TOPIC 7 - GI system Flashcards

(45 cards)

1
Q

GI diseases

A

GERD, hiatal hernia, peptic ulcer disease, gastritis, gastroenteritis, inflammatory bowel disease (crohns and colitis), diverticulitis, IBS, intestinal or bowel obstruction, bowel surgery, ostomies, bariatric surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

symptoms of GERD

A

heart burn, burning, tight sensation under lower sternum, spreading towards throat or jaw, felt intermittently

chest pain can mimic angina, relieved with antacids

resp : wheezing, coughing, dyspnea, nocturnal discomfort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

predisposing factors of GERD

A

incompetent lower esophageal sphincter
decreased LES pressure
increased intraabdominal pressure
hiatal hernia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

GERD complications

A

Barretts esophagus
esophageal varices
esophageal ulcers
respiratory (from irritation of upper airway) : cough, bronchospasm, laryngospasm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

diagnostic tests

A

upper GI endoscopy
ambulatory esophageal pH monitoring
radionuclide tests

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

meds

A

PPIs, H2R blockers, acid protective, pro kinetic drugs, antacids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

PPIs

A

promote esophageal healing
ex : omeprazole
s/e : headache

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

H2R blockers

A

decrease secretion of HCl
reduce symptoms and promote esophageal healing
ex : famotidine
uncommon s/e

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

acid protective

A

cytoprotective properties
ex : sucralfate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

prokinetic drugs

A

promote gastric emptying
reduce risk of reflux
ex: metoclopramide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

antacids

A

quick but short lived relief
neutralize HCl
taken 1-3 hours after meals
ex : maalox, mylanta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

interventions for GERD

A

elevate HOB 30 degrees
do not lie down for 2-3 hours after eating
avoid smoking, alcohol, acidic foods
stress reduction
weight reduction
small, frequent meals

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

surgical therapy for …

A

patients with med intolerance, barretts meaplasia, esophageal stricture and stenosis, chronic esophagitis, failure of conservative therapy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

nissen fundoplication

A

treat more than 1 clinical condition

fundus of stomach is wrapped around distal esophagus and sutured to itself

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

hiatal hernia - sliding

A

stomach slides through hiatal opening in diaphragm when patient is supine, goes into abdominal cavity when patient is standing upright

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

hiatal hernia - rolling

A

fundus and greater curvature of stomach stomach roll up through diaphragm, forming a pocket alongside the esophagus
Paraoesophageal junction remains in normal position
Acute paraoesophageal hernia is a medical emergency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

complications of hiatal hernia

A

GERD, esophagitis, hemorrhage, stenosis, ulcerations of herniated portion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

diagnostics

A

esophagogram (show protrusion of mucosa)
endoscopy (visualize lower esophagus)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

surgical therapy

A

gastropexy : anti-reflux procedure, attachment of stomach sub-diaphragmatically
herniotomy : reduction of herniated stomach, excision of hernia sac
herniorrhaphy : closure of hiatal defect

20
Q

first indication in geriatric population

A

esophageal bleeding or respiratory complications

21
Q

invasive imaging for diagnostics patient prep

A

NPO, bowel prep, take normal meds, IV access for sedation, cardiac monitor, labs: electrolytes and CBC,
monitor LOC, RR, O2, vitals

risk for perforation

22
Q

causes of peptic ulcer disease

A

Hydrochloric acid & pepsin
Helicobacter Pylori
Medications (aspirin, NSAIDS, corticosteroids, anticoagulants, SSRIs)
Lifestyle (excessive ETOH, coffee, smoking, stress)

23
Q

peptic ulcer disease :

A

erosion of GI mucosa from action of HCl, affecting lower esophagus, stomach, and duodenum

24
Q

acute peptic ulcer disease

A

superficial erosion, minimal inflammation, short duration, resolves when cause treated

25
chronic peptic ulcer disease
erodes through the muscular wall, present continuously for years or intermittent through life, most common type of ulcers
26
diagnostic studies for peptic ulcer disease
endoscopy with biopsy, invasive tests (endoscopic procedure or biopsy), noninvasive (urea breath test), stool antigen test, serum or whole blood antibody test (IgG - wont distinguish between past and current infection) barium contrast study (patients who cant undergo endoscopy, not for superficial ulcers, diagnose gastric outlet obstruction) gastric analysis (content for acidity and volume, NG tube, analyze for HCl) labs (CBC, liver enzymes, serum amalyse, stool exam)
27
meds for peptic ulcer
PPIs H2R blockers Antibiotics Antacids Anticholinergics Cytoprotective therapy
28
antibiotics for peptic ulcer
Eradicates H. pylori infection No single agent has been effective in eliminating H. pylori Prescribed concurrently with a PPI for 7 to 14 days
29
complications of ulcers
hemorrhage (hematemesis, melena, EGD, type and screen, PRBC give and hold, IV access, O2) perforation (sudden, sharp, epigastric pain, peritonitis, rebound tender, rigid) gastric outlet obstruction (edema, inflammation, pain worse at end of day, burping, constipation)
30
surgical interventions and complications
partial gastrectomy, vagotomy, pylorplasty dumping syndrome, posprandial hypoglycemia, bile reflux gastritis
31
gastritis
Breakdown of the normal mucosal barrier Causes: Drugs, Diet, Microorganisms, Environment, diseases, and other factors Nausea, vomiting, anorexia, epigastric tenderness, feeling of fullness, gi bleed Chronic Management: remove causes, manage symptoms, treat pernicious anemia for stomach tissue atrophies
32
gastroenteritis
Inflammation of the mucosal lining Causes: bacterial, viral, food contamination Table 42-1, Harding et al… Sudden diarrhea, nausea, vomiting, fever, abdominal cramping Self-limiting Management: fluids replacement, antipyretics
33
foods that trigger exacerbations
Lactose intolerance High-fat foods Cold foods High-fiber foods
34
goals of treatment of IBD
Rest the bowel Control inflammation Combat infection Correct malnutrition Alleviate stress Relieve symptoms Improve quality of life
35
nutritional therapy during acute exacerbations
regular diet not tolerated liquid enteral feedings (high calorie, lactose free, easily absorbed, low fiber)
36
goal of drug treatment
induce and maintain remission
37
meds
Aminosalicylates Antimicrobials Corticosteroids Immunosuppressants Biologic and targeted therapy
38
bowel obstruction causes
mechanical : tumor, adhesion, stricture vascular : interference of blood supply
39
bowel obstruction assessment
vomiting, abdominal distention, hyperactive proximal to blockage, colicky type pain, fluid/electrolyte imbalance
40
barium enema only used after what is ruled out
perforation
41
bowel obstruction complications
infection, ischemia-infaraction, perforation, severe dehydration, electrolyte imbalance
42
bowel obstruction treatment
NG tube - decompression surgery - ileostomy or colostomy
43
bowel obstruction interventions
NPO, IV fluids, abdominal assessment, I+O, monitor CBC,BMP, pain meds, anti-emetics, oral care
44
purpose of bariatric surgeries
decrease client weight as safely as possible, decrease risks of obesity related diseases
45
who should adjust NG tube placement
only provider