Thorax and Lungs Ch. 19 Flashcards

(68 cards)

1
Q

What is the sternal angle and what is its signifcants?

A

It is a useful place to start counting ribs as it is continuous with the 2nd rib. Palpate the sternal angle, and slide down to the 2nd intercostal. Each intercostal space is numbered by the rib above it.
This is also where the trachea bifurcates

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2
Q

What is the coastal angel

what is the costoverterberal (CVA)

A
  • the angel under the anterior ribs, should be 90 degrees (>90 = barrel chest)
  • where the ribs meet the spine, location of fist percussion
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3
Q

what is the supra aspect of teh lungs called

A

apices

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4
Q

describe the lobes of the right and left lung

A

right: 3 lobes, RML is at the 4th and 5th ICS
left: 2 lobes
apices are above the clavical, anterior LL end at the 6th ICS, posterior LL end at teh 9th ICS

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5
Q

Important health history questions for respiratory (12)

A

Coungh (productive, dry), SOB, allergies (asthma), past lung injury, URIs, current meds (PMH), O2 at home, last TB sink test/ X-ray, PNA vaccine (>65, or immuno comp), flu vaccine, family history (CF, asthma), smoking (present/ past, pacs/ yr), chindren exposure to 2nd/ 3rd hand smoke?

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6
Q

5 A’s to help smokers quit

A

1) Ask- about his or her tobacco use status at every visit
2) Advise- Give clear, nonjudgmental, and personalized suggestions for quitting
3) Assess- each person’s readiness for and interest in quitting
4) Assist- each person with a specific cessation plan that includes medications, behavioral modification, exercise programs, or referrals
5) Arrange- follow-up visits

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7
Q

Problem based question to ask regarding a cough (3)

A

OPQRSTU
Constant, or does it come and go? Associated with specific activities, positions, or other symptoms i.e. fever?Describe your cough (Dry, Congested, Wet, Barking, Hacking)
Are you coughing up sputum

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8
Q

What questions are asked about sputem and what it means

A
What is color of sputum: 
clear= vital
white
green= bacterial
bloody= ulcer

Consistency of sputum:
thick, thin, frothy (PE, heart failure, ARDS)

Does it have an odor? yes= bacterial

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9
Q

what is bloody sputem called

A

hemoptysis

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10
Q

Problem based question to ask regarding SOB (4)

A

OPQRSTU
How many stairs or blocks can you walk before you become SOB?
Does it wake you up at night? (Paroxysmal nocturnal dyspnea), How many pillows do you use? (orthopnea)
Does anything seem to trigger episodes or make it worse?
What do you do to relieve symptoms?

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11
Q

Inability to breath when lying down:

A

orthopnea

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12
Q

An attack of severe shortness of breath and coughing that generally occurs at night

A

Paroxysmal nocturnal dyspnea

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13
Q

Pleuritis
what it is
s/s

A

(inflammation of pleura) = Pleural friction rub
can hear the rub with breathing. Can also occure in the heart; have the pt. hold theit breath while ascultating, sound will stop if it is the lungs and continue if it is the heart.
severe angina

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14
Q

Chostochondritis
what it is
s/s

A

Inflammation of the cartilage that connects a rib to the sternum. Pain might mimic that of a heart attack

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15
Q

physical exam: inspection of overall appearence (3)

A

1) General appearance (LOC may mean acute hypoxia), posture (tripoding, not wanting to lay down), breathing effort (symmetry in chest expansion, pursed liped breathing, nostral flaring)
2) Quality of respirations (rate, pattern, effort)
3) Nails, skin, lips (cyanosis, pallor)

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16
Q

Quality of respirations (3)

A

Rate (older adult breathes after, babies may breath irregular)
Breathing pattern
Effort (SOB, retractions, accessory muscles)

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17
Q
Breating patterns:
Cheyne Stoke
Tachypnea
Kussmaul
Hypoventilation
Bradypnea
A

1) Cheyne Stoke: tachypnea followed by apnea
2) Tachypnea: fast and shallow breaths (anything that causes SOB)
3) Kussmaul respirations= Hyperventilation: anxiety, DKA (diabetic ketoacidosis)
4) Hypoventilation: shallow slow irregular breaths (sedative (opioid) overdose, abd pain)
5) Bradypnea: slow breathing (least common), late sign of increased ICP

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18
Q

Physical exam: Anterior chest and posterior thorax

A

IPPA for both

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19
Q

Physical exam: Anterior chest INSPECTION (7)

A

Symmetry, Retractions, Accessory muscle use, Muscle development, Lesions, Anteroposterior (AP) diameter, Costal angle

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20
Q

What is the Anteroposterior (AP) diameter
what is it compared to
how is it documented
what can it be a sign of

A

measurement from anterior to posterior. AP should be > transvers
(documented: AP>T, 1:2)
If AP=T could be a sign of barrel chest (which is a sign of COPD)

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21
Q

What can asymmetry of the chest mean?

A

PNA, pneumonthorax, plural effusion

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22
Q

Physical exam: Anterior chest PALPATION

A
Symmetric chest expansion
Tenderness, lumps, masses?
Skin temp, moisture
Crepitus 
Tactile Fremitus
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23
Q

What can Crepitus signify?

A

= subcutaneous emphysema, feels like how rice crispy cereal sounds

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24
Q

Tactile Fremitus palpation
how to palpate
how to assess what you feel
where are vibrations the strongest

A

Use ball of fingers or ulnar edge of hand to feel for vibrations, while patient says “99” or “blue moon”. Using both hands start on top of shoulders, and then palpate at 3 locations down the midclavicular line, ending at the 6th ICS (where the lung ends)

Assess bilaterally; symmetry is most important (asymmetry could mean consolidation, PNA)

Vibrations stronger around sternum where major bronchi are closest to chest wall

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25
How does consolidation effect breath sounds
Consolidation enhances the transmission of breath and voice sounds!!
26
what is Rhonchal Fremitus
palpable vibrations with inhalation (may be caused by mucous in brachioli)
27
Physical exam: Anterior chest PERCUSSION and ASCULTATION | how to percuss/ ascultate
start at the supraclavicular moving in a digital 2, and then palpate at 4 locations down the midclavicular line the 5th is closer to the axillary line, ending at the 6th ICS (where the lung ends).
28
how to assess what you hear during percussion
Resonance is the normal finding. Hyperresonance= decreased density and increased air (eg: emphasema). Dullness= organs or fluid.
29
how to assess what you hear during ascultation
Broncheal (tracheal) Loud, hollow Inspiration < Expiration Bronchovesicular (midsternal line) Moderate amplitude Inspiration = Expiration Vesicular (between ICS: supreclavicular to 6th ICS) Soft, rustling Inspiration > Expiration Regardless of the type of normal breath sounds you hear, we report and document normal lung sounds as “CLEAR” (no adventitious sounds)
30
what can Broncheal in the LL mean (think about it!!)
PNA, b/c Consolidation enhances the transmission of breath and voice sounds!!
31
PNA what it is what causes it Clinical findings:
pneumonia an infection that inflames the air sacs in one or both lungs. The air sacs may fill with fluid or pus (purulent material), causing cough with phlegm or pus, fever, chills, and difficulty breathing. A variety of organisms, including bacteria, viruses and fungi, can cause pneumonia clinical findings: tachypnea, asymmetric chest expansions, fever, elevated vitals, increased fremitus at LL, coarse crackles.
32
physical exam: posterior thorax (IPPA)
Inspect: symmetry, shape (kyphosis or scoliosis limiting thoracic excursion?), skin Palpate: chest expansion, tactile fremitus (avoid scapula bones and spine... vibrations will be strongest between scapula) Percuss: same areas you palpated for fremitus. Should hear resonance! Auscultate: should hear vesicular, clear lung sounds Lungs extend to the 9th ICS
33
what are the 6 adventitious breath sounds
1) Coarse crackles (rales) 2) Fine crackles (rales) 3) Pleural friction rub 4) Wheeze (high-pitched) 5) Wheeze (low-pitched) 6) Stridor
34
Coarse crackles (rales):
Low-pitched bubbling sounds; heard on inspiration and/or expiration; may decrease with cough/suctioning. Could mean mucous, PNA, pulm fibrosis, chronic bronchitis. upperairway.
35
Fine crackles (rales):
High-pitched crackling, popping sounds upon inspiration. Could mean Pulm edema (HF), PNA, chronic bronchitis. Fluid filled alvioli.
36
Pleural friction rub:
Grating, low-pitched (like walking on snow). Could mean pleuritis.
37
Wheeze (high-pitched):
High-pitched, musical, squeaking; usually expiratory. Could mean Airway obstruction from tumor, asthma, emphysema.
38
Wheeze (low-pitched):
AKA rhonchi; Snoring, moaning sounds. Could mean Bronchitis (upper airway).
39
Stridor:
EMERGENCY: High-pitched crowing sound; louder in neck. Could mean croup, obstructed airway.
40
physical exam: infants and children (4)
- Obligate nose breathers until 3 months - Newborn RR 30-40 bpm and may be irregular; count for full minute - AP diameter of 1:1 is a normal finding in individuals <6yo - Thin chest walls may cause you to hear louder bronchovesicular breath sounds throughout the lung fields; Percussion may yield hyperresonance
41
Developmental Competence: Pregnancy (4)
- Enlarging uterus elevates diaphragm which decreases vertical diameter of the thorax; compensates by widening horizontally - Increase in tidal volume (TV) to meet increased oxygen demand (deeper respirations) - Wider costal angle - Some report mild dyspnea
42
Developmental Competence: older adult (3)
- Costal cartilages become calcified, and lungs are more rigid causing lung bases become less ventilated and alveoli become closed off and thus less surface area is available for gas exchange and increased risk of postoperative pulmonary complications (atelectasis and infections) - Kyphosismay make thoracic expansion more difficult. - Older people fatigue more quickly during auscultation.
43
Atelectasis what it is Clinical findings risk
collapsed alveoli (may be one section or entire lung); caused by external pressure from tumor, fluid, or foreign body. Most common post opp. complication. Clinical findings: Affected lobe has diminished or absent breath sounds. Oxygen saturation may be low. Decreased fremitus. Dullness with percussion. Can turn into PNA
44
Chronic Bronchitis what it is cause Clinical findings
AKA smokers cough. Characterized by hypersecretion of mucus by goblet cells of trachea and bronchi resulting in productive cough. Caused by irritants such as cigarette smoke and air pollution or by infection. Clinical findings: Productive cough, increased mucus production (green mucous= bacterial infection on top of the bronchitis), and dyspnea. When mucus occludes alveoli, coars crackles or wheeze (low pitch) may also be heard.
45
Emphysema what it is cause Clinical findings
destruction of pulmonary connective tissue that causes permanent abnormal enlargement of air spaces (hyper-inflated lung). Cause: long-term exposure to irritants that damage your lungs and the airways (smoking) Clinical findings: Classic appearance: underweight with barrel chest and SOB. Exam findings: diminished breath and voice sounds;possible wheezing on auscultation; decreased tactile fremitus; hyperresonant tones on percussion
46
Asthma/ RAD what it is Clinical findings
RAD= Reactive Airway Disease Hyperreactive airway disease characterized bybronchoconstriction, airway obstruction, and inflammation. Clinical findings: Tachypnea with prolonged expiration. Audible wheeze. Dyspnea; Anxiety; Cough. Use of accessory muscles. Auscultation: wheezy and diminished
47
COPD
Chronic Obstructive Pulmonary Disease. | chronic emphasema and bronchitis
48
Pleural Effusion what it is Clinical findings
accumulation of serous fluid in pleural space (outside of the lung) Clinical findings: If large, there may be dyspnea or decreased chest wall movement. Decreased tactile fremitus. Percussion: dull (over fluid). Decreased breath sounds
49
PE what it is Clinical findings
Pulmonary Edema fluid inside of the lung caused by a heart condition (back flow from the left heart). Other causes include PNA.
50
Pneumothorax what it is Clinical findings
Partial or complete lung collapse resulting from air in pleural spaces. Clinical findings vary, depending on amount of lung collapse. Pt may be slightly SOB, anxious or in severe distress. Decreased chest wall movement Hyper-resonance on percussion. Breath sounds decrease or absent on affected side. Tracheal displacement
51
TB what it is cause Clinical findings
tuberculosis: contagious (airbourn), bacterial infection caused by Mycobacterium tuberculosis. Primarily in lungs, but kidney, bone, lymph node, and meninges can also be involved. Clinical Findings: usually asymptomatic early on. May develop fatigue, anorexia, weight loss, fever, night sweats, crackles in apices, and cough (possible blood tinged/rust sputum)
52
Lung cancer cause Clinical findings
Most fatal of all cancers: 85% caused by tobacco smoking Clinical findings: Most common initial symptom reported is a persistent cough. Weight loss, hemoptysis,SOB, Nail clubbing. If tumor is causing partial obstruction can cause atelectasis or wheezing. Percussion may be dull over a large tumor.
53
ARDS what it is cause Clinical findings
Acute Respirtory Distress Syndrome a type of respiratory failure characterized by rapid onset of widespread inflammation in the lungs. High mortality. results from trauma or shock (but most often sepsis) leading to increased permeability of pulmonary capillaries. Leads to pulmonary edema. Clinical findings: Restlessness, dyspnea, Ventilatory support often required; suctioning will reveal frothy sputum
54
What are the possible causes: | Upon inspection on the pt. you notice unequal chest expansion:
severe atelectasis, PNA, effusion, pneumothorax, trauma
55
What are the possible causes: | Upon inspection on the pt. you notice they have a barrel chest, pursed lip breathing and hypertrophied muscles
COPD, emphysema
56
What are the possible causes: | Upon palpation of the pt. you notice increased fremitus
consolidation due to PNA
57
What are the possible causes: | Upon palpation of the pt. you notice decreased fremitus
obstruction, effusion, pneumothorax, emphysema
58
What are the possible causes: | Upon palpation of the pt. you notice crepitus
subcutaneous emphysema
59
What are the possible causes: | Upon percussion of the pt. you notice dullness
increased density of the lungs- PNA, effusion, atelectasis, tumor
60
What are the possible causes: | Upon percussion of the pt. you notice hyperresonance
increased air- pneumothorax, emphysema
61
What are the possible causes: | Upon ascultation of the pt. you hear crackles
PNA, bronchitis, pulm fibrosis, pulm edema (ARDS)
62
What are the possible causes: | Upon ascultation of the pt. you hear wheezes
asthma, emphysema
63
What are the possible causes: | Upon ascultation of the pt. you hear stridor
epiglottitis, croup
64
What are the possible causes: | Upon ascultation of the pt. you hear plural effusion rub
pleuritis
65
Hypercapnia
increased CO2 in the blood
66
Egophony how is the test done normal abnormal
(Greek: “the voice of a goat”) Auscultate the chest while the person phonates a long “ee-ee-ee-ee” sound Normally you should hear “eeeeeeee” through your stethoscope Over area of consolidation or compression the spoken “eeee” sound changes to a bleating long “aaaaa” sound
67
Bronchophony how is the test done normal abnormal
Ask the person to repeat “ninety-nine” while you listen with the stethoscope over the chest wall; listen especially if you suspect pathology Normal voice transmission is soft, muffled, and indistinct; you can hear sound through the stethoscope but cannot distinguish exactly what is being said Pathology that increases lung density enhances transmission of voice sounds; you auscultate a clear “ninety-nine” The words are more distinct than normal and sound close to your ear
68
Whispered Pectoriloquy how is the test done normal abnormal
Ask the person to whisper a phrase such as “one-two-three” as you auscultate The normal response is faint, muffled, and almost inaudible With only small amounts of consolidation, the whispered voice is transmitted very clearly and distinctly, although still somewhat faint; it sounds as if the person is whispering right into your stethoscope, “one-two-three”