treatment based classification Flashcards

(76 cards)

1
Q

criteria for manipulation

A

no symptoms below the knee

recent onset of sym (<16 days)

low FABQW score

hypomobilty of the lumbar spine

hip internal rotation ROM (>35)

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2
Q

criteria for stablization

A

younger (<40)

greater flexibility

instability catch or aberrant movements during lumbar flexion and extension

positive prone instability test

postpatrum things

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3
Q

aberrant movements

A

deviate
from the typical or expected movement
pattern, are associated with low back dysfunction

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4
Q

criteria extension

A

symptoms distal to the buttock

sym centralize with lumbar extension

sym peri with lumbar flexion.

directional perference of extension

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5
Q

criteria flexion

A

older then 50

directional preference for flexion

imaging for lumbar spinal stenosis

stiff achy back

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6
Q

criteria lateral shift

A

visible frontal plane deviation of the shoulders relative to the pelvis

directional preference for lateral translation movements of the pelvis

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7
Q

traction criteria

A

signs and symptoms of nerve root compression

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8
Q

Symptoms of Radiculopathy

A

Tingling or numbness in the fingers or hand.

Weakness in arm, shoulder or hand.

Decreased motor skills.

Loss of sensation.

Pain associated with neck movement or straining.

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9
Q

Instability catch

A

any sudden acceleration or deceleration of trunk movement or movement occurring outside the primary plane of motion

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10
Q

Transversus Abdominis action

A

“Support & compress abdominal viscera; assist in forced expiration, decreases infrasternal angle

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11
Q

Rectus Abdominis action

A

Flexes vertebral column; can posteriorly rotate pelvis when thorax is fixed

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12
Q

Quadratus Lumborum

A

Fixes last rib so diaphragm acts more efficiently during inspiration;

hikes hip;

ipsilateral side bending when pelvis is fixed

Acting bilaterally forms guy wire support to stabilize lumbar spine in frontal plane”

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13
Q

Blocked Extension Principle

A
  • Flexion worsens status
  • Not able to test “Extension Hypothesis”
  • Typically, not a “Mobilization Principle” candidate
  • Pt: flex brought on their initial problem; we need to restore their ext to get them to centralize
  • You cannot go into extension – this motion is blocked
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14
Q

Unstable Status (“Volatile”)

A
  • Rapid worsening with any flexion movement or position
    – Flexion – peripheralization
  • May or may not achieve a rapid improvement with extension
    Pt: have inflammation that is interplaying with mech issues
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15
Q

Stable Status (“Mechanical”)

A
  • Gradual improvement with sustained or repeated extension postures
    – Clear extension bias
  • Status will worsen with sustained or repeated flexion
  • Pt: have acute LBP and fall in line with textbook def
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16
Q

multifudus

A

Extension

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17
Q

Iliocostalis Lumborum, pars Lumborum
and
Longissimus Thoracis, pars Lumborum

A

Back extension, creates posterior shear forces to counteract anterior shear forces

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18
Q

External Abdominal Oblique action

A

“Support & compress abdominal viscera;
assist in forceful expiration
Acting bilaterally:

flexes spine

Lateral fibers: acting bilaterally, posteriorly rotates pelvis; acting unilaterally, laterally flexes spine
Anterior fibers: acting unilaterally, flex & contralaterally rotate spine”

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19
Q

malaise

A

geneally feeling unwell often accompanied by fatigue and diffused pain

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20
Q

what are myotomes testing for

A

you are looking for muscle weakness of a particular group of muscles.

Results may indicate lesion to the spinal cord nerve root, or intervertebral disc herniation pressing on the spinal nerve roots.

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21
Q

emergent medial red flags

A

extensive neurological involvement

non MSK conditions - red flags

fracture or something that need imaging work up

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22
Q

symptom modulation

A

preventing worsening status - moderating factors

signs of active inflammation

treatment:
- directional preference
- manipulation
- traction
- active rest

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23
Q

movement control - stage 2

A

impairment driven - improve dynamic movement control

improve basic functional movement patterns, break faulty compensatory patterns

the symptoms are now under control

treatment:
- flexibility exercises
- stabilization exercises
- sensorimotor exercises

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24
Q

functional optimization - stage 3

A

return to work or sport - increase functional capacity and tolerance

symptoms are low

treatment:
- strength and conditioning exercises
- work or sport specific tasks
- aerobic exercises
- general fitness exercises

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25
radiculopathy from an acute disc herniation - history and age
30 - 55 acute or reccurrent episodes
26
radiculopathy from an acute disc herniation - pain pattern
pain and or numbness radiating towards LE below the knee normally increases with lumbar flexion
27
radiculopathy from an acute disc herniation - neuro exam
sensory and/or motor changes diminished/absent deep tendon reflexes unilat
28
radiculopathy from an acute disc herniation - ROM
guarded and limited
29
spinal stenosis - age and history
>60 insidous onset of chronic progressive LBP more recent onset of LE pain
30
spinal stenosis - pain patterns
LE symptoms increase with ext and are relieved by flexion
31
spinal stenosis - neuro exam
sensory and motor changes
32
spinal stenosis - ROM
pain and limited ext
33
what is the start back for
screening for yellow flags
34
ospro is for what
screening red flags
35
what is the oswestry
standard screening for low back pain
36
ospro-YF
looking at specific yellow flags fear avoidance, depression, anxiety
37
what is the most significant neurological sign
babinski
38
what does peri mean in term of low back pain
pain or paresthesia moves distally away from the spine para is produced previously where it was not seen sym increase and sustain for at least 30 secs
39
what is centralization
pain or para moves centrally - towards the spine paresthesia that was present is abolished sym is diminished or abolished any movement that causes ccentralization should be included in treatment
40
what is paresthesia
an abnormal sensation, typically tingling or pricking (“pins and needles”), caused chiefly by pressure on or damage to peripheral nerves.
41
status quo
neither centralization or peri is produced there is a tranisent increase of decrease in pain pbserved with movement if this is seen - think of doing moblization, traction, or immoblization
42
what is active rest
when the pt inflammation is so bad that most movement leads to pain only ADLs for 48 -72 hours this is a acute issue may be using modalities to get pain under control
43
what is a right lateral shift
the shoulder are shifted right and the pelvis is going left
44
what is directional perferance
situation in which movement in one direction improves pain and limitation of ROM
45
when do you have the most intradiscal pressure
standing with flexion more when you add a weight supine
46
internal disc distruption
with the internal arch of the internal disc is disrupted even though its external apperence remains noraml
47
why do people get injured in the fully flexed position
support responsibilities shift from the muscle to the disc leads to creation of shearing force
48
disc herniation and fully flexed spine
posterior protustion due to annular failure associated with a fully flexed spine
49
pathomechanics of a lateral shift
disc herniation protective muscle spasm segmental instablity
50
lumbar radiculopathy history
presence of sciatica LE pain greater then back pain dermatome distribution of pain
51
weakness seen with lumbar radiculopathy
PF, DF, great toe extension, quads L4, L5, S1, S2
52
what is Ischemia
when blood flow is restricted to a part of the body
53
do people with spinal stenosis have pain when seated
normal no are in pain when they are up on their feet
54
lumbar spinal stenosis population
greater then 65
55
do people with spinal stenosis have pain with flexion or extension
extension
56
traction in extension criteria
younger worsen with flexion ISQ with extension prefer to stand and walk compared to sitting alternative: auto-traction to extend
57
traction in flexion criteria
older worsen with extension ISQ with flexion prefer sitting vs standing and walking alternative: de weighted ambulation
58
the flexion principle - what kind of spine
stenotic, degenerative,
59
flex prin - type of spine
flat back, sway back, hyperlordoic the load is going to the posterior segments
60
flex prin - special cases
spondylothesis adherent nerve roots syndromes
61
what is the mechanism of onset for flex principle
no specific mech often has a gradual onset
62
flex prin - demographic
stiff achy back - often with radiating features variable but late 50s and older exhibit claudicant behavior - sense of weakness when standing, they do not want to get on their feet
63
what is the common path of degeneration in the back
the disc degenerates and then the facets joints
64
what is lumbar spinal stenosis
any narrowing of the lumbar spinal canal, nerve root canal, or intervertbral foramen that produces compression on the neural elements
65
primary lumbar stenosis
the bone did not form consistent with expectations
66
secondary spinal stenosis
degenerative chnages post op changes fracture tumor systematicc disease
67
what makes up degenerative stenosis
facet joint arthrosis ligamentum flavum thickening intervertebral disc bulging spondylolethesis
68
structural component of spinal stenosis
degenerative changes degenerative spondylolethesis
69
what does flexion do to CSA
it increases CSA -loading
70
what does extension do to CSA
decrease CSA - unloading
71
narrowing and spinal extension
greater structure narrowing of the spinal canals --> greater narrowing during ext
72
Myelopathy
an injury to the spinal cord caused by severe compression that may be a result of spinal stenosis, disc degeneration, disc herniation, autoimmune disorders or other trauma.
73
shopping cart sign and graded treamill
both of these are putting in pt in flexion their preferred position the shopping cart is also supporting their trunk wieght
74
are we treating spinal stenosis with flexion pt
no more treating the impairments caused by the stenosis
75
surgery and spinal stenosis
complication rates are high
76
what is the treatment program for flex prin pt's
flex oriented exercise de-weighted treadmill ex for individual impairments