Trauma Management 2 Flashcards
Explain the difference bw head trauma, head injury and TBI.
Head trauma- general term inclusive of both head injury and TBI
Head Injury- traumatic insult to head that results in injury of soft tissue of scalp or skull, does not include face.
TBI- impairment of brain fxn caused by external force that causes physical/social/emotional changes
What are the major regions of the brain?

What are the fxns of the major regions of the brain?
- cerebrum
- cerebral cortex
- cerebellum
- brainstem
- diencephalon
- pons
- medulla
- corpus callosum
Cerebrum- largest portion of brain, responsible for higher fxn/thought
Cerebral cortex- largest part of cerebrum, regulates voluntary skeletal movement, level of awareness
Cerebellum- maintains posture and equilibrium, skilled motions
Brainstem- crucial for vitals fxns (RAS-awareness)
Diencephalon-relays motor and sensory signals
Pons- regulates breathing and REM
Medulla- controls automatic fxns like HR and RR
What are the 4 different lobes and their fxns?
Frontal lobe: voluntary motions, personality and judgment
Parietal lobe: processes sensory info from skin and joints, responsible for proprioception
Temporal lobe: speech center, hearing, taste, smell, long term memory
Occipital lobe: processes visual information from optic nerve
What are the meninges?
They are a protective layer around the entire brain and spinal cord.
Dura mater –>Arachnoid –> Pia mater
Explain epidural hematoma.
Occurs bw dura mater and skull
Usually caused by a rupture of middle meningeal artery
Explain subdural hematoma
Occurs bw dura and arachnoid
Usually caused by rupture of bridging veins in bw these layers
Explain subarachnoid hemorrhage.
Occurs below arachnoid membrane.
Bleed directly into brain.
What do we look for in assessing a basilar skull fx?
- Blood and CSF leaking from ears, nose, both
- Raccoon eyes*
- Battle Sign*
*late signs
Cervical plexus
C1-5
innervates the diaphragm
Brachial plexus
C5-T1
Controls upper extremities
Lumbar plexus
L1-L4
Supplies skin and muscles of abdominal wall, external genitalia, part of lower limbs
Sacral plexus
L4-S4
Supplies buttocks, perineum, most of lower limbs
SCI at of below ___ may disrupt flow of sympathetic stimulation communication.
T6
How do we handle pt assessment for head and spine trauma? Any special considerations?
ABCDE
consider C spine
consider aggressive airway
consider neurogenic shock
do neuro exam
assess for ICP/abnormal posturing
consider backboarding
always place hands on pt for assessment
Signs of cerebral herniation
Unequal pupils
bilateral fixed dilated pupils
Decerebrate posturing/no motor response to px
GCS <9 that drops by 2+ points
What are the S/S of head injury?
DCAP
Visible fx
Battle sign
Raccoon eyes
CSF ears/nose
What EtCO2 do we ventilate head injury pt at?
30-35mm Hg
When would you want to start an IV in the case of head or SCI and use lots of fluids?
- establish 18g IV with LR
- do not give glucose unless known hypoglycemia
- only administer fluids on needed basis when hypotensive <90
- neurogenic pt’s may not require fluids so much as vagolytic drugs (atropine) and pressors or TCP
- watch for pulmonary edema
What are some specific assessments we do with SCI?
CMS in all extremeties
AVPU/serial GCS
Spinal immobilization
Pupils
check for chest trauma/fx
check for posturing
watch for hypo/hypertension
Stroke neuro exam
ask about sensation/pins and needles/numbness
**if pt unresponsive, but responsive to painful stimuli
–>grimaces, flexes limbs not likely to have SCI
Dermatome map

Explain skull fx types
Basilar- fx to base of skull.
Linear- closed, non displaced fx usually due to blunt trauma
Depressed- multiple fx in one area as result of blunt trauma with depression or dent in skull. can be concurrent with basilar fx. usually profound deficits seen
Open- brain tissue exposed
Closed- brain tissue not exposed
What is the difference bw primary and secondary injury? Give some MOI examples.
Primary- actual injury to brain as direct result of insult. Aka GSW, blunt trauma.
Secondary- damage to brain tissue as result of primary injury that bleeds into brain and swelling. can also include abscess, infection, hypoxia, etc.
Explain ICP
Skull is not capable of expanding for swelling brain post injury. The brain swells regardless of skulls capacity to expand, and it begins to take up what available space there is in the cranium. It takes up CSF space, occludes small blood vessels, increasing ICP. As pressure grows, brain recognizes it is hypoxic as a result. To combat this brain sends order to increase BP. In contrast carotid sinuses recognize that BP is increasing they did not call for, and order HR to slow down. This cycle continues, and pressure in skull worsens as the brain tries to maintain CP. As pressure grows, brain has no choice but to herniate out foramen magnum. Breathing centers and HR are sacrificed, ventilation is impaired, acidosis grows. Pupils will change. Pt will vomit out of nowhere with no previous complaint of nausea. Cushings triad is seen.

