TOPIC 14 - Integumentary : diseases of skin, eye, and ear Flashcards

(68 cards)

1
Q

skin cancer major causes

A

Overexposure to sunlight is the major cause of skin cancer, although other factors are associated.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

most common skin cancer types

A

actinic or solar keratosis, squamous cell carcinoma, basal cell carcinoma, and melanoma. These are involved in cellular regulation of growth.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what is a nevus

A

a mole, is a benign growth of the pigment-forming cells classified according to their location within the layers of the skin.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what treatment is being tried to treat metastatic melanoma

A

experimental therapy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

who is skin cancer likely to develop in

A

light-skinned races
individuals older than 60 years of age
outdoor workers
higher altitudes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

ABCDE

A

asymmetry
border
color
diameter
evolving

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

how often should TSSE be performed

A

monthly

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

surgeries for skin cancer

A

Cryosurgery
Curettage and electrodesiccation
Excision
Mohs’ surgery
Wide excision

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

nonsurgical therapy for skin cancer

A

radiation
medications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

malignant melanoma

A

a tumor arising in melanocytes, the cells producing melanin. Melanoma causes the majority of skin cancer deaths.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

where can melanoma occur

A

in the eyes, ears, gastrointestinal tract, and oral and genital mucous membranes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is it called when melanoma begins in the skin

A

cutaneous melanoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

characteristics of melanoma

A

can be Irregular color, surface, and border Variegated color, including red, white, blue, black, gray, brown, *Flat or elevated. Eroded or ulcerated, Often <1 cm in size

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

most common sites of melanoma in male vs female

A

Most common sites in males are trunk, head and neck. In females are legs, then back metastasize to any organ, including the brain and heart.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

treatment of melanoma

A

Wide surgical excision and possible sentinel lymph node evaluation
Possible use of adjuvant therapy after surgery if lesion >1.5 mm in depth
Correlation between survival rate and depth of invasion
Poor prognosis unless diagnosed and treated early
Spreading by local extension, regional lymphatic vessels, and bloodstream

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

psoriasis

A

Sharply demarcated silvery scaling plaques on reddish colored skin commonly on the scalp, elbows, knees, palms, soles, and fingernails

Itching, burning, pain

Localized or general, intermittent or continuous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

topical treatments for psoriasis

A

Corticosteroids, tar, salicylic acid,
Intralesional injection of corticosteroids for chronic plaques

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

systemic treatments for psoriasis

A

Natural or artificial UVB. PUVA (UVA with topical or systemic photosensitizer

Antimetabolite (methotrexate)

immunosuppressant (cyclosporine)

biologic therapy (adalimumab [Humira], etanercept [Enbrel], infliximab [Remicade]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

shingles

A

activated varicella zoster virus
Potentially contagious to anyone who has not had varicella or who is immunosuppressed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

clinical manifestations

A

Linear distribution along a dermatome of grouped vesicles and pustules on erythematous base resembling chickenpox
Usually unilateral on trunk, face, and lumbosacral areas
Burning, pain, and neuralgia preceding outbreak
Mild to severe pain during outbreak

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

topical treatment for herpes

A

Wet compresses, silver sulfadiazine (Silvadene) to ruptured vesicles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

systemic treatment for herpes

A

Antiviral agents within 72 hr to prevent postherpetic neuralgia
Analgesia. Mild sedation at bedtime
Gabapentin (Neurontin) to treat postherpetic neuralgia
Usually heals without complications, but scarring and postherpetic neuralgia possible
Vaccine (Zostavax) to prevent shingles for adults ≥50 yr

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

cataract

A

Opacity within the lens in the eye of the patient
Cataract may be in one or both eyes
If in both eyes, one eye may affect the person’s vision more than the other

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

causes of cataracts

A

Age
Blunt or penetrating trauma
Congenital factors such as maternal rubella
Radiation exposure or ultraviolet (UV) light (sunlight) exposure
Certain drugs such as systemic corticosteroids or long-term topical corticosteroids
Ocular inflammation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
signs and symptoms of cataracts
Decrease in vision Abnormal color perception Glare (may be significantly worse at night when the pupil dilates) Visual decline is gradual, but the rate of cataract development varies from patient to patient.
26
nonsurgical therapy for cataracts
Change in glasses prescription Strong reading glasses or magnifiers Increased lighting Lifestyle adjustment
27
preoperative care for cataracts surgery
Mydriatic, cycloplegic agents Nonsteroidal anti-inflammatory drugs Topical antibiotics Antianxiety medications
28
surgery for cataracts involves
* Removal of lens * Phacoemulsification * Extracapsular extraction * Correction of surgical aphakia * Intraocular lens implantation (most frequent type of correction) * Contact lenses
29
postoperative care for cataracts surgery
Topical antibiotic Topical corticosteroid or other anti-inflammatory agent Mild analgesia if necessary Eye patch or shield and acti­vity as prescribed by patient's surgeon Activity restriction: Activities that increase IOP: bending, stooping, coughing, or lifting
30
education for patients after eye surgery
Proper hygiene and eye care techniques to ensure that medications, dressings, and/or surgical wound is not contaminated during eye care How to monitor pain, take pain medication, and report pain not relieved by medication Importance of continued follow-up as recommended to maximize potential visual outcomes Signs and symptoms of infection and when and how to report these to allow for early recognition and treatment of possible infection Importance of complying with postoperative restrictions on head positioning, bending, coughing, and Valsalva maneuver to optimize visual outcomes and prevent increased intraocular pressure How to instill eye medications using aseptic techniques and adher­ence with prescribed eye medication routine to prevent infection
31
two types of macular degeneration
age related or wet
32
risk factors for macular degeneration
genetics, family history, white ethnicity, chronic inflammatory conditions, smoking, hypertension
33
which type of macular degeneration is most common
age related
34
dry macular degeneration
90% of all cases close vision tasks are becoming more difficult. macular cells start to atrophy slowly progressive and painless vision loss.
35
wet macular degeneration
more severe form more rapid onset development of abnormal blood vessels in or near the macula
36
etiology of macular degeneration
Related to retinal aging Genetic factors Family history Gene responsible Long-term exposure to UV light Hyperopia cigarette smoking light-colored eyes Nutritional factors: Vit C, E, and zine decreases progression
37
what vitamin supplements decrease the progression of advanced macular degeneration
vitamin C, vitamin E, beta-carotene, and zinc
38
manifestations of macular degeneration
Blurred and darkened vision, scotomas (blind spots in the visual field), Distortion of vision.
39
treatment for macular degeneration
 Limited treatment Medications that are injected directly into the vitreous cavity.  “Cold” laser to excite the dye.  People at risk for developing advanced AMD should consider supplements of vitamins and minerals (in consultation with their health care provider).  
40
retinal detachment
A retinal detachment is a separation of the sensory retina with fluid accumulation between the two layers.
41
causes of retinal detachment
Increasing age Severe myopia Eye trauma Cataract surgery Family or personal history of retinal detachment
42
signs symptoms of retinal detachment
Patients with a detaching retina describe symptoms that include photopsia (light flashes); floaters; and a “cobweb,” “hairnet,” or ring in the field of vision. Once the retina has detached, the patient describes a painless loss of peripheral or central vision, “like a curtain” coming across the field of vision.
43
medical treatment for retinal detachment
Laser Photocoagulation & Cryopexy Scleral Buckling Pneumatic retinopexy Vitrectomy
44
glaucoma
Increased intraocular pressure, optic nerve atrophy, resulting in peripheral visual field losses
45
eye drop medications
B-Adrenergic blockers, a-Andrergic Agonists
46
normal IOP vs acute angle closure glaucoma vs open angle glaucoma
normal is 10 to 21 mm Hg In open-angle glaucoma, IOP is usually between 22 and 32 mm Hg. In acute angle-closure glaucoma, IOP may be over 50 mm Hg.
47
POAG progression
primary open angle glaucoma develops slowly and without symptoms of pain or pressure. The patient usually does not notice the gradual visual field loss until peripheral vision has been severely compromised. Eventually the patient with untreated glaucoma has “tunnel vision” in which only a small center field can be seen, and all peripheral vision is absent.
48
acute angle closure glaucoma symptoms
causes definite symptoms, including sudden, excruciating pain in or around the eye. This is often accompanied by nausea and vomiting. Visual symptoms include seeing colored halos around lights, blurred vision, and ocular redness.
49
chronic open angle glaucoma drug therapy
*β-Adrenergic blockers *α-Adrenergic agonists *Cholinergic agents (miotics) *Carbonic anhydrase inhibitors
50
acute angle closure glaucoma therapy
* Topical cholinergic agent * Hyperosmotic agent * Laser peripheral iridotomy * Surgical iridectomy
51
external otitis
Inflammation or infection of the epithelium of the auricle and ear canal
52
causes of external otitis
Swimmer’s ear: due to chemical and contaminated water Using a sharp object in ear Bacteria: Pseudomonas aeruginosa Fungi: Candida Albicans and Aspergillus
53
symptoms of external otitis
Pain is the first sign Discomfort may be during chewing, moving the auricle, pressing on the tragus May have serosanguineous or purulent drainage Fever
54
treatment of external otitis
Exam C & S may be done Moist heat, mild analgesics, and topical anesthetic drops to control pain Antibiotics and corticosteroids
55
when will patients see improvement with antibiotics
within 48 hours still need to complete course (7-10 days)
56
acute otitis media
infection of the tympanum, ossicles, and space of the middle ear
57
causes of acute otitis media
Swelling of the auditory tube from colds or allergies can trap bacteria Pressure from the inflammation pushes on the TM, causing it to become red, bulging, and painful. Infection can be due to viruses or bacteria.
58
otitis media with effusion
an inflammation of the middle ear with a collection of fluid in the middle ear space.
59
chronic otitis media and mastoiditis
Repeated attacks of otitis media may lead to chronic otitis media
60
symptoms of chronic otitis media and mastoiditis
purulent exudate and inflammation that can involve the ossicles, auditory tube, and mastoid bone Painless, hearing loss, nausea, and episodes of dizziness can occur. Hearing loss is a complication from inflammatory destruction of the ossicles, a TM perforation, or accumulation of fluid in the middle ear space
61
treatment for chronic otitis media and mastoiditis
IV or oral antibiotics Tympanoplasty (myringoplasty) involves reconstruction of the TM and/or the ossicles
62
post op care for Tympanoplasty
Impaired hearing is expected A cotton ball dressing is used for the incision Instruct the patient to change the cotton packing as needed.
63
Ménière's disease symptoms
episodic vertigo Tinnitus fluctuating sensorineural hearing loss aural fullness.
64
when do symptoms usually begin for Ménière's disease 
age 30-60
65
possible causes of Ménière's disease 
excessive accumulation of endolymph in the membranous labyrinth
66
symptoms during attacks of Ménière's disease 
Attacks may be preceded by a sense of fullness in the ear, increasing tinnitus, and muffled hearing. The patient may experience the feeling of being pulled to the ground (“drop attacks”). Some patients report that they feel like they are whirling in space. Attacks may last hours or days and may occur several times a year. The clinical course of the disease is highly variable.
67
diagnostic tests for Ménière's disease 
Hearing (Audiometric) Tests Vestibular Tests for balance Inner ear assessment: electronystagmography, glycerol test
68
acute nursing interventions for Ménière's disease 
Bed rest Quiet dark room Avoid sudden head movements and position changes Avoid fluorescent lights & flashing lights Sedation Ambulate with assistance only, not independent Call light within reach Side rails up Fall precautions Monitor Input & Output Antiemetics – Prochlorperazine Antivertigo – Meclizine Verbal & tactile reassurance and support