Vestibular disorders Flashcards

(46 cards)

1
Q

Sensation of inappropriate movement – Spinning

A

Vertigo

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2
Q

_______ is usually “ear” generated and causes a sensation of spinning

A

Nystagmus

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3
Q

main role of ear is to

A

keep eye fixed on target

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4
Q

Ear is constantly sending info to brain on baseline activty… if both ears are in same state means

A

you’re not moving

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5
Q

When you turn right, your right ear will send signals, the response is:

A

eyes accomidate to movement

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6
Q

loss of function of left ear: you don’t get normal tonic input so more input comes from right… brain intereprets this as..

A

you turning your head right = Unilateral hypofunction

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7
Q

Stimulation of a semicircular canal generates eye movements in the plane of that canal

A

Ewalds first law

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8
Q

Ewalds first law

A

Stimulation of a semicircular canal generates eye movements in the plane of that canal

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9
Q

Horizontal canal cauases eye movements to go

A

left and right

ampullopetal flow

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10
Q

Superior canal causes eyes

A

to go obqlique and off to the side

ampullofugal flow

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11
Q

Posterior canal causes eyes to

A

go oblique and to the back

ampullofugal flow

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12
Q

semicircular canals are at

A

right angle

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13
Q

stimulate the right horizontal canal, eyes move

A

left

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14
Q

stimulate the right superior canal, eyes move

A

eyes move UP, then rotate left

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15
Q

stim the right posterior canal, eyes move

A

eyes move down, then rotate left

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16
Q

Nystagmus occurs

A

Opposite the direction evoked by canal excitation

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17
Q

Nystagmus is a Corrective mechanism

A

– Rapidly bring eyes back to where they belong

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18
Q

if right horizontal canal is more active, nystagmus causes eyes

A

to the left

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19
Q

right superior canal is more active, nystagmus causes

A

eyes to go down and left

20
Q

With nystagmus the eyes drift to the

A

weak side… then jerk back to more active side

21
Q

if i stim right horizontal canal, i will see nystagmus that

A

beats to right… beats in plane of stimulated canal or towards more active side

22
Q

if someone comes in and complains of dizziness but no nystagmus…

A

CANNOT be ear related

23
Q

Three tests to uncover nystagmus

A

Head thrust
Gaze evoked nystagmus
head shake test

24
Q

Most important vestibular reflex
• Maintains eye position during motion
• Extremely fast responses
• Disturbances are demonstrated by eye examination

A

Vestibulo ocular reflex

25
Loss of VOR on left side when you do a head shake:
eyes will pulse to the right
26
Gaze in the direction of the fast phase of nystagmus increases amplitude and frequency
alexanders law
27
Clinical situation: patient looks left and see a slow, solid nystagmus patient looks right and see it's more active diagnosis?
something is going on with the left eye
28
3 days post op left acoustic neuroma pt no longer has visible nystagmus but i can bring out a stronger nystagmus by having:
having patient look right | left is still hypofunctioning
29
Excitatory response for angular VOR are greater than inhibitory responses
Ewalds 2nd law | turing towards a side activates that side more then turning away from a side's inhibtion
30
What happens to a normal patient during head shake exam?
nothing.. not nystagumus because summed up equally on both sides
31
during a head shake exam with a patient that has left side weakness... what happens when you're done
see right beating nystagmus
32
loss of bone covering over superior canal is
superior canal dehiscence
33
you can mimic superior canal dehiscence by:
tones, exercise, pressure
34
• Posterior canal canalithiasis • Posterior canal activated by movement – Otoconia move in canal simulating movement (crystals) • Nystagmus is toward affected ear and rotary in nature – Geotropic beating (toward the ground)
Benign Paroxysmal Positional Vertigo BPPV
35
To diagnose BPPV
Dix-Hallpike Testing | chaning a lightbulb or getting the bed spins
36
• Head thrust and head shake can uncover a
weakness
37
Gaze can point to the
more active canal
38
Sound can stimulate a
dehiscent superior canal
39
Loss of vestibular function in all canals • Can elicit signs of unilateral weakness • Hearing loss • Viral or bacterial in origin – Assess history of URI or otitis media
Labyrinthisis
40
Vestibular Neuronitis affects:
• Superior Vestibular Nerve – Horizontal and superior canals – Posterior canal spared (BPPV)
41
During Acute Phase of vestibular neuronitis
– Nystagmus beating away from affected ear
42
During Chronic Phase of vestibular neuronitis
>6monthsafterinitialattack • Weakness in balance function on one side • Sensitive to rapid head movements • Rare to have recurrence of initial attack • May develop BPPV • 30%developanxiety/”fearofdizziness”
43
``` 1st week • Sudden and intense vertigo and imbalance • Need to stay still • Nausea and vomiting • Typically seen in ER ```
Vestibulo neuronitis
44
Question: I suspect vestibular neuritis in a patient with a single long vertigo attack 1 year ago. To identify a unilateral vestibular weakness and which ear was affected, my exam should include: A. Head thrust test B. Head shake exam
Both A and B – Head thrust may show refixation saccades with thrust to the weak ear – Head shake should uncover asymmetry with nystagmus beating to the good ear (away from the affected ear)
45
Inner ear fluid imbalance Episodic vertigo Fluctuating hearing loss
Menieres disease
46
Meniere’s Disease | Episodic Vertigo
``` Recurrent and episodic • Vertigo • Last 30 minutes to ~4 hours • Minimal imbalance between attacks • Unilateral balance weakness – fast turns or head rotations ```