAA in the shoots

A

Promotes cell elongation
IAA synthesises in the shoot tips and diffuses into the growing regions
Light causes movement of IAA from the light side to shaded side
Higher conc of IAA on the shaded side
Causes directional growth as there is greater cell elongation on shaded side
Shaded side grows faster and causes shoot to bend towards the light

50
Q

IAA in the roots

A

Inhibits cell elongation
IAA synthesised in the root tips ans then diffuses into the growing regions
Light causes movement of IAA from light side to shaded side and towards gravity
Higher conc of IAA on the shaded side
Cell elongation inhibited on shaded side
Results in directional growth away from light and towards gravity

51
Q

Binding of GANA causes chloride ions to enter post synaptic neurones
Explain how this inhibits transmission of nerve impulses

A

Neurone becomes more negatibe
More Na+ required to reach threshold
For ACTION POTENTIAL/DEPOLARISATION